1 is the physiologically relevant component of blood calcium.

2 hium treatment was associated with increased blood calcium (+0.09 mmol/L, 95% CI 0.02-0.17, p=0.009),

3 The most common adverse effect was decreased blood calcium (68.9% vs 59.8%).

4 s include both PTH, a hormone that regulates blood calcium and phosphate, and PTH-related protein (PT

5 hat PTHrP is an important regulator of fetal blood calcium and placental calcium transport.

6 totypical class C GPCR that maintains stable blood calcium (Ca(2+)) levels by sensing minute changes

7                            Consequently, the blood calcium, calciotropic hormones, and skeleton appea

8 aSR) expressed by parathyroid cells controls blood calcium concentration by regulating parathyroid ho

9                    CaSR inhibition increased blood calcium concentration in animals pretreated with a

10 creased renal tubular calcium absorption and blood calcium concentration independent of PTH secretion

11 onclude that CaSR is a direct determinant of blood calcium concentration, independent of PTH, and mod

12 as accompanied by a significant reduction in blood calcium levels and a physiologic increase in parat

13 nt negative effect on bird morphometrics and blood calcium levels and a positive relationship with th

14 tamin D metabolites, and calcitonin regulate blood calcium levels and contribute to the overall calci

15 roxygemini required higher doses to increase blood calcium levels in mice and to suppress INF-gamma r

16 had high levels of monotypic human Igs, high blood calcium levels, increased osteoclast activity, and

17 e hormone calcitonin in response to elevated blood calcium levels.

18       CLDN14 is strongly upregulated by high blood calcium, restricts this paracellular calcium flux,

19 e role of PTHrP in fetal calcium metabolism, blood calcium was measured in mice homozygous (HOM) for