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1 xtinctions, and how many species will likely be lost.
2 time hobbyists, causes migratory behavior to be lost.
3 on about dynamics and cellular heterogeneity is lost.
4 ulation of the intensity of locomotor output is lost.
5 omerular activity, and behavioral attraction is lost.
6 e the function of one of the two gene copies is lost.
7 line, and chromatin accessibility at the URE is lost.
8 that defines the tandem-repeat protein class is lost.
9 specificity of the copper/zinc binding sites is lost.
10 issociated, producing atomic hydrogen, which is lost.
11 y removed, the stemness epigenetic landscape is lost.
12  critical reactions collapsed as H(2) uptake is lost.
13 LFs are retained in adult alveoli while AMFs are lost.
14 ce characterized entire agricultural regions are lost.
15 etermines which cells expand and which cells are lost.
16 and depressurization, these residual strains are lost.
17 f the ions (>99.8%) in a continuous ion beam are lost.
18 atients) or RCT (527 patients), significance was lost.
19 absence of B cells, R848-mediated protection was lost.
20 d classes where almost all original identity was lost.
21 ) in which the N-terminal lysine residue has been lost.
22 y-related effects, but phase information has been lost.
23 n 41 and 62% of the LIA total ice volume has been lost.
24 p75(-/-) mice half of all geniculate neurons were lost.
25 hen more than half of the rod photoreceptors were lost.
26 GTR) of the achieved coverage after 3 months were lost.
27  16 (64%) survived, 7 (28%) died, and 2 (8%) were lost.
28 tion, 94% of allergen-specific IgE responses were lost.
29 sult in some biologically meaningful signals being lost.
30                                  No implants were lost (100% survival rate).
31                                 If mangroves were lost, 15 million more people would be flooded annua
32 is released, the slower-growing species will be lost (a hypothesis that we call 'feed the faster grow
33 nus, the estrogen-enhanced growth phenotypes are lost, a dependency not observed in monoculture.
34  binding and functional properties of the Fc are lost after deglycosylation or removal of the unique
35 vered after 120 min of cooking, while all RP was lost after 80 min in the absence of bran.
36                           Treatment efficacy was lost after antibiotic treatments with associated spe
37                              This processing was lost after exoglycosidase treatment and after RNAi-b
38 ed MEF2C mRNA and increased MEF2C abundance, was lost after OIP5-AS1 silencing, suggesting that OIP5-
39 -1 Gag-specific CD4 and CD8 T cell responses were lost after transplantation, whereas cytomegalovirus
40  DNA populations but 94% of the circular DNA were lost after ~15 divisions, whereas rDNA circles unde
41                             This enhancement was lost almost completely in gene knockout mice lacking
42 that, by telophase, condensin-mediated loops are lost and a transient folding intermediate is formed
43 key structural components of the kinetochore are lost and centromere structure is changed.
44        By doing so, the underlying guarantee is lost and accuracy is reduced in repetitive genomic re
45 ociously, the proliferative ventricular zone is lost and differentiation markers become expressed thr
46 of RPTPzeta, the reticular structure of PNNs is lost and phenocopies the PNN structural abnormalities
47 s a threshold beyond which coastal land will be lost, and mitigation efforts should shift upstream.
48 tient-matched and derivative cell lines have been lost, and accessing information and datasets is tim
49  and other rodents, the IFIT1 orthologue has been lost, and the closely related Ifit1b has been dupli
50 s, as the mixing end-members here would have been lost, and the measured ratios would have been inacc
51 affected keratinocytes the AP-1 beta subunit is lost, and the gamma subunit is greatly reduced, demon
52  phenotypic instability, the high redundancy was lost, and gid1a exhibited dwarfism that was strongly
53 t recurrences, the Chromogranin A expression was lost, and the Ki-67 index increased to 90%.
54 es as skeletal elements elongated and digits were lost, and yet the developmental mechanisms remain u
55            The physical means by which cilia are lost are poorly understood but are thought to involv
56       Furthermore, CRPC tumors in which Pten is lost are also resistant to eradication by PI3K inhibi
57                            We find that TADs are lost as genes become silenced on the paternal X chro
58  the recovery of carbon that would otherwise be lost as ethanol, for example following hypoxia.
