ライフサイエンスコーパス: be responsible for

1 lness in 9.2 to 35.6 million individuals and is responsible for 12,000 to 56,000 deaths.

2 ike 15 (METTL15), encoded by a nuclear gene, is responsible for 12S mt-rRNA methylation at m(4)C839 b

3 PCV13 serotypes were responsible for 20.1% (n = 4033), while serotype 8

4 Bacteria were responsible for 2202 infections (63%) prevailing th

5 433,371 SVs, from which we estimate that SVs are responsible for 25-29% of all rare protein-truncatin

6 presented only 12.3% of the 73 emm types and were responsible for 31.6% of the 411 clinical cases.

7 demonstrated that the HH103 rkpLMNOPQ operon is responsible for 5-acetamido-3,5,7,9-tetradeoxy-7-(3-h

8 of the MutLalpha subunit MLH1, whose defect is responsible for ~50% of dMMR cancers, results in loss

9 th Organization estimates that air pollution is responsible for 7 million deaths per annum, with 7% o

10 geochemical process in the carbon cycle that is responsible for 70% of global emissions of the potent

11 esses, including the 5'-3' exonuclease Xrn1, are responsible for a cross-talk between the two process

12 Emerging and reemerging viruses are responsible for a number of recent epidemic outbreak

13 robusta, showing that gene family expansions are responsible for a quarter of all 36,254 protein-codi

14 Mosquito-borne helminth infections are responsible for a significant worldwide disease burd

15 Adenoviruses are responsible for a spectrum of pathogenesis including

16 initial hope that a small set of genes would be responsible for a given disorder proved false.

17 upporting the possibility that ovulation may be responsible for a localized inflammatory response.

18 his previously unresolved N-terminal peptide is responsible for a ball-and-chain inactivation mechani

19 Dysregulation of the cGAS-STING pathway is responsible for a broad array of inflammatory and aut

20 e of death and morbidity, heart failure (HF) is responsible for a large portion of healthcare and dis

21 A Gly13 in the histone H3 tail is responsible for a sharp reduction in deacetylase acti

22 an extension of sensitivity and testosterone was responsible for a decrease in the duration of the hy

23 receptor has multiple methylation sites that are responsible for adaptation.

24 matopoietic stem cells (HSCs) are thought to be responsible for age-related changes in haematopoiesis

25 ed NTP-mediated excision mechanisms that may be responsible for aiding in maintaining fidelity (the f

26 2) groups on DA and the CF(2) groups on PVDF is responsible for aligning the PVDF chains and promotin

27 Expansions of simple tandem repeats are responsible for almost 50 human diseases, the majori

28 owth of the parasites during the blood stage is responsible for almost all malaria-associated morbidi

29 Thus, GluD1(R)-channels are responsible for alpha1-A(R)-dependent induction of p

30 Intracellular pathogens are responsible for an enormous amount of worldwide morb

31 These emissions are responsible for an increased number of days when the

32 ction with influenza A virus (IAV) infection is responsible for an estimated 500,000 deaths and up to

33 at the terminal hydroxyls on gamma-Al(2)O(3) are responsible for anchoring Ag species.

34 on holding potential) so this current could be responsible for AP activation during depolarization i

35 RAS G domain, its cysteine-rich domain (CRD) is responsible for association to anionic lipid-rich mem

36 IRT1, FoxO3a, AMPK, and PPAR-alpha signaling are responsible for autophagy impairment but mTOR is inv

37 Greater London reveals that these exchanges were responsible for avoiding emission of 87-156t of CO(

38 membrane of Gram-negative bacteria where it is responsible for barrier function(1,2).

