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1 te key disease features, and pathophysiology is poorly understood.
2 high rates of relative sea level rise (RSLR) is poorly understood.
3 the brain recovers from general anaesthesia is poorly understood.
4 ifferent groups are added to the ascarosides is poorly understood.
5 hemical transformations, the nature of which is poorly understood.
6 cell cycle(2-5) but how this is accomplished is poorly understood.
7 non-centrosomal MTOCs during differentiation is poorly understood.
8 m by which ethanol causes toxicity in muscle is poorly understood.
9 ep-ocean dissolved organic carbon (deep-DOC) is poorly understood.
10 w this variation affects microbial evolution is poorly understood.
11 antigenic variation of LPAIVs in wild birds is poorly understood.
12 ingencies, but its role in behavioral timing is poorly understood.
13 ced under pathologically relevant conditions is poorly understood.
14 ifferent VP cell types and their projections is poorly understood.
15 s encoded within RfaH sequence and structure is poorly understood.
16 es to these diverse IFN-alpha/beta functions is poorly understood.
17 a rare event which significance for immunity is poorly understood.
18 wever, the role of epsins in atherosclerosis is poorly understood.
19 ration of spindle elongation during anaphase is poorly understood.
20 nes which TF complex forms at any given site is poorly understood.
21 ive signaling pathways controlling cell fate is poorly understood.
22 istics of monsoonal precipitation and runoff is poorly understood.
23 ns, yet what governs their proper engagement is poorly understood.
24 use disorder (AUD) and other mental diseases is poorly understood.
25 tamin D status on the development of obesity is poorly understood.
26 ce or susceptibility to intestinal pathogens is poorly understood.
27 on the community structure of microorganisms is poorly understood.
28 , but how these are implemented in the brain is poorly understood.
29 tic basis for the superiority of these drugs is poorly understood.
30 vasive primary melanoma to an invasive state is poorly understood.
31 c organization is established and maintained is poorly understood.
32 nd mousepox, the mechanistic basis for which is poorly understood.
33 entially phosphorylating specific substrates is poorly understood.
34 ever, its role in driving disease phenotypes is poorly understood.
35 influence of seasons on biological processes is poorly understood.
36 rs regulate spinal network output in mammals is poorly understood.
37 uch regularities on decision-making strategy is poorly understood.
38 bution of this function to tumor suppression is poorly understood.
39 How polyphosphate potentiates pathogenicity is poorly understood.
40 The pathogenesis of this condition is poorly understood.
41 hogen growth in the microbiota-colonized gut is poorly understood.
42 have played in the evolution of bat immunity is poorly understood.
43 n's spatial framework to represent landmarks is poorly understood.
44 ) is one of these kinases, although its role is poorly understood.
45 nflammation in otherwise healthy individuals is poorly understood.
46 and their functions at a network-level scale is poorly understood.
47 ich preferentially induces A-to-C mutations, is poorly understood.
48 in combination with non-cytotoxic compounds is poorly understood.
49 how mammalian crossing over is accomplished is poorly understood.
50 signals to regulate interneuron development is poorly understood.
51 quired to sustain the Fe-scavenging strategy is poorly understood.
52 Yet the underlying mechanism is poorly understood.
53 throughout the plant water transport system is poorly understood.
54 al disequilibrium in the subgingival crevice is poorly understood.
55 get site accessibility is controlled in vivo is poorly understood.
56 rved conditions, how Leu regulates autophagy is poorly understood.
57 ir activity is coordinated with Gag assembly is poorly understood.
58 cause of diarrheal illness, but pathogenesis is poorly understood.
59 ts associated with these clinical conditions is poorly understood.
60 ls but their response to ocean acidification is poorly understood.
61 hip between ophthalmologic disorders and ASD is poorly understood.
62 ranching is coordinated with seasonal growth is poorly understood.
63 nd therefore extrasynaptic glutamate actions is poorly understood.
64 detects and responds to secondary structures is poorly understood.
65 s I MHC complex to the lysosomal compartment is poorly understood.
66 tivities and site-selectivities in chromatin is poorly understood.
67 demographics, in norovirus transmissibility is poorly understood.
68 o sustain epidemic transmission among humans is poorly understood.
69 l vulnerability in neurodegenerative disease is poorly understood.
70 ular and cellular regulation of this process is poorly understood.
71 sion is regulated during synaptic plasticity is poorly understood.
72 ssociated transitions in cell-cycle-dynamics is poorly understood.
73 ax6 achieves this deeply homologous function is poorly understood.
74 ly in the ISG family, the mechanism of which is poorly understood.
75 y of sliding and paraesophageal type hernias is poorly understood.
