ライフサイエンスコーパス: bleeding on probing

1 probing) and > or =3 mm diseased (featuring bleeding on probing).

2 ttachment level and proportion of sites with bleeding on probing.

3 as clinical attachment level, recession, and bleeding on probing.

4 robing depth, clinical attachment level, and bleeding on probing.

5 s included gingival index, plaque index, and bleeding on probing.

6 tence of sites with probing depths >4 mm and bleeding on probing.

7 , buccal flap thickness, papilla height, and bleeding on probing.

8 e per-patient percentage of tooth sites with bleeding on probing.

9 ts, probing depths, plaque index scores, and bleeding on probing.

10 ng attachment level, gingival recession, and bleeding on probing.

11 probing depth reduction and the reduction in bleeding on probing.

12 ariables measured: PD, attachment level, and bleeding on probing.

13 comparing strict versus relaxed criteria for bleeding on probing.

14 robing depth, clinical attachment level, and bleeding on probing.

15 nical parameters including probing depth and bleeding on probing.

16 robing depth was 2 mm (SE 0.02), and 17% had bleeding on probing.

17 4) the infected sites had higher plaque and bleeding on probing 0.9 +/- 0.3, 73 +/- 12%, respectivel

18 Signs of infection (51.7%) and bleeding on probing (37.5%) were common signs detected a

19 Pill users had more sites with bleeding on probing (44.0% versus 31.1%; P = 0.017).

20 er percentage of sites with PD >/=4.5 mm and bleeding on probing (9.78% versus 12.69%; P = 0.052).

21 iodontal disease to be present in teeth with bleeding on probing and a probing depth >/=3 mm at one o

22 gnificantly associated with increased extent bleeding on probing and levels of microorganisms Porphyr

23 depths were similar in both groups, whereas bleeding on probing and plaque scores were higher in the

24 score (peak at induction minus baseline) for bleeding on probing and probing depth (PD), the patients

25 In contrast, in the experimental group, bleeding on probing and probing depths were significantl

26 mplete compliers tended to show reduction in bleeding on probing and reduction in plaque index compar

27 nine patients with 4 pockets > or = 5 mm and bleeding on probing and/or suppuration were randomized i

28 t loss >=3 mm, together with the presence of bleeding on probing and/or suppuration), smoking status,

29 ngival sulcus: < or =3 healthy (featuring no bleeding on probing) and > or =3 mm diseased (featuring

30 Gingival status (bleeding on probing) and oral hygiene effectiveness (den

31 ment loss, 13.1% (95% CI, 8.1% to 18.1%) for bleeding on probing, and 0.27 (95% CI, 0.17 to 0.37) for

32 g depth (PD), supragingival plaque, gingival bleeding on probing, and calculus.

33 of periodontal probing depth, plaque score, bleeding on probing, and clinical attachment level (CAL)

34 parameters, including pocket probing depth, bleeding on probing, and clinical attachment level were

35 ere included according to the probing depth, bleeding on probing, and clinical attachment level.

36 ss (AL), the presence of gingival recession, bleeding on probing, and full-mouth radiographic surveys

37 -gingival margin distance (attachment loss), bleeding on probing, and furcation involvement.

38 ng probing depth, clinical attachment level, bleeding on probing, and gingival and plaque indices, we

39 attachment level (CAL), probing depth (PD), bleeding on probing, and gingival index change after tre

40 ded probing depth, clinical attachment loss, bleeding on probing, and gingival index.

41 th, clinical attachment level, plaque index, bleeding on probing, and gingival index.

42 ntly higher mean PD and CAL, more sites with bleeding on probing, and increased levels of CRP compare

43 bing depth, clinical attachment level (CAL), bleeding on probing, and percentage of visible plaque.

44 sessment of probing depth, attachment level, bleeding on probing, and plaque and calculus accumulatio

45 depth (PD), clinical attachment level (CAL), bleeding on probing, and plaque index were measured, and

46 depth (PD), clinical attachment level (CAL), bleeding on probing, and plaque index were selected as o

47 , clinical attachment level, gingival index, bleeding on probing, and plaque index) were recorded at

48 obing depth (PD), clinical attachment level, bleeding on probing, and plaque index.

