ライフサイエンスコーパス: biological marker

1 lesterol stands out as a bright fluorescence biological marker.

2 ocalcitonin, C-reactive protein, sepsis, and biological markers.

3 in (GFP) are extensively studied as powerful biological markers.

4 ent sample groups, and thus can be important biological markers.

5 predict due to lack of reliable clinical and biological markers.

6 ch is now focusing on identifying measurable biological markers.

7 This clinical study did not include biological markers.

8 d identify potential therapeutic targets and biological markers.

9 ing contrast agents specifically targeted to biological markers.

10 metabolic disturbances calls for predictive biological markers.

11 sage, prior psychiatric history, and several biological markers.

12 , but not severity, could be correlated with biological markers.

13 of affected patients using genetic and other biological markers.

14 relation to other prognostic factors such as biological markers.

15 Regional CBF may also provide a useful biological marker across different types of psychopathol

16 In the search for biological markers after a large-scale exposure of the h

17 Examples of compounds marketed as biological markers along with recent advances in definin

18 We review promising clinical, imaging, and biological markers, along with novel designs, for clinic

19 Our study established a link between a biological marker and impulsivity among offenders (and l

20 nerve fibre layer thickness may be used as a biological marker and may help to distinguish between op

21 ofiling in pristine forms of a wide range of biological markers and aid biomedical diagnostics as wel

22 Biological markers and alternative treatment regimens wi

23 The observed correlations between biological markers and brain activation provide new evid

24 cal correlations, stressing the need for new biological markers and consensus regarding updated termi

25 and ML algorithms could be applied to other biological markers and disease phenotypes, to examine th

26 In this study, we investigated the biological markers and established a predictive model fo

27 nce(s), which both expands its repertoire of biological markers and furthers its use as a diagnostic

28 ussed with regard to the utility of in vitro biological markers and genetic models of analgesia.

29 Our objective was to assess the presence of biological markers and pathways related to frailty, agin

30 We measured biological markers and typed human leukocyte antigen gen

31 37.3% (P < .001), independent from age, sex, biological markers, and disease duration.

32 Tumor extension, biological markers, and treatments at initial PNET diagn

33 Biological markers are not yet clinically useful for ide

34 on task measured with fNIRS might serve as a biological marker associated with delirium in ESLD patie

35 We thus have identified a biological marker associated with familial mood disorder

36 Loneliness was also related to biological markers associated with Alzheimer's disease (

37 ing the impact of parenting interventions on biological markers associated with these risks is crucia

38 Together, we provide a set of biological markers at the level of genes, synapses, cell

39 characterized by its symptoms rather than by biological markers because we have only a limited knowle

40 Biological markers (biomarkers) may offer an opportunity

41 on measurements and enables the discovery of biological markers (biomarkers).

42 Integrating relationships between biological markers, brain imaging and clinical parameter

43 c abnormal personality traits, plus numerous biological markers (cognitive, anatomical, and psychophy

44 ssible carryover effect was seen for several biological markers.Conclusions: Subjects with AATD on SD

45 Preliminary tumor response and biological marker data suggest that WT2725 dosing emulsi

46 Changes in biological markers, dietary factors, and microbial taxa

47 Examination of dinoflagellate-specific biological markers (dinosteranes and 4alpha-methyl-24-et

48 cipation and performance may be an important biological marker distinguishing BD from MDD.

49 a discrete trait and a correlated continuous biological marker (e.g., a disease precursor or associat

50 fication with phenotypic characteristics and biological markers (eg, electrophysiological changes) mi

51 showed overall improvements in cognition and biological markers, females did not exhibit improvements

52 ce spectroscopy GABA measures may serve as a biological marker for a subtype of MDD.

53 that MRI findings may potentially serve as a biological marker for bipolar disorder.

54 ole for these Abs, measurable in blood, as a biological marker for clinical activity.

