ライフサイエンスコーパス: beta-oxidation

1 mainly function in mitochondrial fatty acid beta oxidation.

2 f acetyl-CoA from metabolic reprogramming to beta oxidation.

3 on of mitochondrial structure and fatty acid beta-oxidation.

4 on of lipase 3 and enzymes for mitochondrial beta-oxidation.

5 f certain ascarosides for shortening through beta-oxidation.

6 olonic epithelial cells (colonocytes) toward beta-oxidation.

7 otein kinase, as well as enhanced fatty acid beta-oxidation.

8 and muscle tissues, and had reduced rates of beta-oxidation.

9 in organonitrogen metabolism and fatty acid beta-oxidation.

10 that can occur alternatively to the dominant beta-oxidation.

11 ice exhibited no change in peroxisomal C(12) beta-oxidation.

12 plex directs saturated fatty acyl-CoA toward beta-oxidation.

13 peroxisomes are the sole site of fatty acid beta-oxidation.

14 t metabolic fates such as lipid synthesis or beta-oxidation.

15 chondrial substrates to generate ATP through beta-oxidation.

16 pid accumulation due to defective fatty acid beta-oxidation.

17 ence of a functional defect in mitochondrial beta-oxidation.

18 t metabolic fates such as lipid synthesis or beta-oxidation.

19 glyceride accumulation and ex vivo palmitate beta-oxidation.

20 muscle, due in part to incomplete fatty acid beta-oxidation.

21 ns involved in mitochondrial respiration and beta-oxidation.

22 s ATGL expression and free fatty acids (FFA) beta-oxidation.

23 proic acid by microorganisms through reverse beta-oxidation.

24 d oxidative stress, and increased fatty acid beta-oxidation.

25 tabolic switch from glycolysis to fatty acid beta-oxidation.

26 sis from glutamine, and decreased fatty acid beta-oxidation.

27 LDs efficiently supply FAs for mitochondrial beta-oxidation.

28 y other ACSL isoforms were not available for beta-oxidation.

29 rocess is essential for efficient fatty acid beta-oxidation.

30 wn catabolic processes such as lipolysis and beta-oxidation.

31 ta-ketoacyl-CoA esters as part of fatty acid beta-oxidation.

32 ssion of key enzymes required for fatty acid beta-oxidation.

33 otein secretion, and/or attenuation of lipid beta-oxidation.

34 inducing de novo lipogenesis and decreasing beta-oxidation.

35 e is known about the effects of HCV on lipid beta-oxidation.

36 cylglycerol and 46% more into the pathway of beta-oxidation.

37 in acyl-carnitines into the mitochondria for beta-oxidation.

38 octanoyl moieties provided by mitochondrial beta-oxidation.

39 DHA are increased, more DHA is available for beta-oxidation.

40 reased H(2)O(2) as a byproduct of fatty acid beta-oxidation.

41 r enzymes and increased FA/BA metabolism and beta-oxidation.

42 ibitive effects on the enzymes in fatty acid beta-oxidation.

43 agreement with the known OGDH dependence of beta-oxidation.

44 MDV replication does not require fatty acid beta-oxidation.

45 that ech2 phenotypes require efficient core beta-oxidation.

46 efficiency or oxygen consumption rate during beta-oxidation.

47 efect of mitochondrial long-chain fatty acid beta-oxidation.

48 residues to alanine resulted in defective of beta-oxidation.

49 and enlarged peroxisomes suggest compromised beta-oxidation.

50 s were higher (p < 0.05), suggesting reduced beta- oxidation.

51 ins and do not form normally when fatty acid beta-oxidation, a core function of peroxisomes, is impai

52 Due to suppression of autophagy and beta-oxidation, a high-fat diet challenge aggravated ste

53 s indicates a link to central metabolism via beta-oxidation, a non-decarboxylating glutaryl-CoA dehyd

54 or glucose and an altered rate of fatty acid beta-oxidation, accompanied by a decreased pantothenic a

55 h alterations in acylcarnitines, a metric of beta-oxidation, across stages of CKD.