39 NANOG, SOX2, and KLF4, which along with OCT4 are responsible for BCSC specification.

40 l for predicting which residues in a peptide are responsible for binding and for understanding the dr

41 tion of the N-terminal domains of OPG, which is responsible for binding to RANKL, the exact biologica

42 Our results suggest that the SUMO pathway is responsible for both repression of interrupted units

43 investigators concluded that the film weight is responsible for both the film collapse and wrinkling

44 The enzyme lipoprotein lipase (LPL) is responsible for breaking down triglycerides in the bl

45 mismatches in CTG trinucleotide repeats that are responsible for causing neurological diseases such a

46 und that the TLR2/6 heterodimer, not TLR2/1, is responsible for CDT recognition, and that gene pathwa

47 suggesting that differences in break sensing are responsible for cell cycle arrest variation.

48 V. cholerae serogroups O1 and O139 are responsible for cholera outbreaks in developing coun

49 46A1 (CYP46A1) or cholesterol-24-hydroxylase is responsible for cholesterol metabolism and homeostasi

50 (which, in conjunction with serum amyloid P, is responsible for clearing residual hyaluronan and cell

51 abilizing the reactive Rh(-I) species, which is responsible for cleaving the Ar-F bond and is ultimat

52 rons, the so-called concept cells (CCs), may be responsible for complex tasks performed by humans.

53 It is this dense layer that is responsible for conducting Li(+) and insulating elect

54 to FusB binding and show that these changes are responsible for conferring FA resistance.

55 In skin homeostasis, dermal fibroblasts are responsible for coordinating the migration and diffe

56 suggest that a mutation in the WFS1 gene may be responsible for corneal abnormalities similar to kera

57 SARS-CoV-2 is responsible for COVID-19, resulting in the largest pa

58 In humans, it is responsible for cryptococcosis, a deadly invasive fun

59 Molecular motors are known to be responsible for cytoskeletal ordering and force gener

60 Emerging infectious diseases are responsible for declines in wildlife populations aro

61 emonstrating that the CTNNBL1 M466V mutation is responsible for defective SHM induction.

62 inate, hydroxylinalool, and 2-phenyl ethanol was responsible for describing the BRS Rubea wines as fr

63 the first study demonstrating that BPSS2242 is responsible for detoxification of toxic metabolites,

64 hers, which mirror the ones found in mice to be responsible for different egg allergy risk.

65 and vascular and tissue sequestration, which is responsible for disease symptoms and complications, a

66 ced by polycomb repressive complex 2 (PRC2), is responsible for downregulating FBP1 in liver and kidn

67 typically acquired during tetraploidization are responsible for driving tumorigenesis.

68 a lasting increase in GABA release and this was responsible for driving the change in endocannabinoi

69 nctions of these subsets, with recruited MPs being responsible for efficient myelin stripping and cle

70 ith this tool, we show that vibration itself is responsible for eliciting much of the shaking signal'

71 This enzyme is responsible for elimination of unconjugated bilirubin

72 st that cis-regulatory elements in FaEnSpm-2 are responsible for enhanced MYB10-2 expression and anth

73 cation of O-fucose on EGF8 and -12 of NOTCH2 was responsible for enhancement of DLL1-NOTCH2 activatio

74 results suggest that inflammatory processes are responsible for epigenetic changes contributing to l

75 Activity of Na(+) K(+) -ATPase, which is responsible for establishing the sodium gradient nece

76 that global transcription at yeast promoters is responsible for eviction of H2A.Z.

77 tion (ORR), while the inner Ni-N(4) clusters are responsible for excellent activity toward the oxygen

78 intramolecular singlet fission (iSF) process is responsible for excited state deactivation in isoindi

79 We argue that a similar mechanism is responsible for exciting Saturn's hexagonal flow patt

80 The Dzyaloshinskii-Moriya interaction (DMI) is responsible for exotic chiral and topological magneti

81 n with no lysine (K) 1 (WNK1) and WNK4 genes are responsible for familial hyperkalemic hypertension (

82 (L), only one, a leucine to valine mutation, is responsible for fibril formation.