59 e role in choroid plexus and this protection is lost as Abeta accumulates with the disease progressio
60 iological immunomodulatory functions of CD31 are lost at the surface of blood leukocytes in patients
61 l Sequencing data, allowing each mutation to be lost at most a fixed number of times.
62 er new insights about what functions tend to be lost at what stage compared to the control tissues an
63 roductive lifespan, DNA methylation fidelity is lost at a number of CpG islands (CGIs) resulting in C
64 x evolutionary processes but this congruence is lost at the continental to country extents.
65 t outside the fovea, this spatial resolution is lost at the level of ganglion cells.SIGNIFICANCE STAT
66 hout the retina, but that spatial resolution is lost at the level of the ganglion cells.
67       This protective association of alpha-T was lost at higher gamma-T levels.
68 e presence or absence of air; this stability was lost at higher temperatures in the presence of air.
69 is, and 23.1% of the substantia nigra volume was lost at the time of diagnosis, which was in line wit
70          Then, essential amino acid pathways are lost (at ~30-60 mya), which coincides with the incre
71  to this type of data, important information is lost, because they either require all tissues to be a
72  on superficial and deep cells: lamellipodia are lost, blebs appear instead, and collective migration
73  molecule(s), and new MHC-like gene(s) would be lost by neutral drift.
74  and PF-3M2B in the troposphere whereas 2M3P is lost by both photolysis and gas phase reaction with a
75 I) on postnatal day 1 (P1), but this ability is lost by postnatal day 7 (P7).
76  points: 1 week and 1 month, but this effect was lost by 3 months.
77        MYBL1 3'UTR inhibition by miR-221/222 was lost by deletion of a single putative miR-221/222 bi
78 endrocyte differentiation while this support was lost by EVs from aged astrocytes.
79                     Ipsilateral gastrulation was lost by midline ECM and PCD inhibition but restored
80  GPe neurons and subthalamic nucleus neurons were lost by 18 months but not prototypical PARV+ GPe ne
81 ll pool and replenish specialized cells that are lost due to death or usage.
82 ation, though after rewarming these benefits are lost due to increases in cord swelling, ischemia and
83  with nearly 30% of arable land predicted to be lost due to soil salinity in the next 30 years.
84 rophy, and neurodegeneration when regulation is lost due to genetic and/or environmental insults.
85                                   Fewer days were lost due to heart failure hospitalisations and card
86 last follow up and 9 (18.8%) pancreas grafts were lost due to patient death or graft failure after >2
87                                Primary cilia are lost during cancer development, but the mechanism re
88 ion, and noise in short-read data, reads can be lost during mapping, and de novo assembly can be time
89 to recognise further biomolecules thought to be lost during the fossilisation process.
90             Our data suggest that dioecy has been lost during domestication through a rare recombinat
91 ential mitochondrial signal for healing that is lost during aging.
92 act may inadvertently arise if NCP asymmetry is lost during image processing.
93 ty is robust in the neonatal mouse heart but is lost during postnatal development when cardiomyocytes
94                      The acetyl methyl group is lost during the reaction as methane.
95 he information in the sensory representation is lost during the transformation to a decision-level re
96 transported to the airway mucus layer, which is lost during tissue preparation.
97  an important factor for dantrolene's action was lost during the purification procedure of RyR.
98  seasons in subsequent years and more carbon was lost during the rainy season relative to the dry sea
99 sults showed that 28% of the spiked (14)C-AA was lost during the roasting process, presumably by degr
100 , MUC5B adopted a compact conformation which was lost either upon removal of calcium with EGTA, or by
101 stral function, sub- or neofunctionalize, or be lost entirely.
102  potential diminishes with maturity until it is lost entirely.
103 ction, whereby 25% of the gene inventory has been lost, followed by its split into the species-poor e
104                                 The efficacy is lost following depletion of CD8(+) T cells.
105 rate that STEP61 is the molecular brake that is lost following KCC2-dependent disinhibition and that
106 rations of Kdm6a male germline deletion, but is lost following passage through a wild type germ line.
