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1 ion of new crystalline phases not present in beta-cyclodextrin.
2 g a high-surface-area, mesoporous polymer of beta-cyclodextrin.
3 an be prepared from various omega-3 oils and beta-cyclodextrin.
4 mutiwalled carbon nanotubes assimilated with beta-cyclodextrin.
5 ation of aqueous mixtures of omega-3 oil and beta-cyclodextrin.
6 id rafts, similar to positive control methyl-beta-cyclodextrin.
7 rbed from the film and then solubilized with beta-cyclodextrin.
8 aromatic rings of curcumin and the cavity of beta-cyclodextrin.
9 olubility by increasing the concentration of beta-cyclodextrin.
10 uperior host-guest interaction capability of beta-cyclodextrin.
11 after disruption of lipid rafts with methyl-beta-cyclodextrin.
12 epletion fully reversed the effect of methyl beta-cyclodextrin.
13 an aryl sulfonate to promote inclusion into beta-cyclodextrin.
14 eated with 7KCh solubilized in hydroxypropyl-beta-cyclodextrin.
15 following disruption of caveolae with methyl-beta-cyclodextrin.
16 ter incubating dissociated cells with methyl-beta-cyclodextrin.
17 aft domains, similar to the action of methyl-beta-cyclodextrin.
18 the nanoencapsulation of Danube fish oils in beta-cyclodextrin.
19 cubating neonatal cardiomyocytes with methyl-beta-cyclodextrin.
20 cholesterol stores were depleted with methyl-beta-cyclodextrin.
21 e ethyl esters form inclusion complexes with beta-cyclodextrin.
22 d by using aqueous solution of hydroxypropyl-beta-cyclodextrins.
23 otenoids were complexed with 2-hydroxypropyl-beta-cyclodextrin (2-HPbetaCD) in different mass ratios
24 ted with the sterol acceptor 2-hydroxypropyl-beta-cyclodextrin (2-OHCD) confirmed the release of an a
25 e independent methods: 1) exposure to methyl-beta-cyclodextrin, 2) treatment with the HMG-CoA reducta
27 ermore, we demonstrated that 2-hydroxypropyl-beta-cyclodextrin, a clinical agent used to enhance the
28 , the mice were treated with 2-hydroxypropyl-beta-cyclodextrin, a drug previously reported to rescue
29 anges in hysteresis due to interactions with beta-cyclodextrin, a molecule that is known to form stab
30 gradual neurodegeneration, and we reveal how beta-cyclodextrin, a potential therapeutic drug, reverts
31 prevented by treatment with 2-hydroxypropyl-beta-cyclodextrin, a promising therapy currently under c
32 zed following treatment with 2-hydroxypropyl-beta-cyclodextrin, a therapy that reduces pathological f
33 the well-known cholesterol extracting agent, beta-cyclodextrin, acts by catalytically tipping the equ
36 Pharmacological raft disruption using methyl-beta-cyclodextrin also increased CD44-ezrin coprecipitat
37 ing prompted by cholesterol efflux to methyl-beta-cyclodextrin also was prevented, indicating that PM
38 tion reactions of two structurally different beta-cyclodextrins (AmPrbetaCD and AMBnTbetaCD) in the P
42 water (calorimetric effect of 536Jg(-1) for beta-cyclodextrin and 304-422.5Jg(-1) for complexes).
43 r reactivity was only moderately affected by beta-cyclodextrin and bovine serum albumin, taken as mod
44 inct binding epitopes are capable of binding beta-cyclodextrin and cucurbit[6/7]uril (CB) simultaneou
45 into the supramolecular interactions between beta-cyclodextrin and ferrocene-nucleobase derivatives.
