ライフサイエンスコーパス: birth

1 10% of childhood blindness (~1 in 5000 live birth).

2 ered to infants weighing less than 1000 g at birth.

3 s to a more mature but quiescent state after birth.

4 nably improve the quality of data on care at birth.

5 tinuing with direct dosing of the pups after birth.

6 e of calcineurin in promyelinating SCs after birth.

7 angements did not significantly expand after birth.

8 ses of death and morbidity following preterm birth.

9 n (IUGR) and small-for-gestational age (SGA) birth.

10 mined were unrelated to SGA, LBW, or preterm birth.

11 nd pulmonary hypertension (PH) after preterm birth.

12 njugate vaccine at 2, 4, and 11 months after birth.

13 ment and cardiomyocyte maturation soon after birth.

14 ucleus of the pulvinar are well-developed by birth.

15 ytes extracted from cord blood shortly after birth.

16 temporally through tamoxifen treatment after birth.

17 alth but fails to reduce the risk of preterm birth.

18 /or birth weight <1500 g were enrolled after birth.

19 dentify risk factors for sudden death before birth.

20 d lifespan and are depleted near the time of birth.

21 g/kg/day of DEHP from gestation day 11 until birth.

22 developing embryo from embryonic day 9.5 to birth.

23 ment and shape gut microbiota assembly after birth.

24 ceptor CXCR6 and seed meninges shortly after birth.

25 k between maternal HIV infection and preterm birth.

26 d sPTB, 4.0% had pi-PTB and 89.3% had a term birth.

27 gulatory T-cell populations were measured at birth.

28 t delays robust Foxl2 expression until after birth.

29 om the general population by sex and year of birth.

30 ced by adult hemoglobin S at about 1 y after birth.

31 on were no fetal heartbeat heard or imminent birth.

32 re common in the exit survey after caesarean birth.

33 aged 14-24 years (n = 1,033) with singleton birth.

34 ) is the commonest diagnosis after premature birth.

35 ride Study, which has followed a cohort from birth.

36 e LSt group is sexually dimorphic soon after birth.

37 vidual variation in brain development before birth.

38 supported fetal development to term and live birth.

39 al mortality ratio was 36.2 per 100,000 live births.

40 dystrophy (DMD) affects 1 in 3500 live male births.

41 0% versus 50% to 84% for spontaneous vaginal births.

42 % versus 2% to 30% for episiotomy in vaginal births; 3% to 30% versus 1% to 7% for instrumental vagin

43 n = 292 healthy newborn infants (mean age at birth = 39.9 weeks) with regional patterns of gene expre

44 ht, length/height, and head circumference at birth, 5 mo, 12 mo, and 5 y were included in a path mode

45 Glyburide-exposed neonates were heavier at birth (58.20 g, 95% confidence interval [CI] 10.10-106.3

46 Among the 150 patients who gave birth (76.9%; 170 babies), pregnancy complications and c

47 At birth, a higher proportion of activated regulatory T cel

48 al gestational weight gain (GWG) and preterm birth according to pre-pregnancy body mass index (BMI) a

49 ce intervals (CIs) for SGA, LBW, and preterm birth across tertiles (or categories) of DBP biomarker c

50 us was significantly associated with preterm birth (age-adjusted odds ratio [aOR], 1.50; 95% confiden

51 term birth, particularly spontaneous preterm birth among nulliparous women.

52 There was a higher risk of preterm birth among women exposed to gabapentin either late (RR,

53 efects, 409 of 5426 (8%) resulted in preterm birth and 333 of 5426 (6%) in low birth weight.

54 s of South America, often become infected at birth and account for up to 95% of newly reported chroni

55 er time, and to breastfeed immediately after birth and at hospital discharge.

56 passed from mothers to their progeny during birth and breastfeeding.

57 nvestigated the relationship between mode of birth and childhood infection-related hospitalisation in

58 996 to December 31, 2015 using record-linked birth and hospitalisation data from Denmark, Scotland, E

59 the entire width of the muscle cell both at birth and in mature muscle.

