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1 sent protein aggregates that contain several blood proteins.
2 ytic activity may influence cross-linking of blood proteins.
3 ed from fragments of extracellular matrix or blood proteins.
4 state-of-the-art methods available mostly on blood proteins.
5 of proteases working collectively to degrade blood proteins.
6                                     Abundant blood proteins adducted by active electrophiles are exce
7                                              Blood proteins adsorb onto the surface of nanoparticles
8   These initial efforts were centered around blood protein adsorption on biomaterials and related mec
9 T cells to peptide-MHC complexes, results in blood protein Ags having a profound effect on thymic T c
10                                    The major blood protein albumin is constitutively transcytosed bid
11                     We study exchange of two blood proteins, albumin and fibrinogen, adsorbing to and
12                      We studied inflammatory blood proteins (Alpha-1-acid glycoprotein-A1AG1; Albumin
13           Beta-2-glycoprotein I (B2GPI) is a blood protein and the major antigen in the autoimmune di
14 s in our understanding of control of soluble blood proteins and blood cell receptors by functional di
15 h as extreme dilutions and interactions with blood proteins and cells.
16                                  Circulating blood proteins and enzymes are maintained within normal
17 gested that alterations in brain structures, blood proteins and inflammation potentially contribute t
18 whether DNA is either bound to aggregates of blood proteins and lipid micelles or intrinsically assoc
19 ics of this versatile interface, interfering blood proteins and potential interferences associated wi
20 t a >25 million-fold excess of contaminating blood proteins and represents a 4 order of magnitude imp
21  lead to undesired peptide interactions with blood proteins and self-aggregation due to exposed hydro
22 strongly conserved among vitamin K-dependent blood proteins and, in addition to a high relative conte
23 cursors to neuropeptides, synaptic proteins, blood proteins, and transporters.
24                  The majority of non-albumin blood proteins are post-translationally modified with si
25  comparable to or smaller than many globular blood proteins are unknown.
26 ise the integrity of this barrier, and allow blood proteins as large as albumin to gain access to the
27               This cohort study found 7 cord blood proteins associated with birth weight and growth t
28            Proteomic profiling identified 35 blood proteins associated with chronic histopathologic l
29  results demonstrate that dynamic changes in blood proteins associated with disease severity can pote
30 evaluated the association between a panel of blood proteins at initial presentation of bacteremia and
31 -PSMA-1007 demonstrated significantly higher blood protein binding and bone uptake than the other tra
32                   Albumin, the most abundant blood protein, binds heme specifically and bypasses the
33                  Our study aimed to identify blood protein biomarkers associated with progression of
34                                      Several blood protein biomarkers have been associated with prost
35  previously, including over 200 cleavages of blood proteins by aminopeptidases.
36                          We present multiple blood protein-cancer risk associations, including many d
37                                              Blood protein concentrations are clinically useful, pred
38 ls that predict TEG curve outputs from input blood protein concentrations, to facilitate treatment de
39                                 Digestion of blood proteins depends on the biphasic expression of ser
40 we aimed to determine whether levels of cord blood proteins differ between the offspring of asthmatic
41 gut extracts, while in vitro fluorescent and blood protein digestion assays revealed important substr
42 A production from glutamic acid derived from blood protein digestion.
43                       Spectra indicated that blood proteins displaced the nanotube coating of synthet
44  effect of sample matrix (animal tissues and blood proteins, etc.).
45 ion has been paid to serum albumin, the main blood protein, even though blood disposal is a severe pr
46           Our findings not only suggest that blood protein expression is influenced by polymorphisms
47 ral network prediction head can generate 206 blood protein expression levels.
48                                              Blood protein extravasation through a disrupted blood-br
49 , in which brain tissue becomes permeable to blood proteins, extravascular fibrin deposition correlat
50                        Here we show that the blood protein fibrinogen induces rapid microglial respon
51                       Here, we show that the blood protein fibrinogen induces spine elimination and p
52                        Here we show that the blood protein fibrinogen is an inhibitor of neurite outg
53                        Here we show that the blood protein fibrinogen, which leaks into the CNS immed
54 y quenching with acetonitrile to precipitate blood proteins followed by analysis on an LC-electrospra
55 ales have access to human hosts that provide blood proteins for egg development, conspecific males th
56 st report on determining adduction levels of blood proteins for long-term exposure to CEs.
57 entation of various extracellular matrix and blood proteins generates antiangiogenic substances that
58                       First, mouse and human blood proteins had insignificant affinity for PEG-NPs.
59 s identified in humans involves the abundant blood protein haptoglobin.
60 oach is demonstrated through the assays of a blood protein human alpha-thrombin and an oncoprotein hu
61 arriers, degradation of hemoglobin and other blood proteins, immune evasion, and activation of inflam
62  blood-brain barrier, the potential role for blood proteins in repair processes remains unknown.