56 The marker of fatty acid beta-oxidation, adipate, is mostly decreased by the shor

57 muscle (i.e., increased AMP kinase activity, beta-oxidation and -uncoupling, and decreased triglyceri

58 protein enhances catabolic pathways, such as beta-oxidation and autophagy, to generate ATP, and inhib

59 rase-like enzyme, is required for fatty acid beta-oxidation and cardiolipin remodeling, essential for

60 , and on hepatocyte mitochondrial fatty acid beta-oxidation and cell survival.

61 ing this pathway are decreased mitochondrial beta-oxidation and decreased energy expenditure.

62 improvement of the redox status via enhanced beta-oxidation and decreased glucose uptake, leading to

63 These data implicate a mismatch of beta-oxidation and fatty acid uptake as a mechanism lead

64 ed hypertrophic myocardial cells, fatty acid beta-oxidation and heart function were substantially str

65 at Astragaloside IV can stimulate fatty acid beta-oxidation and improve mitochondrial function, which

66 or alpha (PPARalpha) to stimulate fatty acid beta-oxidation and increase cardiac energy production, i

67 er were consistent with decreased fatty acid beta-oxidation and increased triglyceride storage.

68 vement of their gene products in peroxisomal beta-oxidation and initial seedling growth.

69 together with the downregulation of hepatic beta-oxidation and ketogenesis in the neonatal chicken.

70 ive mitohormetic pathway to increase hepatic beta-oxidation and mitochondrial complex content and act

71 sociated with the induction of mitochondrial beta-oxidation and mitochondrial fusion.

72 of long-chain fatty acids for mitochondrial beta-oxidation and nuclear receptor activation.

73 450) family and genes involved in fatty acid beta-oxidation and omega-oxidation.

74 e peroxisomes, organelles housing fatty acid beta-oxidation and other critical metabolic reactions.

75 periportal zonation of the enzymes mediating beta-oxidation and oxidative phosphorylation resulted in

76 strongly dependent on the rate constants for beta-oxidation and oxidative phosphorylation.

77 provide evidence that redox reactions within beta-oxidation and the electron transport system serve a

78 ved altered rates of pyruvate and fatty acid beta-oxidation and the likely re-directing of glutamine

79 tance may be linked to incomplete fatty acid beta-oxidation and the subsequent increase in acylcarnit

80 ng with more modest reductions in enzymes of beta-oxidation and the tricarboxylic acid cycle.

81 obilizes lipid stores, resulting in enhanced beta-oxidation and viral replication.

82 R represses the fad genes of FA degradation (beta-oxidation) and activates the fab genes of FA synthe

83 f enoyl-CoA hydratase involved in fatty acid beta-oxidation) and tub-1 (an ortholog of the human TUBB

84 s revealed dysfunction of purine metabolism, beta oxidation, and antioxidants, which were differentia

85 ing hepatic steatosis, increasing fatty acid beta-oxidation, and activating 5'adenosine monophosphate

86 Bacterial lipid import, beta-oxidation, and glyoxylate shunt genes were required

87 transport, mitochondria and peroxisomes for beta-oxidation, and lysosomes for lipid hydrolysis and r

88 concentrations of free fatty acids, maximal beta-oxidation, and mitochondrial abnormalities.

89 mitochondrial energy metabolism, fatty acid beta-oxidation, and mitochondrial biogenesis and their k

90 ard cells express all the genes required for beta-oxidation, and we showed that light-induced stomata

91 T3 deacetylation target; improved fatty acid beta-oxidation; and ameliorated liver steatosis and gluc

92 ; instead, fatty acid synthesis and reversed beta-oxidation are manipulated to synthesize medium-chai

93 rimary metabolic pathways such as fatty acid beta-oxidation are unclear.

