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1 g treatment with 50 nM Compound 49b, a novel beta-adrenergic receptor agonist.
2 This is reversed by isoproterenol, a beta-adrenergic receptor agonist.
3 or-mediated activation with isoproterenol, a beta-adrenergic receptor agonist.
4 ve agonist, or isoprenaline, a non-selective beta-adrenergic receptor agonist.
5 also sensitizes the channel to activation by beta-adrenergic receptor agonists.
6 was threefold greater than that elicited by beta-adrenergic receptor agonists.
7 solated neonatal cardiac myocytes exposed to beta-adrenergic receptor agonists.
8 F by preventing enhanced atrial responses to beta-adrenergic receptor agonists.
9 n the myocardium to exogenous and endogenous beta-adrenergic receptor agonists after burn injury may
10 haracteristics in response to treatment with beta-adrenergic receptor agonists and activators of aden
11 everal currently available therapies such as beta-adrenergic receptor agonists and antagonists, phosp
13 all measured in response to isoproterenol, a beta-adrenergic receptor agonist, and carbachol, a choli
15 sed to cold temperatures or treated with the beta-adrenergic receptor agonist CL316,243 and that its
18 ion can inhibit the Cl- current activated by beta-adrenergic receptor agonists in guinea-pig ventricu
22 diated electrophysiological responses to the beta-adrenergic receptor agonist isoprenaline (Iso) in C
23 n/Hsp27 complex in response to the selective beta adrenergic receptor agonist isoproterenol, was subs
24 following intra-LS injections of either the beta-adrenergic receptor agonist isoproterenol (10 mug o
28 ultures, we show that norepinephrine and the beta-adrenergic receptor agonist isoproterenol also inhi
29 ction were conducted using forskolin and the beta-adrenergic receptor agonist isoproterenol as agonis
30 er administration of either the nonselective beta-adrenergic receptor agonist isoproterenol or the be
32 ions, ATP and UTP were not additive with the beta-adrenergic receptor agonist isoproterenol, but were
33 tion, either alone or in the presence of the beta-adrenergic receptor agonist isoproterenol, failed t
34 during periodic pacing in the presence of a beta-adrenergic receptor agonist isoproterenol, was sign
35 ibited enhanced inotropic sensitivity to the beta-adrenergic receptor agonist isoproterenol, with imp
40 es were incubated with forskolin or with the beta-adrenergic receptor agonist, isoproterenol, to stim
42 following administration of isoproterenol, a beta-adrenergic receptor agonist known to induce cardiac
44 activity by forskolin, cAMP analogs, or the beta-adrenergic receptor agonists norepinephrine and iso
46 ce remained responsive to stimulation by the beta-adrenergic receptor agonist, (S)-isoproterenol.
49 change after treatment with isoproterenol, a beta-adrenergic receptor agonist that causes turnover of
51 ary epithelial bilayer, when stimulated with beta adrenergic receptor agonists, vasointestinal peptid
52 reated some cells with Compound 49b, a novel beta-adrenergic receptor agonist we have reported previo
53 ions of isoproterenol (a peripherally-acting beta-adrenergic receptor agonist) were administered in a
54 rly phase of LTP by pairing isoproterenol, a beta-adrenergic receptor agonist, with a weak train, sub