83 Subpopulations of ribosomes are responsible for fine tuning the control of protein s

84 nd concluded that sodium channel block alone is responsible for flecainide's efficacy in CPVT.

85 These TbD1-deleted "modern" lineages are responsible for globally-spread tuberculosis epidemi

86 is "fitness." A single mutation in the virus was responsible for greater fitness, enabling high growt

87 mycetes fungal species Colletotrichum, which is responsible for heavy yield losses in chilli and toma

88 conserved only in YfdC-alpha, were found to be responsible for high energy barriers for the anions t

89 of PKM2 with unchanged PKM1 expression might be responsible for higher pyruvate kinase activity in db

90 eveal that tandem condensin-histone activity is responsible for higher order chromosomal structures,

91 The 4-Cl substituent was responsible for higher rates of light absorption and

92 ownregulation of N/OFQ levels in the CeA may be responsible for hyper-GABAergic tone in the CeA that

93 the dynamic hot spots recently identified to be responsible for imatinib's high selectivity toward Ab

94 mentarity-determining region 3 (CDR3), which is responsible for imparting specific antibody-antigen i

95 ly unclear which bacterial virulence factors are responsible for increased clinical severity.

96 e observed previously that partial oxidation was responsible for increased TK(high) activity, while l

97 ch, if any, of these NET-affiliated proteins is responsible for inducing the metastatic phenotype.

98 owever, the Aspergillus fumigatus PAMPs that are responsible for inflammasome activation are not know

99 g that the EBV gL N-glycosylation site might be responsible for inhibiting the binding of gH/gL to Ep

100 rather than inhibitory receptor expression, was responsible for inhibition of T-cell proliferation.

101 s, rather than plasmacytoid dendritic cells, are responsible for interferon production prior to clini

102 We identify SMAX1 domains that are responsible for its nuclear localization, KAR-induce

103 veal that just 70-140 neurons in this region are responsible for its sensitivity to opioids.

104 CYP102A1 architecture has been postulated to be responsible for its extraordinary catalytic prowess.

105 h pattern of mesenchymal subtype tumor cells is responsible for its poor outcome.

106 AK3 in a myristoylation-dependent manner and is responsible for its targeting to and enrichment in th

107 A C-terminal IQGAP1 region was responsible for its association with OX40, and TNFR-

108 ,991 MtC) of the region's carbon in 2016 but were responsible for just 10% (-130 MtC) of the net chan

109 ication, DNA polymerase epsilon (Polepsilon) is responsible for leading strand synthesis, whereas DNA

110 mechanism (hippocampal theta-band activity) is responsible for learning in these settings, whereas a

111 ge frugivores, such as tapirs and peccaries, were responsible for less than 21% of frugivory events.

112 The peptidoglycan layer is responsible for maintaining bacterial cell shape and

113 um verticillioides is a fungal pathogen that is responsible for maize ear rot and stalk rot diseases

114 Associated aberrant signaling is responsible for many forms of cancer and disorders of

115 rane tension caused by the adsorption of NPs is responsible for mechanical deformation, leading to ce

116 d suggests that tumor-derived exosomes could be responsible for mediating systemic immunosuppression

117 n binding demonstrated the Valphabeta-domain is responsible for MHC-independent B12A recognition of i

118 sative agent of pulmonary tuberculosis (TB), is responsible for millions of infections and deaths ann

119 piratory syndrome coronavirus 2 (SARS-CoV-2) is responsible for millions of infections and hundreds o

120 ncestral genome, horizontally acquired genes are responsible for more crucial abilities, including vi

121 RNA repeat expansions are responsible for more than 30 incurable diseases.

122 ne diseases, such as dengue and chikungunya, are responsible for more than 50 million infections worl

123 ispensable for a sustained process, where it is responsible for more than 92% of the final H(2) yield

124 in a variety of plant genera, the genes that are responsible for morphological differences remain mos

125 The affluent citizens of the world are responsible for most environmental impacts and are c

126 Peanut, tree nut, and sesame allergies are responsible for most life-threatening food-induced a

127 strated that variations in noncoding regions are responsible for most of the common variation heritab

128 the analysis of KO cells suggested that TET2 is responsible for most 5fC formation.