107    The perivalvular antithrombotic phenotype was lost following genetic deletion of FOXC2 or femoral
108 on processes and cellular activity, and both were lost following blockade of GluR2-endocytosis.
109            The resulting spatial segregation is lost for weak growth-migration trade-offs and a lack
110 atment success, a total of 3 pancreas grafts were lost for immunological reason.
111 imulations leads to a model in which protons are lost from amino acid that will serve as OEC ligands
112 ver, we show that the mono(ADP-ribose) scars are lost from the chromatin of ARH3-defective cells in t
113 lly when protective calcium binding proteins are lost from the cytosol.
114 encing (perpetuated) phase, the PHD proteins are lost from the nucleation region and silencing is lik
115 nes - genes with small fitness benefits that are lost from the population without HGT - can be collec
116 and ACS recovers carbon that would otherwise be lost from the plant as ethanol.
117 f pine (Pinus palustris Mill.) ecosystem has been lost from the Coastal Plain of the southeastern Uni
118                             These genes have been lost from the genomes of metazoans but are otherwis
119 that carbon fixed by plant photosynthesis is being lost from ecosystems to the atmosphere more quickl
120 ine-free selection, although exogenous mtDNA is lost from metabolically impaired, mtDNA-intact (rho+)
121  study can be prohibitive, lots of potential is lost from MS datasets when uncharacterized PTMs are f
122                                  When a base is lost from RNA, the remaining ribose is found as a clo
123 ne-induced senescence (OIS), heterochromatin is lost from the nuclear periphery and forms internal se
124           In absence of Shrub function, Mega is lost from the SJ and becomes trapped on endosomes coa
125 zes to unattached kinetochores and like MPS1 is lost from these structures upon microtubule attachmen
126 E-containing enhancers increased, whereas AR was lost from cAREs.
127                One graft in this later group was lost from recurrence 141 months post transplant.
128 less, nearly one-half billion tons of carbon were lost from both ITs and PNAs (-434 MtC and -423 MtC,
129                            Afferent synapses were lost from inner hair cells throughout the aged coch
130 beyond which the majority of intolerant taxa were lost from streams.
131                                   As species were lost from the communities, the probability of taxa
132 her results, may explain why clinical effect is lost if SLIT is discontinued at this point.
133         Given a significant number of islets are lost immediately following transplantation, pFUS has
134                           Similarly, lncRNAs are lost in diabetic humans.
135 hesis and SA perception pathways, since they are lost in isochorismate synthase1/salicylic acid induc
136 rt of the G-protein networks; however, these are lost in many monocots.
137     Topologically associating domains (TADs) are lost in meiotic prophase, suggesting that assembly o
138                   Surprisingly, such effects are lost in per (0) mutants, supporting a PER-dependent
139 in mice, and the majority of male germ cells are lost in the meiotic defect of first wave spermatogen
140 Although NAFLD genomics sometimes appears to be "lost in translation," we envision clinical utility i
141 rafficking, and function, a feature that may be lost in JNCL.
142 s seed dispersal of large seeded plants, can be lost in large continuous forests due to the rarity of
143   Thus, tonic PVN NPY inhibition of LSNA may be lost in obese males as a result of a decrease in NPY
144  and srg-37), which were previously shown to be lost in population-dense laboratory cultures.
145 rproliferation, and that this regulation can be lost in the development or progression of breast canc
146 e might predict that that HSR function would be lost in the reproductive queens.
147                 Up to 84 000 tons of dye can be lost in water, and 90 million tons of water are attri
148        Alternatively, this function may have been lost in Arabidopsis.
149 ilies are present across eukaryotes but have been lost in humans.
150 i that share features with animals that have been lost in most other fungi.
151 ) are an ancestral eukaryotic trait that has been lost in several lineages like flowering plants.
152 ascular plant groups but is believed to have been lost in the ancestor of leptosporangiate ferns.
153 77 mammal and bird species on the brink have been lost in the last century.
154 sic life-history theory, but appears to have been lost in translation in recent developments of life-
155 se gene families is highly lineage-specific, being lost in some descendant lineages, but undergoing e
156 tly, the anti-proliferative activity of IRF1 is lost in cell lines expressing T181A mutant.