49 Plaque index, bleeding on probing, and probing depth were recorded at

50 l status was assessed by probing depth (PD), bleeding on probing, and radiographic alveolar bone loss

51 significant correlations with plaque scores, bleeding on probing, and RF-IgA.

52 s including probing depth, attachment level, bleeding on probing, and root caries remineralization we

53 s are superior to a single strip in reducing bleeding on probing, and that local delivery of tetracyc

54 baseline in probing depth, attachment level, bleeding on probing, and the Modified Gingival Index (MG

55 ved in both groups for: 1) PIss; 2) PDss; 3) bleeding on probing; and 4) relative CAL.

56 with at least 6 mm LOA; number of sites with bleeding on probing; and deepest probing depth per perso

57 tooth with > or =4 mm loss of attachment and bleeding on probing as an indicator of inflammation), or

58 robing depth, clinical attachment level, and bleeding on probing, as well as crevicular levels of int

59 attachment level, probing depth, plaque, and bleeding on probing at 6 sites per tooth.

60 erdental bleeding index, probing depths, and bleeding on probing at interdental sites and underwent a

61 asuring attachment level, probing depth, and bleeding on probing at monthly examinations at qualifyin

62 logistic regression models, with tooth-level bleeding on probing at sites with attachment loss<or=2 m

63 In the presence of peri-implantitis, only bleeding on probing at the adjacent dentate sites was id

64 1) health, all probing depth (PD) <3 mm and bleeding on probing (BOP) <10%; 2) gingivitis, all PD <3

65 of patients showed no probing depths > 5mm, bleeding on probing (BOP) <=25%, no pain and satisfactor

66 : P = 0.002; 6 and 9 months: P = 0.001), and bleeding on probing (BOP) (3 months: P <0.01; 6 months:

67 ociated to worsened SRP outcomes in terms of bleeding on probing (BOP) (OR = 1.02, P <0.05) and mean

68 ment loss (AL), and percentage of sites with bleeding on probing (BOP) and clinical AL >/=5 mm.

69 ined at baseline, 2 weeks and 12 months, and bleeding on probing (BOP) and clinical attachment level

70 severe periodontitis, and the combination of bleeding on probing (BOP) and clinical attachment loss (

71 ontal outcomes were percentage of teeth with bleeding on probing (BOP) and combination of BOP and att

72 bular quadrants were evaluated for calculus, bleeding on probing (BOP) and loss of gingival attachmen

73 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP) and microbiological assays (PC

74 hs (PPDs), clinical attachment levels (CAL), bleeding on probing (BOP) and plaque scores (PI) recorde

75 dontal probing depth (PD) and assessments of bleeding on probing (BOP) and radiographic alveolar bone

76 ng explorer-detectable supragingival plaque, bleeding on probing (BOP) and relative clinical attachme

77 In each patient four pockets > 5 mm with bleeding on probing (BOP) and/or suppuration were studie

78 (PD), clinical attachment levels (CAL), and bleeding on probing (BOP) as well as gingival crevicular

79 t one site with probing depth (PD) >4 mm and bleeding on probing (BOP) at 12 months post-therapy.

80 of sites with a probing depth (PD) >4 mm and bleeding on probing (BOP) at the 3-month reevaluation.

81 ed into two groups based on probing depth or bleeding on probing (BOP) at the site of collection of t

82 nt loss (CAL), gingival recession (REC), and bleeding on probing (BoP) being measured.

83 to evaluate possible predictors influencing bleeding on probing (BoP) changes at 24-month follow-up.

84 rs, including visible plaque index (VPI) and bleeding on probing (BOP) from six sites per tooth.