46 lloprotease inhibitor batimastat, and methyl-beta-cyclodextrin and filipin, which block lipid raft fo
47 ies of ferrocene-based guests with CB[7] and beta-cyclodextrin and provide a coherent view of the rol
48 n/activation was also decreased after methyl-beta-cyclodextrin and statin treatment but increased in
49 ts showed a cross-peak between H-3 proton of beta-cyclodextrin and the aromatic rings group of curcum
51 pyrene-modified beta-cyclodextrin rings, the beta-cyclodextrin and/or pyrene-modified beta-cyclodextr
52 nzene units leads to the dissociation of the beta-cyclodextrin and/or pyrene-modified beta-cyclodextr
53 through cholesterol-scavenging drugs (methyl-beta-cyclodextrin) and the enzymatic breakdown of sphing
54 acid, sodium dodecyl sulfate, hydroxypropyl-beta-cyclodextrin, and NaCl served as model medium const
59 their acute cholesterol depletion by methyl-beta-cyclodextrin as a tool to describe the physiologica
61 micellar electrokinetic chromatography using beta-cyclodextrin as the chiral selector and sodium taur
62 f curcumin through inclusion complexation by beta-cyclodextrin as well as the topology and geometry o
63 and occupancy of binding sites; (ii) methyl-beta-cyclodextrin, as a FA acceptor, to observe the diss
66 S faces, functionalized with a pH-responsive beta-cyclodextrin-based supramolecular nanovalve on the
69 ap, 4, that is tethered via amide bonds to a beta-cyclodextrin (beta-CD) and a dodecyl chain was achi
70 on-sensitive host-guest crosslinkers between beta-cyclodextrin (beta-CD) and ferrocene (Fc) and iron
71 f the tricyclic antidepressant doxepin using beta-cyclodextrin (beta-CD) as a buffer additive is inve
74 ructural and thermodynamic insights into the beta-cyclodextrin (beta-CD) encapsulation of key compoun
76 t decreases with increasing concentration of beta-cyclodextrin (beta-CD) in an aqueous solution, and
77 with mono-6-deoxy-6-((2-mercaptoethyl)amino)-beta-cyclodextrin (beta-CD) in order to produce a select
78 d ensemble has been developed by introducing beta-cyclodextrin (beta-CD) into polymer-grafted mesopor
79 that has been chemically functionalized with beta-cyclodextrin (beta-CD) is extremely effective in af
80 le of drug delivery based on the function of beta-cyclodextrin (beta-CD) nanovalves that are responsi
81 ized nanoparticles consist of a monolayer of beta-cyclodextrin (beta-CD) rings positioned selectively
86 between rutin and four cyclodextrins, namely beta-cyclodextrin (beta-CD), (2-hydroxypropyl)-alpha-cyc
87 ole (AN) with alpha-cyclodextrin (alpha-CD), beta-cyclodextrin (beta-CD), hydroxypropyl-beta-cyclodex
88 nclusion complexes of 2-nonanone (2-NN) with beta-cyclodextrin (beta-CD), were prepared by a co-preci
94 unds in aqueous solution was investigated at beta-cyclodextrin (beta-CD)/silver nanoparticle (AgNPs)
96 ivery of CAB formulated with 2-hydroxypropyl-beta-cyclodextrin (betaCAB) and examined the pharmacokin
97 ptide LyeTxI and association compound LyeTxI/beta-cyclodextrin (betaCD) against multispecies biofilms
99 a-hemolysin (alphaHL) to map the presence of beta-cyclodextrin (betaCD) at a substrate pore opening.
100 isperse amphiphilic oligoethyleneimine (OEI)-beta-cyclodextrin (betaCD) clusters have been prepared,
101 d yellow fluorescent proteins (Fc-YFPs) onto beta-cyclodextrin (betaCD) molecular printboards was cha
102 vailable sport beverage, with and without 2% beta-cyclodextrin (betaCD) under light and darkness cond
103 of orthogonal supramolecular interactions of beta-cyclodextrin (betaCD)-adamantyl (Ad) host-guest and
109 m to improve the response to 2-hydroxypropyl-beta-cyclodextrin, by restoring a functional NPC1 to the
110 alent capture of olive pheromones inside the beta-cyclodextrin cavity leads to the reduction of reson
111 characterization of the rosmarinic acid (RA)-beta-cyclodextrin (CD) complex in aqueous solution by (1
112 ed carbonization (TPC) of Cu(2+)- and Fe(3+)-beta-cyclodextrin (CD) complexes, a rapid reduction of C
114 between a carbohydrate-based molecule and a beta-cyclodextrin (CD) hydrophobic cavity for asymmetric
115 the particles were loaded with cargo, bulky beta-cyclodextrin (CD) molecules were noncovalently asso
116 phosphate-buffered saline (PBS), 0.1% methyl-beta-cyclodextrin (CD), or CD plus cholesterol (0.1%, CD
118 Dynamin inhibitors, chlorpromazine, methyl-beta-cyclodextrin, chloroquine, and concanamycin A drama
119 but following cholesterol efflux with methyl-beta-cyclodextrin, clusters containing zona-binding mole
121 h excess cholesterol by a cholesterol/methyl-beta-cyclodextrin complex, phenocopying SR-BI KO oocytes
123 ridged biguanidyl adamantine (Ad-SS-GD) with beta-cyclodextrin-conjugated low-molecular-weight polyet
124 Multivalent host-guest interactions between beta-cyclodextrin-conjugated superparamagnetic iron oxid
125 ta-cyclodextrin (RAMEB) and a low methylated-beta-cyclodextrin (CRYSMEB) were investigated in aqueous
130 at disrupting lipid raft formation by methyl-beta-cyclodextrin decreased NO production and apoptosis.