94 es involved in oxidative phosphorylation and beta-oxidation are up-regulated in the daw mutants, indi

95 eam pathways (i.e., fatty acid synthesis and beta-oxidation) are differentially regulated by KISS1, a

96 hus, our results identify bulk autophagy and beta-oxidation as important energy providers during acut

97 nockout mutants were impaired in peroxisomal beta-oxidation as shown by developmental arrest of seedl

98 oA reductase (DECR1), an auxiliary enzyme of beta-oxidation, as a clinically relevant biomarker for C

99 lysis and glutaminolysis, but not fatty acid beta-oxidation, as an essential energy source for the re

100 aloside IV switched glycolysis to fatty acid beta-oxidation, as confirmed by reduced anaerobic glycol

101 respiratory capacity, heightened fatty acid beta-oxidation-associated mitochondrial reactive oxygen

102 ed hepatic lipogenesis and increased hepatic beta-oxidation at organ programming peak in early life (

103 appearance, and correlated with up-regulated beta-oxidation at the expense of lipogenesis.

104 a activity resulting in decreased fatty acid beta-oxidation, augmentation of translation of fatty aci

105 ine and short chain acylcarnitine, increased beta-oxidation but diminished incomplete fatty acid oxid

106 of hepatic lipoprotein output, activation of beta oxidation by muscle, and regulation of the producti

107 ses which alter the allosteric inhibition of beta-oxidation by acetyl-CoA.

108 These changes suggested saturation of beta-oxidation by ozone in exercising humans.

109 e periodically expressed coincident with the beta-oxidation byproduct histone crotonylation.

110 y acid (FA) oxidation in concert with higher beta-oxidation capacity to reduce the accumulation of IR

111 tant correction of acylcarnitine profile and beta-oxidation capacity, two hallmarks of the disorder.

112 conclusion that incomplete muscle fatty acid beta-oxidation causes acylcarnitine accumulation and ass

113 te energy substrate flux through glycolysis, beta-oxidation, citric acid (TCA) cycle, and oxidative p

114 Interpreted in the context of beta-oxidation, CoA inhibition would prevent Acot-mediat

115 frees amino acids, and lipid degradation via beta-oxidation contribute in parallel to energy maintena

116 nes involved in mitochondrial biogenesis and beta-oxidation (Cox4, Nrf1, Pgc1alpha, Pgc1beta and Tfam

117 Peroxisomal beta-oxidation cycles are required for the biosynthesis

118 Peroxisomal beta-oxidation cycles shorten the side chains of ascaros

119 ases, which catalyze the first step in these beta-oxidation cycles, form different protein homo- and

120 ases, which catalyze the first step in these beta-oxidation cycles, have different side chain-length

121 a potential therapeutic modality to correct beta-oxidation deficiencies.

122 x of pyruvate-derived acetyl-CoA relative to beta-oxidation-derived acetyl-CoA, are suggested to impa

123 ure also increased circulating mitochondrial beta-oxidation-derived metabolites, such as acylcarnitin

124 on of transcripts encoding peroxisomal-based beta-oxidation did not change in response to day : night

125 ided the first in vivo evidence for enhanced beta-oxidation during HCV infection because HCV-infected

126 regulates lipid mobilization and fatty acid beta-oxidation during seed germination and seedling esta

127 Adipose-specific knockout of the peroxisomal beta-oxidation enzyme acyl-CoA oxidase 1 (Acox1-AKO) was

128 esults support a biosynthetic model in which beta-oxidation enzymes act directly on the CoA-thioester

129 in Arabidopsis employs the same core set of beta-oxidation enzymes as in the synthesis of indole-3-a

130 nes upregulated included putative fatty acid beta-oxidation enzymes.

131 ylcarnitine ratio, a marker of efficiency of beta-oxidation, exhibited a graded decrease from stage 2

132 termined that T cells switch from fatty acid beta-oxidation (FAO) and pyruvate oxidation via the tric

133 ate peroxisomal and mitochondrial fatty acid beta-oxidation (FAO) in HEK-293 cells, we identified ess

134 y, and relevance of mitochondrial fatty acid beta-oxidation (FAO) in the brain are highly controversi

135 Fatty acid beta-oxidation (FAO) is the main bioenergetic pathway in

136 Mitochondrial fatty acid beta-oxidation (FAO) is the major pathway for the degrad

137 he electron transfer chain (ETC), fatty acid beta-oxidation (FAO), and the tricarboxylic acid cycle.