55 indicating that FOXP3 expression is a valid biological marker for human T(reg) cells even in the tum

56 assay may prove to be a useful intermediate biological marker for identifying subjects at increased

57 Rectal pain sensitivity has been called a biological marker for irritable bowel syndrome, but this

58 ermining collagenase 3 levels as a potential biological marker for OA, we developed highly selective

59 We are proposing a translational clinical biological marker for patients diagnosed with Bipolar Di

60 studies are needed to establish an objective biological marker for potential diagnostic usage in seve

61 Thus, PAI-1 may be a biological marker for severe COVID-19.

62 d by background noise that make its use as a biological marker for speech processing challenging.

63 proach has practical potential as a scalable biological marker for sports-related concussion and othe

64 Biological markers for anxiety disorders may further und

65 s part of a growing effort to identify early biological markers for ASD.

66 teogenic differentiation and correlated with biological markers for CAVD progression.

67 uest has been stymied by a lack of validated biological markers for characterizing and distinguishing

68 ws great potential for providing much-needed biological markers for diagnosing, tracking, and predict

69 The scarcity of validated biological markers for diagnosis, prediction, or treatme

70 The molecular changes in the TME are used as biological markers for diagnosis, prognosis, and respons

71 y important roles in vaccine development, as biological markers for disease diagnosis, and for analys

72 ingle-photon emission computed tomography as biological markers for following the progression of Park

73 The studies of biological markers for genetic vulnerability to schizoph

74 primitive neuroectodermal tumours prognostic biological markers have been identified that are undergo

75 mbination of clearing protocol, tissue type, biological marker, imaging modality and biological quest

76 ortance of studying the relationship between biological markers, impulsivity and criminal behavior.

77 lung, and skin tumors, suggest its use as a biological marker in cancer.

78 uid where it has the potential to serve as a biological marker in human neuronal disorders.

79 elationship is the first instance in which a biological marker in T-lineage ALL has been unequivocall

80 as reveals associations between chemical and biological markers in an exemplified perturbed aquatic e

81 rial production of drugs and pesticides, and biological markers in blood and urine for early diagnosi

82 signal the onset of diseases and be used as biological markers in diagnostics.

83 erved in trials using intermediate end point biological markers in humans, in which beta-carotene has

84 ET) and computed tomography (CT), identified biological markers in mice and patients that provide a r

85 Moreover, the utility of biological markers in predicting threshold levels is unc

86 r2, alters toxin-induced expression of known biological markers in the AhR signaling pathway.

87 ghting the importance of understanding early biological markers in the natural history of PASC.

88 ue and/or other discriminatory properties of biological markers in the saliva of patients with COVID-

89 Biological markers including the neutrophil-lymphocyte r

90 Long-lived men also exhibited several biological markers indicative of greater insulin sensiti

91 However, the exploration of molecular and biological markers is still at an early stage.

92 robe targeted at alpha(v)beta(3) integrin, a biological marker known to modulate angiogenesis, was de

93 We correlated biological marker levels with negative control (C) asymp

94 ated further by looking at valuable specific biological markers like troponin I and natriuretric pept

95 In future trials, baseline plasma biological markers may help identify patients who are mo

96 Adult telomere length (TL) is a biological marker of aging associated with vascular heal

97 IONALE: Leukocyte telomere length (LTL) is a biological marker of aging, and shorter LTL is associate

98 Leukocyte telomere length (LTL) is a biological marker of aging, and shorter LTL is associate

99 ronic stress exposure and allostatic load, a biological marker of chronic physiological dysregulation

100 nic stress is measured by allostatic load, a biological marker of cumulative wear and tear on the bod

101 It is unknown whether PMP22 can be used as a biological marker of disease progression and therapy eff

102 contributes to VEE virus virulence and is a biological marker of epizootic potential.

103 recent attention as a sensitive and specific biological marker of ethanol consumption.

104 tion in studies of DNA adduct formation as a biological marker of exposure to carcinogens and for env

105 vels of procollagen peptide III (PCP III), a biological marker of fibroproliferation, and with increa

106 amocortical mechanisms and could represent a biological marker of illness.

107 eukocyte telomere length (LTL) is a putative biological marker of immune system age, and there are de

108 The search for a unique biological marker of language-based learning disabilitie

109 Semen quality has been suggested to be a biological marker of long-term morbidity and mortality;

110 A self-reported intake in conjunction with a biological marker of macronutrient intake was required a

111 SD and SLI-history groups), but may act as a biological marker of persisting SLI.

112 , a measure of platelet size, is a potential biological marker of platelet function.