85 Participants with bleeding on probing (BOP) in <10% of sites were classifi

86 aque (PI), calculus (CI), gingival (GI), and bleeding on probing (BOP) indices were used to assess gi

87 The absence or presence of bleeding on probing (BOP) is a sign of periodontal healt

88 Bleeding on probing (BOP) is widely interpreted as a sig

89 bing pocket depth (PPD), plaque indices, and bleeding on probing (BOP) measured at baseline, intermed

90 r of sites with probing depth (PD) >4 mm and bleeding on probing (BOP) per patient was the primary ou

91 = 0.31 to 0.96) and lower odds of increased bleeding on probing (BOP) percentage values (OR = 0.62,

92 ) reduction, clinical attachment level gain, bleeding on probing (BOP) reduction, radiographic bone f

93 were defined using probing depths (PDs) and bleeding on probing (BOP) scores.

94 clinical attachment level (CAL), and reduced bleeding on probing (BOP) to a greater extent than SRP a

95 level (VAL), visual plaque score (VPS), and bleeding on probing (BOP) were assessed and compared.

96 CB samples from those patients with adequate bleeding on probing (BOP) were collected on special bloo

97 Gingival index (GI) and bleeding on probing (BOP) were evaluated in addition to

98 leeding index (GBI), probing depth (PD), and bleeding on probing (BOP) were measured in implants and

99 gingival index (GI), probing depth (PD), and bleeding on probing (BOP) were measured, and gingival cr

100 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP) were observed both short and l

101 with probing depths (PD) of > or =5 mm with bleeding on probing (BOP) were randomized into the test

102 achment level (CAL), probing depth (PD), and bleeding on probing (BOP) were recorded as the clinical

103 vel (CAL), modified gingival index (GI), and bleeding on probing (BOP) were recorded at baseline and

104 ), gingival recession, plaque index, GI, and bleeding on probing (BOP) were recorded at baseline, 3 a

105 gingival index (GI), plaque index (PI), and bleeding on probing (BOP) were recorded on fully erupted

106 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP) were recorded.

107 teeth, restorations, probing depth (PD) and bleeding on probing (BOP) were recorded.

108 al attachment level, and percentage of sites bleeding on probing (BOP) were significantly higher in t

109 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), and gingival index (GI) at ba

110 ingival recession (GR), gingival index (GI), bleeding on probing (BOP), and horizontal and vertical b

111 Gingival crevicular fluid, bleeding on probing (BOP), and interleukin-1beta were te

112 Probing depth (PD), attachment level, bleeding on probing (BOP), and interproximal plaque inde

113 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), and plaque index were measure

114 Peri-implant plaque index (PI), bleeding on probing (BOP), and probing depth (PD) were e

115 Peri-implant plaque index (PI), bleeding on probing (BOP), and probing depth (PD) were r

116 cal attachment level, furcation involvement, bleeding on probing (BOP), and suppuration.

117 nt level (CAL), recession (REC), presence of bleeding on probing (BOP), and supragingival plaque (PL)

118 attachment level (RAL), probing depths (PD), bleeding on probing (BOP), and the full-mouth plaque sco

119 Periodontal probing depth (PD), gingival bleeding on probing (BOP), clinical attachment level (CA

120 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingival index (GI), and peri

121 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingival index (GI), and plaq

122 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingival index (GI), plaque i

123 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingival index (GI), plaque i

124 riables included probing pocket depth (PPD), bleeding on probing (BoP), gingival margin level, dentin

125 Clinical periodontal parameters included bleeding on probing (BOP), mean probing depth (PD), and

126 uding probing depth (PD), plaque index (PI), bleeding on probing (BOP), mucosal redness (MR), suppura

127 an plaque indices (PI), probing depths (PD), bleeding on probing (BOP), or relative clinical attachme

128 tes of a novel lateral-flow immunoassay with bleeding on probing (BOP), oral hygiene, and periodontal

129 Plaque index (PI), GI, bleeding on probing (BOP), PD, and attachment gain were

130 were plaque index (PI), gingival index (GI), bleeding on probing (BOP), PD, gingival crevicular fluid

131 and periodontal data [visible plaque (VPI), bleeding on probing (BOP), periodontal probing depth (PP

132 , baseline periodontal parameters, including bleeding on probing (BOP), periodontal probing depths (P

133 Positive correlations were found between bleeding on probing (BOP), plaque index (PI) scores, and

134 probing depth (PD), gingival recession (GR), bleeding on probing (BOP), plaque index (PI), and count

135 gingivitis demonstrated significantly higher bleeding on probing (BOP), plaque index (PI), and gingiv

136 ded probing depth, clinical attachment loss, bleeding on probing (BOP), plaque index (PI), and tooth

137 with CAL loss or gain > or =2 mm, changes in bleeding on probing (BOP), plaque index, and mobility.