138 pogenesis, impaired mitochondrial fatty acid beta-oxidation (FAO), changes in fat distribution, alter

139 ar acidification rate (ECAR), and fatty acid beta-oxidation (FAO)-mediated OCR assays for metabolic f

140 ctivity, and marked inhibition of fatty acid beta-oxidation (FAO).

141 ism but have an increased rate of fatty acid beta-oxidation (FAO).

142 yl coenzyme A (CoA) (a product of fatty acid beta-oxidation [FAO]), or dichloroacetate, a compound th

143 ncreased the expression of genes involved in beta-oxidation: fibroblast growth factor 21 and peroxiso

144 yzes the last 3 steps of mitochondrial lipid beta-oxidation for cellular energy production.

145 we show that the retina also uses fatty acid beta-oxidation for energy.

146 bility shift assays revealed that Msn4 binds beta-oxidation gene promoters.

147 d respiration that accelerating flux through beta-oxidation generates a corresponding increase in mit

148 h plays a dominant role in the expression of beta-oxidation genes after ligand-induced activation, wa

149 tabolic cycle (YMC) and find that fatty acid beta-oxidation genes are periodically expressed coincide

150 has increased occupancy on the promoters of beta-oxidation genes in glucose-depleted conditions, and

151 drial biogenesis, respiration and fatty acid beta-oxidation genes is significantly reduced in the liv

152 In cultured macrophages, lipid import and beta-oxidation genes were required for bacterial replica

153 y during developmental progression, but more beta-oxidation genes were upregulated in early C5s compa

154 tion of lipogenic genes, lower expression of beta-oxidation genes, greater reduction in AMP-activated

155 e and had reduced transcript levels of major beta-oxidation genes.

156 Peroxisomal beta-oxidation has recently been shown to contribute to

157 rnitine conjugated lipid levels, and altered beta-oxidation have been observed.

158 of TAG catabolism and downstream fatty acid beta-oxidation have not been characterised in diatoms.

159 ns involved in mitochondrial and peroxisomal beta-oxidation, have an increased rate of fatty acid oxi

160 Under the conditions of impaired lipid beta-oxidation, host cells were less responsive to the a

161 hen the cells do not depend on mitochondrial beta-oxidation.IMPORTANCE Viruses can manipulate host ce

162 increasing oxygen consumption and fatty acid beta-oxidation in adipocytes.

163 4-HNE provides a novel mechanism for altered beta-oxidation in ALD, and these data demonstrate for th

164 The importance of peroxisomal FA beta-oxidation in algal physiology was shown by the impa

165 d opposite views on the role of flux through beta-oxidation in causing insulin resistance.

166 ic mobilization of lipids leads to increased beta-oxidation in DENV-infected cells.

167 betaine is likely due to the stimulation of beta-oxidation in liver and the effects on PL metabolism

168 was manipulated by varying flux rate through beta-oxidation in muscle mitochondria minus/plus pharmac

169 Stimulating fatty acid beta-oxidation in neonatal hearts may present a novel ca

170 or carnitine-dependent long-chain fatty acid beta-oxidation in neural stem cells of the developing ma

171 GDNF also enhanced mitochondrial fatty acid beta-oxidation in primary mouse and rat hepatocytes, and

172 s stored in lipid droplets via mitochondrial beta-oxidation in response to neuronal activity and turn

173 tty acyl coenzyme As (CoAs) into peroxisomal beta-oxidation in the intestine blunts the effects of ne

174 endoplasmic reticulum followed by 1 round of beta-oxidation in the peroxisomes.

175 iminished by inhibition of mitochondrial FFA beta-oxidation in vivo.

176 oxidative stress, and inducers of fatty acid beta-oxidation, including sirtuin 1 (SIRT1), sirtuin 3 (

177 ibits a decreased mitochondrial capacity for beta-oxidation, increased accumulation of intracellular

178 vere peroxisomal defects, including impaired beta-oxidation, inefficient matrix protein import, and d

179 CETSA identified 18 proteins and fatty acid beta-oxidation inhibition pathways that were significant

180 es exposed to palmitate in the presence of a beta-oxidation inhibitor.

