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1 eability and active efflux of ampicillin and benzylpenicillin.
2 of beta-lactamase from Escherichia coli with benzylpenicillin.
3 yperbolic dependence on the concentration of benzylpenicillin.
4 -Ala-D-Ala-carboxypeptidase interacting with benzylpenicillin.
5   The limit of quantitation was 10mug/kg for benzylpenicillin, 20mug/kg for cloxacillin, 25mug/kg dic
6 nt than the position observed in the E. coli benzylpenicillin acyl-enzyme complex.
7 attached to the cleaved beta-lactam rings of benzylpenicillin and cephaloridine are located in a simi
8 a-lactam ring, when the rigid fused rings of benzylpenicillin and cephaloridine each form different i
9             Modeled Michaelis complexes with benzylpenicillin and cephaloridine show that the perturb
10  complexes with the traditional beta-lactams benzylpenicillin and cephalosporin C were determined for
11 is and initiation of antibiotic therapy with benzylpenicillin and clindamycin as seen in this case is
12 ed in 90 (12%) infants who received procaine benzylpenicillin and gentamicin (reference), 76 (10%) of
13  a day for 7 days; or intramuscular procaine benzylpenicillin and gentamicin once a day for 2 days fo
14 ned infants to either intramuscular procaine benzylpenicillin and gentamicin once a day for 7 days (r
15 r 7 days (group B) or intramuscular procaine benzylpenicillin and gentamicin once per day for 2 days,
16 tandard treatment was intramuscular procaine benzylpenicillin and gentamicin once per day for 7 days
17 assigned the reference treatment of procaine benzylpenicillin and gentamicin, 816 (751 per protocol)
18 arted on a standard antimicrobial regimen of benzylpenicillin and gentamicin.
19 sequently, the kcat values for hydrolysis of benzylpenicillin and nitrocefin have been reduced by 10(
20 uctures of two acyl-enzyme adducts, one with benzylpenicillin and the other with cephaloridine, have
21 ch group, some infants received antibiotics (benzylpenicillin and/or gentamicin), while others did no
22 ree and substrate-bound forms of TEM-1 (with benzylpenicillin) and PSE-4 (with carbenicillin) were re
23    It exhibits no detectable activity toward benzylpenicillin, and 10(5)-fold reduction of kcat for n
24      In both cases, the catalytic rates with benzylpenicillin are reduced by 10(4) compared with the
25 in the literature and (with the exception of benzylpenicillin) are less than the maximum residue limi
26 peptidomics approach was applied to identify benzylpenicillin (BP)-modified peptide ligands within th
27            The kinetics of the hydrolysis of benzylpenicillin catalyzed by cobalt substituted beta-la
28 d BcII, the pH dependence of k(cat)/K(m) for benzylpenicillin, cephalexin, and cefoxitin similarly in
29 triaxone, lidocaine, omalizumab, cefuroxime, benzylpenicillin, clindamycin, amoxicillin/clavulanate,
30 e was made, and treatment was initiated with benzylpenicillin, clindamycin, metronidazole and vancomy
31 tion of four penicillin antibiotic residues (benzylpenicillin, cloxacillin, dicloxacillin and oxacill
32 were stimulated once a week for 4 weeks with benzylpenicillin coupled to human serum albumin.
33 ature autologous dendritic cells loaded with benzylpenicillin coupled to human serum albumin.
34 ctive efflux by AcrAB was more effective for benzylpenicillin due to the stronger affinity and high d
35 illin G benzathine (also known as benzathine benzylpenicillin) every 4 weeks for 2 years or no prophy
36      The fairly high Kd value indicates that benzylpenicillin fits rather poorly into the protein act
37  and 99 (13%) of those treated with procaine benzylpenicillin, gentamicin, and amoxicillin (risk diff
38 17 (753 per protocol) were assigned procaine benzylpenicillin, gentamicin, and amoxicillin.
