ライフサイエンスコーパス: bisphenol

1 cations and the limits of detection for each bisphenol.

2 anged after additionally adjusting for other bisphenols.

3 ylhexyl phthalate (DEHP) and polychlorinated bisphenol 153 (PCB153), were detected in adult dog teste

4 Bisphenol A (2,2-bis[4-hydroxyphenyl]propane, BPA), the

5 By combination with an antibody directed to bisphenol A (alphaBPA), this conducting polymer-based bi

6 hinylestradiol (EE2)] and two xenoestrogens [bisphenol A (BPA) and 4-n-nonyl-phenol (NP)] in surface

7 The thermal degradation of bisphenol A (BPA) and bisphenol S (BPS) was investigated

8 al was to provide a definitive evaluation of bisphenol A (BPA) and explain disparities between tradit

9 Bisphenol A (BPA) and its brominated derivative tetrabro

10 the impact of serially assessed exposure to bisphenol A (BPA) and phthalates on measures of kidney f

11 ence regarding associations for exposures to bisphenol A (BPA) and phthalates with type 2 diabetes (T

12 is growing concern that prenatal exposure to bisphenol A (BPA) and phthalates, which are widely used

13 The endocrine disruptor bisphenol A (BPA) and the pharmaceutical 17alpha-ethinyl

14 Phthalates and bisphenol A (BPA) are endocrine disruptors, and previous

15 Phthalates and bisphenol A (BPA) are used in some personal care product

16 Phthalates and bisphenol A (BPA) are widely used industrial chemicals t

17 Increasing concern over bisphenol A (BPA) as an endocrine-disrupting chemical an

18 cale sequencing batch reactors (SBRs), using bisphenol A (BPA) as the TOrC.

19 erns, variability, and predictors of urinary bisphenol A (BPA) concentrations in 337 children from th

20 ective optical sensor to detect and quantify Bisphenol A (BPA) contamination.

21 ivity, bisphenol S (BPS) as a substitute for bisphenol A (BPA) could actually increase the risk of en

22 ent study revealed that prenatal exposure to bisphenol A (BPA) disrupted the transcriptome profiles o

23 A positive association between Bisphenol A (BPA) exposure and coronary heart disease ha

24 n expert panel reviewed associations between bisphenol A (BPA) exposure and reproductive health outco

25 We used in utero bisphenol A (BPA) exposure as a model environmental expo

26 Prenatal bisphenol A (BPA) exposure has been associated with adve

27 ticipants previously randomly selected for a bisphenol A (BPA) exposure study who had unusually high

28 Human health risks due to bisphenol A (BPA) exposure through canned food consumpti

29 Early-life exposure to bisphenol A (BPA) has been implicated to play a role in

30 Bisphenol A (BPA) has been linked with pediatric asthma

31 Exposure to bisphenol A (BPA) has been reported to alter global gene

32 Prenatal exposure to nonylphenol (NP) and/or bisphenol A (BPA) has been reported to be associated wit

33 widespread environmental disrupting chemical bisphenol A (BPA) has estrogenic activity, but its impli

34 Pressures to ban bisphenol A (BPA) has led to the use of alternate chemic

35 Although prenatal exposure to bisphenol A (BPA) has recently been associated with the

36 We report on an innovative heterogeneous bisphenol A (BPA) immunoassay based on an electrolyte-ga

37 try, has been developed for determination of bisphenol A (BPA) in canned tomatoes.

38 The determination of bisphenol A (BPA) in foods and beverages sold in Turkish

39 s increasingly been used as a substitute for bisphenol A (BPA) in some "BPA-free" consumer goods and

40 ectrochemical biosensor for the detection of Bisphenol A (BPA) in water has been developed by immobil

