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1  hereditary immunodeficiency syndrome called bare lymphocyte syndrome.
2 everely affected in the FZA patient with the bare lymphocyte syndrome.
3 ization of CIITA and one form of the type II bare lymphocyte syndrome.
4                                          The bare lymphocyte syndrome, a severe combined immunodefici
5  expression on B cells of patients with rare bare lymphocyte syndrome and prevention of self-antigen
6                                 AIDS and the bare lymphocyte syndrome (BLS) are severe combined immun
7                                              Bare lymphocyte syndrome (BLS) is an autosomal recessive
8                                          The bare lymphocyte syndrome (BLS) is characterized by the a
9          The severe immunodeficiency type II bare lymphocyte syndrome (BLS) lacks class II MHC gene t
10                                       In the bare lymphocyte syndrome (BLS), which is a human severe
11 up A(II) of MHC class II immunodeficiency or bare lymphocyte syndrome (BLS).
12 ree different HLA-DR allelic proteins in the bare lymphocyte syndrome cell line, BLS-1, revealed that
13 utant B-cell lines, which represent distinct bare lymphocyte syndrome class II genetic complementatio
14 lass II expression and that the mutations in bare lymphocyte syndrome complementation groups B and D
15  independently established B cell lines from bare lymphocyte syndrome complementation groups B and D.
16 ived from class II- plasma B-cells, and from bare lymphocyte syndrome group II cell lines, RJ2.2.5 an
17                   MHC class II deficiency or bare lymphocyte syndrome is a severe combined immunodefi
18 mmunodeficiency (CID), also known as type II bare lymphocyte syndrome, is an autosomal recessive gene
19 n groups identified in the class II-negative bare lymphocyte syndrome or corresponding laboratory mut
20 NA-binding defects in the protein RFX5 cause bare lymphocyte syndrome or major histocompatibility ant
21             MHC class II deficiency found in bare lymphocyte syndrome patients results from the absen
22 he study of genetic defects in cell lines of bare lymphocyte syndrome patients, as well as in cell li
23 oblasts derived from two recently identified bare lymphocyte syndrome patients, EBA and FZA, were fou
24 findings offer one explanation of why in the Bare Lymphocyte Syndrome, which is a congenital severe c
25              In addition, a patient with the bare lymphocyte syndrome who could not express MHC class