60 CI are diagnosed early, generally soon after birth and in some cases before birth by fetal ultrasound

61 ot achieving viral suppression before giving birth and increased mother-to-child transmission of HIV.

62 Preterm birth and LBW were assessed using maternal reports from

63 s Health (ALSWH) for the analyses of preterm birth and LBW, respectively.

64 entified sex differences that are present at birth and maintained throughout life, and those that are

65 ulting from p62 deficiency is manifest after birth and obesity subsequently develops despite normal f

66 delta TCR repertoires develop directly after birth and present important differences among gammadelta

67 es the association between maternal place of birth and severe pre-eclampsia in the PreCARE cohort of

68 in the association between maternal place of birth and the development of severe pre-eclampsia.

69 ociation between cord serum interleukin-7 at birth and the trajectories of children's anxiety-depress

70 union (cohabitation or marriage), and first birth and used logistic regression to show the change in

71 iBAT has two different growth phases between birth and weaning: increase of BAs size and number in th

72 th higher AAMRs had significantly fewer live births and a younger ALB.

73 Data Collection we compared rates of preterm births and small-for-gestational-age infants born in Aus

74 nual incidence of MNM was 7.2 per 1,000 live births and the intra-hospital maternal mortality ratio w

75 5 exposures during the final week on preterm birth, and departures from additive joint effects were a

76 ne the timing of parturition, the process of birth, and how they are coordinated with fetal developme

77 nfected during the third trimester had given birth, and no infants with apparent abnormalities, inclu

78 lonized vaginally with serotype-Ia or III at birth, and their healthy infants were eligible as matche

79 turation of the mammalian brain occurs after birth, and this stage of neuronal development is frequen

80 epidemics on networks are approximated by a Birth-and-Death process which keeps track of the number

81 30% versus 1% to 7% for instrumental vaginal births; and 42% to 70% versus 50% to 84% for spontaneous

82 le and higher family socioeconomic status at birth are strong and consistent predictors of lower phys

83 Birth asphyxia constitutes a major global public health

84 th 95% confidence intervals (CIs) of preterm birth associated with smoking status and the number of c

85 Skilled, high-quality health providers and birth attendants are important for reducing maternal mor

86 lity rates may relate to the availability of birth-attending and primary care providers.

87 ss index during 5 age periods (conception to birth, birth to age 3 months, ages 3-12 months, ages 12

88 ly for microcephaly infants, were poor after birth but showed improvement beyond 4 months of life.

89 of infants with cCMVi have normal hearing at birth, but are at risk of developing late-onset SNHL.

90 cing has transformed genetic diagnosis after birth, but its usefulness for prenatal diagnosis is stil

91 In urban children birth by cesarean section is associated with food allerg

92 ly soon after birth and in some cases before birth by fetal ultrasound.

93 Interruption to gestation through preterm birth can significantly impact cortical development and

94 lthough the epicardium becomes dormant after birth, cardiac injury reactivates developmental gene pro

95 In the "Children of 1997" birth cohort (n = 8,327), we used adjusted multivariable

96 ge 70 to age 79, in the longitudinal Lothian Birth Cohort 1936 study.

97 of 3079 participants of the Northern Finland Birth Cohort 1966 who had reported LBP over the previous

98 at age 6-10 years of a multicentre European birth cohort based was undertaken using an online parent

99 The HE and PE estimates in the 1995 birth cohort for HPV18/31/33 were significant in the gen

100 n the Generation R Study, a population-based birth cohort in Rotterdam, Netherlands (enrollment 2002-

101 tibiotic exposure were assessed monthly in a birth cohort of 271 children aged 0-24 months.

102 evaluated this association in a prospective birth cohort setting.

103 Important lessons have been learned from birth cohort studies examining viral infections and subs

104 B study-an ongoing, multi-ethnic prospective birth cohort study in Bradford.

105 nrolled in the Wisconsin Infant Study Cohort birth cohort study.

106 We will prospectively follow-up a birth cohort to obtain incidence data on RSV acute respi

107 loratory case-control study within LIFECODES birth cohort was performed.

