ライフサイエンスコーパス: blood alcohol

1 a-III excretion correlating with the rise in blood alcohol.

2 by ethanol (EtOH) at concentrations seen in blood alcohol after binge drinking, and diminished ALDH1

3 king that was associated with high levels of blood alcohol and a reduction in somatic symptoms of eth

4 ol doses; namely, a placebo dose [equivalent Blood Alcohol Concentration (BAC) = 0.00%], a low dose o

5 Blood alcohol concentration (BAC) and rate of alcohol el

6 a significant difference in relation to the blood alcohol concentration (BAC) as a function of gende

7 effects of alcohol vary between limbs of the blood alcohol concentration (BAC) curve (i.e., whether B

8 Lowering the permitted blood alcohol concentration (BAC) for drivers is a commo

9 Blood alcohol concentration (BAC) is unable to be determ

10 At a calibrated blood alcohol concentration (BAC) of 0.02%, SDLP was inc

11 As such, the peak blood alcohol concentration (BAC) of low and high dose o

12 ragastrically with the ethanol solution, the blood alcohol concentration (BAC) was found to peak late

13 dents aged 18 years or older with a positive blood alcohol concentration (BAC; ie, >=0.01 g/dL).

14 wer body temperature (p = 0.045), and higher blood alcohol concentration (p = 0.012); vascular fillin

15 ge of 8.5 standard alcohol drinks (estimated blood alcohol concentration [eBAC]=0.115 g/dl) versus 3.

16 hat it is a criminal offense to drive with a blood alcohol concentration above the state's legal limi

17 ing as significant at doses as low as 0.026% blood alcohol concentration and increasing to moderate/l

18 nitoring chronic alcohol abstinence, whereas blood alcohol concentration and urinary EtG are the most

19 ect to demographic factors, injury type, and blood alcohol concentration and urine toxicology results

20 Determination of blood alcohol concentration is commonly used in cases of

21 cle crashes was lower when laws specifying a blood alcohol concentration of 0.08 g/dl per se (laws st

22 usly administered 6% v/v ethanol to a target blood alcohol concentration of 0.08% or placebo between

23 tar rats exposed to alcohol vapor had a mean blood alcohol concentration of 176.2 mg/dl during the la

24 se decreased as both the child's age and the blood alcohol concentration of the child's driver increa

25 tion in the laboratory, the maximum achieved blood alcohol concentration was influenced in an allele-

26 Mean blood alcohol concentration was not significantly differ

27 ubation controls, (3) 3 g/kg/day of alcohol (blood alcohol concentration {BAC}=139.9 mg/dl), (4) 4 g/

28 dramine (50 mg), alcohol (approximately 0.1% blood alcohol concentration), or placebo, given at weekl

29 and peer norms) on alcohol expenditure, peak blood alcohol concentration, alcohol dependence, and acc

30 Patients were screened using a blood alcohol concentration, gamma glutamyl transpeptida

31 al aldehyde dehydrogenase, in part determine blood alcohol concentration, thereby contributing to the

32 ma Scale, lower body temperature, and higher blood alcohol concentration.

33 ol concentrations equivalent to 0.05 to 0.08 blood-alcohol concentration-levels that would be mildly

34 C, CeMPV and EW was significantly related to blood alcohol concentrations (BAC).

35 binge alcohol drinking (intake resulting in blood alcohol concentrations (BACs) >/=80 mg% within a 2

36 Elevated blood alcohol concentrations (BACs) are found in about o

37 Of 302 injury deaths, blood alcohol concentrations (BACs) were available for 2

38 t rodent models do not result in binge-level blood alcohol concentrations (BACs), and to better under

39 cantly reduced their alcohol consumption and blood alcohol concentrations achieved.

40 cant differences in vital signs or estimated blood alcohol concentrations between PT150 non-exposed a

41 quate to intoxicating through to supralethal blood alcohol concentrations in humans and provides an i

42 Alcohol-attributable fractions based on blood alcohol concentrations were used to assess acute p

43 We found that, at equivalent blood alcohol concentrations, HDs reported lower subject

44 ol drinking access that achieved significant blood alcohol concentrations.

45 f the non-motor-carrier drivers had positive blood alcohol concentrations.

46 Native American heritage, despite equivalent blood alcohol concentrations.

47 previous studies have focused on deficits at blood-alcohol concentrations (BACs) above about 0.04%.

48 had blood alcohol measured were intoxicated (blood alcohol content >22 mmol/L [100 mg/dL]).

49 Breathalyzers estimate Blood Alcohol Content (BAC) from the concentration of et

50 ale (P < 0.0001), Black (P < 0.0001), have a blood alcohol content above 80 mg/dL (P < 0.0001), and s

51 Male sex, Black race, and elevated blood alcohol content and penetrating injury are associa

52 At admission, 36.7% of e-scooter users had a blood alcohol content higher than the legal threshold (n

53 supported than opposed policies to lower the blood alcohol content limit for driving, prohibit alcoho

54 ld) and cortex (up to 50-fold), depending on blood alcohol content.