157                     This requirement for PP1 is lost in cells expressing CDK1 phosphorylation-defecti
158                            This adaptability is lost in cultured adult endothelial cells, which do no
159 stantial heterogeneity in protein age, which is lost in iPSC-derived neurons lacking the lysosomal pr
160               Glucose-dependent excitability is lost in islets from K(ATP)-knockout (K(ATP)-KO) mice,
161                  One of these is FAM46C that is lost in more than 10% of patients with multiple myelo
162  This ability to impart left-sided character is lost in parapineal cells lacking Sox1a function, desp
163                                Shh signaling is lost in reactive astrocytes at the lesion site, but p
164  provide key insights into how cell identity is lost in some germ cell tumors.
165                   While caspase-8 expression is lost in some tumors, it is increased in others, indic
166  decreases after approximately 10% of weight is lost in the surgical group and approximately 20% in t
167 anding of how and where stimulus information is lost in the visual system under crowding.
168 we identified an immunoreactive antigen that was lost in a putative glycosyltransferase mutant, sugge
169 rum viremia was achieved-an observation that was lost in anti-TNF-alpha-treated IFNAR(-/-) animals.
170 g C(org) m(-2) year(-1) , respectively), but was lost in bare areas.
171 C/EBP-alpha-mediated regulation of claudin-5 was lost in blood vessels from tissue biopsies from pati
172                                This response was lost in bone marrow-derived macrophages from mice de
173          Hypoxia-mediated induction of ATG9A was lost in cells lacking HIF-1.
174  chronic nitroglycerin model, an effect that was lost in Dlx-DOR mice.
175 DB overexpressers compared with WT parasites was lost in gp91-phox (-/-) macrophages, underscoring th
176 terial-derived histamine on BAL inflammation was lost in HDC-deficient mice.
177                       This close correlation was lost in heart failure myocytes from both species.
178                  Notably, miR-223 expression was lost in microdissected ductal carcinoma in situ (DCI
179        Itch sensation to histamine injection was lost in most symptomatic patients.
180                                       FNDC3A was lost in some multiple myeloma cell lines.
181 tumor effect induced by anti-MerTK treatment was lost in Sting(gt/gt) mice, but not in Cgas(-/-) mice
182 injury scores) was found in the controls but was lost in TBI patients.
183                           We show that Cdc14 was lost in the common ancestor of angiosperm plants but
184 dentified several proteins whose acetylation was lost in the Deltakatms strain, and whose transcript
185 ry sequence, and the expression of this gene was lost in the developing gut of mice that lacked the I
186  for placebo; P = .03), but the significance was lost in the pooled analyses (P = .058).
187 ddition, the anti-allodynic effects of SNC80 were lost in a model of opioid induced hyperalgesia/medi
188 during the diversification of eukaryotes but were lost in a subgroup of Fungi.
189 s remained in place in SHAM and OVX ewes but were lost in all ZOL ewes.
190 hesis of the essential vitamin B1 (thiamine) were lost in an ancestor of a yeast lineage, the Wickerh
191 ontractility and skeletal muscle hypertrophy were lost in beta-arrestin 1 knockout mice, implying tha
192 troduced thousands of ancestral alleles that were lost in Eurasian populations before introgression.
193 e ACs coupled to RGCs via gap junctions that were lost in glaucoma, whereas uncoupled ACs were largel
194 g endurance, and beneficial glycemic effects were lost in Il13(-/-) mice.
195                                  These bonds were lost in MGL(short) H259T, thus explaining its lower
196             The salutary effects of the drug were lost in Parkin knockout mice, implicating Parkin-me
197         All or majority of the key ATG genes were lost in several fungal groups with unique lifestyle
198 nsposable elements in Brassica oleracea, but were lost in the majority of the Bot1 elements in Brassi
199                  We confirm that Bmp16 genes were lost independently in at least three lineages (mamm
200 ch as ischemia-reperfusion, phosphorylations are lost, leading to maximum ETC flux, DeltaPsi(m) hyper
201                           Editing sites have been lost many times during evolution by C-to-T mutation
202 esents compelling evidence that smaller RGCs are lost more rapidly than larger RGCs despite the dynam
203 m, as dopamine neurons from the ventral tier are lost more rapidly than those of the dorsal tier.
