ライフサイエンスコーパス: blood group A

1 g complex lactoside carbohydrates resembling blood group A.

2 Omicron variants, exhibited specificity for blood group A.

3 Among 925 LT recipients, 406 were blood group A, 94 group B, 380 group O, and 45 group AB.

4 Blood groups A, AB and B significantly reduced IE bindin

5 3)-N-acetylgalactosamine residues from human blood group A and AB mucin glycoproteins, Forssman hapte

6 We show that mAb F77 can also bind to blood group A and B analogs but with lower intensities.

7 rase (GTB) catalyze the final step in ABO(H) blood group A and B antigen synthesis through sugar tran

8 Recombinant E-ABase not only destroyed the blood group A and B antigenicity of human type A and B e

9 Histo-blood group A and B antigens are oligosaccharide antigen

10 stimulated to secrete substantial amounts of blood group A and B antigens linked to von Willebrand fa

11 lates gastrointestinal mucosal expression of blood group A and B antigens) (rs602662, P=3.4x10(-5)).

12 Clusters enriched for aminoglycosides, blood group A and B antigens, glycolipids, ganglio-serie

13 anti-blood group antibodies interacting with blood group A and B antigens.

14 e enzymes responsible for synthesis of histo-blood group A and B antigens.

15 nal step of the biosynthesis of ABO(H) human blood group A and B antigens.

16 glycosyltransferases that includes the histo blood group A and B enzymes.

17 glycosyltransferases that includes the histo-blood group A and B glycosyltransferases, Forssman glyco

18 ha1-->2)Gal) from glycoconjugates containing blood group A and B glycotopes, respectively.

19 igands supported this preference over type 1 blood group A and B oligosaccharides.

20 Four of five patients were blood group A and had no significant comorbid conditions

21 nthesis of oligosaccharides similar to human blood group A and may participate in the synthesis of th

22 cause in-hospital mortality in patients with blood group A and O who were transfused.

23 wing immunocytochemical markers were tested: blood group A and precursors of blood antigens; laminin

24 s such as pregnancy-induced hypertension and blood groups A and AB as compared to blood group O and R

25 The carbohydrate antigens, blood groups A and B, and the alpha-gal epitope (Galalph

26 d no longer than 7 days, both in patients of blood groups A and O and all patients.

27 ytes, but also released A-Tri and B-Tri from blood group A(+)- and B(+)- containing glycoconjugates.

28 l for studying the structure and function of blood group A- and B-containing glycoconju-gates as well

29 functionalized with immunoglobulin G against blood group A (anti-A IgG) by forming a self-assembled m

30 e perfusion (HMP) strategies, we demonstrate blood group A antigen loss of approximately 80% in as li

31 Particularly, we identified oligosaccharide blood group A antigen tetraose 6 (BGA6) as a biologicall

32 In analogy with histo-blood group A antigen, Forssman (Fs) antigen terminates

33 bodies against alpha-gustducin and the human blood group A antigen.

34 ncogene at codon 12 and all tumors expressed blood group A antigen.

35 vonifractor plautii to enzymatically convert blood group A antigens from the renal vasculature of hum

36 monstrating its versatility for synthesis of blood group A antigens.

37 n for anitgen-negative donors, 6) to resolve blood group A, B, and D typing discrepancies, 7) to dete

38 ound sulfated disaccharides as well as human blood groups A, B, and H on both N-glycans and linear gl

39 Incompatibility of the major blood groups A, B, and O has been an absolute contraindi

40 al GSL identified were GSL with the terminal blood groups A, B, H, Le(a), Le(b), Le(x), Le(y), P1, an

41 ythrocytes was insensitive to differences in blood groups A, B, O, or MN.

42 Blood groups A, B, or AB, were at 2.85 times (95% CI = 2

43 Preincubation of blood group A cells with a blood group-binding galectin

44 Acbeta3Galalpha4Galbeta4Glcbeta1Cer), i.e. a blood group A determinant on a type 4 core chain.

45 -binding galectin specifically inhibited the blood group A enhancement of SARS-CoV-2 infection, where

46 ulfated and methylated variants of the histo-blood group A epitope.

47 recombinant lectin, rCNL, agglutinates human blood group A erythrocytes and is specific for the uniqu

48 o displayed a preferential ability to infect blood group A-expressing cells.

49 B6 wild-type (WT) mice were sensitized with blood group A-human erythrocytes; others received passiv

50 To cleave blood group A immunodeterminants from erythrocytes, we p

51 Not only did each RBD recognize blood group A in a glycan array format, but each SARS-Co

52 nical evolution of COVID-19 and confirm that blood group A individuals are at greater risk of infecti

53 AB mucin glycoproteins, Forssman hapten, and blood group A lacto series glycolipids.

54 des by remodeling blood group B mimicry into blood group A mimicry.

55 The accuracy for detecting blood group A (n=12), B (n=13), AB (n=9), O (n=14), and

56 Thr residues in the tandem repeat domains of blood group A-negative porcine submaxillary gland mucin.

57 asite glycans structurally distinct from the blood group A oligosaccharides on the hemocyte surface m

58 that CvGal1 preferentially recognizes type 2 blood group A oligosaccharides.

59 on-associated carbohydrate antigens, such as blood group A or B and the alpha-gal epitope (Gal(alpha)

60 pes of the same lines transfected with histo-blood group A or B genes.

61 zyme-linked immunosorbent assay (ELISA) with blood group A or B human red cell membranes, as solid ph

62 Blood group A or B patients had lower antibody levels th

63 sitization, donor blood group O to recipient blood group A or B transfer, and ciclosporin treatment h

64 elated antigens LewisX, Y, and B, as well as blood group A or B were not bound.

65 lood stored 7 days or less (in patients with blood group A or O: 0.92, 0.74-1.15, p=0.48; in all 0.90

66 blood product use history (in patients with blood group A or O: hazard ratio 0.94, 95% CI 0.73-1.20,

67 ence (STD) NMR confirmed RgGH98 affinity for blood group A over blood group B and H antigens.

68 al blood type donor (P = .01), recipient non-blood group A (P = .02), and donor cause of death other

69 ults demonstrated that SARS-CoV-2 can engage blood group A, providing a direct link between ABO(H) bl

70 ors for parasite invasion, such as the Duffy blood group, a receptor for Plasmodium vivax, and the Ge

71 for all B recipients, even shorter than for blood group A recipients (median waiting times of A2/A2B

72 Retrospectively, blood group A recipients of O grafts with PLS were compa

73 lated specificity, such as B cells with anti-blood group A specificity.

74 ereus galectin shows particular affinity for blood group A structures.

75 Blood group A subjects had higher levels of anti-pig IgM

76 galectin for N-acetyllactosamine, the human blood group A tetrasaccharide and Galbeta1,3GalNAc relat

77 and LC-FD-MS/MS that RgGH98 is specific for blood group A tetrasaccharide type II (BgA II).

78 up-specific analysis showed a higher risk in blood group A than in other blood groups (odds ratio, 1.

79 Histo-blood group A transferase produces A antigens and transf

80 A bacterial version of human blood group A transferase was identified and found to be

81 osylceramide) were identified as well as the blood group A type 1 hexaosylceramide.

82 Finally, blood group A was more frequent in COVID+ (45.5%) than C

83 Recipients with blood group A were also more likely to develop IEC-defin

84 aeroallergen sensitization, cat at home, and blood group A were associated with increased odds of alp