76 nterface with CMG to execute these functions is poorly understood.
77 mechanism of the illusory contour perception is poorly understood.
78 r amidase activation, but the process itself is poorly understood.
79 s diseases worldwide, yet how Las causes HLB is poorly understood.
80 the relative importance of these two factors is poorly understood.
81 atients' long-term risk after removal of SPs is poorly understood.
82 sinophil recruitment and contributing to AAI is poorly understood.
83 re that gives rise to these quantum emitters is poorly understood.
84 disparities in cancer incidence and outcome is poorly understood.
85 y and functional diversification of proteins is poorly understood.
86 date, and the natural history of the disease is poorly understood.
87 functionally assembled during embryogenesis is poorly understood.
88 ich these adaptors regulate dynein transport is poorly understood.
89 treatment failure for Mycoplasma genitalium is poorly understood.
90 ng motor neurons to OPCs with distinct fates is poorly understood.
91 the canine population and yet, its etiology is poorly understood.
92 rted into proliferation-quiescence decisions is poorly understood.
93 negative regulator of mTOR/protein kinase B, is poorly understood.
94 ever, its mechanism of transport into plants is poorly understood.
95 bial translocation, and inflammation in PHIV is poorly understood.
96 mplicated; however, the genetic architecture is poorly understood.
97 and how this may contribute to tumorigenesis is poorly understood.
98 variants on protein function and physiology is poorly understood.
99 igation is limited and functional importance is poorly understood.
100 signals to produce a given behavioral output is poorly understood.
101 bariatric surgery in this patient population is poorly understood.
102 omain dynamics and the function of a protein is poorly understood.
103 The etiology of autism spectrum disorder is poorly understood.
104 mal complex is assembled upon binding to IFN is poorly understood.
105 role of scaffold proteins in these processes is poorly understood.
106 s in mediating autophagy to suppress viruses is poorly understood.
107 entry, gene expression, assembly, and egress is poorly understood.
108 the pathophysiology of Lassa fever in humans is poorly understood.
109 ar fates are triggered by environmental cues is poorly understood.
110 ll-derived exosomes in metabolic homeostasis is poorly understood.
111 er, the role of the thalamus in this process is poorly understood.
112 cumulation is regulated or impacts longevity is poorly understood.
113 ef ecosystems, yet their formation mechanism is poorly understood.
114 ntarctic Ice Sheet (EAIS), to global warming is poorly understood.
115 ism of myosin 1s distinctive lipid targeting is poorly understood.
116 erm association of plasmids with their hosts is poorly understood.
117 ed to modulate specific biological functions is poorly understood.
118 ur in the brain as this learning takes place are poorly understood.
119 ational modifications, and disease phenotype are poorly understood.
120 d indefinitely, and the results of cessation are poorly understood.
121 the molecular determinants of its catabolism are poorly understood.
122 chronic neuropathic pain, but its mechanisms are poorly understood.
123 er it predicts the response to immunotherapy are poorly understood.
124 chanisms governing seeding in distal tissues are poorly understood.
125 sitional identity of these neural stem cells are poorly understood.
126 ive effects on males, but effects on females are poorly understood.
127 ction of GPR139 and its signaling mechanisms are poorly understood.
128 ion, distribution, metabolism, and excretion are poorly understood.
129 sses leading to the frequent outcome of ESKD are poorly understood.
130 3, is well supported but its effects on fish are poorly understood.
131 er and coordinate movement of multiple cells are poorly understood.
132 lmonellosis and leading Salmonella serotypes are poorly understood.
133 e underlying implications for agenda setting are poorly understood.
134 ular actors involved in COVID-19-driven ARDS are poorly understood.
135 echanisms underlying its antigenic diversity are poorly understood.
136 ng the regulation of glial and neuronal size are poorly understood.
137 tion levels, processing, and functionalities are poorly understood.
138 nd its distribution among gut microorganisms are poorly understood.
139 he development of the central nervous system are poorly understood.
140 ovements; however, the underlying mechanisms are poorly understood.
141 istinct, but their origins and relationships are poorly understood.
142 e in pancreatic ductal adenocarcinoma (PDAC) are poorly understood.
143 ntrolling organ size, especially in legumes, are poorly understood.
144 and of the transitions between these steps, are poorly understood.
145 assembly and maintenance by mechanisms that are poorly understood.
146 f cells to complex viscoelastic environments are poorly understood.
147 tion(1), although their biological functions are poorly understood.
148 however, their spatial and temporal dynamics are poorly understood.
149 but the mechanisms underlying these effects are poorly understood.
150 on, particularly across large spatial scales are poorly understood.