41 Bisphenol A (BPA) is a high-production-volume chemical a

42 Bisphenol A (BPA) is an endocrine disrupting compound th

43 Bisphenol A (BPA) is an endocrine disruptor and potentia

44 Bisphenol A (BPA) is an endocrine disruptor frequently d

45 Bisphenol A (BPA) is an endocrine disruptor that affects

46 Bisphenol A (BPA) is an endocrine-disrupting compound wi

47 Bisphenol A (BPA) is an environmental endocrine disrupto

48 The plastic additive bisphenol A (BPA) is commonly found in landfill leachate

49 Bisphenol A (BPA) is found in polycarbonate plastic and

50 Bisphenol A (BPA) is known to be biologically active in

51 The plastic monomer bisphenol A (BPA) is one of the highest production volum

52 ure to the environmental endocrine disruptor bisphenol A (BPA) is ubiquitous and associated with the

53 Bisphenol A (BPA) is widely recognized being an endocrin

54 Bisphenol A (BPA) is widely used in the manufacture of p

55 Laboratory studies show that bisphenol A (BPA) leaches from bisphenol A-glycidyl meth

56 products from store shelves after reports of bisphenol A (BPA) leaching from baby bottles, reusable d

57 oprobe is designed for the quantification of bisphenol A (BPA) levels in the blood after human exposu

58 rly-life exposure to the endocrine disruptor bisphenol A (BPA) may contribute to the development of o

59 tory actions and public concerns, the use of bisphenol A (BPA) may decrease, while the use of BPA alt

60 Early-life exposure to bisphenol A (BPA) may increase childhood obesity risk, b

61 1.0 microM) and a response time of 250 s for bisphenol A (BPA) monitoring.

62 Certain phthalates and bisphenol A (BPA) show reproductive effects in animal st

63 The lowest concentration of bisphenol A (BPA) that induces a response was ~0.1 mg/L

64 Humans are exposed to low-dose bisphenol A (BPA) through plastic consumer products and

65 However, bisphenol A (BPA) was identified by chemical:gene covari

66 In this study, bisphenol A (BPA) was selected as a model compound.

67 The electrochemistry of bisphenol A (BPA) was studied by voltammetric methods at

68 to replace the endocrine disrupting chemical bisphenol A (BPA) while exerting estrogenic effects of c

69 scharge of the endocrine disrupting compound bisphenol A (BPA) with wastewater treatment plant (WWTP)

70 sed on the MS results these were assigned to bisphenol A (BPA), 2,4-di-tert-butylphenol, and a possib

71 Bisphenol A (BPA), a chemical incorporated into plastics

72 t chronic exposure of synthetic xenoestrogen bisphenol A (BPA), a component of consumer plastic produ

73 Bisphenol A (BPA), a component of some dialysis membrane

74 Bisphenol A (BPA), a plastics component, has been descri

75 Bisphenol A (BPA), a prevalent endocrine-disrupting chem

76 Bisphenol A (BPA), a ubiquitous endocrine disruptor that

77 al models suggests that prenatal exposure to bisphenol A (BPA), a ubiquitous endocrine-disrupting che

78 S (BPS) are increasingly used to substitute bisphenol A (BPA), a widespread environmental endocrine

79 Bisphenol A (BPA), an environmental contaminant with wea

80 to investigate the carcinogenic property of Bisphenol A (BPA), an environmental estrogen, by long-te

81 tions have been reported between phthalates, bisphenol A (BPA), and child behavior.

82 Phthalate esters (PEs), bisphenol A (BPA), and parabens (PBs), which are used in

83 We examined the association of bisphenol A (BPA), benzophenone-3 (BP-3), triclosan (TCS

84 ion of bisphenol F diglycidyl ether (BFDGE), bisphenol A (BPA), bisphenol B (BPB), bisphenol F (BPF)

85 e promising lignin-derivable alternatives to bisphenol A (BPA), but limited bioassay data are availab

86 Three selected chemicals, bisphenol A (BPA), diethylhexyl phthalate (DEHP) and min

87 of environmental contaminants, particularly bisphenol A (BPA), is understudied with regard to PTB.

88 vestigated the direct effects of exposure to bisphenol A (BPA), mono-n-butyl phthalate (Pht), and pol

89 in (GEN), and in industrial chemicals, e.g., bisphenol A (BPA), present in plastics and other product

90 With the pressure to ban or limit the use of bisphenol A (BPA), production of alternatives such as bi

91 on ROS scavengers inhibited the oxidation of bisphenol A (BPA), suggesting the possible involvement o

92 te (ICC: 0.55), whereas the ICC was 0.39 for bisphenol A (BPA).