129 of pharmacological inhibition of p53, which is responsible for motor neuron death and 4-AP, results

130 logical systems, these fascinating molecules are responsible for movement of molecules, organelles, c

131 pportunistic pathogen Pseudomonas aeruginosa is responsible for much of the morbidity and mortality a

132 uronal gene transcription repressor protein, is responsible for NE gene expression in infected endoth

133 iency virus type 1 (HIV-1) via breastfeeding is responsible for nearly half of new infections of chil

134 s two proteins, NPC1 and NPC2, whose defects are responsible for Niemann-Pick disease type C (NPC).

135 idues (triK), which were previously shown to be responsible for nonspecific binding to DNA.

136 Hematopoiesis is responsible for numerous functions, ranging from oxyg

137 The inferotemporal (IT) cortex is responsible for object recognition, but it is unclear

138 tiple factors in host-like environments that are responsible for observed changes in susceptibility.

139 iable linker region, and a hub domain, which is responsible for oligomerization.

140 CD4(+) Th cells are responsible for orchestrating diverse, pathogen-spec

141 nuclear factor kappaB ligand (RANKL), which is responsible for osteoclast differentiation and activa

142 This clade has been responsible for outbreaks of urethritis in multiple

143 Repeat expansions are responsible for over 40 monogenic disorders, and und

144 oxidative stress in Atg7 (Delta/Delta) mice was responsible for p53 activation, Atg7 was deleted in

145 dioactive nuclides on particle surfaces, may be responsible for part of PM toxicity.

146 find that variation in a single gene, MPK4, is responsible for population overyielding through a mec

147 erstone of the human adaptive immune system, being responsible for processing antigens that allow kil

148 ells (NSCs) are multipotent progenitors that are responsible for producing all of the neurons and mac

149 mor necrosis factor alpha converting enzyme) is responsible for producing active EGFR family ligands

150 d that a proteinaceous factor other than NOS is responsible for producing diformazan in aldehyde-trea

151 virus infectivity controller 1 (vic1) locus, is responsible for production of antiretrovirus Abs in I

152 e to the transmembrane subunits are found to be responsible for proton pumping.

153 Stereochemical configuration of the drug is responsible for racemic switch with enantiomers in pr

154 ponent within the DNA damage response, as it is responsible for recognizing and repairing double-stra

155 The serine-threonine kinase PINK1 is responsible for recruiting Parkin to mitochondria, bu

156 substrate binding protein (SBP) MppA, which is responsible for recycling peptidoglycan fragments in

157 Histone deacetylase 1 (HDAC1) and HDAC2 are responsible for reducing H4 acetylation as chromatin

158 The catalytic synergy is responsible for reduction of the oxidation temperatur

159 in a transcriptional activator (Aro80) that is responsible for regulating the use of aromatic amino

160 such as hyaluronidase 1 and hyaluronidase 2, are responsible for regulation of the glycocalyx thickne

161 cilitates the complete maturation of VI, and is responsible for releasing the lytic protein from the

162 Notably, we found that carboxypeptidase D is responsible for removal of a C-terminal RKRR motif(2)

163 of the foot-and-mouth disease virus (FMDV), is responsible for replication of viral genomes.

164 ce for a common remapping mechanism that may be responsible for representing stimulus identity and st

165 amines nor thyroid-stimulating hormone (TSH) are responsible for sarcolipin expression or FAO stimula

166 elix-turn-helix conformation of the MPER-TMD is responsible for sequestering cholesterol.