131 sgk1 with the apical surface, whereas methyl-beta-cyclodextrin decreased the association of sgk1 with
132 Here we measured the rate at which methyl-beta-cyclodextrin depletes cholesterol from a supported
133 sulfobutyl ether-beta-cyclodextrin (SCD), a beta-cyclodextrin derivative carrying ionizable groups t
134 dazim using gold nanorods derivatized with a beta-cyclodextrin derivative to bind this fungicide.
135 dical bTbK in complex with captisol (CAP), a beta-cyclodextrin derivative, host-guest assembling offe
137 Using this method, we determined that methyl-beta-cyclodextrin differentially extracts cholesterol fr
138 re synthesized by varying the molar ratio of beta-cyclodextrin:diphenyl carbonate (beta-CD:DPC; 1:2,
139 m parasites with cholesterol-specific methyl-beta-cyclodextrin disrupts both membrane liquid order an
146 that the primary and secondary OH groups of beta-cyclodextrin exhibit markedly different dynamics, s
150 ent of HSV-1-infected Vero cells with methyl beta-cyclodextrin from 2 to 9 h postentry reduced plaque
151 ic strategy for controlled synthesis of thio-beta-cyclodextrin functionalized graphene/gold nanoparti
153 ctrochemical sensor using graphene oxide and beta-cyclodextrin functionalized multiwalled carbon nano
154 ultiwalled carbon nanotubes assimilated with beta-cyclodextrin/glassy carbon electrode exhibited cata
157 eties) via controlled esterification or with beta-cyclodextrins (host moieties) through amidation.
158 yclodextrin (HP-alpha-CD), (2-hydroxypropyl)-beta-cyclodextrin (HP-beta-CD) and (2-hydroxypropyl)-gam
161 tly, the cycloheptaglucoside 2-hydroxypropyl-beta-cyclodextrin (HP-beta-CD) has shown efficacy as a p
162 he compact and hard HS tissue, hydroxypropyl-beta-cyclodextrin (HP-beta-CD) was added into sodium hya
163 CNS of the npc1(-/-) mouse, 2-hydroxypropyl-beta-cyclodextrin (HP-beta-CD), a compound known to prev
164 , beta-cyclodextrin (beta-CD), hydroxypropyl-beta-cyclodextrin (HP-beta-CD), randomly methylated-beta
168 lation combines this drug with hydroxypropyl-beta-cyclodextrin (HPbetaCD) to improve its solubility a
169 ies reported ototoxicity of 2-hydroxypropyl- beta-cyclodextrin (HPbetaCD), a cholesterol chelator and
172 Furthermore, we utilized 2-hydroxypropyl-beta-cyclodextrin (HPbetaCD, an emerging therapeutic) ad
173 In preclinical testing, 2-hydroxypropyl-beta-cyclodextrins (HPbetaCD) significantly delayed cere
175 contact time, using sequential hydroxypropyl-beta-cyclodextrin (HPCD) extractions in soils amended wi
177 e, the disruption of SM-rich rafts by methyl-beta-cyclodextrin impaired myosin activation and clot re
179 lowed by CE-LIF using 0.5 mM hydroxyl propyl-beta-cyclodextrin in borate buffer [80 mM, pH 9.3].
