ライフサイエンスコーパス: bacterial translocation

1 poptosis, intestinal barrier disruption, and bacterial translocation.

2 y a single site on this protein required for bacterial translocation.

3 ters and disorders associated with increased bacterial translocation.

4 hat intestinal epithelial hypoxia influences bacterial translocation.

5 mucosal injury, luminal fluid secretion, and bacterial translocation.

6 There was no bacterial translocation.

7 IL-1alpha significantly reduced the rates of bacterial translocation.

8 ) disrupts the epithelial barrier, promoting bacterial translocation.

9 y to the intestine, and an increased rate of bacterial translocation.

10 ls prevented an increase in MPO activity and bacterial translocation.

11 ith subsequent increases in permeability and bacterial translocation.

12 ntestinal inflammation, gut permeability and bacterial translocation.

13 eading to increased mucosal permeability and bacterial translocation.

14 nce, intestinal mucus and mucin content, and bacterial translocation.

15 bowel allograft rejection without increasing bacterial translocation.

16 verely increased intestinal permeability and bacterial translocation.

17 sis, aggravated by systemic inflammation and bacterial translocation.

18 mune response triggered by gut dysbiosis and bacterial translocation.

19 to inflammatory bowel disease and subsequent bacterial translocation.

20 t modifier mediating the systemic effects of bacterial translocation.

21 itated by microbial dysbiosis and associated bacterial translocation.

22 al integrity leading to gut permeability and bacterial translocation.

23 mical barrier that might function to prevent bacterial translocation.

24 intestinal barrier, which leads to increased bacterial translocation.

25 pathogen by inhibiting goblet cell-mediated bacterial translocation.

26 he density of mucosa-associated bacteria and bacterial translocation.

27 that experimental liver fibrosis depends on bacterial translocation.

28 ores epithelial barrier function and reduces bacterial translocation.

29 sulfonic acid-induced colitis and associated bacterial translocation.

30 at CD4 T cell dysfunction might be caused by bacterial translocation.

31 n intestinal T cell suppression and enhanced bacterial translocation.

32 rvested under aseptic conditions to quantify bacterial translocation.

33 gesting an important role for PAFR-dependent bacterial translocation.

34 , including microbiome changes and increased bacterial translocation across a compromised gastrointes

35 ent of processed foods and that can increase bacterial translocation across epithelia in vitro, might

36 ing epithelial permeability and facilitating bacterial translocation across the gut barrier.

37 We hypothesized that bacterial translocation across the intact barrier occurs

38 L-1alpha reduces intestinal permeability and bacterial translocation after burn and sepsis.

39 were examined for viability, apoptosis, and bacterial translocation after exposure to a series of in

40 e reports of leakage of LPS from the gut and bacterial translocation after injury in animal models, t

41 this study was to test whether iNOS mediates bacterial translocation after intestinal ischemia-reperf

42 ups than in controls, supporting the role of bacterial translocation and activation of the inflammato

43 CXCL11, and TRANCE) correlated strongly with bacterial translocation and can be used to predict and d

44 eem to indicate that there is little risk of bacterial translocation and contamination from the conju

45 Advanced liver disease predisposes to bacterial translocation and endotoxaemia which can contr

46 ypothesis that altered gut permeability with bacterial translocation and endotoxaemia would be increa

47 Plasma markers of bacterial translocation and gut barrier dysfunction (lip

48 acterial challenge model, was used to assess bacterial translocation and gut colonization.

49 pendent gut tissue damage, body weight loss, bacterial translocation and gut dysbiosis.

50 out mice were resistant to endotoxin-induced bacterial translocation and ileal mucosal damage.

51 helial permeability and leads to exaggerated bacterial translocation and increased mortality during p

52 tility may be involved in the development of bacterial translocation and infection in patients with l

53 nistic factors in liver cirrhosis leading to bacterial translocation and infectious complications.

54 thobiont Enterococcus gallinarum facilitates bacterial translocation and initiation of inflammation.

55 severe shock states is often associated with bacterial translocation and intestinal barrier dysfuncti

56 zonulin family peptides (ZFP) as markers of bacterial translocation and intestinal permeability and

57 disrupts the epithelial barrier to initiate bacterial translocation and liver inflammatory responses

58 Protein malnutrition was not associated with bacterial translocation and measurement of enteroadheren

59 Given the strong association between bacterial translocation and mucosal inflammatory disease

60 t to intestinal ischemia-reperfusion-induced bacterial translocation and mucosal injury than wild-typ

61 nuously present in cirrhotic patients due to bacterial translocation and patients' susceptibility to

62 hine use or abuse results in significant gut bacterial translocation and predisposes patients to seri

63 of the intestinal epithelial barrier allows bacterial translocation and predisposes to destructive i

64 protected against lipopolysaccharide-induced bacterial translocation and prevented the lipopolysaccha

65 anslocation and that IVIG treatment resolves bacterial translocation and restores CD4 T cell function

66 Bacterial translocation and subsequent sepsis were minim

67 stance of mice irradiated with gamma-rays to bacterial translocation and subsequent sepsis.