93 for label-free and single step detection of Bisphenol A (BPA).

94 sh register receipts, contain high levels of bisphenol A (BPA).

95 e electrochemical biosensor for detection of bisphenol A (BPA).

96 ting endocrine-disrupting chemicals, such as bisphenol A (BPA).

97 hesis-based studies to assess the effects of bisphenol A (BPA).

98 oplasia from early-life exposure to low-dose bisphenol A (BPA).

99 the formation of chlorinated derivatives of Bisphenol A (ClxBPA), that have higher estrogenic activi

100 ribe a mixture of phthalates, pesticides and bisphenol A (mixture N1) detected in pregnant women of t

101 Upon bisphenol A addition, the competitive dissociation of th

102 PA), 2,4-di-tert-butylphenol, and a possible bisphenol A analog.

103 n sample age groups for case study chemicals bisphenol A and 2,5-dichlorophenol.

104 Bisphenol A and adiposity in an inner-city birth cohort.

105 e strain CBDB1 completely converted TBBPA to bisphenol A and BPB to phenol red with a stepwise remova

106 With the exceptions of bisphenol A and DEET, all TOrCs that were detected in th

107 Combined effects of high-dose bisphenol A and oxidizing agent (KBrO3) on cellular micr

108 Human exposure to phenols, including bisphenol A and parabens, is widespread.

109 cals as EDCs, and we use diethylstilbestrol, bisphenol A and perchlorate as examples to illustrate th

110 Urinary concentrations of bisphenol A and phthalate metabolites measured during pr

111 Recent fast food consumption and bisphenol A and phthalates exposures among the U.S. popu

112 minent plastic-associated chemicals, such as bisphenol A and phthalates, have been thoroughly studied

113 ants of emerging concern were performed with bisphenol A and sulfamethoxazole.

114 l molecules, as we demonstrated with a novel bisphenol A aptamer.

115 We demonstrate that bisphenol A as the template in combination with phthaloy

116 ained from horse radish peroxidase (HRP) and bisphenol A assays were 12.5 ng/ml (2.84x10(-10) M ) and

117 The adsorption and partition of bisphenol A can be quantified using a dual-mode model of

118 fication and temporal variability in urinary bisphenol A concentrations among couples in Utah-the HOP

119 e were no measured chlorinated byproducts of bisphenol A despite occasionally high concentrations of

120 We present here a proof-of-concept for bisphenol A detection; the device could be readily adapt

121 Epoxy resins incorporating bisphenol A diglycidyl ether (BADGE) and novolac glycidy

122 A), bisphenol B (BPB), bisphenol F (BPF) and bisphenol A diglycidyl ether (BADGE) down to 0.50ng/mL;

123 Bisphenol A diglycidyl ether (BADGE)- and bisphenol F di

124 ly lacquered with epoxy resins (ER) based on bisphenol A diglycidyl ether (BADGE).

125 against similarly filled methacrylate-based bisphenol A diglycidyl ether dimethacrylate / triethylen

126 PPARgamma antagonists (bisphenol A diglycidyl ether or GW9662) blunted the effe

127 cerol dimethacrylate (PMGDM) and ethoxylated bisphenol A dimethacrylate (EBPADMA) at mass ratio of 1:

128 xanediol dimethacrylate (HDDMA), ethoxylated bisphenol A dimethacrylate (EBPADMA), and urethane dimet

129 d respiratory health, with ethyl-paraben and bisphenol A exhibiting some consistency across respirato

130 Bisphenol A exposure alters placentation and causes pree

131 ne the multigenerational effects of maternal bisphenol A exposure on mouse pancreatic islets.

132 which factor, the dimethacrylate component, bisphenol A glycerolate dimethacrylate (BisGMA) or photo

133 2-hydroxyethyl methacrylate (HEMA) and 5% of bisphenol A glycidyl dimethacrylate (BisGMA) (PEHB).