204 phylococcus and found that mRNA conservation was lost mostly downstream of the coding sequence (CDS),
205 resent in almost all oxygen-redox compounds, is lost on charging, driven in part by formation of mole
206                        Systemic inflammation was lost on expression of a non-self-reactive BCR or los
207 own, such regenerative capacity (plasticity) is lost once supporting cells undergo maturation.
208 at there are conditions in which sporulation is lost or selected against.
209                Rare N-gly-negative subclones were lost or negligible from successive events, in contr
210 pendent transmission, acquired immunity that is lost over time, and recrudescence.
211  CD4(+) T cell activation, a difference that was lost over the first 3 years of infection as activati
212    Only 4% of the originally deposited PdNPs were lost over 90 days of continuous operation.
213 Over the 10-year period, these initial gains were lost over time with a final VA change of +3 letters
214                                   While TADs are lost, physically separated A and B compartments are
215 show that low temporal frequency information is lost primarily between the cones and the LGN, whereas
216  the LGN, whereas high-frequency information is lost primarily within the cones, with a small additio
217 f PRDM9 and the mechanism by which Prdm9 can be lost remain unclear.
218 information about periodic light modulations is lost, signal-to-noise ratios were compared across sim
219           Thus, catalysis and pore stability were lost simultaneously in vertebrate TRPM2 channels.
220 e toxin once genes encoding a TA system have been lost, such as following failure to inherit a plasmi
221  (presumably faster) corticospinal terminals are lost, suggesting a significantly altered cortical mo
222  proliferative activity and that EOMES+ SSCs are lost through proliferative exhaustion in Plzf (lu/lu
223  in the hypertension care continuum patients are lost to care is sparse.
224 p in the diabetes care continuum individuals are lost to care, and how this varies between countries
225                       Although many patients are lost to follow-up, the evidence indicates that subth
226 -molecular-weight atmospheres; instead, they are lost to space alongside H(2) This early volatile dep
227  postdocs and their knowledge and skills may be lost to academia without intervention.
228 om other enzymatic systems, since enzyme can be lost to proteolytic degradation.
229 though data were lacking because of patients being lost to follow-up.
230 ctron transfer (EET) within biofilms without being lost to the environment has been a long-standing q
231 in more than 5,500 kilometres of lithosphere being lost to the mantle).
232 recycled within aerobic ecosystems, but some is lost to the system and is sequestered in the crust fo
233 d up to 18% of the bobcat population in Ohio is lost to vehicle-strikes annually.
234 urden, severe pre-existing comorbidity], one was lost to follow-up, one developed of Graves disease,
235 efore imaging showed PD, and one participant was lost to follow-up.
236 olonies were disbanded and the CHS cat model was lost to the research community before the causative
237 on (<1%) of the total processed API quantity was lost to the wastewater, and despite the small percen
238 urther, three and four patients respectively were lost to clinical follow-up (ie, survival status was
239  patients met criteria for analysis (38 eyes were lost to follow up at 3 months; 68 at 12 months; 146
240           Among 743 NP patients, 64 died, 13 were lost to follow up; therefore, a total of 666 patien
241 ts (83.7%) completed the study and 21 (5.9%) were lost to follow-up after completing protocol therapy
242 ment group), but at the 10-year follow-up 14 were lost to follow-up and 12 withdrew, leaving 3031 to
243 g the 304 patients without SVR12 results, 48 were lost to follow-up and 256 had no SVR12 results but
244 a normal ECG, different cardiac diseases, or were lost to follow-up and exited the study.
245 d 14 from the APBI group withdrew consent or were lost to follow-up at this stage, so 2039 and 2093 p
246                                Five patients were lost to follow-up because of emigration (99.9% foll
247      Nine patients discontinued treatment or were lost to follow-up before completion; 1 is continuin
248                                Two practices were lost to follow-up but provided some outcome data.
249                                Four patients were lost to follow-up during the clinical follow-up.
250                          No patients died or were lost to follow-up during the study.
251                                 Two patients were lost to follow-up in complete remission at 12 month
252                             Participants who were lost to follow-up or did not provide biochemical va
253 ificantly more eyes in the aflibercept group were lost to follow-up within 12 months (21% vs. 9% rani
254 ere available for 406 (64%) participants, 25 were lost to follow-up, and two participants no longer w
255 onsent for participation in the trial or who were lost to follow-up, leaving a total of 680 patients,
256 eat population, excluding those patients who were lost to follow-up, with an equivalency test on the
257                                  No patients were lost to follow-up.
258                               No communities were lost to follow-up.
259 antation, 115 (31.9%) failed, and 83 (23.1%) were lost to follow-up.