138 Bleeding on probing (BOP), plaque index, and probing dep

139 Full-mouth plaque index (PI), bleeding on probing (BOP), probing depth (PD) >/=4 mm, a

140 tal parameters, including plaque index (PI), bleeding on probing (BOP), probing depth (PD) >3 mm, cli

141 Plaque index (PI), bleeding on probing (BOP), probing depth (PD), and attac

142 Demographic and biologic data, bleeding on probing (BOP), probing depth (PD), and clini

143 Plaque index (PI), bleeding on probing (BOP), probing depth (PD), and clini

144 essed included plaque index, gingival index, bleeding on probing (BOP), probing depth (PD), and clini

145 ta, including approximal plaque index (API), bleeding on probing (BOP), probing depth (PD), and clini

146 ull-mouth periodontal examination, including bleeding on probing (BOP), probing depth (PD), and clini

147 uth periodontal examination with a record of bleeding on probing (BOP), probing depth (PD), and clini

148 Periodontal examinations of bleeding on probing (BOP), probing depth (PD), and clini

149 Peri-implant plaque index (PI), bleeding on probing (BOP), probing depth (PD), and mesia

150 ect: plaque index (PI), gingival index (GI), bleeding on probing (BOP), probing depth (PD), clinical

151 Bleeding on probing (BOP), probing depth (PD), relative

152 ewly diagnosed PCOS had increased sites with bleeding on probing (BOP), probing depth, clinical attac

153 s were evaluated for periodontal parameters (bleeding on probing (BoP), probing pocket depth (PPD), C

154 hment level (CAL), gingival recession (REC), bleeding on probing (BOP), suppuration (SUP), and suprag

155 uded CAL, probing depth, gingival recession, bleeding on probing (BOP), visible plaque, supragingival

156 plaque index (PI), gingival index (GI), and bleeding on probing (BOP), were compared for M2 at basel

157 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP), were performed, and subgingiv

158 ites with a probing depth (PD) >/= 5 mm with bleeding on probing (BOP).

159 pth (PD), clinical attachment loss (AL), and bleeding on probing (BOP).

160 grouped based on adjacent probing depth and bleeding on probing (BOP).

161 achment level (CAL), probing depth (PD), and bleeding on probing (BOP).

162 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP).

163 e attachment level (RAL), probing depth, and bleeding on probing (BOP).

164 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP).

165 s of > 2 mm, probing depth (PD) >/=5 mm with bleeding on probing (BOP).

166 2 mm, probing pocket depth (PD) >/=5 mm with bleeding on probing (BOP).

167 proximal pocket during PMT with a history of bleeding on probing (BOP).

168 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP).

169 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP).

170 indices, including: 1) plaque index (PI); 2) bleeding on probing (BOP); 3) probing depth (PD); and 4)

171 ding: 1) plaque index; 2) gingival index; 3) bleeding on probing (BOP); 4) probing depth; and 5) atta

172 7% (95% CI = -34.27 to 24.53%, P = 0.75) for bleeding on probing (BOP); a WMD of 0.36 mm (95% CI = 0.

173 with: 1) periodontal probing depth (PD); 2) bleeding on probing (BOP); and 3) attachment loss (AL).