134 The monomer mixture consisted of bisphenol A glycidyl methacrylate (BisGMA), hexanediol d

135 als composed of a trimethacrylate monomer or bisphenol A glycidyl methacrylate, which is a monomer st

136 Several functional substitutes of bisphenol A have been proposed in the literature, produc

137 The change in the amount of bisphenol A in all of these food, based on expiration da

138 In this study, the sorption and release of bisphenol A in marine water were investigated through la

139 osensor capable of detecting the presence of bisphenol A in ultra-low concentrations, yielding a wave

140 Finally, we observe that bisphenol A induces rapid and parallel modulation of 5S

141 e proved to be a persistent source releasing bisphenol A into aquatic environments.

142 Bisphenol A is an oil-derived, large market volume chemi

143 However, bisphenol A is not considered safe due to its endocrine

144 Bisphenol A led to a wide range of stress responses, and

145 method was applied for the determination of bisphenol A migrated from some polycarbonate plastic pro

146 The binding of bisphenol A onto the polymer film was successfully chara

147 hromic activation of spiropyran in high T(g) bisphenol A polycarbonate.

148 The trends between bisphenol A pregnancy level and increased asthma and bro

149 lently modified with an aptamer specific for bisphenol A recognition.

150 , the UV filter benzophenone-2 (BP2) and the bisphenol A substitute bisphenol S (BPS) was deciphered

151 w and comparison of the hormonal activity of bisphenol A substitutes.

152 intramolecular glycosylations using a rigid bisphenol A template supplemented with linkers of variou

153 ith FEV1% (beta=-0.59; 95% CI: -1.24, 0.05); bisphenol A tended to be associated with increased rates

154 at levels ranging from 0.6 to 4.6ngg(-1) and bisphenol A was detected in all the samples at concentra

155 dichlorophenols, triclosan, benzophenone-3, bisphenol A) and 11 phthalate metabolites were measured

156 for rapid detection of estrogenic substance (bisphenol A) has been proposed.

157 f other xenoestrogens (e.g., alkylphenols or bisphenol A) that are sometimes found in surface water.

158 compounds detected, one plastics component (bisphenol A), three pharmaceuticals (carbamazepine, sulf

159 micals such as phthalates and phenols (e.g., bisphenol A).

160 mmonly targeted EDC and their isobars (e.g., bisphenol A, (Z)- and (E)-diethylstilbestrol, hexestrol,

161 water consumption was associated with higher bisphenol A, 2,4-dichlorophenol, and 2,5-dichlorophenol

162 Ethyl-paraben, bisphenol A, 2,5-dichlorophenol, and DIDP tended to be a

163 e concentrations of 8 phthalate metabolites, bisphenol A, 25 polychlorinated biphenyls (PCBs), 6 orga

164 the approach allows the direct detection of bisphenol A, a low molecular weight (228 Da) target usua

165 an environmentally relevant concentration of bisphenol A, a nearly ubiquitous environmental contamina

166 Bisphenol A, an endocrine disrupting compound, is widely

167 b copper, 5 ppb cadmium, 10 ppb lead, 10 ppb bisphenol A, and 1 ppb paraoxon, tested as safety limits

168 environmental contaminants (benzo[a]pyrene, bisphenol A, and acrolein) in human serum via a competit

169 to exhibit the highest sorption capacity for bisphenol A, and it was almost irreversible, probably du

170 hyl paraben, propyl paraben, benzophenone-3, bisphenol A, and triclosan in urine collected during 199

171 lphenol, octylphenol, ibuprofen, diclofenac, bisphenol A, and triclosan.

172 hemical detection of the endocrine disruptor bisphenol A, as well as the capture of living nonadheren

173 Exposure to environmental phenols (e.g., bisphenol A, benzophenone-3, and triclosan) and parabens

174 erum testosterone concentrations and urinary bisphenol A, benzophenone-3, triclosan, and paraben leve

175 Bisphenol A, bisphenol S, and 4-hydroxyphenyl 4-isoproox

176 s had overall false positive rates of </=5%, bisphenol A, caffeine, NP, OP, and triclosan had false p

177 d disinfection byproducts of salicylic acid, bisphenol A, gemfibrozil, naproxen, diclofenac, technica

178 ct on pbPPARG, while hexabromocyclododecane, bisphenol A, oxychlordane, and endosulfan were weak anta

179 or by four major endocrine disruptor classes:bisphenol A, polychlorinated biphenyls, organophosphates

180 ed for phthalates and to a lesser extent for bisphenol A, synthetic musks, some pesticides, and PAHs.