108 The ALSPAC birth cohort was used for independent replication.

109 In a population-based birth cohort we measured mite, cat, and dog allergen lev

110 lthy Infant Longitudinal Development (CHILD) birth cohort were studied.

111 ffspring of mothers from the Danish National Birth Cohort who filled out a food-frequency questionnai

112 ong 887 participants from a population-based birth cohort with severe wheeze exacerbations confirmed

113 In this high-allergy-risk birth cohort, some profiles of HMOs were associated with

114 285 mothers enrolled in a high-allergy-risk birth cohort, the Melbourne Atopy Cohort Study.

115 Among the birth cohort, we measured biomarkers of gut inflammation

116 atory health/allergy at 8 years in the PARIS birth cohort.

117 n Prospective Studies on Asthma in Childhood birth cohort.

118 ancy, and childhood in a UK population-based birth cohort.Methods: Individual exposures to source-spe

119 g data from three population-based Brazilian birth cohorts (analytical samples: n = 2740 for 1982 coh

120 Using 3 nationally representative British birth cohorts, we investigated whether the duration of o

121 how age patterns change over time and across birth cohorts.

122 rred in RTN neurons precisely at the time of birth, coinciding with exposure to the external environm

123 had significantly increased odds of preterm birth compared with adequate GWG in underweight women ag

124 Similarly adjusted, preterm birth (compared to full-term birth) was associated with

125 utable to a prenatal etiology with secondary birth complications.

126 potential pitfalls, of the approach using a birth-death model with both synthetic and experimental d

127 We use a piecewise-constant birth-death model, combined with both Gaussian Markov ra

128 Therefore birth-death models are central to macroevolutionary as w

129 Sufficiently flexible time-varying birth-death models are still lacking.

130 productive numbers (Re) were estimated using birth-death models for large clusters that expanded >=2-

131 ecreased monotonically by 0.03 degrees C per birth decade.

132 tal abnormalities were classified as a major birth defect according to the European Concerted Action

133 lying causes of child deaths were congenital birth defects (39 [13%] of 304 deaths), lower respirator

134 s the most common infectious cause of infant birth defects and an etiology of significant morbidity a

135 understanding of the etiology of associated birth defects and medical conditions among those with tr

136 ging translational evidence of genitourinary birth defects and their impact on male infertility.

137 Recently, birth defects have been observed in patients with varian

138 ng illness in infected adults and congenital birth defects in infants born to mothers infected during

139 of Biologists Workshop 'Understanding Human Birth Defects in the Genomic Age' held in the UK in Nove

140 ) signaling and aberrant HH signaling causes birth defects or cancers.

141 which 12 (4%; 2-6) were adjudicated as major birth defects potentially related to DOAC exposure.

142 essed conditions and is the leading cause of birth defects related to infectious disease.

143 ter the exclusion of pregnancies affected by birth defects, 409 of 5426 (8%) resulted in preterm birt

144 population, is a leading cause of congenital birth defects, and poses serious risks for immuno-compro

145 livery, maternal adverse effects, congenital birth defects, childhood cancer.

146 (CP) is one of the most common craniofacial birth defects, impacting about 1 in 800 births in the US

147 79A and R695H in a heterozygous state caused birth defects, it would be via haploinsufficiency of MYR

148 s the most common infectious cause of infant birth defects, resulting in permanent neurological disab

149 NAD synthesis as potential causes of complex birth defects.

150 derlies human diseases, including cancer and birth defects.

151 es regarding the potential causes of cardiac birth defects.

152 Hepatitis B birth dose coverage of 90% is unlikely to be reached und

153 le-income countries (LMICs) receive a timely birth dose.

154 hat the location of the VWFA is earmarked at birth due to its connectivity with the language network,

155 al complications, including preterm and twin birth, eclampsia and toxemia, shorter period of breastfe

156 gy for fetal protection and delay of preterm birth elicited by sterile stimuli.

157 d-race, intercensal population data and live birth estimates were used as denominators.