151 their origins and evolutionary relationships are poorly understood.
152 and elevation of intraocular pressure (IOP) are poorly understood.
153 cle and give rise to their unique properties are poorly understood.
154 es of intermediate filaments in this process are poorly understood.
155 he neural mechanisms underlying this process are poorly understood.
156 ife originates from a lifeless chemical soup are poorly understood.
157 onsequences of the various extracellular tau are poorly understood.
158 echanisms for early metastatic dissemination are poorly understood.
159 pearance of PAX7+ cells during embryogenesis are poorly understood.
160 is common, but its evolutionary consequences are poorly understood.
161 how it impacts flanking chromosomal regions are poorly understood.
162 es of shifts and their underlying mechanisms are poorly understood.
163 rix to reach taste receptors, processes that are poorly understood.
164 the pathological mechanisms governing TMJ OA are poorly understood.
165 GMP phosphodiesterases (PDEs) RmdA and RmdB, are poorly understood.
166 l communities and complex chemical processes are poorly understood.
167 tory mechanisms that underlie this selection are poorly understood.
168 ring cardiac neovascularization after injury are poorly understood.
169 castration-resistant prostate cancer (CRPC) are poorly understood.
170 eractions in the lung in the context of ARDS are poorly understood.
171 lations in these hard-to-access environments are poorly understood.
172 ticulates, yet subsequent removal mechanisms are poorly understood.
173 , the mechanisms underlying their maturation are poorly understood.
174 olecular mechanisms underlying these effects are poorly understood.
175 r progression, but the underlying mechanisms are poorly understood.
176 nderlying the pre-metastatic niche formation are poorly understood.
177 f beta-cell compensation to metabolic stress are poorly understood.
178 r HA traits necessary for pandemic potential are poorly understood.
179 clei fuse in the first mitosis of the embryo are poorly understood.
180 ctosylceramide, and the origin of psychosine are poorly understood.
181 sion and function of uPA in the mature brain are poorly understood.
182 e addiction propensity, but whose mechanisms are poorly understood.
183 enesis and underlying transcriptomic changes are poorly understood.
184 or offspring, but the underlying CNS changes are poorly understood.
185 chanisms of cell-stress-induced inflammation are poorly understood.
186 rivers on the persistence of soil carbon (C) are poorly understood.
187 et stimuli while ignoring distractor stimuli are poorly understood.
188 nal epithelial cell (IEC) barrier properties are poorly understood.
189 ion, including the role of the meat pathway, are poorly understood.
190 utionary dynamics of, the Cambrian Explosion are poorly understood.
191 le in coagulation and phenotypic composition are poorly understood.
192 the associated changes in the nervous system are poorly understood.
193 sequelae of infant C. difficile colonization are poorly understood.
194 ular pathways by which sleep need is encoded are poorly understood.
195 he removal of ubiquitin from target proteins are poorly understood.
196 )(6) but the underlying molecular mechanisms are poorly understood.
197 contributor to poor outcomes, yet its causes are poorly understood.
198 functional relevance of these modifications are poorly understood.
199 spatial positioning of each MICOS subcomplex are poorly understood.
200 mmunity and their interactions with bacteria are poorly understood.
201 (s) by which inflammation induces metaplasia are poorly understood.
202 lamic patterning and cell fate specification are poorly understood.
203 e brainstem pathways that drive MOC function are poorly understood.
204 factors that determine substrate reactivity are poorly understood.
205 uting factors and strategies to address this are poorly understood.
206 , but the cues promoting Trm cell generation are poorly understood.
207 t cooperate with known somatic driver events are poorly understood.
208 The mechanisms underlying this disruption are poorly understood.
209 ver, the mechanisms that govern this process are poorly understood.
210 g functional properties of cortical circuits are poorly understood.
211 s by which TBC1D23 regulates cargo transport are poorly understood.
212 omic events that drive metastatic recurrence are poorly understood.
213 chronic obstructive pulmonary disease (COPD) are poorly understood.
214 ell characterized, the underlying mechanisms are poorly understood.
215 in postoperative prescribing among providers are poorly understood.
216 owever, the patterns for grants and grantees are poorly understood.
217 sport and dyke propagation through the crust are poorly understood.
218 cal for its entry, replication, and assembly are poorly understood.
219 However, ASD pathogenic mechanisms are poorly understood.
220 swallowing performance, the mechanics of TBR are poorly understood.
221 but the underlying causes of this variation are poorly understood.
222 mechanical properties of the mineral phases are poorly understood.
223 impair NHE6 activity and endosomal function are poorly understood.
224 iron homeostatic mechanisms during pregnancy are poorly understood.