181 actone, genistein, daidzein, benzophenone-3, bisphenol A, the sum of parabens (methyl, ethyl, and pro

182 n significantly reduce the teratogenicity of bisphenol A, triclosan and 17alpha-ethinyl estradiol wit

183 pathways have been relatively well studied, bisphenol A, triclosan, and ibuprofen.

184 e, phthalates, fatty acids, alkylphenols and bisphenol A, were quantified.

185 cles can serve as transportation vectors for bisphenol A, which may constitute an ecological risk.

186 gel-filled, and water-filled (most labeled "bisphenol A-free"), were collected from the U.S. market

187 ies show that bisphenol A (BPA) leaches from bisphenol A-glycidyl methacrylate (bisGMA)-based dental

188 co-crystallized with an alkyl derivative of bisphenol A.

189 ahistidine peptide for the quantification of bisphenol A.

190 midispersive solid phase extraction of eight bisphenols (A, AF, AP, C, BP, G, M, and Z) from river wa

191 ndocrine disrupting chemicals (EDCs) such as bisphenol-A (BPA) and diethylstilbestrol (DES).

192 health hazards related to persisting use of bisphenol-A (BPA) are well documented.

193 In contrast, Bisphenol-A (BPA) elicited a non-monotonic response.

194 Bisphenol-A (BPA) is an environmentally ubiquitous estro

195 Bisphenol-A (BPA) is widely used in the manufacture of p

196 Bisphenol-A (BPA), a potent endocrine disrupting compoun

197 ny endocrine disrupting chemicals, including bisphenol-A (BPA), induce non-monotonic dose response (N

198 t famous member of this family of compounds, bisphenol-A (BPA), when spiked (at 1 mug.g(-1) concentra

199 Linking Academic and Regulatory Insights on Bisphenol-A (CLARITY-BPA) is a rare collaboration of gui

200 in diastolic BP were observed with maternal bisphenol-A concentrations (0.7 mm Hg [95% CI: 0.1 to 1.

201 Gestational bisphenol-A in male mice primed macrophages in adulthood

202 that gestational, but not adult, exposure to bisphenol-A increased the development of experimental au

203 Early life exposure to bisphenol-A may represent an environmental consideration

204 factor blocked susceptibility to disease in bisphenol-A mice.

205 Bisphenol-A's rising concentrations in the environment r

206 trogen response after in utero xenoestrogen (bisphenol-A) exposure.

207 We investigated whether bisphenol-A, a prominent contaminant with endocrine-disr

208 A (BPA), production of alternatives such as bisphenol AF (BPAF) and bisphenol S (BPS) are increasing

209 such as BPA analogues bisphenol S (BPS) and bisphenol AF (BPAF) in production of consumer products;

210 s, bisphenol S (BPS), bisphenol F (BPF), and bisphenol AF (BPAF), in 616 archived samples collected f

211 uture exposure trends to BPA and some of its bisphenol alternatives.

212 our ethanol-solubilised complexes with amine bisphenol, aminoalcohol bisphenol or salan ligands are e

213 the urinary concentrations of BPA and three bisphenol analogs, bisphenol S (BPS), bisphenol F (BPF),

214 A series of triphenylethylene bisphenol analogues of the selective estrogen receptor m

215 hirogenic diphosphazanes incorporating bulky bisphenol and 1,1'-bi-2-naphthol-derived substituents vi

216 approximately 10-fold less amounts than were bisphenols and benzophenones.