174 < 0.01), gingival index (G]; P < 0.01), and bleeding on probing (BOP; P < 0.05) scores increased ove

175 Full-mouth plaque-index (PI), bleeding-on-probing (BOP), probing depth (PD), clinical

176 (PI), gingival index (GI), and percentage of bleeding on probing (%BOP) were observed in placebo-trea

177 inal bone loss [MBL], plaque index [PI], and bleeding on probing [BOP] in shamma users and non-users

178 cal attachment level, recession, plaque, and bleeding on probing [BOP]) and a sampling of gingival cr

179 recession, mobility, plaque index [PI], and bleeding on probing [BOP]) and defect (vertical and hori

180 th [PD], clinical attachment loss [CAL], and bleeding on probing [BOP]) were performed.

181 th presenting probing depth [PD] >/=5 mm and bleeding on probing [BOP]) were selected and randomly al

182 ual pockets (probing depth [PD] >/=5 mm with bleeding on probing [BOP]).

183 one diseased site (probing depth [PD] >4 mm, bleeding on probing [BOP], and clinical attachment level

184 PD], plaque index [PI], gingival index [GI], bleeding on probing [BOP], and clinical attachment level

185 periodontal parameters (probing depth [PD], bleeding on probing [BOP], and clinical attachment loss

186 ve teeth with probing depth [PD] > or =5 mm, bleeding on probing [BOP], and clinical attachment or ra

187 t soft tissue parameters (plaque index [PI], bleeding on probing [BOP], and probing depth [PD] >/=4 m

188 t inflammatory variables (plaque index [PI], bleeding on probing [BOP], probing depth (PD) and cresta

189 st two diseased interproximal papillae (with bleeding on probing [BOP], probing depth [PD] > or =4 mm

190 Periodontal parameters (plaque index [PI], bleeding on probing [BOP], probing depth [PD], clinical

191 Periodontal parameters (plaque index [PI], bleeding on probing [BOP], probing depth [PD], clinical

192 inflammatory conditions (plaque index [PI], bleeding on probing [BOP], probing depth [PD], marginal

193 depth > 2 mm, filled and decayed teeth, and bleeding on probing by smoking history were not signific

194 laque index, probing depth, attachment loss, bleeding on probing, calculus index, and furcation invol

195 oth loss; dental caries; periodontal status (bleeding on probing, calculus, and attachment loss); and

196 ge in probing depth was the primary outcome, bleeding on probing, clinical attachment level, gingival

197 Probing depth, gingival bleeding on probing, clinical attachment loss (CAL), and

198 0.78 +/- 0.30) had a significant decrease in bleeding on probing compared to baseline.

199 Sites with subgingival calculus and bleeding on probing demonstrated more LCAL and PD, and s

200 Plaque index and bleeding on probing did not change.

201 measurements, including probing depth (PD), bleeding on probing, gingival index, and plaque index (P

202 Probing depth, bleeding on probing, gingival index, and plaque index cl

203 ed probing depth, clinical attachment level, bleeding on probing, gingival index, and plaque index.

204 ing pocket depths, clinical attachment loss, bleeding on probing, gingival index, fasting glucose lev

205 uated were plaque (full-mouth plaque score), bleeding on probing, gingival recession, probing depth (

206 > 50 years (OR = 1.98), smoking (OR = 3.51), bleeding on probing >30% sites (OR = 4.10), and the inte

207 redictor of periodontal disease progression, bleeding on probing has low sensitivity owing to a high

208 days moderately increased the appearance of bleeding on probing in a population that had > or = 20%

209 PTX3 levels correlated with plaque index and bleeding on probing in the CP group (P <0.05).

210 ill have persistent periodontal pockets with bleeding on probing, indicating ongoing inflammation.

211 According to gingival and bleeding on probing indices, 84 children were classified

212 ng clinical outcomes were tested: plaque and bleeding on probing indices, pocket probing depth (PD),

213 Clinical parameters, including bleeding on probing, mobility, suppuration, mucosal rece

214 However, comparisons of other parameters (bleeding on probing, modified bleeding index, GI, probin

215 l attachment level (P <0.05), and sites with bleeding on probing (not significant).

216 depth, clinical attachment level (CAL), and bleeding on probing on all teeth except third molars and

217 healthy (no clinical attachment loss [AL] or bleeding on probing) or as having early periodontitis (c

218 bone loss, gingival inflammation measured as bleeding on probing, or a combination of these measures.