181 able to utilize exogenous myristate and form beta-oxidation intermediates, suggesting that ATF parasi

182 he end products of glycolysis and fatty acid beta-oxidation into the reducing equivalents NADH and FA

183 s experimental evidence that a plant-type FA beta-oxidation involving H2 O2 -producing acyl-CoA oxida

184 However, placental beta-oxidation is affected by high glucose and reduced i

185 ence that lipid synthesis is attenuated, and beta-oxidation is enhanced in these cells.

186 n of attenuated lipid synthesis and enhanced beta-oxidation is not conducive to lipid accumulation, y

187 to palmitate without further degradation via beta-oxidation is still unknown.

188 that catalyzes the first step in peroxisomal beta-oxidation, is enriched in liver and further increas

189 Partial inhibition of beta-oxidation led to persisting TRAS in Pten(-/-) mice

190 acyl-CoA esters suggests potential roles in beta-oxidation, lipid biosynthesis, signal transduction,

191 This suggests that interference with lipid beta-oxidation may assist the virus in the establishment

192 d expression of genes involved in fatty acid beta-oxidation mediated by PGC1alpha.

193 otype with aerobic glycolysis and fatty acid beta-oxidation-mediated oxidative (glyco-oxidative) meta

194 the wild type, illustrating the potential of beta-oxidation mutants for algal biotechnology.

195 00 components from wild-type and peroxisomal beta-oxidation mutants including (omega - 1)-linked acyl

196 are suppressed in combination with the core beta-oxidation mutants mfp2 or ped1, and ech2 mfp2 seedl

197 These data represent the first evidence of beta-oxidation occurring in specialized proresolving med

198 ived but may also be produced by the partial beta oxidation of dietary 18:1 t11.

199 tion, trimethylamine-N-oxide production, and beta oxidation of fatty acids (FDR < 0.1) that differed

200 C-EPA per hour as (13)CO2 and the cumulative beta-oxidation of (13)C-EPA did not differ between young

201 units for chain elongation are derived from beta-oxidation of [1,2,3,4-(13)C4]palmitic acid.

202 Peroxisomal beta-oxidation of C26:0 was normal, but beta-oxidation o

203 which play key roles in gluconeogenesis and beta-oxidation of fatty acid, respectively.

204 phosphorylation (metformin, oligomycin) and beta-oxidation of fatty acids (etomoxir) enhanced the an

205 gh-fat diet, smaller fat deposits, increased beta-oxidation of fatty acids (FAO) and oxygen consumpti

206 ances the capacity of hepatocytes to mediate beta-oxidation of fatty acids and minimizes lipid accumu

207 resent in peroxisomes and positively affects beta-oxidation of fatty acids and protoauxins.

208 ofibrate also induced autophagy and promoted beta-oxidation of fatty acids and stimulated gene expres

209 e novo pyrimidine biosynthesis and defective beta-oxidation of fatty acids in the absence of NAT1.

210 reactions, many of which are related to the beta-oxidation of fatty acids or fatty acid-related meta

211 , using isotopologue analysis, we found that beta-oxidation of fatty acids with varying chain lengths

212 isposal of glucose and AAs and more complete beta-oxidation of fatty acids) compared with CR.

213 ays affected in exercise physiology, such as beta-oxidation of fatty acids, glycolysis, and glycogeno

214 athways is related to alkane degradation and beta-oxidation of fatty acids.

215 genase (SCAD), involved in the regulation of beta-oxidation of fatty acids.

216 improved energy metabolism through increased beta-oxidation of fatty acids.