217 the chemical replacements, however, are also bisphenols and may have similar physiological effects in

218 We examined associations between bisphenols and morbidity due to pediatric asthma.

219 tween concentrations or detection of urinary bisphenols and morbidity outcomes and assessed heterogen

220 ethylhexyl) tetrabromophthalate, halogenated bisphenols and phenols, and hydroxylated PBDEs were foun

221 Common environmental contaminants such as bisphenols and phthalates and persistent contaminants su

222 amount of the sum of nine bisphenols (Sigma9 bisphenols) and 5 benzophenones (Sigma5 benzophenones) l

223 additives (organophosphate esters (OPEs) and bisphenols) and other related organic compounds (perfluo

224 enyl ethers (PBDEs), halogenated phenols and bisphenols, and their metabolites.

225 Prenatal exposures to phthalates and bisphenols are associated with impaired brain developmen

226 ting readily available amines, anilines, and bisphenols as starting materials.

227 entrations of 18 phthalate metabolites and 8 bisphenols at < 18, 18-25, and > 25 wks of gestation.

228 diglycidyl ether (BFDGE), bisphenol A (BPA), bisphenol B (BPB), bisphenol F (BPF) and bisphenol A dig

229 Parabens (p-hydroxybenzoic acid esters), bisphenols, benzophenone-type UV filters, triclosan, and

230 bisalkylsulfonyl fluorides (AA monomers) and bisphenol bis(t-butyldimethylsilyl) ethers (BB monomers)

231 Here we identify the bisphenols bithionol and hexachlorophene as potent, sAC-

232 the toxicity and teratogenic effects of the bisphenols BPA, BPS, BPF, and BPAF in zebrafish embryo-l

233 ent difluoromethylation of a panel of eleven bisphenols (BPs) for their enhanced detection and identi

234 benchmark partial agonist triphenylethylene bisphenol (BPTPE), and antagonists 4-hydroxytamoxifen an

235 ential critical windows of susceptibility to bisphenols by comparing the exposure patterns of newborn

236 BB monomers were obtained from silylation of bisphenols by t-butyldimethylsilyl chloride.

237 effects and potencies between the different bisphenol chemicals.

238 Migration of bisphenol compounds (only BPA and BADGE.2H(2)O detected)

239 simultaneous migration of several metals and bisphenol compounds from coated tinplate cans into veget

240 All bisphenol compounds induced estrogenic responses in Tg(E

241 e trimester-specific associations of urinary bisphenol concentrations with birth weight, birth length

242 , are uncommon antioxidants bearing isomeric bisphenol cores substituted with allyl functions.

243 ssive than simulant B, and concentrations of bisphenols decreased with consecutive exposure to simula

244 BN was applied to concentrate bisphenol derivatives in spiked water samples and the co

245 se extraction method for the enrichment of 5 bisphenol derivatives using hexagonal boron nitride (BN)

246 Furthermore, bisphenol derivatives were analyzed in spiked and non-sp

247 Furthermore, each derivatized bisphenol exhibits unique retention times vastly differe

248 he association between prenatal phthalate or bisphenol exposure and cognition have produced mixed fin

249 maternal urinary biomarkers of phthalate or bisphenol exposure and nonverbal intelligence quotient (

250 did not observe that maternal biomarkers of bisphenol exposure are associated with nonverbal IQ.

251 FDGE), bisphenol A (BPA), bisphenol B (BPB), bisphenol F (BPF) and bisphenol A diglycidyl ether (BADG

252 Bisphenol F (BPF) and bisphenol S (BPS) are increasingly

253 Bisphenol F (BPF) and bisphenol S (BPS), which have been

254 Bisphenol S (BPS) and bisphenol F (BPF) are two such BPA substitutes.

255 its structural analogs bisphenol S (BPS) and bisphenol F (BPF) is associated with asthma morbidity re

256 to structurally similar compounds including bisphenol F (BPF) or bisphenol S (BPS) even at much high

257 three bisphenol analogs, bisphenol S (BPS), bisphenol F (BPF), and bisphenol AF (BPAF), in 616 archi

258 We evaluated tetramethyl bisphenol F (TMBPF) using in vitro and in vivo assays.

259 ery sensitive, allowing for determination of bisphenol F diglycidyl ether (BFDGE), bisphenol A (BPA),

260 Bisphenol A diglycidyl ether (BADGE)- and bisphenol F diglycidyl ether (BFDGE)-based epoxy resins

261 omatography-tandem mass spectrometry, BADGE, bisphenol F glycidyl ether (BFDGE), 3-ring NOGE, and eig

262 BPA and one petroleum-derived BPA analogue (bisphenol F, BPF), and the incorporation of more methoxy

263 ssarily safer and support the removal of all bisphenols from consumer merchandise.

