ライフサイエンスコーパス: blood flow velocity

1 a frame count method was used to quantitate blood flow velocity.

2 ative angiography and Doppler measurement of blood flow velocity.

3 essels, smaller lumenal diameters, and lower blood flow velocity.

4 od pressure, electrocardiogram, and cerebral blood flow velocity.

5 meliorated pericardial effusion and restored blood flow velocity.

6 ndependently of beta-cell expansion or islet blood flow velocity.

7 between arterial blood pressure and cerebral blood flow velocity.

8 hy scan and rapidly increasing mean cerebral blood flow velocities.

9 In 21 patients, coronary blood flow velocity (0.014-inch Doppler flow wire), hear

10 a substantially larger increment in coronary blood flow velocity (0.51 versus 0.14 m/s, P <0.001).

11 4% +/- 24 [adenosine]; P = .80) and coronary blood flow velocity (21% +/- 16 [hypercapnia] vs 26% +/-

12 udy demonstrates the importance of decreased blood flow velocity accompanying activated NF-kappaB sig

13 udy demonstrates the importance of decreased blood flow velocity accompanying activated NF-kB signali

14 age reductions of mean (+/- s.e.m.) cerebral blood flow velocity after 10 min upright tilt were small

15 nsive visualization to the quantification of blood flow velocities and flow volumes.

16 Fluid simulations revealed lower LAA blood flow velocities and increased thrombotic risk in c

17 In the middle cerebral artery blood flow velocities and vasomotor reactivity were meas

18 Restriction of vessel diameter increased blood flow velocity and caused reduction in vascular ACE

19 on depth information regarding blood volume, blood flow velocity and direction, vascular architecture

20 e PTCA significantly improve distal coronary blood flow velocity and DSVR but not CFR.

21 emia: NE evoked similar decreases in femoral blood flow velocity and femoral vascular conductance (FV

22 LAD coronary blood flow velocity and free-breathing myocardial BOLD M

23 with controls, CADASIL patients showed lower blood flow velocity and higher pulsatility index within

24 olor Doppler imaging, suggest that decreased blood flow velocity and increased vascular resistance ar

25 hemia was quantified by temporally resolving blood flow velocity and oxygenation (SvO(2)) in superfic

26 ([Cl] 98 mmol/L, Baxter Healthcare) on renal blood flow velocity and perfusion in humans using magnet

27 lia and centrum semiovale perforating artery blood flow velocity and pulsatility, vascular reactivity

28 encement of infusion to measure renal artery blood flow velocity and renal cortical perfusion.

29 encement of infusion to measure renal artery blood flow velocity and renal cortical perfusion.

30 althcare, Thetford, United Kingdom] on renal blood flow velocity and renal cortical tissue perfusion

31 f 0.9% saline results in reductions in renal blood flow velocity and renal cortical tissue perfusion.

32 poral sequence of changes of cerebral artery blood flow velocity and systemic arterial pressure in 15

33 In addition, quantitative measurements of blood flow velocity and tissue perfusion will be feasibl

34 e measured as the product of the transaortic blood flow velocity and transesophageal echocardiographi

35 Blood-flow velocity and flow reserve were assessed with

36 ng from increased blood pressure, changes in blood flow velocity, and a progressive stiffening of pul

37 t changes in retinal and ONH morphology, ONH blood flow velocity, and retinal and cortical pattern-de

38 ecuff occlusion, resistivity index, baseline blood flow velocity, and SvO(2) were evaluated.

39 n OCT, 3 independent graders categorized the blood flow velocities as low, medium, or high.

40 , the systolic (PSV), end-diastolic and mean blood flow velocities as well as pulsatility and resista

41 Lower peak-systolic, end-diastolic, and mean blood flow velocities at baseline were associated with h

42 se apparent viscosity significantly, slowing blood flow velocity at arterial oxygen tension even with

43 recordings of ascending aortic pressure and blood flow velocity at rest and with supine bicycle exer

44 udy was to assess serial changes in coronary blood flow velocity before and after Rotablator atherect

45 laser speckle contrast imaging (LSCI) ocular blood flow velocity (BFV) and five birth parameters: ges

46 Coronary artery blood flow velocity (BFV) in diastole was not different

47 ntified the coupling between BP and cerebral blood flow velocity (BFV) using transfer function analys

48 High mean cerebral blood flow velocities can be apparent before the presenc

49 Using a set of artificial cerebral blood flow velocities (CBFV) generated from a well-known

50 Bilateral middle cerebral artery blood flow velocities (CBFV) were measured with transcra

51 anscranial doppler (TCD) to measure cerebral blood flow velocities (CBFv).