180 response to postprandial micelles or methyl-beta-cyclodextrin in cultured enterocytes, and it is req
181 clodextrin is more potent than hydroxypropyl-beta-cyclodextrin in reducing both cholesterol and bis(m
184 lic acid (PAA) nanofibres (NF) incorporating beta-cyclodextrin inclusion complex (beta-CD-IC) of quer
185 s of glutathione thiyl radical (GS(*)); (ii) beta-cyclodextrins increase the kinetic stability of the
186 esults of this study show that carboxymethyl-beta-cyclodextrin increased lipoplexes' encapsulation ef
188 t depleting cellular cholesterol with methyl-beta-cyclodextrin increased the resilience of stromal ce
190 he cells with cholesterol-deficient media or beta-cyclodextrins induced changes in cholesterol distri
191 iously described a technique in which methyl-beta-cyclodextrin-induced lipid exchange is used to prep
193 revealed that the therapeutic drug candidate beta-cyclodextrin induces the subplasmalemmal location o
194 In 3T3L-1 adipocytes, apoAI, HDL, and methyl-beta-cyclodextrin inhibited chemotactic factor expressio
195 traction of membrane cholesterol with methyl-beta-cyclodextrin inhibited infection by virions but had
197 Recent studies have shown that hydroxypropyl-beta-cyclodextrin injections in npc1(-/-) mice are parti
201 Acute exposure of LLC-PK1 cells to methyl beta-cyclodextrin led to parallel decreases in cellular
204 with the cholesterol-extracting agent methyl-beta-cyclodextrin (MbetaCD) not only disrupted the DRM l
209 To test this hypothesis, we used methyl-beta-cyclodextrin (MbetaCD) to load mouse peritoneal mac
210 agasi promastigotes were treated with methyl-beta-cyclodextrin (MbetaCD), a sterol-chelating reagent,
211 cholesterol levels were reduced using methyl-beta-cyclodextrin (mbetaCD), as confirmed by Amplex Red
213 pid rafts, we pretreated animals with methyl-beta-cyclodextrin (MbetaCD), which disrupts lipid rafts.
215 etreatment with lipid raft disruptor (Methyl-beta-cyclodextrin, MbetaCD) and oxidative stress inhibit
217 te depletion of cholesterol with 5 mm methyl-beta-cyclodextrin (MCD) caused a substantial increase in
218 with that of a simple sterol carrier, methyl-beta-cyclodextrin (MCD), when STARD4 and MCD were overex
221 Membrane cholesterol depletion with methyl-beta-cyclodextrin mimicked the effects of AC6 silencing
222 rrelated with the increasing availability of beta-cyclodextrin moieties over the surface of the devic
224 esterol depletion of macrophages with methyl-beta-cyclodextrin normalized FC content between the two
227 feasibility of synthesizing NS derived from beta-cyclodextrin of high crystallinity for the encapsul
229 hort-term cholesterol chelation using methyl-beta-cyclodextrin or by stable knockdown of caveolin-1 a
231 Depletion of cholesterol through methyl-beta-cyclodextrin or cholesterol oxidase abolished the p
233 domains by acute exposure of cells to methyl-beta-cyclodextrin or chronic exposure to different stati
234 ted by pretreatment of the cells with methyl-beta-cyclodextrin or Filipin III, hence implicating memb
236 Correspondingly, pharmacological (methyl-beta-cyclodextrin) or genetic disruption of caveolae (Ca
237 toxoids or pretreatment of cells with methyl-beta-cyclodextrin) or osmotic protection of target cells
238 ore, cholesterol lowering by statins, methyl-beta-cyclodextrin, or filipin also activates PKA and, co
239 her in vitro, by treatment with 25 mM methyl-beta-cyclodextrin, or in vivo, by subjecting animals to
243 linkers that attached the drug payloads to a beta-cyclodextrin-PEG copolymer to form self-assembled n
244 ic blockers, namely per-6-S-(3-amino) propyl-beta-cyclodextrin, per-6-S-(3-aminomethyl) benzyl-alpha-
245 cyclodextrin, per-6-S-(3-aminomethyl) benzyl-beta-cyclodextrin, per-6-S-(3-aminomethyl) benzyl-gamma-
246 io from intact cells does not reflect methyl-beta-cyclodextrin plasma membrane extraction properties.