264 ethod for simultaneous determination of five bisphenols in canned energy drinks by UPLC with fluoresc

265 to determine contamination levels from these bisphenols in forty energy drinks of different brands, c

266 Continued biomonitoring of these bisphenols in populations and further investigations on

267 lished the migration and stability of twelve bisphenols in two food simulants (C: 20% ethanol, and B:

268 We assessed the potential EA of both a new bisphenol monomer used to synthesize polymeric coatings

269 complexes with amine bisphenol, aminoalcohol bisphenol or salan ligands are equally or more effective

270 ng agents such as 4,4'-dihydroxybiphenyl and bisphenol P.

271 potential of environmental pollutants (e.g., bisphenols, polycyclic aromatic hydrocarbons, and synthe

272 Recoveries of the eleven bisphenols ranged from 76% to 122% among the different f

273 erstand how BPA and the reputedly more inert bisphenol S (BPS) affect the placenta, C57BL6J mouse dam

274 le absorption of BPA or the BPA alternatives bisphenol S (BPS) and 4-hydroxyphenyl 4-isoprooxyphenyls

275 of alternate chemicals such as BPA analogues bisphenol S (BPS) and bisphenol AF (BPAF) in production

276 Bisphenol S (BPS) and bisphenol F (BPF) are two such BPA

277 er exposure to BPA or its structural analogs bisphenol S (BPS) and bisphenol F (BPF) is associated wi

278 alternatives such as bisphenol AF (BPAF) and bisphenol S (BPS) are increasing, but little is known on

279 Bisphenol F (BPF) and bisphenol S (BPS) are increasingly used to substitute bi

280 Given its hormonal activity, bisphenol S (BPS) as a substitute for bisphenol A (BPA)

281 lar compounds including bisphenol F (BPF) or bisphenol S (BPS) even at much higher concentration.

282 Bisphenol S (BPS) has increasingly been used as a substi

283 enone-2 (BP2) and the bisphenol A substitute bisphenol S (BPS) was deciphered in eight human and zebr

284 thermal degradation of bisphenol A (BPA) and bisphenol S (BPS) was investigated in water and fish (co

285 than the accepted human daily exposure) and bisphenol S (BPS), a common analog used in BPA-free prod

286 trations of BPA and three bisphenol analogs, bisphenol S (BPS), bisphenol F (BPF), and bisphenol AF (

287 Bisphenol F (BPF) and bisphenol S (BPS), which have been substituted for BPA i

288 Bisphenol S and F: a systematic review and comparison of

289 otics (triclosan, tetrabromobisphenol A, and bisphenol S) in liver microsome and cell models.

290 Bisphenol A, bisphenol S, and 4-hydroxyphenyl 4-isoprooxyphenylsulfon

291 ed compounds including 2,6-dinitro-p-cresol, bisphenol S, clonixin, and triclopyr.

292 The bisphenols S, F, and AF (BPS, BPF, and BPAF) are used to

293 ompounds in surface water, like caffeine and bisphenol-S, five dihydroxydiphenylmethane isomers were

294 The total amount of the sum of nine bisphenols (Sigma9 bisphenols) and 5 benzophenones (Sigm

295 o other soil properties correlated well with bisphenol sorption.

296 Metabolites identified for all three bisphenols support degradation pathways that include met

297 s of BPF were generally lower than for other bisphenols, the 95th percentile concentration of BPF was

298 Maternal urinary concentrations of bisphenols were not associated with child nonverbal IQ.

299 Bisphenols were stable in dried (eight weeks, -20 degree

300 Eleven bisphenols were used as targeted compounds.