113 uroblastomas and is the single most powerful biological marker of poor prognosis in this disease.

114 T, the catalytic subunit of telomerase, is a biological marker of progression in several cancers.

115 lomere length (TL), another well-established biological marker of psychological stress and show that

116 The most common biological marker of suicide is reduced concentrations o

117 aspartate receptors, represents a precocious biological marker of the disease.

118 R levels at admission could provide an early biological marker of the outcome of cerebral malaria.

119 n 30 and 70% because no clinically validated biological marker of the SOZ exists.

120 y useful platform for rapidly validating any biological marker of this common disease.

121 IL-6 is a biological marker of ventilator-associated lung injury t

122 hbourhood deprivation and allostatic load, a biological marker of wear and tear, taking into account

123 alization is both an early psychological and biological marker of worse later psychiatric outcomes.

124 ative to angiopoietin-1 with physiologic and biological markers of activated endothelium.

125 ese findings suggest that the 2 measures are biological markers of AD pathophysiology.

126 The development of biological markers of aging has primarily focused on adu

127 fect on chronological longevity and many key biological markers of aging in the absence of environmen

128 Chronological aging independent of biological markers of aging is the primary risk factor f

129 As one of the most extensively studied biological markers of aging, telomere length (TL) provid

130 erived from land use regression modeling and biological markers of airway inflammation.

131 a represents a viable avenue for identifying biological markers of antidepressant treatment response.

132 , the estimated energy balance, clinical and biological markers of cachexia, and survival.REE was mea

133 ermetabolism is correlated with clinical and biological markers of cancer cachexia and is associated

134 Biological markers of childhood allergy are associated w

135 cies of CD8(+) CD28(null) T cells are useful biological markers of compromised immunocompetence, iden

136 ted dimethylarginines may serve as important biological markers of deleterious outcome in alcoholic h

137 There are few biological markers of delirium, perhaps related to the e

138 Robust biological markers of dietary exposure are essential in

139 n-targeted metabolite profiling can identify biological markers of dietary exposure that lead to a be

140 e currently being explored as more sensitive biological markers of disease activity.

141 Moderate to weak correlation was found with biological markers of endothelial activation.

142 le studies connecting human experiences with biological markers of energetic stress.

143 and health is hindered by the lack of robust biological markers of food exposure.

144 a virus vaccine responses and can be used as biological markers of immunosenescence.

145 The identification of biological markers of impulsivity may lead to a better u

146 ides a potential strategy for development of biological markers of lipid modification of proteins fol

147 toward identification of early clinical and biological markers of long-term risk as well as avenues

148 e-body protein balance (WBPB) and associated biological markers of metabolic response.

149 on the activation of nuclear factor-kappaB, biological markers of neuronal injury, and neurobehavior

150 in vivo magnetic resonance imaging (MRI) and biological markers of oxidative stress and inflammation,

151 esearch has focused on the identification of biological markers of response to antidepressant treatme

152 isorders, longitudinal high-risk research on biological markers of risk has become a priority.

153 oblematic for researchers aiming to identify biological markers of schizophrenia or psychosis.

154 ses were evaluated according to clinical and biological markers of severity and multi-organ manifesta

155 ponsible for susceptibility to CHD, specific biological markers of stress are increasingly being meas

156 The identification of biological markers of successful treatment response may

157 20% and 80%, because no clinically validated biological markers of the epileptogenic zone exist.

158 letal function described here will be useful biological markers of the functional effects of BCR/ABL

159 The identification of blood-based biological markers of the therapeutic response would ena

160 ponse in individual patients, review MRI and biological markers of treatment response, and summarise

161 al clinical disorders, they might constitute biological markers of vulnerability, linking exposure to

162 ts but are also associated with quantitative biological markers or quantitative risk factors.

