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4 The receptors were stably expressed in rat basophilic leukemia 2H3 cells and characterized for rece
5 lize the level of LAT phosphorylation in rat basophilic leukemia 2H3 cells show that Erk2 phosphoryla
6 duces an increase in 5-HT uptake Vmax in rat basophilic leukemia 2H3 cells that is enhanced by pretre
11 retion from transiently transfected RBL (rat basophilic leukemia)-2H3 mast cells (a tumor analog of m
16 ransfected the unique domain of Lyn into rat basophilic leukemia-2H3 mast cells and examined the cons
17 E-loaded receptors (IgE-FcepsilonRI) on rat basophilic leukemia-2H3 mast cells in contact with fluid
18 rgent sensitivity of this association on rat basophilic leukemia-2H3 mast cells, and we compare the c
19 or genetically encoded Ca(2+) sensors in rat basophilic leukemia and bone marrow-derived rat mast cel
20 release-activated Ca2+ (CRAC) channel in rat basophilic leukemia and other hematopoietic cells to rel
24 in a similar manner, we studied a stable rat basophilic leukemia cell line (RBL-CR1) transfected with
25 ere therefore examined using transfected rat basophilic leukemia cell lines (RBL-2H3) that expressed
26 blasts (as a model of adherent cell) and rat basophilic leukemia cells (as a model of nonadherent cel
28 ctive channels in the plasma membrane of rat basophilic leukemia cells (RBL-2H3 m1), an immortalized
29 donoyl ethanolamide (anandamide, AEA) in rat basophilic leukemia cells (RBL-2H3) has been proposed to
30 investigated the regulation of CXCR4 in rat basophilic leukemia cells (RBL-2H3) stably transfected w
31 alcium mobilization and degranulation in rat basophilic leukemia cells and cytokine production (IL-6
32 gated through patch-clamp experiments on rat basophilic leukemia cells and fluorometric imaging plate
33 , 5-LO is localized to the cytoplasm; in rat basophilic leukemia cells and human alveolar macrophages
34 o image Annexin 2-GFP in live, secreting rat basophilic leukemia cells and in cells performing pinocy
35 s across intact cell-attached patches in rat basophilic leukemia cells and rat megakaryocytes reveale
36 on-dependently inhibited CRAC current in rat basophilic leukemia cells and thapsigargin-induced Ca(2+
37 cts more effectively with FcepsilonRI(+)-rat basophilic leukemia cells at 37 degrees C compared with
40 that the mitotic clock of unsynchronized rat basophilic leukemia cells has a marked precision with 80
41 inity receptor for IgE (Fc epsilonRI) on rat basophilic leukemia cells is regulated by its initiating
42 sured the extent and rate of adhesion of rat basophilic leukemia cells preincubated with anti-dinitro
43 Recent studies in Jurkat T cells and in rat basophilic leukemia cells revealed an Mg(2+)-inhibited c
44 ster ovary cells and alpha4 integrins on rat basophilic leukemia cells showed little or no associatio
45 iating Ca(2+) waves are observed in most rat basophilic leukemia cells stimulated with soluble Ag and
47 gh affinity receptors (Fc epsilon RI) on rat basophilic leukemia cells using both single particle tra
48 we describe a study in which we incubate rat basophilic leukemia cells with a fluorescently labeled c
49 cepsilonRI-mediated calcium signaling in rat basophilic leukemia cells, a property dependent on the S
52 ar characteristics to that recorded from rat basophilic leukemia cells, namely a similar time course
54 degranulation assay in C3aR-transfected rat basophilic leukemia cells, two were prominent agonists (
56 om HeLa cells expressing SERT and intact rat basophilic leukemia cells, we show that agents such as N
71 een identified in T-lymphocytes and RBL (rat basophilic leukemia) cells and named MagNuM (for Mg(2+)-
72 ibitor of mast cell degranulation in the rat basophilic leukemia mast cell model, in the passive cuta
73 ng enlargement of these vesicles in both rat basophilic leukemia mast cells and Jurkat T leukemia cel
74 find that SNAP-23 is phosphorylated when rat basophilic leukemia mast cells are triggered to degranul
76 lation mutants inhibited exocytosis from rat basophilic leukemia mast cells, demonstrating that phosp
79 STZ mitigated degranulation of RBL-2H3 (rat basophilic leukemia) mast cells induced by compound 48/8
83 ion of the IgE high affinity receptor on rat basophilic leukemia RBL-2H3 cells results in activation
84 a monoclonal antibody raised against the rat basophilic leukemia RBL-2H3 cells, we identified that pl
85 man chemoattractant receptor regulation, rat basophilic leukemia RBL-2H3 cells, which are thrombin-re
89 Coimmunoprecipitation experiments in rat basophilic leukemia (RBL 2H3) cells show that SHIP-2 int
92 itro with human and mouse mast cells and rat basophilic leukemia (RBL) cells and in vivo in mice.
93 ocessed appropriately, and functional in rat basophilic leukemia (RBL) cells following retroviral tra
95 parallel studies of FcepsilonRI-bearing rat basophilic leukemia (RBL) cells transfected with constru
96 ion channel, in human and rat platelets, rat basophilic leukemia (RBL) cells, and phorbol myristate a
97 and capacitative Ca(2+) influx, we used rat basophilic leukemia (RBL) cells, vascular smooth muscle
99 ysteinyl leukotriene type I receptors in rat basophilic leukemia (RBL)-1 cells, large amplitude Ca(2+
100 to analyze published experiments on the rat basophilic leukemia (RBL)-2H3 cell line that seem to con
101 d, disassembled, and soluble vimentin in rat basophilic leukemia (RBL)-2H3 cells expressing human CCR
102 ist of (125)I-C3a radioligand binding to rat basophilic leukemia (RBL)-2H3 cells expressing the human
103 Surprisingly, overexpression of SphK1 in rat basophilic leukemia (RBL)-2H3 mast cells impaired degran
107 lation of the CCR1 chemokine receptor, a rat basophilic leukemia (RBL-2H3) cell line was modified to
109 IV-1 infection, monocytes and MAGIC5 and rat basophilic leukemia (RBL-2H3) cell lines co-expressing t
112 of 60 phosphate units was identified in rat basophilic leukemia (RBL-2H3) mast cells using specific
113 are expressed either in the cytoplasm of rat basophilic leukemia (RBL-2H3) mucosal mast cells or anch
114 d receptor, Mas-related gene X2 (MrgX2), rat basophilic leukemia, RBL-2H3 cells, and murine BMMCs do