戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ast, factor XIII was lower in AKI (increased bleeding tendency).
2 ructure and function resulting in a variable bleeding tendency.
3 n severely reduced thrombin generation and a bleeding tendency.
4 bosis, but its inhibition causes only a mild bleeding tendency.
5 ysfunction is a third possible mechanism for bleeding tendency.
6 zed by low platelet count that may result in bleeding tendency.
7 terozygotes may display a distinct, if mild, bleeding tendency.
8 hould be evaluated carefully for symptoms of bleeding tendency.
9 d with peptic ulcer disease and an increased bleeding tendency.
10 er traumatic brain injury without increasing bleeding tendencies.
11                       History of hemorrhagic/bleeding tendencies (93%) was the most common reason for
12 e disorder in which oculocutaneous albinism, bleeding tendency and a ceroid-lipofuscin lysosomal stor
13 isorder that is characterized by a life-long bleeding tendency and complications in wound healing.
14 w that mutant mice lacking CD148 exhibited a bleeding tendency and defective arterial thrombosis.
15    Affected patients present with a moderate bleeding tendency and have Factor V and Factor VIII leve
16 nia and lack of natural killer (NK) cells, a bleeding tendency and neurologic abnormalities.
17 fected individuals, including differences in bleeding tendency and VWF quantity, activity, and multim
18 czema, frequency and severity of infections, bleeding tendency, and autoimmunity and reduction in dis
19 er characterized by oculocutaneous albinism, bleeding tendency, and lysosomal ceroid storage disease,
20                       Gingival inflammation, bleeding tendency, and supragingival plaque were clinica
21 leeding time measurements revealed no severe bleeding tendency as a consequence of GP VI deficiency.
22  effect of annexin V was not associated with bleeding tendency, as judged by the amount of blood abso
23 III or LAD-1/variant) present with increased bleeding tendency because of the lack or dysfunction of
24                              Analyses of the bleeding tendency by means of the bleeding score (BS) ha
25 cy is a rare bleeding disorder manifesting a bleeding tendency caused by low FX activity levels.
26 onstitutive JAK2(V617F) expression exhibit a bleeding tendency combined with the accelerated formatio
27 g models, f10low mice displayed an increased bleeding tendency compared with f10WT mice.
28 emely challenging in patients with increased bleeding tendency, detection and aggressive treatment of
29                                 Importantly, bleeding tendency did not correlate with plasma VWF leve
30 ptoms, clinical manifestations can vary, and bleeding tendency does not always correlate with FXI pla
31 of factor V which shed light on the variable bleeding tendencies in severe factor V deficiency.
32 ADP receptor gene (P2RY12) contribute to the bleeding tendency in 92 index cases enrolled in the Euro
33        To assess the factors responsible for bleeding tendency in AKI, we performed a prospective stu
34 ince been questioned on the absence of clear bleeding tendency in mice lacking the platelet-specific
35 ssociated with a thrombocytopathy inducing a bleeding tendency in the homozygous mutated patients.
36 de clear explanations for the thrombotic and bleeding tendency in these MPDs, suggesting the need for
37 -V13A12 resulted in a significantly improved bleeding tendency in VWD type 1 mice when using the saph
38 bocytopathy, which likely contributes to the bleeding tendency in vWD-type 2B.
39                                              Bleeding tendency is associated with heterogeneous clini
40                           This suggests that bleeding tendencies may likewise vary among patients wit
41                     GPS is associated with a bleeding tendency, myelofibrosis, and splenomegaly.
42 individuals (n = 97), and between those with bleeding tendency (n = 50) and without (n = 24).
43                       There was no increased bleeding tendency, no neurologic abnormalities, and no i
44 e data suggest that a ketogenic diet-related bleeding tendency occurs in about one third of treated p
45                              The significant bleeding tendencies of individuals homozygous for prothr
46 l assessments (Plaque Index, Gingival Index, Bleeding Tendency, pocket depth, and coronal staining) w
47 causes of morbidity and mortality in FA is a bleeding tendency, resulting from low platelet counts.
48 y had significantly lower Gingival Index and Bleeding Tendency scores at the one-, three-, six-, and
49                      Measurement of gingival bleeding tendency should be an integral part of a compre
50 ease f9a transcripts, potentially leading to bleeding tendencies similar to disseminated intravascula
51 usually lead to integrin nonexpression and a bleeding tendency termed Glanzmann thrombasthenia.
52 h Factor XI (FXI) deficiency have a variable bleeding tendency that does not correlate with FXI:C lev
53             Patients with vWD-type 2B have a bleeding tendency that is linked to loss of vWF multimer
54 rhosis might be responsible for an increased bleeding tendency, the current theory posits a rebalance
55                         Apart from increased bleeding tendency, the pathophysiological consequences o
56                          The severity of the bleeding tendency varies among patients and is related t
57 n ITP patients account for this variation in bleeding tendency, we conducted a single-center, cross-s
58 tients referred for assessment of a possible bleeding tendency will eventually be diagnosed with a mi
59 ive of impaired platelet function (increased bleeding tendency), without differences in von Willebran