219 robing depth, percentage of sites exhibiting bleeding on probing, or plaque and calculus accumulation

220 t transparency, mucosal recession, mobility, bleeding on probing, or suppuration (n = 40) at 48 month

221 ons tended to show a reduction in plaque and bleeding on probing over time.

222 PD was related to plaque and bleeding on probing (P < 0.001) in both sexes and also t

223 ent level; blood glucose was related only to bleeding on probing (P <0.05).

224 0.006), probing depth 4 to 6 mm (P = 0.016), bleeding on probing (P = 0.001), and radiographic bone l

225 with high probing depth (P = 0.02) and high bleeding on probing (P = 0.008).

226 cases than controls had plaque (P = 0.004), bleeding on probing (P = 0.013), and probing depths of >

227 higher levels of plaque index (P = 0.01) and bleeding on probing (P = 0.03) and higher severity of pe

228 multiple strip group significantly decreased bleeding on probing (P = 0.05) compared to all other tre

229 olar teeth with probing depth > or =5 mm and bleeding on probing participated in this split-mouth tri

230 mber of teeth with probing depth >=4 mm with bleeding on probing (PD/BOP) or with clinical attachment

231 buted 198 gingival papillae: 158 'diseased' (bleeding-on-probing, PD > 4 mm, and AL >/= 3 mm) and 40

232 requency of flossing, periodontal diagnosis, bleeding on probing percentage, number of missing teeth

233 mination measuring probing depth, recession, bleeding on probing, plaque index (PI), and GE.

234 odontal attachment loss (AL), probing depth, bleeding on probing, plaque index (PI), and gingival ind

235 th probing depth, clinical attachment level, bleeding on probing, plaque index scores, and keratinize

236 clinical parameters (Probing Pocket Depths, Bleeding On Probing, Plaque Index) and marginal bone los

237 l recession; and 4) percentage of sites with bleeding on probing, plaque, PD >/=5 mm, and CAL >/=3 mm

238 e newly forming peri-implant sulcus depth or bleeding on probing prevalence.

239 uction in the percentage of sites exhibiting bleeding on probing (primary outcome) and lower levels o

240 At baseline, at day 14 and day 42 bleeding on probing (primary outcome), gingival index, p

241 h visits, gingival index (GI), plaque index, bleeding on probing, probing depth (PD), and attachment

242 Recordings of plaque, bleeding on probing, probing depth (PD), and clinical at

243 The waist/hip ratio (WHR), plaque index, bleeding on probing, probing depth (PD), and clinical at

244 Recordings of plaque, bleeding on probing, probing depth (PD), and clinical at

245 ere positively associated with the extent of bleeding on probing, probing depth, and clinical attachm

246 Plaque and gingival indices, bleeding on probing, probing depth, and clinical attachm

247 iodontal assessment was performed, including bleeding on probing, probing depth, and clinical attachm

248 six sites on each premolar included plaque, bleeding on probing, probing depth, and relative attachm

249 evels, periodontal parameters (plaque index, bleeding on probing, probing depth, attachment loss, and

250 ts were assessed for periodontal parameters (bleeding on probing, probing depth, clinical AL, oral hy

251 dontal disease severity was measured through bleeding on probing, probing depth, clinical attachment

252 e following clinical periodontal parameters: bleeding on probing, probing depth, clinical attachment

253 Periodontal parameters (plaque index, bleeding on probing, probing depth, clinical attachment

254 ing parameters were evaluated: plaque index, bleeding on probing, probing depth, gingival recession,

255 e recorded: suppuration on probing, modified bleeding on probing, probing depth, implant mobility, bo

256 eters measured included plaque accumulation, bleeding on probing, probing depths (PDs), and clinical

257 Recordings of bleeding on probing, probing depths, and clinical attach

258 Similarly, changes in bleeding on probing, probing pocket depth, and suppurati

259 'diseased' gingival papillae (n = 241; with bleeding-on-probing, probing depth >/= 4 mm, and clinica