68 rived K. pneumoniae that was associated with bacterial translocation and susceptibility to T(H)17-med

69 Bacterial translocation and systemic endotoxemia have be

70 observed in CVID patients is associated with bacterial translocation and that IVIG treatment resolves

71 al barrier dysfunction may lead to secondary bacterial translocation and the development of the multi

72 Whereas gut bacterial translocation and the loss/dysfunction of peri

73 the overall inflammatory state via increased bacterial translocation and the presence of bacterial pr

74 ntial regulatory mechanism implicated (i.e., bacterial translocation), and its role in conditions of

75 enhanced intestinal epithelial permeability, bacterial translocation, and elevated colonic lymphocyte

76 y impaired C. difficile clearance, increased bacterial translocation, and elevated levels of endotoxi

77 central pathophysiological mechanisms of PH, bacterial translocation, and inflammation, that are all

78 markers of disease progression, gut damage, bacterial translocation, and inflammation.

79 lantation contributing to graft dysmotility, bacterial translocation, and possibly, acute rejection.

80 al colonization of mucosal surfaces, reduces bacterial translocation, and protects mice from alcohol-

81 Fibrosis, intestinal permeability, bacterial translocation, and serum endotoxemia were meas

82 tion against systemic inflammation driven by bacterial translocation, and to assess whether BTK inhib

83 al suppression, gut damage and the resultant bacterial translocation are associated with body composi

84 immune system of intestinal permeability and bacterial translocation are distinct.

85 increased postburn barrier permeability and bacterial translocation associated with deranged neutrop

86 partially explain the increased frequency of bacterial translocation associated with tissue ischemia.

87 e resistant to such therapies as a result of bacterial translocation at the time of transplantation,

88 he membrane cycling of SecA, the cytoplasmic bacterial translocation ATPase, and in the stabilizing o

89 ulation, and its severity has been linked to bacterial translocation (BT) and endotoxemia.

90 Liver cirrhosis is associated with bacterial translocation (BT) and endotoxemia.

91 Bacterial translocation (BT) has been suggested to be re

92 1) and exogenous HMGB1 is able to induce gut bacterial translocation (BT) in normal mice; therefore,

93 Here, we investigated the impact of bacterial translocation (BT) to MLNs on TNF-alpha produc

94 Bacterial translocation (BTL) drives pathogenesis and co

95 in pharmacologic antagonists of PAFr blocked bacterial translocation by as much as 80 +/- 6%.

96 se studies indicate that enhanced intestinal bacterial translocation caused by burn injury could be r

97 Sham operation did not induce bacterial translocation, change cecal bacterial populati

98 The aims of our study were to investigate bacterial translocation, changes in the enteric microbio

99 inal barrier permeability and an increase in bacterial translocation compared to infection with curli

100 c response without increased inflammation or bacterial translocation compared with controls.

101 ileal leakage of horseradish peroxidase, and bacterial translocation compared with food deprivation (

102 The crystal structure of a bacterial translocation complex describes the binding st

103 Intestinal inflammation and bacterial translocation contribute to liver fibrosis via

104 ccurred after SMAO than in the mice in which bacterial translocation did not occur after SMAO.

105 sham SMAO or mice subjected to SMAO in which bacterial translocation did not occur.

106 Bacterial translocation did not reduce TER.

107 HMGB1 neutralization is associated with bacterial translocation during APAP hepatotoxicity.

108 nal epithelial barrier as well as monitoring bacterial translocation during infection.

109 Our data suggest that bacterial translocation, endotoxaemia, inflammation and

110 preserved gut barrier integrity and reduced bacterial translocation, epithelial inflammation, and ox

111 r, it is unclear whether clinically relevant bacterial translocation even occurs in humans, much less

112 In contrast, a second temporally distinct bacterial translocation event resulted in successful hep

113 derlying compromised gut immune function and bacterial translocation following morphine treatment.

114 and closure of alveoli (group 2) facilitated bacterial translocation from the alveoli to the bloodstr

115 has been shown to reduce hemorrhage-induced bacterial translocation from the gut in mice and rats.

116 Bacterial translocation from the gut is considered the k

117 This is potentially associated with bacterial translocation from the gut leading to local an

118 Bacterial translocation from the gut microbiota is a sou

119 These mice also had increased enteric bacterial translocation from the gut to the mesenteric l

120 bial activity of Paneth cells (PCs), causing bacterial translocation from the gut to various organs.