167 omeostasis and alterations in this mechanism are responsible for several pathological conditions, inc

168 very strong basal expression of STAT6, which is responsible for signaling following exposure to IL-4

169 ed that plants but not microbial communities were responsible for significantly higher soil respirati

170 /Tolloid family metalloprotease Tolkin (Tok) is responsible for Slit proteolysis in vivo and in vitro

171 uggest that different isoforms are likely to be responsible for some of the variation observed in FA

172 ction through exposure to an exogenous virus is responsible for some cases of nonprimary maternal inf

173 ntity and role of cell surface proteins that are responsible for sperm-egg recognition, binding, and

174 y mesenchyme cells (PMCs) are the cells that are responsible for spicule formation.

175 t the increased Notch3 signaling in mgR mice was responsible for structure alterations in the lungs,

176 s the critical Dab2 residues within SBP that are responsible for sulfatide binding.

177 tinction, a type of inhibitory learning that is responsible for suppressing learned behaviors when th

178 The TLM indicates that hydrolyzed adsorbates are responsible for surface-potential inversions, and we

179 filamentous actin (F-actin)-binding domains are responsible for Tarp-mediated invasion efficiency.

180 tentials, further suggesting that those taxa are responsible for the bioelectroactivity.

181 niform structures and identify the ones that are responsible for the catalyst performance is a crucia

182 Fungal diseases are responsible for the deaths of over 1.5 million peopl

183 that Tyr-1292 and Tyr-1387 but not Tyr-1325 are responsible for the effect of leptin.

184 Antibiotic use and bacterial transmission are responsible for the emergence, spread and persistenc

185 Particularly, it is unclear which defects are responsible for the emergent magnetic interfaces.

186 ria: small iterative monomodular T1PKSs that are responsible for the enormously diverse fungal natura

187 stimulation field; and which brain networks are responsible for the evolution of these symptoms?

188 eacting with MA(+) in the precursor solution are responsible for the formation of gap states.

189 utions of different signaling pathways which are responsible for the generation of the innate immune

190 r results show that both OATP1B1 and OATP1B3 are responsible for the hepatic uptake of major circulat

191 These compounds are responsible for the high durability of wood and may

192 Cities are responsible for the largest anthropogenic CO(2) emis

193 MYB, RET, KRT8, MUC1, and ERBB2-genes which are responsible for the luminal signature in breast canc

194 asis and emergence of therapeutic resistance are responsible for the majority of prostate cancer mort

195 icate that divergent pathways from the avBST are responsible for the mnemonic effects of avBST inhibi

196 es in firing, confirming that these channels are responsible for the molecular adaptation of ChIs to

197 not only initiate the rearrangement but also are responsible for the nature of substituents at positi

198 main regarding the molecular mechanisms that are responsible for the pathological calcium release, re

199 identified as FN1(+) mesenchymal cells which are responsible for the precocious astrogenesis.

200 sidues in the UGT2B7 cofactor binding domain are responsible for the preferential binding of UDP-GlcU

201 to explore the immunological components that are responsible for the proliferative alterations in the

202 nce of regulatory agencies and local factors are responsible for the reduction and recovery.

203 mes, and show that a number of these enzymes are responsible for the selective addition of specific m

204 that changes in Ca(2+) and Na(+) homeostasis are responsible for the surprisingly modest action poten

205 domain-containing F-box (KFB) proteins that are responsible for the ubiquitination and degradation o

206 Coding variants in TMEM175 are likely to be responsible for the association in the TMEM175/GAK/DG

207 nd South America, and its oral secretion may be responsible for the damage it causes in soybean (Glyc

208 nstrated epigenetic modifications that might be responsible for the different organoid gene expressio

209 tigen receptor (TCR)-dependent mechanism may be responsible for the earliest T(FH) cell-fate decision

210 to be a direct target of miR-3085-3p and may be responsible for the early inhibition of NFkappaB sign

211 candidates, whose product dysfunction might be responsible for the ectopic distribution of CSF-cNs.