158 outside the United States, four or more live births, exposure to secondhand tobacco smoke, and ever p

159 at the effect could be mediated by labor and birth exposures.

160 adjusted for sex, education, age, country of birth, father's occupation, ever-consumed alcohol, and a

161 ccine were given at 2, 3, and 4 months after birth followed by a booster at 11 months and a 10-valent

162 DBS are collected at birth for the child.

163 All mothers who gave birth from 2008 to 2016 while living within 15 km of LAX

164 l membranes (FM) collected immediately after birth from women delivering preterm, p-IRAK1 was signifi

165 1997, and April, 2018, we identified 716 478 births from 338 223 women in 123 Demographic and Health

166 cohort study of all recorded singleton live births from January 1, 1996 to December 31, 2015 using r

167 study included 3422 and 3508 singleton live births from the Australian Longitudinal Study on Women's

168 close proximity to a greener environment at birth has a protective effect on the development of alle

169 aimed to compare two questionnaires: a full birth history module with additional questions on pregna

170 Survivors following very premature birth (i.e., <= 32 weeks gestational age) remain at high

171 r second trimester of pregnancy with preterm birth in a large-scale population-based retrospective co

172 ation-based study is a secondary analysis of Birth in Brazil study, conducted in 266 maternity units

173 as atrioventricular bundle hypoplasia after birth in heterozygous mice.

174 Furthermore, the timings of ovulation and birth in some species occurs within a very short interva

175 born health-care coverage around the time of birth in survey data and routine facility register data.

176 n-based EPICE cohort study (all very preterm births in 19 regions from 11 European countries, 2011-20

177 cial birth defects, impacting about 1 in 800 births in the USA.

178 person-years (or 831 deaths per 100,000 live births) in the first year postpartum.

179 stages from 10.5 days after conception until birth, including transcriptomes, methylomes and chromati

180 Numbers of preterm births increased in association with heatwave exposures

181 At birth, infants who remained healthy had significantly hi

182 y risk and outcomes (such as Apgar score and birth injuries) and 7 found limited evidence for an asso

183 Infection-induced preterm birth is a major cause of neonatal mortality and morbidi

184 gnificant clinical relevance because preterm birth is the leading cause of infant and under 5 year ol

185 2), whose expression decreases in mice after birth, is essential for nuclear envelope breakdown prior

186 was associated significantly with country of birth, language spoken at home, and marital status.

187 njury to the sensory receptors shortly after birth leads to predictable pattern alterations at all le

188 Higher birth length tended to increase risk of premenopausal br

189 lycemic control in mothers and birth weight, birth length, macrosomia, and large for gestational age

190 parity data, and not having had at least two births (livebirth or stillbirth) in the 5 years before t

191 Preterm birth (<37 weeks of gestation) and its complications are

192 repregnancy BMI, previous history of preterm birth, marital status, infant sex, and initiation of pre

193 Maternal age at birth, maternal level of education, household income, as

194 rt-term reduction in women's CD risk after a birth may reflect biological effects of pregnancy.

195 ampsia suggest that the incidence of preterm birth might also be decreased, particularly if initiated

196 in female fertility as observed by maternal birth month and whether this change was correlated with

197 delay and medical refractory seizures since birth most recently presented with continuous simple par

198 I proteins to silence transposons but, after birth, most post-pubertal pachytene piRNAs map to the ge

199 icant improvement in maternal mortality when birthing mothers share race with their physician.

200 , including hydrops, respiratory distress at birth, need for supplemental oxygen, neonatal ventilator

201 At birth, neonatal triceps and subscapular skinfold thickne

202 n countries with higher rates of spontaneous birth (nulliparous: rho = 0.65).

203 understanding the pulse behavior during the birth of an ultrafast mode-locked laser pulse and the st

204 Since the birth of biotechnology, hundreds of biotherapeutics have

205 uction of lithium into widespread use at the birth of modern psychopharmacology, that medication rema

206 ks the 50th anniversary of Earth Day and the birth of the modern environmental movement.