121 at SCI increases intestinal permeability and bacterial translocation from the gut.

122 cular macrophages correlating with increased bacterial translocation from the lamina propria into the

123 emic immune system abnormalities lead to gut bacterial translocation (GBT) and bacterial infection.

124 scites is associated with a high rate of gut bacterial translocation (GBT) and spontaneous bacterial

125 - and NO3- were highest in the mice in which bacterial translocation had occurred.

126 In the last few years, bacterial translocation has revealed itself as playing a

127 Gut dysbiosis and bacterial translocation have been implicated in PSC-IBD,

128 ects from HIV infection, including increased bacterial translocation, immune activation, and presence

129 ral nutrition and elemental diets both cause bacterial translocation, immune dysfunction, and increas

130 Intestinal ischemia-reperfusion caused bacterial translocation in 72% of the iNOS+/+ mice but o

131 IBD and reduced intestinal permeability and bacterial translocation in a severe model of the disease

132 Intravenous TPN caused greater bacterial translocation in all small intestinal segments

133 s systemic immune activation associated with bacterial translocation in both disorders.

134 estinal inflammation, barrier disruption and bacterial translocation in CCl(4) -cirrhosis (1).

135 The mechanisms for bacterial translocation in cirrhosis that predisposes pa

136 an interplay of gut barrier dysfunction and bacterial translocation in colorectal carcinogenesis.

137 sing marker to elucidate the contribution of bacterial translocation in CRC.

138 s by cirrhotic plasma, suggesting a role for bacterial translocation in driving B-cell changes in cir

139 In view of the suggested role played by bacterial translocation in liver disease and obesity, we

140 the role of TLRs, intestinal microbiota and bacterial translocation in liver fibrosis, alcoholic liv

141 ased colonization of the small intestine and bacterial translocation in mice lacking Cd1d, an MHC cla

142 dulates the gut microbiome and might prevent bacterial translocation in patients with cirrhosis.

143 sts may prevent epithelial deterioration and bacterial translocation in patients with impaired bile f

144 tes mellitus and underscore the role of oral bacterial translocation in placental tissues during preg

145 Gastrointestinal (GI) bacterial translocation in sepsis is well known, but the

146 y cytokines in vitro and in vivo, aggravated bacterial translocation in TG mice under DSS treatment,

147 The incidence of bacterial translocation in the DuP753-treated animals wa

148 Studies of bacterial translocation in two models of human skin indi

149 pools to offer better protection against gut bacterial translocations in patients with CVID.

150 s that did not express TLR2 also had reduced bacterial translocation, indicating that TLR2 expression

151 ere changes in markers of T-cell activation, bacterial translocation, inflammation, and alpha and bet

152 independently, correlated with biomarkers of bacterial translocation/inflammation.

153 of plasma biomarkers linked to inflammation (bacterial translocation, inflammatory response, and endo

154 ysbiosis leads to gut barrier disruption and bacterial translocation, initiating local gut inflammati

155 cus within the abdominal cavity, followed by bacterial translocation into the blood compartment, whic

156 ile the molecular mechanisms responsible for bacterial translocation into the heart have been elucida

157 rences in histology, cytokine expression and bacterial translocation into the mesenteric lymph node.

158 Bacterial translocation is a shared phenomenon in common

159 Thus, in this article, we showed that bacterial translocation is associated with rPSC after LT

160 In acute pancreatitis (AP) bacterial translocation is considered as the key event l

161 In CVID, bacterial translocation is linked to systemic immune act

162 A new component of the bacterial translocation machinery, YidC, has been identi

163 ses, and this work supports the concept that bacterial translocation may adversely affect host defens

164 implications on understanding how intestinal bacterial translocation may affect infectious complicati

165 To determine whether bacterial translocation may contribute to these effects,

166 lunts mastocytosis in ileal villi as well as bacterial translocation, measured as numbers of mesenter

167 n both the iNOS+/+ and iNOS-/- mice in which bacterial translocation occurred after SMAO than in the

168 mesenteric artery occlusion (SMAO) in which bacterial translocation occurred had cecal bacterial pop

169 Bacterial translocation occurred prior to changes observ

170 ation and one (endotoxin challenge) in which bacterial translocation occurs and intestinal morphology

171 hese data suggest a novel mechanism by which bacterial translocation occurs and suggest a critical ro

172 mic sepsis, although the mechanisms by which bacterial translocation occurs remain largely unknown.

173 Bacterial translocation of E. coli C-25 from the mucosal

174 t here a novel model of chemotherapy-induced bacterial translocation of E. coli.

