ライフサイエンスコーパス: bladder capacity

1 in the rat urothelium in vivo and increased bladder capacity.

2 isplayed bladder hyperactivity and decreased bladder capacity.

3 0.1-50 mg kg(-1), i.v.) which also increased bladder capacity.

4 of vesicoureteral reflux, and posttransplant bladder capacity.

5 or kidney transplant recipients with a small bladder capacity.

6 d to a significant OAB phenotype and reduced bladder capacity.

7 r, hydronephrosis, renal failure and reduced bladder capacity.

8 e in micturition frequency and a decrease in bladder capacity.

9 sets: nocturnal polyuria, storage or reduced bladder capacity, 24-h polyuria, and sleep-associated no

10 a distinct bladder volume threshold (74% of bladder capacity) above which flow-evoked bladder contra

11 jected to VCR exhibited augmented functional bladder capacity accompanied by frequent non-void contra

12 little change in heart rate between maximum bladder capacity and after micturition when compared to

13 le subjects exhibited a significantly larger bladder capacity and compliance compared to females.

14 d detrusor underactivity (DU) with increased bladder capacity and compliance in aged subjects.

15 One patient with decreased bladder capacity and increased PVR had a stroke and was

16 urinary frequency with increased functional bladder capacity and non-void contractions.

17 ected rats also showed significantly reduced bladder capacity and postvoid residual volume than diabe

18 rition intervals and voided volumes, lowered bladder capacity, and induced relative bladder wall hype

19 bladders of SK3T/T had significantly greater bladder capacity, and urine output exceeded the infused

20 inuous inhibitory stimulation that increases bladder capacity (BC) can reduce the efficiency of subse

21 and propofol) on urodynamic (Deltapressure, bladder capacity, bladder compliance, non-voiding contra

22 lter filling pressure, threshold pressure or bladder capacity, but micturition pressure was elevated

23 by decreased voiding frequency and increased bladder capacity, but normal bladder pressures.

24 1), i.v.) significantly (P < 0.05) increased bladder capacity during saline distension but not during

25 tivity and significantly (P <0.05) increased bladder capacity during slow infusion of saline or 0.25%

26 ferent sensory input and maintain sufficient bladder capacity during the night sleep period.

27 hable from that in age-matched controls, but bladder capacity in old animals was only approximately 5

28 At 26 weeks after subtotal cystectomy, bladder capacity in young animals was indistinguishable

29 ior cohort of four consecutive patients with bladder capacities < or =30 ml showed that three of four

30 e, girls and patients with reflux had larger bladder capacities (P < .01).

31 s is yet unknown, but may involve changes in bladder capacity rather than simple fluid management.

32 One patient with reflux and decreased bladder capacity refused treatment.

33 eased peak voiding pressure, voiding volume, bladder capacity, residual volume, and number of non-voi

34 tivity and significantly (P < 0.001) reduced bladder capacity to 14.9 +/- 10.3% of the saline control

35 usion PNS significantly (P < 0.05) increased bladder capacity to 167.7 +/- 27.1% at 1T and 196.0 +/-

36 der overactivity and significantly increased bladder capacity to 68.0 +/- 31.3% at 1T (P < 0.05) and

37 erocystoplasty (BAE) is utilized to increase bladder capacity to preserve renal function using autolo

38 Four patients with decreased bladder capacity underwent hydrodistention.

39 transplant bladder and 1 year posttransplant bladder capacity was 14.5% and 84% of expected, respecti

40 both in awake and in anesthetized mice, the bladder capacity was decreased ~fourfold when cystometry

41 Deltapressure was greatest with propofol, bladder capacity was highest with alpha-chloralose, non-

42 s, the epithelium was markedly thickened and bladder capacity was reduced relative to controls.

43 nnel antagonist Brilliant Blue FCF increased bladder capacity, whereas i.v. administration did not.

44 Clozapine increased the bladder capacity while reducing the micturition volume t

45 to increased voiding frequency and decreased bladder capacity with cystitis.