39  1170 infants to receive injectable procaine benzylpenicillin-gentamicin and 1163 infants to receive
40 was as efficacious as an injectable procaine benzylpenicillin-gentamicin combination for 7 days for s
41 in for 7 days (group B); injectable procaine benzylpenicillin-gentamicin for 2 days, then oral amoxic
42 cated infants to receive injectable procaine benzylpenicillin-gentamicin for 7 days (group A, referen
43 ens were as effective as injectable procaine benzylpenicillin-gentamicin for 7 days on an outpatient
44 dequate follow-up in the injectable procaine benzylpenicillin-gentamicin group and 1145 (98%) infants
45                              In the procaine benzylpenicillin-gentamicin group, 234 infants (22%) fai
46 pital, to receive either injectable procaine benzylpenicillin-gentamicin once per day or oral amoxici
47 us as the combination of injectable procaine benzylpenicillin-gentamicin.
48 y ceftriaxone (46%), metronidazole (28%), or benzylpenicillin-gentamycin (23%).
49 intermittently with a subtherapeutic dose of benzylpenicillin had significantly more colony-forming u
50 ce of naive CD4(+) T lymphocytes specific to benzylpenicillin in healthy donors.
51 antibiotic resistance to either multidrug or benzylpenicillin in S. aureus isolates.
52                                Resistance to benzylpenicillin increased slightly but significantly (p
53                     The carboxylate group of benzylpenicillin interacts with the side chain of Gln237
54  in the k2/Kd value as compared with that of benzylpenicillin is mostly attributable to the decreased
55 ly, product complex structures of hydrolyzed benzylpenicillin-, methicillin-, and oxacillin-bound NDM
56 strated high-level resistance to penicillin (benzylpenicillin MIC >= 256 mug/ml), beta-lactam/beta-la
57 strated high-level resistance to penicillin (benzylpenicillin minimum inhibitory concentration [MIC]
58 affected by prior acylation of the enzyme by benzylpenicillin; nor did it inhibit reaction at that si
59 ith psr did not modify the susceptibility to benzylpenicillin or the growth and cell autolysis rates.
60 n of women who had treatment with benzathine benzylpenicillin out of those who tested positive for sy
61 how a trend that explains the preference for benzylpenicillin over cephaloridine in the class A beta-
62 e beta-lactam antibiotics, ampicillin (AMP), benzylpenicillin (PEG), cephalexin (CFX), cefazolin (CFL
63 alyses the cleavage of the amide bond in the benzylpenicillin (penicillin G) side-chain to produce ph
64 tir bar sorptive extraction and detection of benzylpenicillin (penicillin G, PEN G).
65                                      For the benzylpenicillin phenotyping analysis, RBF SVM, MLP neur
66 tine sputum culture, and that treatment with benzylpenicillin remains appropriate for clinical failur
67 ing the previous four, were able to classify benzylpenicillin resistant and susceptible strains with
68 GSH restores defective swarming motility and benzylpenicillin sensitivity in a cydD mutant and also b
69 cillin sensitivity in a cydD mutant and also benzylpenicillin sensitivity in a gshA mutant defective
70 er rate constants (ka) of PBP2a acylation by benzylpenicillin showed a hyperbolic dependence on the c
71 (+) ) CD4(+) T lymphocytes showed that these benzylpenicillin-specific CD4(+) T lymphocytes belonged
72               Results showed the presence of benzylpenicillin-specific CD4(+) T lymphocytes in 9 of 1
73  this study was to evaluate the frequency of benzylpenicillin-specific CD4(+) T lymphocytes in health
74                                 Frequency of benzylpenicillin-specific naive CD4(+) T lymphocytes was
75                                      For the benzylpenicillin the diagnostic systems achieved up to (
76 stered intermittent subtherapeutic dosing of benzylpenicillin, the strain with a Pro601Leu amino acid
77 , we have been able to convert penicillin G (benzylpenicillin) to deacetoxycephalosporin G.
78 ains had identical fitness in the absence of benzylpenicillin treatment.
79 own number of bacteria radiolabeled with [3H]benzylpenicillin were separated by sodium dodecyl sulfat
80 th pneumococci of intermediate resistance to benzylpenicillin, which comprised 28% of pneumococcal is