225 molecular mechanisms underlying ECM layering are poorly understood.
226 lying increased susceptibility to infections are poorly understood.
227 to water-stress are shaped by host genotype are poorly understood.
228 he forces driving their emergence and spread are poorly understood.
229 between specific dietary fats and cell fates are poorly understood.
230 ute nicotine withdrawal on cognitive control are poorly understood.
231 ion of epithelial cells to the host response are poorly understood.
232 tal factors to variation in immune responses are poorly understood.
233 e mechanisms underlying diverse morphologies are poorly understood.
234 omer-specific effects on receptor activation are poorly understood.
235 ue specificity of their function is achieved are poorly understood.
236 ical determinants of recognition specificity are poorly understood.
237 ndothelial-to-hematopoietic transition (EHT) are poorly understood.
238 g; however, the adaptive mechanisms involved are poorly understood.
239 early risk factors for adulthood fatty liver are poorly understood.
240 ional members of AGO, the functions of which are poorly understood.
241 tly, the structural basis of MraY inhibition was poorly understood.
246 sms by which SCN8A variants lead to epilepsy are poorly understood, although heterologous expression
247 neuronal cell type diversity within the VPH is poorly understood, an impediment to deconstructing th
250 eatment; the underlying molecular mechanisms are poorly understood and warrant investigation given th
251 molecular rules driving TCR cross-reactivity are poorly understood and, consequently, it is unclear t
252 e extracellular complex glycocalyx and mucus is poorly understood and a future biochemical challenge.
253 urobiological heterogeneity in schizophrenia is poorly understood and confounds current analyses.
254 n of bioactive small molecules from the diet is poorly understood and poses a substantial obstacle to
255 microbiological TCE and ClO(4)(-) reductions is poorly understood and seldom addressed in the literat
256 al functions and targets of DNA interference are poorly understood, and the mechanisms of DNA guide g
257 underlying the painful manifestations of PN are poorly understood, and therapies are restricted to u
258 photosensitive functions of Opn3 in mammals are poorly understood, and whether Opn3 has a role in fa
259 is, the etiology of the cognitive impairment is poorly understood, and no satisfactory cognitive trea
261 ss-of-function mutations in the gene ABCA12, is poorly understood, and to date, no satisfactory treat
262 the Polycomb system selects its target genes is poorly understood, and whether its histone-modifying
263 mic status (SES) in transplantation outcomes is poorly understood because of limitations of conventio
264 phagy) is implicated in human neuropathy but is poorly understood beyond a few autophagosomal recepto
265 t and space use on wildlife disease dynamics are poorly understood, but could help to predict disease
266 term health outcomes for children born by CS are poorly understood, but limited data suggest that CS
268 However, the molecular mechanisms in CRSsNP are poorly understood compared with those of polypoid CR
270 volution of stresses within volcanic systems is poorly understood, despite its importance for magma t
271 m aqueous film-forming foam (AFFF) ingestion is poorly understood due to the complexity of AFFF mixtu
272 stem cells (HSCs) within the leukemic niche are poorly understood, especially in the human context.
278 prion diseases or equivalent animal diseases are poorly understood, in part because cell models of sp
279 cteristics underlying this high productivity are poorly understood including the mode of photosynthes
280 such barrier, whose mechanism for overcoming is poorly understood, is access to recombination hot spo
281 , because the function of APE2 in DNA repair is poorly understood, it is unclear why BRCA-deficient c
283 mplex, which forms the core of the particle, is poorly understood, likely due to inherent dynamics.
284 dictate the metal utilized by metalloenzymes are poorly understood, limiting our ability to manipulat
285 y in regulating multiple ecosystem functions is poorly understood, limiting our ability to predict ho
286 by the ser/thr protein phosphatase 2A (PP2A) is poorly understood, limiting our understanding of PP2A
288 lization; however, its adaptive consequences are poorly understood, particularly among large mammalia
289 mpairment (MCI) and Alzheimer's disease (AD) is poorly understood, particularly at early stages prece
291 of spatial bias in hybridization efficiency is poorly understood, particularly in IHC and in-gel imm
292 a limited array of G proteins and effectors is poorly understood, particularly in native cell system
294 signaling network and thus their regulation is poorly understood; this also applies to all other euk
295 isms underlying this paradoxical association are poorly understood, we aimed to identify transcriptom
297 variation across and within vegetation types is poorly understood, which hinders our capacity to accu
298 l predictors of BE progression to HGD or EAC are poorly understood, with multiple contradictory studi
299 sex hormones affect non-reproductive organs is poorly understood, yet highly relevant to health give
300 urrently, the source of exported assemblages is poorly understood, yet this information may be critic