163 Use of biological markers, or biomarkers, potentially offers a

164 example, an abnormal circulating level of a biological marker pertinent to the study drug; and c) pa

165 Numerous reports suggest that biological markers predict survival in patients undergoi

166 Although the prevailing view is that basic biological markers regulate this circadian modulation, b

167 This study helps to characterize biological markers related to angiogenesis, growth facto

168 rther studies with physiologic, genetic, and biological markers related to these phenotypes will be n

169 monstrating potential clinical, imaging, and biological markers relating to BsAbs are growing.

170 Because AF identifies these characteristic biological markers so specifically, autofluorescence met

171 going efforts toward identification of early biological markers specific to subphenotypes of ASD.

172 ld-type (WT) mice in the following important biological markers: spontaneous eschar separation, thinn

173 In addition, the biological marker studied here provided additional predi

174 We review clinical features and biological markers studied in relation to outcome of lon

175 Of all the biological markers studied, only increased density of CD

176 may make this group a homogeneous sample for biological marker studies.

177 aining the poor predictive value of isolated biological markers such as ferritin level.

178 Putative schizophrenia-associated biological markers, such as abnormal evoked response, oc

179 omprising DNA methylation markers with other biological markers, such as AFP, provide an option to fu

180 Given the lack of concrete biological markers, such as laboratory tests or imaging

181 Objective biological markers, such as those based on brain imaging

182 exia nervosa after recovery could indicate a biological marker that alters the normal motivation to e

183 espite extensive research there is as yet no biological marker that can aid in its diagnosis and cour

184 A biological marker that helps to objectively define the d

185 hite matter abnormalities may be a heritable biological marker that indicates increased vulnerability

186 Identification of a biological marker that might improve these odds could ha

187 te significant effort, there is no universal biological marker that serves as a metric for metastatic

188 A biological marker that would allow clinicians to determi

189 Currently there are no prognostic biological markers that accurately predict conversion of

190 The availability of robust quantitative biological markers that are correlated with qualitative

191 A more reliable approach would be to use biological markers that are specific for SHS exposure an

192 Universal biological markers that clearly identify potentially met

193 The identification of biological markers that define subtypes of major depress

194 se who are sensitized, there are no specific biological markers that differentiate between allergic a

195 isorder and those with pure ADHD to identify biological markers that distinguish these clinically ove

196 ific therapeutic interventions benefits from biological markers that match the intervention.

197 social factors in cancer and concentrates on biological markers that may mediate such relationships.

198 Indeed, emerging clinical and biological markers that might be used to quantify pre-sy

199 This research field critically needs biological markers that specifically identify the reside

200 esign of clinical trials, the development of biological markers, the advent of genetic animal models,

201 In the absence of biological markers, the development of new diagnostic cr

202 the deformation of the tissue directly from biological markers, thus providing 3D cellular scale inf

203 A widely used biological marker to identify the active form of ATM is

204 ea, NAAT negative), we aim to discover novel biological markers to distinguish between these cohorts.

205 There is a lack of biological markers to evaluate the effectiveness of anti

206 e intra-alveolar space suggests the need for biological markers to guide response to therapy.

207 ing candidates for applications ranging from biological markers to organic light-emitting diodes (OLE

208 hysiology of these disorders and the lack of biological markers to stratify and individualize patient

209 nd to collect cerebrospinal fluid to develop biological markers to track disease stage and progressio

210 nt for many common diseases, but established biological markers to track stress and guide investigati

211 notypic targets, such as symptom severity or biological markers, to enhance gene discovery and the tr

212 Allergy measures included biological markers (total serum immunoglobulin E (tIgE),

213 t GRN mutation status and could be used as a biological marker, we optimized a GRN ELISA and studied

214 ndition and identify potential molecular and biological markers, we analyzed demographic information,

215 g would be easier if individually predictive biological markers were available.

216 Biological markers were measured before challenge.

217 Neuroinflammatory and lipid-based biological markers were then assessed.

218 We suggest that a low HVA level is a biological marker with modest association to the diagnos

219 orts to develop tissue-sensitive imaging and biological markers with diagnostic and prognostic utilit

220 ic foci provide a clinically relevant set of biological markers with potential importance for develop

221 re used to assess the association of pleural biological markers with septation presence and severity