217 pment; low carnitine, which is essential for beta-oxidation of fatty acids; alterations in glutathion

218 saturated C16-C20 FFAs coupled with impaired beta-oxidation of FFAs and inverse partitioning into com

219 ck glycolysis, glutaminolysis, or fatty acid beta-oxidation of host cells to provide the energy and m

220 e role of mTOR in lipogenesis, adipogenesis, beta-oxidation of lipids, and ketosis of carbohydrates,

221 ndrial trifunctional protein (TFP) catalyzes beta-oxidation of long chain fatty acyl-CoAs, employing

222 rated two compartment mitochondrial model of beta-oxidation of long-chain fatty acids and main energy

223 ivated receptor gamma (PPAR-gamma)-dependent beta-oxidation of microbiota-derived short-chain fatty a

224 utational approach, we comparatively analyze beta-oxidation of palmitoyl CoA (PCoA) in isolated heart

225 mitoyl-carnitine stimulated IS, showing that beta-oxidation of palmitoyl-carnitine is not required fo

226 omal beta-oxidation of C26:0 was normal, but beta-oxidation of pristanic acid was reduced.

227 DECR1 knockdown selectively inhibited beta-oxidation of PUFAs, inhibited proliferation and mig

228 nerate electron equivalents as FADH2 through beta-oxidation of saturated fatty acids, while COD:N of

229 per min; SHAM, 84.3+/-4.9; P=0.0212), as was beta-oxidation of TG.

230 lso expressed a different set of enzymes for beta-oxidation of the resultant fatty acids depending on

231 sis, whereas no components related to either beta-oxidation or catalase were present.

232 n key genes involved in de novo lipogenesis, beta-oxidation, or lipolysis.

233 degradation of 4-hydroxynonanedioic acid via beta-oxidation originating from C-9 of HNA breaking down

234 mitochondrial matrix are poised to mitigate beta-oxidation overload and maintain CoA availability.

235 how the family of matrix Acots can mitigate beta-oxidation overload and prevent CoA limitation.

236 he link between incomplete muscle fatty acid beta-oxidation, oxidative stress, inflammation, and insu

237 , the reaction catalyzed by an enzyme in the beta-oxidation pathway (3-hydroxyacyl-CoA dehydrogenase)

238 e IF, site IIIQo, and perhaps site EF in the beta-oxidation pathway account for most of the remainder

239 dium chain fatty acyl-CoA generated from the beta-oxidation pathway and convert them to versatile med

240 me of the very-long-chain fatty acid (VLCFA) beta-oxidation pathway in peroxisomes and leads to H(2)O

241 upregulated glutathione pathway, whereas the beta-oxidation pathway is inhibited, leading to increase

242 cardial structural components and fatty acid beta-oxidation pathway were upregulated.

243 nalyses suggested the presence of a modified beta-oxidation pathway with the key intermediate 3-hydro

244 izers (esterases and enzymes involved in the beta-oxidation pathway) as well as the molecular respons

245 decrease was localized to the mitochondrial beta-oxidation pathway, as Sirt5KO mice exhibited no cha

246 enhance the burning of fat by activating the beta-oxidation pathway.

247 e glucose sensing, signaling, and fatty acid beta-oxidation pathways are evolutionarily conserved thr

248 tly or indirectly associated with fatty acid beta-oxidation pathways being especially important.

249 utants by screening for inefficient seedling beta-oxidation phenotypes.

250 purine and pyrimidine metabolism, fatty acid beta-oxidation, phospholipid catabolism, arachidonic aci

251 a of key life processes including fatty acid beta-oxidation, photorespiration, synthesis of hormones,

252 erophospholipid metabolism and mitochondrial beta-oxidation played important roles in the progression

253 e substrates requires peroxisomal fatty acid beta-oxidation plus additional enzyme activities.

254 ty acids that serve as energy substrates for beta-oxidation, precursors for membrane lipids and signa

255 n myristoylation and a potential peroxisomal beta-oxidation product.

256 etabolites including omega-carboxylation and beta-oxidation products, as well as N-acetylcysteine, ta

257 ights the differential expression of sets of beta-oxidation proteins to overcome steric hinderance fr

258 ked in Pld1(-/-) hepatocytes, with decreased beta-oxidation rate, reduced oxidation-related gene expr

259 n coupling resulting from a lower fatty acid beta-oxidation rate.

