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1 mmation in the lung of patients with chronic beryllium disease.
2 ould serve as a possible therapy for chronic beryllium disease.
3 ify both beryllium sensitization and chronic beryllium disease.
4 at position 69 (HLA-DPB1(Glu69)) in chronic beryllium disease.
5 response to beryllium and in progression to beryllium disease.
6 orted to be strongly associated with chronic beryllium disease.
8 osure in the workplace can result in chronic beryllium disease, a granulomatous lung disorder charact
9 for sarcoidosis and, in contrast to chronic beryllium disease, a non-E(69)-containing allele, HLA-DP
10 1) idiopathic pulmonary fibrosis, 2) chronic beryllium disease and sarcoidosis, 3) control subjects w
12 time between those who progressed to chronic beryllium disease and those who remained sensitized with
13 mong BAL cells of patients with sarcoidosis, beryllium disease, and hypersensitivity pneumonitis.
15 biopsies to determine progression to chronic beryllium disease as evidenced by granulomatous inflamma
16 LA DPB1 locus, a SNP associated with chronic beryllium disease, as well as HLA DPA1 alleles using the
17 ls with beryllium sensitization have chronic beryllium disease at the time of their initial clinical
18 me (ACE) genotype is associated with chronic beryllium disease (CBD) and disease severity, we studied
19 B1 gene (Glu(69)) is associated with chronic beryllium disease (CBD) and possibly beryllium sensitiza
20 ic and clinical similarities between chronic beryllium disease (CBD) and sarcoidosis suggest that sim
21 suggests a genetic predisposition to chronic beryllium disease (CBD) and sarcoidosis, which are clini
39 rom HLA-DP2-expressing patients with chronic beryllium disease (CBD), a debilitating granulomatous lu
40 CII allele, HLA-DP2, are at risk for chronic beryllium disease (CBD), a debilitating inflammatory lun
41 ecific granulomatous inflammation of chronic beryllium disease (CBD), and compared it with that in he
42 ies demonstrate associations between chronic beryllium disease (CBD), beryllium sensitization (BeS),
43 beryllium-specific CD4+ T cells and chronic beryllium disease (CBD), which is characterized by the p
52 in idiopathic pulmonary fibrosis and chronic beryllium disease lung tissues and interacted with hepar
54 ryllium-specific CD4(+) T cells from chronic beryllium disease patients remain CD28-dependent, while
55 lar lavage (BAL) CD4(+) T cells from chronic beryllium disease patients to identify possible therapeu
56 blood and bronchoalveolar lavage of chronic beryllium disease patients up-regulate 4-1BB expression,
57 cell lines derived from the lungs of chronic beryllium disease patients, beryllium presentation to th
58 (+) T cells recruited to the lung in chronic beryllium disease recognize beryllium in an Ag-specific