207 e issues, we herein leverage the presence at birth of two types of locomotor-like movements, spontane

208 n is triggered upon adding a fixed size from birth), or a Sizer-Adder combination.

209 nal data, and report estimates stratified by birth order.

210 smoking only was not associated with adverse birth outcomes but was associated with a higher risk of

211 f trimester-specific DBP exposure on adverse birth outcomes in humans are inconsistent.

212 eview, 21 focused on diabetes, 15 on adverse birth outcomes, 8 on cardiovascular disease, 3 each on o

213 tion and meteorological variables on adverse birth outcomes.

214 y is associated with a lower risk of preterm birth, particularly spontaneous preterm birth among null

215 odontal disease as a risk factor for preterm birth, preeclampsia, and fetal growth restriction.

216 The birth prevalence of pseudostrabismus and the subsequent

217 Less than half of human zygotes survive to birth, primarily due to aneuploidies of meiotic or mitot

218 couples are more likely to cohabit and give birth prior to marriage and less likely to marry at all.

219 Preterm birth (PTB) affects nearly 15 million infants each year.

220 major depressive disorder (MDD) and preterm birth (PTB), and prenatal depression associates with PTB

221 quantify changes in the incidence of preterm birth (PTB), term low birth weight (TLBW), autism spectr

222 ve PKU, with global prevalence 1:23,930 live births (range 1:4,500 [Italy]-1:125,000 [Japan]).

223 butions, to approximate arbitrary changes in birth rate through time.

224 e Australian gecko family Pygopodidae (where birth rates are interpretable as speciation rates), the

225 m HIV subtype A in Russia and Ukraine (where birth rates are interpretable as the rate of accumulatio

226 disparities using intergenerationally linked birth records of 379,794 California-born primiparous mot

227 cases were identified by linking the Medical Birth Register to the National Patient and Cause of Deat

228 822,843 individuals from the Swedish Medical Birth Register, born in Sweden between January 1, 1982,

229 pharmacoepidemiology study uses Scandinavian birth registry data to estimate the association between

230 ll and by clinical type) occurring after the birth-related discharge date were identified in children

231 ile number, size and quality of offspring at birth remained unaffected.

232 Preterm birth remains a common cause of neonatal mortality, with

233 ing gammadelta T (Tgammadelta17) cells, from birth resulted in spontaneous, highly penetrant AD with

234 review is to comprehensively investigate the birth season and female fertility relationship.

235 We examined how birth size and growth in infancy and childhood were asso

236 associations between four outcomes (preterm birth, small-for-gestational age, continuous gestational

237 of Ascl1 yielded a similar delay in neuronal birth, suggesting that Ascl1 cannot rescue the loss of N

238 tal anomaly with the follow-up starting from birth that were published in the English language as pee

239 trypsinogen in 78 organ donor pancreata from birth through adulthood in control subjects and those at

240 erence growth and adiposity acquisition from birth through infancy.

241 ed oral microbiome communities shortly after birth, through adulthood, and up to 1 y of life in a con

242 e (11 pm to 7 am), age combined with preterm birth, time after weaning from supplemental oxygen or fl

243 by age (12 to <18 years, 2 to <12 years, and birth to <2 years) via interactive response technology.

244 easurements of elemental concentrations from birth to 10 years.

245 verify the associations between growth from birth to 18 y and intelligence and schooling in a cohort

246 Patients from birth to 22 years of age with AT/RT were eligible.

247 Human infants from birth to 24 months of age who did not receive any sedati

248 Infants (N = 1871) were followed from birth to 6 months of age and visited weekly to detect pn

249 87 Malian mother-infant pairs, followed from birth to 6 months of age.

250 x during 5 age periods (conception to birth, birth to age 3 months, ages 3-12 months, ages 12 months-

251 of 324 healthy infants were followed up from birth to age 3 years.