260 W7647 treatment increased cardiac fatty acid beta-oxidation rates before and after ischemia, which re

261 y to the heart resulting from low fatty acid beta-oxidation rates.

262 a pathological state with reduced fatty acid beta-oxidation, reduced mitochondrial proton gradient, d

263 ed that hepatocytes displayed malfunctioning beta-oxidation, reflected by increased acylcarnitines (A

264 ered statistically significant phenotypes in beta-oxidation-related processes in mutants for 20 of 27

265 esis) and overexpression of FoxA2 (increased beta-oxidation) resulted in a significant disruption of

266 that interfering with intestinal peroxisomal beta-oxidation results in a modest global transcriptiona

267 last enzyme of the mitochondrial fatty acid beta-oxidation spiral, and thus is important for energy

268 irst step of the peroxisomal fatty acid (FA) beta-oxidation spiral.

269 -3-hydroxyacyl-CoA intermediates to the core beta-oxidation substrate (S)-3-hydroxyacyl-CoA.

270 fied included isoforms of enzymes related to beta-oxidation, such as acyl-CoA thioesterase2, acyl-act

271 ons previously unknown to be associated with beta-oxidation, such as Indigoidine synthase A, Senescen

272 GAT2), and (b) decreases in lipolysis and FA beta-oxidation that paralleled a prolonged drop in adipo

273 le cycle of de novo fatty acid synthesis and beta-oxidation that potentiates WAT oxidative capacity a

274 roplets (LD) due to a decrease in fatty acid beta-oxidation, that leads to a reduction of oxidative p

275 ts toward decreased mitochondrial fatty acid beta-oxidation, the process required to fuel high energy

276 esis, adipose tissue lipolysis, and impaired beta-oxidation), these factors could increase the risk o

277 conclude that diatoms utilise mitochondrial beta-oxidation; this is in stark contrast to the peroxis

278 PTEN down-regulation may promote beta-oxidation through beta-catenin, whereas hypertrophy

279 p-regulated the genes involved in fatty acid beta-oxidation through peroxisome proliferator-activated

280 like nematodes, employ conserved peroxisomal beta-oxidation to edit ascarosides and change their mess

281 ay in cardiac muscle changes from fatty acid beta-oxidation to glycolysis.

282 gests that n-alkane degradation occurred via beta-oxidation to oxygenated transformation products wit

283 d fatty acids are metabolized by peroxisomal beta-oxidation to produce ATP required for stomatal open

284 gulation of lipogenic and/or upregulation of beta-oxidation transcripts, and differentially modulated

285 y acids from membrane lipids for peroxisomal beta-oxidation under prolonged dark treatment.

286 We find that sustained beta-oxidation via activation of the conserved triglycer

287 stically in directing FAs toward peroxisomal beta-oxidation via TAG intermediates, thereby maintainin

288 transcription of predicted genes involved in beta-oxidation was induced.

289 ration were lowered when FFAR1 or fatty acid beta-oxidation was inhibited.

290 how that in HCV-infected Huh7.5 cells, lipid beta-oxidation was significantly attenuated.

291 volved in in situ lipogenesis and fatty acid beta-oxidation were analyzed.

292 e-specific deletion models, lipid uptake and beta-oxidation were increased in cultured cells, whereas

293 ipts encoding enzymes involved in fatty acid beta-oxidation were mostly down-regulated.

294 receptor alpha (PPARalpha) target genes and beta-oxidation, which regulate hepatic lipid degradation

295 evealed that HCV impairs mitochondrial lipid beta-oxidation, which results in low lipid combustion.

296 n would prevent Acot-mediated suppression of beta-oxidation, while providing a release valve when CoA

297 We determined whether stimulating fatty acid beta-oxidation with GW7647, a peroxisome proliferator-ac

298 both can be prevented by the stimulation of beta-oxidation with l-carnitine.

299 CAD protein and exhibit deficient fatty acid beta-oxidation, with S-nitroso-N-acetylcysteine induced

300 Degradation of unusual fatty acids through beta-oxidation within transgenic plants has long been hy