175 Ileal myeloperoxidase activity and bacterial translocation of Enterococcus faecalis were as

176 Model systems that allow bacterial translocation of ExoS have found ExoS to have

177 Bacterial translocation of ExoS into epithelial cells is

178 eceptor (PAFR), which is known to potentiate bacterial translocation of gram-positive bacteria, was s

179 Further analyses of bacterial translocation of HopM1 and AtMIN7 stability in

180 y effect intestinal tissue damage leading to bacterial translocation of indigenous E. coli.

181 inal decontamination also suggest a role for bacterial translocation on TLR-4 activation in PFC after

182 inflammatory cytokine secretion, ameliorated bacterial translocation on treatment with dextran sulfat

183 Zinc supplementation diminished bacterial translocation only in lactose-challenged under

184 a substantially impaired ability to support bacterial translocation, particularly from blood to brai

185 creased enterocyte apoptosis and E. faecalis bacterial translocation (postburn day 3).

186 disease progression, epithelial gut damage, bacterial translocation, proinflammatory cytokines, and

187 mice versus CTRL+BDL mice, suggesting higher bacterial translocation rate.

188 However, the key factors that dictate bacterial translocation remain unclear.

189 ses normal intestinal barrier mechanisms and bacterial translocation results.

190 Despite comparable levels of bacterial translocation, serum cytokine profiling reveal

191 This eradication prevented bacterial translocation, significantly reduced serum end

192 12(+)CD38(+) iNOS(+) M (M1M ) located in the bacterial translocation site (mesenteric lymph nodes [ML

193 VID exhibited differential host responses to bacterial translocation stimuli in vivo and ex vivo, wit

194 ly attenuated pathogenic immune responses to bacterial translocation stimuli.

195 ing, we evaluated their association with PH, bacterial translocation, systemic inflammation, and circ

196 disruptions of the mucous barrier facilitate bacterial translocation that may contribute to the onset

197 al mucosal surface is an important factor in bacterial translocation, that intestinal mucus modulates

198 al barrier function are thought to result in bacterial translocation, the presence of bacterial produ

199 ed in LRP5/6(LysM) mice are due to increased bacterial translocation to extraintestinal sites and mic

200 Gut colonization and bacterial translocation to liver, spleen, and mesenteric

201 We demonstrate significant bacterial translocation to mesenteric lymph node (MLN) a

202 regimen for ethyl pyruvate also ameliorated bacterial translocation to mesenteric lymph nodes and le

203 Twenty-four hours after reperfusion, bacterial translocation to mesenteric lymph nodes, ileal

204 Bacterial translocation to mesenteric lymph nodes, liver

205 d both ileal mucosal permeability to FD4 and bacterial translocation to mesenteric lymph nodes.

206 ction and PC antimicrobial activity, causing bacterial translocation to organs and subsequent polymic

207 d to alter microbial composition and promote bacterial translocation to other tissues.

208 We also outline possible mechanisms of bacterial translocation to the intrauterine environment

209 mice, epithelial proliferation decreased and bacterial translocation to the liver and spleen was dete

210 rmeability, presence of plasma endotoxin and bacterial translocation to the liver.

211 s of mucosa-associated bacteria and enhances bacterial translocation to the mesenteric lymph nodes an

212 The incidence of bacterial translocation to the mesenteric lymph nodes wa

213 ic intestinal inflammation, with evidence of bacterial translocation to the mesenteric lymph nodes, m

214 Results were correlated with bacterial translocation to the mesenteric lymph nodes.

215 T1D, which correlated with the abrogation of bacterial translocation to the PLNs.

216 Bacterial translocation to the spleen was demonstrated 2

217 Moreover, intestinal dysbiosis and bacterial translocation trigger hepatic and systemic inf

218 rial adherence, mucus bacterial binding, and bacterial translocation, two models were used.

219 ral nutrition and enteral diets may pre-vent bacterial translocation via the preservation and augment

220 dy, the role of macrophages in rejection and bacterial translocation was evaluated by depleting macro

221 wild-type mice following bile duct ligation; bacterial translocation was facilitated by TNFRI-mediate

222 Bacterial translocation was investigated in the liver, s

223 Bacterial translocation was measured using agar cultures

224 disruption of the intestinal epithelium and bacterial translocation was no longer observed.

225 of enteroadherent bacteria was increased and bacterial translocation was observed.

226 LPS-induced bacterial translocation was reduced by aminoguanidine.

227 Bacterial translocation was significantly enhanced in Ta

228 Bacterial translocation was significantly increased afte

229 Bacterial translocation was significantly increased in t

230 Liver fibrosis and bacterial translocation were assessed in Toll-like recep

231 factor receptor (PAFr) in hypoxia-associated bacterial translocation, wherein pharmacologic antagonis

232 lteration of intestinal barrier, and finally bacterial translocation, which can trigger proinflammato

233 sbiosis, intestinal barrier dysfunction, and bacterial translocation, which trigger the state of pers