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1 or receptor (EGFR) and the adhesion receptor beta5-integrin.
2 sm for regulation of beta1, but not beta3 or beta5 integrins.
3 eated VSMCs, a process mediated, in part, by beta5 integrins.
4 cubation of Ad particles with soluble alphav beta5 integrin also inhibited subsequent Ad internalizat
5 ic vasculature such as alpha(v)beta3/alpha(v)beta5 integrins, aminopeptidase N, and aminopeptidase A.
6 alance between the interactions of annexin V/beta5 integrin and annexin V/active PKCalpha play a role
7 V, which binds to the cytoplasmic domain of beta5 integrin and protein kinase C alpha (PKCalpha), st
8 epletion of SNX17 led to a loss of beta1 and beta5 integrins and associated a subunits from HeLa cell
9 forms a complex with alphaV/beta1 and alphaV/beta5 integrins and mediates the activation of integrin-
11 locking antibodies against beta1, beta3, and beta5 integrins and ST6Gal-I targets EGFR, tumor necrosi
13 , strongly suggesting that neither beta3 nor beta5 integrins are essential for neovascularization.
14 w that ligand-activated EGFR, Grb2, Src, and beta5-integrin are captured by clathrin coated-structure
17 inds avidly to the alpha(v)beta3 and alpha(v)beta5 integrins but does not bind to other closely relat
18 nhibited TGF-beta-inducible transcription of beta5 integrin by an interaction with a TGF-beta respons
19 chondrocytes and that binding of annexin Vto beta5 integrin controls these interactions and ultimatel
20 otherapy and DNA damage by beta1/alphaVbeta3/beta5 integrin cross-talk, but efficient radiosensitizat
22 ted RPE phagocytosis and retinal function in beta5 integrin--deficient mice, which specifically lack
23 interaction or global knockout of Angptl4 or beta5 integrin delayed recovery from peak proteinuria in
25 esistance, we targeted beta1 and alphaVbeta3/beta5 integrins, essential extracellular matrix receptor
26 Ralpha ligands on TGF-beta-induced beta3 and beta5 integrin expression and potential interaction betw
28 we investigate the involvement of beta3 and beta5 integrins in the development and progression of ma
29 mice that antibody-mediated blockade of the beta5 integrin inhibits and recombinant MFGE8 promotes P
31 Periostin activated alphaV, beta1, beta3 and beta5 integrins located in the cardiomyocyte cell membra
34 nt was not inhibited by expression of mutant beta5 integrin, nor was alphavbeta5 integrin-mediated en
35 ce lacking beta3 integrins or both beta3 and beta5 integrins not only support tumorigenesis, but have
37 RPE expressed both alpha(v)beta3 and alpha(v)beta5 integrins, only alpha(v)beta3 was expressed at bir
38 ected s.c. into mice lacking beta3- or beta3/beta5-integrins or various selectins show enhanced tumor
39 integrin but not neutralizing antibodies to beta5 integrin prevented the hypoxia-induced increase in
40 A, 50 micromol/L) inhibited TGF-beta-induced beta5 integrin protein expression by 72+/-6.8% and 73+/-
41 monstrate that this process requires alpha(v)beta5 integrin, rather than alpha(v)beta3 integrin utili
42 EBV infection increases alpha(v), beta3 and beta5 integrin subunit mRNAs as well as upregulates the
44 a peptide containing a alpha(v)beta3/alpha(v)beta5-integrin targeting domain fused to a proapoptotic
45 vide a signaling platform that link EGFR and beta5-integrin through Src-mediated phosphorylation.
46 PARalpha activators inhibit TGF-beta-induced beta5 integrin transcription in VSMCs through a novel in
48 s the Ad coreceptor alpha(v)beta3 or alpha(v)beta5 integrins, yet these cells are efficiently infecte