247 ences and then reacted with per-O-methylated beta-cyclodextrin (PMbetaCD) caps, to form supramolecula
248 omimetic material, namely, poly(p-phenylene) beta-cyclodextrin poly(ethylene glycol) (PPP-CD-g-PEG) c
251 study, we evaluated the performance of three beta-cyclodextrin polymers (CDPs), an anion-exchange (AE
252 ch was to evaluate the performance of porous beta-cyclodextrin polymers (P-CDP) as adsorbents of MPs
253 -guest complexes, but until now cross-linked beta-cyclodextrin polymers have had low surface areas an
254 er m1 or m3 muscarinic receptors with methyl-beta-cyclodextrin produced a loss of localization of pro
255 tudy we demonstrate that quaternary ammonium beta-cyclodextrin (QABCD) fulfils both of these requirem
256 ntimicrobial activity of randomly methylated beta cyclodextrin (RAMEB) encapsulated thyme oil, lemon
257 clodextrin (HP-beta-CD), randomly methylated-beta-cyclodextrin (RAMEB) and a low methylated-beta-cycl
259 ected Ae. aegypti cells with 2-hydroxypropyl-beta-cyclodextrin restores dengue replication in Wolbach
260 lesterol depletion and repletion with methyl-beta-cyclodextrin reversibly altered PI4KIIalpha associa
261 the beta-cyclodextrin and/or pyrene-modified beta-cyclodextrin rings from the stalks, thus opening th
262 the beta-cyclodextrin and/or pyrene-modified beta-cyclodextrin rings will thread onto the azobenzene-
263 the beta-cyclodextrin and/or pyrene-modified beta-cyclodextrin rings, the beta-cyclodextrin and/or py
265 Propofol was complexed with sulfobutyl ether-beta-cyclodextrin (SCD), a beta-cyclodextrin derivative
266 on that is palmitoylated and mediates methyl-beta-cyclodextrin-sensitive self-association of purified
268 mbrane cholesterol is extracted using methyl beta-cyclodextrin, suggesting that lipid raft microdomai
269 ed by the cholesterol-depleting drug, methyl beta-cyclodextrin, suggesting that the physiological fun
270 negatively charged chiral selector, sulfated beta-cyclodextrin (sulfated beta-CD), that migrated away
271 with the cholesterol chelating agent, methyl-beta-cyclodextrin, that is thought to disrupt lipid raft
272 terol-sequestering drugs nystatin and methyl-beta-cyclodextrin, the dynamin-specific inhibitor dynaso
274 cells co-expressing CPM and B1R with methyl-beta-cyclodextrin to disrupt lipid rafts reduced the B1R
275 have overcome these limitations using methyl-beta-cyclodextrin to solubilize VLCFA for rapid delivery
276 turing strategy in which applying methylated beta-cyclodextrin to the culture medium allows the seque
277 y interaction, exemplified by the binding of beta-cyclodextrin to the primary and secondary binding s
278 t has been synthesized by covalently linking beta-cyclodextrin to the surface of N, S codoped carbon
279 Phe residue, (ii) site-specifically adhering beta-cyclodextrin to the surface of ubiquitin, and (iii)
281 entration of a cyclodextrin (sulfobutylether-beta-cyclodextrin) to inhibit crystallization over a 48
282 that depleting endogenous 7-DHC with methyl-beta-cyclodextrin treatment enhances Hedgehog activation
283 rose gradient ultracentrifugation and methyl-beta-cyclodextrin treatment that CLEC-2 translocates to
284 creased to approximately 79 pN/mum by methyl-beta-cyclodextrin treatment to sequester membrane choles
285 mplexes between red bell pepper pigments and beta-cyclodextrin using two different procedures (i.e.,
288 ion of the title compound in the presence of beta-cyclodextrin was examined in different conditions.
289 e cholesterol (cholesterol mixed with methyl-beta-cyclodextrin), we observed an increase in DAT bindi
290 plexes and a physical mixture of extract and beta-cyclodextrin were evaluated by differential scannin
292 between bound water and the secondary OH of beta-cyclodextrin, which exhibited spatial uniformity wi
293 to the ER can be overcome by 2-hydroxypropyl-beta-cyclodextrin, which leads to a marked increase in A
294 uires coadministration with sulphobutylether-beta-cyclodextrin, which may accumulate in patients with
295 e greatest when the gradient is formed using beta-cyclodextrin while directed concentration of cyanom
297 ntagonist or disrupting caveolae with methyl-beta-cyclodextrin, with an associated approximately 30%
298 lusion complexes of Vitamin A Palmitate with beta-cyclodextrins, without the use of organic solvents,
299 n was achieved with the expectation that the beta-cyclodextrin would lead to increased reactivity to