260 an parent age, 31 [SD, 7] years), of whom 18 were lost to follow-up.
261 secondary neoplasms, 81% were alive, and 14% were lost to follow-up.
262 al demise, 672 pregnancy terminations and 87 were lost to follow-up.
263                                     164 (4%) were lost to follow-up.
264 participants did not proceed with surgery or were lost to follow-up.
265  (survival) was 69.3 months, and 14 patients were lost to follow-up.
266  with 104,966 patients, of whom 17,602 (17%) were lost to follow-up: Those lost to follow-up had medi
267      Her accomplishments and astounding life were lost to history for more than one hundred years and
268 ence of rising EBV DNA-emia, including 7 who were lost to long-term follow-up, with a number of them
269  [SD] age: 25.0 [35.8] months), and 76 (43%) were lost to tertiary follow-up (55% male; mean [SD] age
270 1).One-year post surgery, less RYGB-patients were lost-to follow-up (12.1% vs 16.5%, P < 0.001) and R
271            When fast auditory fiber activity is lost, tonic PV(+) interneuron activity is diminished,
272  of VISTA to restrain naive T cell responses was lost under inflammatory conditions.
273 pendent on lipid-free culture conditions and are lost upon lipid exposure, whereas short-term pharmac
274 y, the direct cell killing of mouse IgG3 mAb is lost upon chimerization or humanization.
275 re capable of bioluminescence, so the signal is lost upon ingestion.
276           The communication of these systems is lost upon isolation of the islets, and therefore the
277 e the endogenous ERbeta ligand, 3beta-Adiol, is lost upon long-term ADT, to obtain the beneficial eff
278  an IFN-I response in human monocytes, which is lost upon macrophage differentiation, and differs mec
279 e competitive advantage of hypervesiculation is lost upon pre-adaptation to bile and antimicrobial pe
280 ignal with the anti-Rac1-GTP antibody, which is lost upon silencing of vimentin expression.
281 hal infection in naive mice, full protection was lost upon neutrophil depletion.
282                        In agreement, pausing was lost upon replacement of the TFIID complex with TATA
283 y also arise about whether information could be lost when transitioning to a new technique.
284 information can be extracted from noise that is lost when averaging multiple measurements.
285  that although up to 70% of ecosystem carbon is lost when mangroves are converted to shrimp ponds, so
286 guided therapy, as observed in these trials, is lost when patients undergo surgery first.
287 om pathogen colonization, but the protection is lost when the ability to respire oxygen under micro-a
288                         However, this effect was lost when COX1 was deleted from both ECs and platele
289 d improved survival, whereas this protection was lost when Enos (also known as Nos3) was also deleted
290               This suppressive effect of EVs was lost when they were obtained from AMs exposed to all
291 f alternatively spliced genes, many of which were lost when ARGLU1 was absent.
292 of DR neurons (ventrolateral part of the DR) was lost, whereas the number of serotonergic neurons in
293 ts, a subset of pS22-Lamin A/C-binding sites were lost, whereas new pS22-Lamin A/C-binding sites emer
294 ic homogenization, where specialist endemics are lost while generalists persist.
295                  This compensatory mechanism is lost with acute irradiation injury, resulting in a pa
296                Despite this, KRT9 expression is lost with volar keratinocyte passaging, despite stabl
297 t promoters of stem/pluripotent genes, which was lost with endocrine therapy.
298 tissue-specific diurnal rhythms under LD and were lost with exposure to LLbright, except IL-6 in hypo
299 hibited abnormal dynamics in young mice that were lost with the P301L mutation, including elevated pr
300  initial conformations of small proteins can be lost within tens of milliseconds.

 
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