212 e low-index facets investigated so far could be responsible for the experimentally observed OER activ

213 The C-terminal tail of CENH3 is confirmed to be responsible for the interaction of CENH3 and histone

214 or generated by triggered El Nino events may be responsible for the late winter weakening of the Aleu

215 Leaf trichomes on soybeans were not found to be responsible for the observed increase of insecticidal

216 ms in caries development are hypothesized to be responsible for the observed population distributions

217 by the same Co(-I) active species appears to be responsible for the observed Z-selectivity.

218 rt for the hypothesis that hybridisation may be responsible for the occurrence of nonmonophyletic mul

219 ermediate volatility organic compounds could be responsible for the rest.

220 Ubiquitous pathological triggers cannot be responsible for the selective neuron loss.

221 emained unclear what physical mechanisms can be responsible for the spatial stability of the magnon c

222 ve agent of typhoid fever, and is thought to be responsible for the symptoms of severe disease.

223 , further confirming the oxygen vacancies as being responsible for the memristive behavior.

224 O-GlcNAc transferase (OGT) is responsible for the addition of GlcNAc moieties to ta

225 ata suggested that a nucleation-growth model is responsible for the adsorption-induced squeeze-out of

226 at the heterodimeric FLA8/10 kinesin-2 alone is responsible for the atypically fast IFT in C. reinhar

227 ARS-CoV-2) emerged in December 2019(1,2) and is responsible for the coronavirus disease 2019 (COVID-1

228 y syndrome coronavirus 2 (SARS-CoV-2), which is responsible for the current coronavirus disease 2019

229 Histone acetyltransferase 1 (HAT1) is responsible for the cytosolic diacetylation of newly

230 a mitochondrial-associated gene (Mrpl3) that is responsible for the decrepit phenotype.

231 SARS-CoV-2 is responsible for the development of coronavirus diseas

232 6-methylguanine-DNA methyltransferase (MGMT) is responsible for the direct repair of the main TMZ-ind

233 the intoxicating constituent of cannabis and is responsible for the drug's reinforcing effects.

234 portant process in nature and technology and is responsible for the emergence of exotic functionaliti

235 reduction with increasing disk rotation rate is responsible for the enhanced CO2RR selectivity.

236 Cytochrome P450 2J2 (CYP2J2) is responsible for the epoxidation of endogenous arachid

237 nd no evidence that a single receptor domain is responsible for the Fls3-sustained reactive oxygen sp

238 C-terminal leucine rich repeat (LRR) domain is responsible for the gain of powdery mildew resistance

239 acture of products from jabuticaba fruit, it is responsible for the generation of large amounts of re

240 re acute respiratory syndrome coronavirus 2) is responsible for the global coronavirus disease 2019 p

241 ily of hepatic cytochrome P450 (CYP) enzymes is responsible for the intrinsic clearance of the majori

242 uct in commercial formulations of rigosertib is responsible for the microtubule-destabilizing activit

243 Dengue virus (DENV) is responsible for the most prevalent and significant ar

244 The receptor CRM1 is responsible for the nuclear export of many tumor-supp

245 own to be able to create a twist moment that is responsible for the observed non-classical morphologi

246 t the energy barrier of the phase transition is responsible for the observed thickness beyond the pre

247 literature) about the SARS-CoV-2 virus that is responsible for the pandemic.

248 ), a novel evolutionary divergent RNA virus, is responsible for the present devastating COVID-19 pand

249 , located downstream of the luxS gene, which is responsible for the production of the quorum sensing

250 ated, and the N-H proton of the aniline ring is responsible for the protonophore activity.

251 ling, likely photoinduced electron transfer, is responsible for the quenching effect of the dihydropy

252 uris, a multidrug-resistant fungal pathogen, is responsible for the recent global outbreaks in hospit

253 entify a specific sequence within Abeta that is responsible for the recognition of the peptide by PrP

254 d deduced a dual function in which carbonate is responsible for the removal of free sulfur dioxide fr

255 toreceptor phosphodiesterase (PDE6) activity is responsible for the speed, sensitivity, and recovery

256 final proton transfer step in the mechanism is responsible for the stereoselectivity observed in exp

257 ls from the primary tumor to distant organs, is responsible for the vast majority of cancer deaths.