260 n available, a 'healthy' papilla (n = 69; no bleeding-on-probing, probing depth </= 4 mm, and clinica

261 Salivary-ProCT levels were correlated with bleeding-on-probing (r = 0.45, p = 0.05), as well as wit

262 Percent of sites bleeding-on-probing reduced in all groups, but the effec

263 eduction (PDR), attachment level gain (ALG), bleeding on probing reduction (BOP), and plaque index we

264 gain was 1.65 mm (95% CI: 1.17-2.13 mm), and bleeding on probing reduction was 45.8% (95% CI: 38.5%-5

265 h reduction, clinical attachment level gain, bleeding on probing reduction, and mucosal recession.

266 graphic, and behavioral risk variables, only bleeding on probing remained significantly associated wi

267 statistically significantly higher levels of bleeding on probing, root calculus, and dental plaque th

268 evel, probing depth, plaque, gingivitis, and bleeding on probing scores) and significant decreases in

269 1) and attachment loss (P = 0.006) and fewer bleeding on probing sites (P = 0.001).

270 sulcus depth: 1 to 3 mm (normal), 3 mm with bleeding on probing (slight disease), 3 to 6 mm (moderat

271 pth, recession, width of keratinized mucosa, bleeding on probing, suppuration, implant mobility, plaq

272 ch implant, probing depths, mucosal redness, bleeding on probing, suppuration, plaque index, and marg

273 Plaque, gingivitis, bleeding on probing, suppuration, probing depth, and cli

274 1.8, 6 months 5.84 +/- 1.71, P < 0.001), and bleeding on probing (test BL 86%, 6 months 41%, control

275 .002); and for every 10 sites that exhibited bleeding on probing, the clinical attachment gain was 0.

276 tal examination consisting of probing depth, bleeding on probing, tooth mobility, gingival index, and

277 aque index versus no reduction, reduction in bleeding on probing versus no reduction, reduction in th

278 : measurements included periodontal indexes (bleeding on probing, visible plaque index, root calculus

279 ers measured at six sites per tooth included bleeding on probing, visible plaque, probing depth, and

280 D), clinical attachment level (CAL), plaque, bleeding on probing, visual gingival inflammation, and c

281 graphic parameters, including probing depth, bleeding on probing, visual inspection, and radiographic

282 Bleeding on probing was a prerequisite for target sites

283 depth, gingival recession, plaque index, and bleeding on probing was performed in 75 Indonesians with

284 1.38 mm (compared to 1.01 mm by SRP alone), bleeding on probing was reduced by 25.2% (compared to 13

285 Bleeding on probing was significantly associated with TG

286 sensitivity and specificity of the test for bleeding on probing were 71.8% and 77.5%, respectively (

287 d at every visit whereas IPD, recession, and bleeding on probing were assessed at 8, 12, 16, 24 week.

288 the clinical parameters of probing depth and bleeding on probing were compared in 15 patients with mo

289 robing depth, clinical attachment level, and bleeding on probing were evaluated through a full-mouth

290 Probing depth (PD) and bleeding on probing were measured at six sites per tooth

291 on cLCAL/cPD, while subgingival calculus and bleeding on probing were negatively associated with cLCA

292 and width, amount of keratinized tissue, and bleeding on probing were obtained.

293 s with 4 periodontal pockets > or = 5 mm and bleeding on probing were randomized into four groups of

294 ce of supragingival plaque accumulation, and bleeding on probing were recorded.

295 obing depth, clinical attachment levels, and bleeding on probing were used to determine the periodont

296 nt level [CAL], and percentage of sites with bleeding on probing) were evaluated.

297 evel [FST], papilla index, plaque index, and bleeding on probing) were measured, and periapical radio

298 ng periodontal probings and an assessment of bleeding on probing, were made using an NIDR probe at 3

299 achment loss were positively associated with bleeding on probing, with stronger associations among me

300 No significant differences were found for bleeding on probing (WMD = 10.77; 95% CI = -3.43 to 24.9