52 Average cerebral blood flow velocity (CBFV) changes (defined by an iterat

53 e of ticagrelor versus prasugrel on coronary blood flow velocity (CBFV) during increasing doses of in

54 changes in blood pressure (BP) and cerebral blood flow velocity (CBFV) in 13 healthy subjects (30 +/

55 ssociation between NLR and elevated cerebral blood flow velocity (CBFv) in CABM-patients.

56 nutes after indomethacin ingestion, cerebral blood flow velocity (CBFV) in the middle cerebral artery

57 neuronal Ca(2+) fluorescence and 3D cerebral blood flow velocity (CBFv) in vascular networks in the m

58 be gained when considering the raw cerebral blood flow velocity (CBFV) recordings.

59 ts indicated a morning reduction in cerebral blood flow velocity (CBFV) relative to values from the p

60 Coronary blood flow velocity (CBFV) was measured by using transth

61 of arterial blood pressure (BP) and cerebral blood flow velocity (CBFV).

62 In contrast, cerebral-blood-flow velocity (CBFv) in arteries and veins fluctua

63 Coronary blood flow velocity (CBV; Duplex Ultrasound), heart rate

64 anial Doppler ultrasound was used to measure blood flow velocity (CFV) in the middle cerebral artery

65 Inherent difficulties in measuring blood flow velocity close to the arterial wall have prev

66 nteric artery revealed a twofold increase in blood flow velocity compared with tumor-free mice (P < 0

67 els were calculated by using a mean cerebral blood flow velocity criterion of >120 cm/sec.

68 Resting middle cerebral artery blood flow velocity did not change significantly from pr

69 The blood flow velocity distribution indicated that most of

70 Vessel size and blood flow velocity distributions were evaluated and ass

71 R) was assessed intermittently from brachial blood flow velocity (Doppler ultrasound) divided by mean

72 essure (Finapres) divided by brachial artery blood flow velocity (Doppler ultrasound).

73 lood pressure (Finapres) divided by brachial blood flow velocity (Doppler) and cardiac responses from

74 baboons there was increased carotid arterial blood flow velocity during late systole and diastole.

75 Reductions of cerebral blood flow velocity during lower body suction were signi

76 tions, one optimized for arterial and venous blood flow (velocity encoding range, +/-50 cm/sec) and t

77 Elevated time averaged mean maximum blood flow velocity, especially when velocity is 200 cm/

78 ective: To evaluate the potential of retinal blood flow velocity estimation by structural OCT.

79 motion phases, contrast-agent dynamics, and blood flow velocity fields.

80 methodologies for simultaneously quantifying blood flow (velocity, flux, hematocrit and shear rate) i

81 in children with elevated cerebral arterial blood flow velocity, further study is required to determ

82 c vasospasm, the mean time for mean cerebral blood flow velocities >120 cm/sec was 7.0 +/- 3 days (p

83 However, intracoronary blood flow velocity has not been compared with the angio

84 lso quantify the impacts of the size of MAs, blood flow velocity, hematocrit and RBC stiffness and ad

85 gnetic resonance imaging measured in vivo 3D blood flow velocities in 60 AF patients and 15 controls.

86 d was better at detecting high mean cerebral blood flow velocities in patients with symptomatic vasos

87 with pH-stat strategy and decreases cerebral blood flow velocities in survivors.

88 Blood flow velocities in the conjunctival microcirculati

89 Because it is commonly assumed that blood flow velocities in these vessels are prohibitively

90 strongest negative correlation with the mean blood flow velocity in anterior cerebral arteries (p < 0

91 E/A (ratio of left ventricular-filling peak blood flow velocity in early diastole [E wave] to that i

92 f stroke, which increases steeply with lower blood flow velocity in either region.

93 Here, we found that decreased blood flow velocity in peritubular capillaries by kidney

94 Here, we found that decreased blood flow velocity in peritubular capillaries by kidney

95 Blood flow velocity in pial and cortical penetrating ves

96 FR) was calculated as the ratio of pulmonary blood flow velocity in response to Ach relative to basel

97 Doppler ultrasonography to demonstrate that blood flow velocity in the anterior cerebral artery is h

98 transcranial Doppler sonography showed that blood flow velocity in the anterior cerebral artery is s

99 peritubular capillaries and decreased kidney blood flow velocity in the congestive kidney.

100 the ratio of hyperemic to baseline coronary blood flow velocity in the left anterior descending coro

101 Blood flow velocity in the middle cerebral artery (MCAv)

102 Cerebral blood flow velocity in the middle cerebral artery was me

103 g in all microvessels, we detected decreased blood flow velocity in venules of all diameters.

104 environment, rapidly increased microvascular blood-flow velocity in the dentate gyrus.