252 pulations: children in early childhood (from birth to age 6) and parents in early adulthood (in their

253 We show that, from birth to hearing onset, the auditory system relies on a

254 before 32 weeks' gestation and randomized at birth to receive early high-dose recombinant human eryth

255 The study population consisted of all births to women in Western Australia from 1980 to 2015,

256 , which naturally undergoes regression after birth, to gain mechanistic insights that could be therap

257 re likely to have skin-to-skin contact after birth, to have it for a longer time, and to breastfeed i

258 potential confounders (age, race, country of birth, total people per household, US region, and histor

259 y showing that any amplification under death-Birth updating is necessarily bounded and transient.

260 Premature birth was identified in 15.0% of EXPECT infants and 11.3

261 EOS risk per 1000 live births was 1.48; 0.76 in term and 15.52 in preterm infan

262 Neonatal mortality rate per 1000 live births was 11.4.

263 justed, preterm birth (compared to full-term birth) was associated with lower FEV(1)/FVC and FEF(25-7

264 s with gestational age (GA) <32 weeks and/or birth weight <1500 g were enrolled after birth.

265 triction (aOR, 1.17; 95% CI, 1.01-1.37), low birth weight (aOR, 1.91; 95% CI, 1.33-2.76), and fetal d

266 s were collected from premature infants with birth weight (BW) <= 1800 g, estimated gestational age (

267 e incidence of preterm birth (PTB), term low birth weight (TLBW), autism spectrum disorder (ASD), and

268 aimed to determine if prematurity and lower birth weight are associated with poorer lung function in

269 ge, maternal smoking, sex-gestation-specific birth weight centile, gestational age, 5-minute Apgar sc

270 ransplanted mothers born preterm or with low birth weight compared with similar controls.

271 e design and interpretation of MR studies of birth weight investigating effects of fetal growth on la

272 n later-life cardiometabolic disease because birth weight is only a crude indicator of fetal growth,

273 The paternal transmitted birth weight score was significantly associated with red

274 A 250-g increase in birth weight was associated with 5.2 mum (95% confidence

275 Associations for birth weight were stronger in boys for FEV(1) (boys: 0.3

276 Controlling for gestational age, birth weight, and BPD severity, MR-EI was associated wit

277 ed with intrauterine growth restriction, low birth weight, and fetal death, but findings are limited

278 aturity, as assessed via gestational age and birth weight, as well as with reduced cognition as measu

279 uate GWG and glycemic control in mothers and birth weight, birth length, macrosomia, and large for ge

280 f ZIKV on the prevalence of prematurity, low birth weight, small-for-gestational-age, and fetal death

281 ted with increased risks of prematurity, low birth weight, small-for-gestational-age, or fetal death.

282 Thirty-three studies (n = 4,733) reported birth weight.

283 in preterm birth and 333 of 5426 (6%) in low birth weight.

284 In extremely-low-birth-weight infants, a higher hemoglobin threshold for

285 des (HMOs) and late-onset sepsis in very-low-birth-weight infants, and to describe the composition an

286 tic formulations on outcomes of preterm, low-birth-weight neonates, we found moderate to high evidenc

287 -threshold group (relative risk adjusted for birth-weight stratum and center, 1.00; 95% confidence in

288 tric anal sphincter injury following vaginal birth were found in countries with higher rates of spont

289 h PM2.5 exposure to increase risk of preterm birth, which adds new evidence to the current understand

290 WG, had significantly higher odds of preterm birth, which increased with maternal age (1.80 [1.16-2.7

291 ences in early microbial exposure by mode of birth, which should be investigated by mechanistic studi

292 ect against oxidative stress associated with birth while ensuring energy availability to the neonate.

293 future behavior with brain microstructure at birth will reveal structural basis of behavioral emergen

294 Because approximately 80% of births worldwide take place in facilities, standardising

295 ccounting for individual matching on sex and birth year were used to estimate the risk of XFS in pati

296 k and Sweden, who were matched for sex, age, birth year, and place of residence.

297 ction, with single-year categories of infant birth year, maternal age, and age-specific HPV vaccinati

298 and proportions by sex, race/ethnicity, and birth year.

299 These studies covered birth years from 1970 to 2015.

300 otal, 22 cohorts were population-based, with birth years ranging from 1991 to 2015.