63 nces allowed the extravasation of endogenous blood proteins, including albumin and immunoglobulin, as
64 omparative functional multiomics showed that blood proteins induce distinct receptor-mediated transcr
65 on of the brain microenvironment by allowing blood proteins into the CNS.
66 and binding.Von Willebrand factor (VWF) is a blood protein involved in clotting and is proposed to be
67 g genes encoded stress response proteins and blood proteins involved in coagulation that were differe
68                        Secretion of multiple blood proteins is a major drain on liver energy and nutr
69 However, direct capillary electrophoresis of blood proteins is challenging due to its high content of
70     We show that initial degradation of host blood proteins is ordered, occasionally redundant, and s
71  attachment to extracellular matrices and to blood proteins is regulated from inside the cell.
72                                Variations in blood protein levels have been linked to numerous comple
73 PGSs for common complex diseases and traits, blood protein levels, and brain and other organ morpholo
74 ants in modifying the traits pulse pressure, blood protein levels, and monocyte count.
75 blood-derived macrophages and thus may alter blood protein levels.
76 he second data set to simulate variations in blood protein levels.
77  barrier, which can lead to extravasation of blood proteins like fibrinogen into the brain.
78 in reversing inactivation by the mixtures of blood proteins, membrane lipids, and fatty acids studied
79  about 70% of circulating Hcy is N-linked to blood proteins, mostly to hemoglobin and albumin.
80 illebrand factor, an ultralarge concatemeric blood protein, must bind to platelet GPIbalpha during bl
81          Basket samples yielded collagen and blood proteins of bovine origin (Bos genus) and a large
82  Using hen egg-white lysozyme, the effect of blood proteins on CD4 thymic cells was examined.
83 d to assess predictive potential of baseline blood proteins on weight loss.
84  the effects of human serum albumin, a major blood protein, on this phase separation.
85 cts from reactions with nucleophilic loci of blood proteins, particularly Cys34 of human serum albumi
86                      Measurement of glycated blood proteins, particularly glycated hemoglobin (HbA1c)
87                                 However, how blood proteins polarize innate immune cells remains larg
88 concentrations of a billion times lower than blood proteins, poses a significant challenge for early
89                                              Blood protein profiling was performed on 59 COVID-19 mil
90                   Using the GWAS results and blood protein quantitative loci, we perform Mendelian ra
91 ib showed a wider spectrum of cardiovascular blood protein reduction, but the protein reduction effec
92 ciated with cardiovascular risks, we studied blood proteins related to inflammation and cardiovascula
93       We consolidate the findings of altered blood proteins relevant for progression to psychotic dis
94  (SWCNT) and use it against a panel of human blood proteins, revealing a specific corona phase that r
95  Willebrand factor (VWF), a large multimeric blood protein, senses changes in shear stress during ble
96                                              Blood protein signatures highly associated with neutroph
97 lar dynamics simulations to demonstrate that blood proteins such as bovine fibrinogen (BFG) can absor
98 ften observed during the binding of drugs to blood proteins such as human serum albumin (HSA).
99                             HNE adduction of blood proteins, such as human serum albumin (HSA), yield
100  von Willebrand factor (VWF) is a multimeric blood protein that acts as a mechanical probe, respondin
101                       Albumin is an abundant blood protein that acts as a transporter of a plethora o
102  of inhibitory proteoglycans is induced by a blood protein that leaks in the CNS after vasculature ru
103     We focus on the complement system of ~40 blood proteins that bind microbes, nanoparticles, and me
104 ource for investigation of the immunology of blood proteins that could support therapeutic targeting
105   We also identified a temporal signature of blood proteins that was significantly different at early
106 siological changes, allowing them to utilize blood protein to develop eggs: clearing excess fluid, di
107  our knowledge, this is the first study with blood proteins to show that, in addition to the aromatic
108 sed of tryptic peptides from human and mouse blood proteins using high-resolution time-of-flight mass
109 ature of the associations between correlated blood proteins utilizing Mendelian randomization.
110                                    The human blood protein vitronectin (Vn) is a major component of t
111 function of the mechanosensitive, multimeric blood protein von Willebrand factor (VWF) is dependent o
112 acid (bis-ANS) to hydrophobic pockets in the blood protein von Willebrand factor (VWF) is enhanced up
113                The mechanosensitive adhesive blood protein, von Willebrand Factor (vWF), interacts wi
114                               A total of 157 blood proteins were quantified by a proximity extension
115                         SNPs influencing six blood proteins were used to evaluate the nature of the a
116  the unique optical anisotropy properties of blood proteins, which undergo structural alterations at
117 ssociations of 368 inflammatory-related cord blood proteins with birth weight or BWR and with early l
118 ars and a-dicarbonyl compounds interact with blood proteins yielding AGEs.
119 and alpha-dicarbonyl compounds interact with blood proteins yielding AGEs.

 
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