105 dies that showed that the time-averaged mean blood-flow velocity in the internal carotid or middle ce

106 f mean arterial pressure and cerebral artery blood flow velocity, in the very low- (0.02-0.07 Hz), lo

107 The mean blood flow velocity index (BFVi) in the optic disc and i

108 RBCs flowing past macrophages for a range of blood flow velocities indicate that the increased blood

109 Lower blood flow velocity, indicating reduced cerebral metabol

110 r-mediated, whereas the increase in coronary blood flow velocity is due to activation of A2 adenosine

111 blood volume without altering microvascular blood flow velocity, leading to a significantly increase

112 he cardiovascular system of interest through blood flow velocity mapping in three spatial dimensions

113 -beat measurements of middle cerebral artery blood flow velocity (MCAv; transcranial Doppler), heart

114 nts, we recorded middle cerebral artery mean blood flow velocity (MCAVm) using transcranial Doppler u

115 lectrocardiogram) and middle cerebral artery blood flow velocities (mean, total, mean/RR interval; Go

116 rast analysis were compared with the retinal blood flow velocities measured by video fluorescein angi

117 Peak aortic blood flow velocity measured at 24 hours was a good disc

118 by intravascular ultrasound), renal Doppler blood flow velocity (measured by a Doppler flow wire in

119 Intracoronary Doppler blood flow velocity measurements were obtained with a Do

120 rovascular blood volume (MBV), microvascular blood flow velocity (MFV), and microvascular blood flow

121 nal arterial replacement is clear, the lower blood flow velocities of small-diameter arteries like th

122 ured rcSO2 using near-infrared spectroscopy, blood flow velocities of the middle cerebral artery, and

123 01 compared with pretreatment angiogram) and blood flow velocity (p < 0.01 compared with pretreatment

124 = 0.005) and reduced middle cerebral artery blood flow velocity (P = 0.002).

125 e were no differences regarding preoperative blood flow velocity parameters among the groups.

126 determine whether changes of cerebral artery blood flow velocity precede or follow reductions of arte

127 Mean blood flow velocity predicted incident depressive sympto

128 action and relaxation, it increases coronary blood flow velocity, predominantly by increasing the dom

129 The cardiac cycle influences the blood flow velocity profiles systematically in retinal a

130 ing, we also quantified the four-dimensional blood-flow velocity profiles associated with the disease

131 Beat-to-beat changes in renal blood flow velocity (RBV; Duplex Ultrasound), mean arter

132 ance: This study suggests that a low retinal blood flow velocity reflects in a visually distinct cont

133 Myocardial blood flow velocity reserve correlated significantly (P<

134 However, myocardial blood flow velocity reserve in patients with no signific

135 ed significantly (P<0.0001) lower myocardial blood flow velocity reserve in vascular territories subt

136 epicardial cross-sectional area and coronary blood flow velocity, resulting in an increase in absolut

137 ity and precise, simultaneous calculation of blood flow velocity, shear stress and drug distribution.

138 tion, and 8 with old myocardial infarction), blood flow velocities through the LVOT were recorded usi

139 ystemic gradient and Doppler measurements of blood flow velocity through the shunt at 1 day.

140 Cerebral blood flow velocities (transcranial Doppler) from middle

141 ll thickness, and a left circumflex coronary blood flow velocity transducer.

142 ood flow was calculated from measurements of blood flow velocity using intracoronary Doppler and coro

143 acute (3-7 d) ischemia-induced increases in blood flow velocity, vessel lumen diameter, and red bloo

144 t impairment of microhemodynamics, including blood flow velocity, volumetric blood flow, and function

145 smaller than SV luminal diameters (P=0.029), blood flow velocity was greater in RA than SV (P=0.008),

146 Cerebral blood flow velocity was measured in 6 patients through t

147 Blood flow velocity was measured in multiple sites in th

148 adenosine; however, the increase in coronary blood flow velocity was not significantly affected.

149 er treatment, Doppler sonography showed that blood flow velocity was preserved in AdCOX-1-treated art

150 interval, chest pain severity, and coronary blood flow velocity were made before and after low-dose

151 transcranial Doppler middle cerebral artery blood flow velocity were measured during 5 min of sponta

152 blood volume (rBV), relative blood flow, and blood flow velocity were quantified.

153 ONH topography and blood flow velocity were serially studied with scanning

154 volume [rBV], relative blood flow [rBF], and blood flow velocity) were measured on both 3D and 2D dat

155 ssessed by measuring the change in pulmonary blood flow velocity with a Doppler-tipped wire and the m

156 ured with intravascular ultrasound and renal blood flow velocity with the aid of an intravascular Dop

157 aditionally required measurement of coronary blood flow velocity with the Doppler wire and, more rece

158 During reactive hyperemia, blood flow velocity yielded peak velocity, time to peak,