258 rotein (vRNP) of the influenza A virus (IAV) is responsible for the viral RNA transcription and repli

259 Regardless of age, pyrazinamide was responsible for the majority of adverse reactions.

260 This butyrate concentration was responsible for the maximum modulatory effects obser

261 ich was affected by the solute concentration was responsible for the velocity difference.

262 c, malic, acetic, gluconic, and lactic acids were responsible for the differentiation between these t

263 at substances released from schistosome eggs were responsible for the observed effects.

264 ded carbon regions of the BDD electrode that were responsible for the voltammetric pH signal.

265 hidden structure, patterns, and motifs that are responsible for their behaviors.

266 motif that is found in two cancer drugs may be responsible for their ability to tackle cancers and f

267 Abnormal vascular development is likely to be responsible for their association.

268 d several other drugs undergoing testing may be responsible for their demonstrated in vitro antiviral

269 on to wild-type plants, indicating that SOT1 is responsible for their formation in planta.

270 beta-catenin pathway in periportal HPCs and was responsible for their expansion and de-differentiati

271 er explore the different driving forces that are responsible for these interactions.

272 The Malat1 RNA is responsible for these effects, as antisense oligonucl

273 The faster action of anti-VEGF agents likely is responsible for these findings.

274 Specific neurotransmitter transporters are responsible for this activity and therefore are esse

275 e junctions at the edges of convection cells are responsible for this behaviour for [Formula: see tex

276 Pathogenic T(H)17 responses are responsible for this neoantigen-induced tumor progre

277 lar, ecological, and evolutionary mechanisms are responsible for this partial reproductive isolation

278 orsional strains present in the trans isomer are responsible for this unexpected relative cis/trans s

279 More than 20 genes are, so far, known to be responsible for this condition.

280 sis that similar biomolecular mechanisms may be responsible for this highly probable electroautotroph

281 n of VCAM1 and hypothesized that LL-37 could be responsible for this response.

282 le spillover event into the human population is responsible for this DRC outbreak.

283 trate that oxoglutarate dehydrogenase (OGDH) is responsible for this remaining glutarylcarnitine prod

284 Brain imaging revealed that the hippocampus was responsible for this interference between statistica

285 TETS) is a so-called "caged" convulsant that is responsible for thousands of accidental and malicious

286 cular crowding resulting from water loss may be responsible for three observed phenomena: a general a

287 RNA-dependent RNA polymerase (FluPol), which is responsible for transcribing and replicating the nega

288 s of the GRN and hypothesize that one module is responsible for translating the extracellular protein

289 re central targets of this response, as they are responsible for translation and subject to lysosomal

290 ader regions of regulated genes and shown to be responsible for translational control.

291 We conclude GmVTL1a is responsible for transport of iron across the SM to ba

292 nses are specific for each patient and could be responsible for treatment failure despite multi-targe

293 aride is recognized by receptors on KCs that are responsible for triggering several pathways that act

294 erall results suggest that 3-mercaptohexanol is responsible for tropical/citrus fruit, TDN for kerose

295 In addition to being responsible for tumorigenesis, CSCs exhibit elevat

296 e with dementia, with Alzheimer disease (AD) being responsible for two-thirds of the total cases.

297 rus replication.IMPORTANCE Influenza viruses are responsible for up to 650,000 deaths per year throug

298 Visceral leishmaniasis is responsible for up to 30,000 deaths every year.

299 Fibrosis can affect any organ and is responsible for up to 45% of all deaths in the indust

300 Bluetongue virus (BTV) epidemics are responsible for worldwide economic losses of up to U