55 es (P<.001) during the ascending limb of the blood alcohol curve and remained significantly above bas

56 (P<.001) and descending (P<.001) limb of the blood alcohol curve.

57 mal enhancement at 16 mM, close to the legal blood alcohol driving limit in most U.S. states (17.4 mM

58 to test the hypothesis that the presence of blood alcohol has a clinically significant impact on GCS

59 To address the hypothesis that elevated blood alcohol increases systemic oxidant stress, we meas

60 concentration six times lower than the legal blood-alcohol intoxication (driving) limit in most state

61 ng methamphetamine had a significantly lower blood alcohol level compared with the control group.

62 s a blood alcohol level of at least 0.05%; a blood alcohol level greater than 0; and a blood alcohol

63 The average peak blood alcohol level in the treatment group was 40 +/- 4

64 ven at concentrations as low as 10 mm (legal blood alcohol level in the United States is below 0.08 g

65 as relevant and comparable to that seen at a blood alcohol level of 0.05%, the legal limit in many co

66 ary seatbelt laws, maximum speed limit laws, blood alcohol level of 0.08, and administrative license

67 ethanol in utero (average maternal and fetal blood alcohol level of 25 mg/dl) promotes premature tang

68 Binge drinking, defined as reaching a blood alcohol level of 80 mg%, carries a risk of negativ

69 vel of at least 2.5 ng per milliliter plus a blood alcohol level of at least 0.05%; a blood alcohol l

70 a blood alcohol level greater than 0; and a blood alcohol level of at least 0.08%.

71 Blood alcohol level of more than 100 mg/dl (OR=9.43, P=0

72 hemselves and others despite having a normal blood alcohol level.

73 Among decedents who had a negative blood-alcohol level (52 in residences and 26 in vehicles

74 ing; whereas, among those who had a negative blood-alcohol level and most likely were awake at the ti

75 A blood-alcohol level greater than 0.01% was present in 56

76 Subjective effects, blood alcohol levels (BAL), and plasma cytokines were me

77 rom the HS/Npt line) to achieve intoxicating blood alcohol levels (BALs) after binge-like drinking.

78 no significant differences between groups in blood alcohol levels (BALs), these results are not due t

79 gnificantly shorter sleeping time and higher blood alcohol levels after regaining reflex than adult r

80 olerance during adolescence and suggest that blood alcohol levels alone do not fully explain ethanol-

81 cts of intoxication, despite greater initial blood alcohol levels and ethanol hypothermia in the form

82 Blood alcohol levels and task performance were similar a

83 concentrations comparable with or less than blood alcohol levels associated with intoxication and at

84 Blood alcohol levels did not differ significantly in wom

85 Whisky promoted highest blood alcohol levels in both sexes.

86 enzymes were associated with 30-fold higher blood alcohol levels in KO mice.

87 of the distilled drinks promoted a spike in blood alcohol levels more quickly than ingestion of the

88 rinking is short-term drinking that achieves blood alcohol levels of 0.08 g/dl or above.

89 Peak blood alcohol levels of 21 mmol/L were attained in each

90 f 1-2 alcoholic beverages in humans leads to blood alcohol levels of approximately 10 mM.

91 ere significantly correlated with changes in blood alcohol levels on both the ascending (P<.001) and

92 In women, beer promoted the lowest blood alcohol levels over the 6h of the experiment.

93 ding low inositol levels in response to high blood alcohol levels support a mechanism of reversible o

94 Blood alcohol levels were determined, and the retinas an

95 3 g/kg b.wt., i.p.) resulted in elevation of blood alcohol levels, ACTH and corticosterone in all gro

96 d the effects of four alcoholic beverages on blood alcohol levels, plasma insulin concentrations and

97 compared to wild-type mice, despite similar blood alcohol levels.

98 Chronic-binge ethanol feeding leads to high blood alcohol levels; thus, this simple model will be ve

99 Binge drinking (blood-alcohol levels >/= 0.08 g% in a 2-h period), is a

100 me alone, and 69 (53%) of 130 adults who had blood alcohol measured were intoxicated (blood alcohol c

101 tine were currently drug dependent, 11.7% of blood alcohol-negative and 3.9% of drug-negative patient

102 While 54.3% of blood alcohol-positive patients were currently alcohol d

103 I (Head Abbreviated Injury Score [AIS]), and blood alcohol testing status were known, were included.

104 e frequently monitored with random urine and blood alcohol tests; patients found positive were exclud

105 A transient elevation of blood alcohol to approximately 120 mg/dL was sufficient

106 f the pursuit response to very low levels of blood alcohol, under the conditions of our study.