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1 oride, sodium), creatinine, urea, and random blood sugar).
2 ate, with higher-GI foods rapidly increasing blood sugar.
3 ed obesity, cholesterol, blood pressure, and blood sugar.
4 variable, accuracy at times of low and high blood sugar.
5 ad (GL), which identify foods that stabilize blood sugar.
6 They too had normal fasting blood sugars.
7 improve outcomes more than tight control of blood sugars.
8 were treated with insulin to normalize their blood sugars.
9 /L; 95% CI: -0.03, 0.28 mmol/L), and fasting blood sugar (-0.16 mmol/L; 95% CI: -0.44, 0.11 mmol/L).
13 The pattern of diabetic care and control of blood sugar among diabetic patients are not well describ
14 eakfast skipping) on lipid profiles, fasting blood sugar and body composition in Ethiopian set up.
16 in addressing the increasing burden of high blood sugar and inform the potential impact of different
17 istration of anti-resistin antibody improves blood sugar and insulin action in mice with diet-induced
18 l obesity, high blood pressure, and abnormal blood sugar and lipid levels, is a growing global health
21 ), respectively; obesity, hypertension, high blood sugar, and regular cigarette smoking were rare.
22 ith the risk of high blood pressure, fasting blood sugar, and triglycerides, with marginal significan
24 on-beta-cells that are capable of modulating blood sugar; and the construction of synthetic 'cells' o
25 t how the counterregulatory responses to low blood sugar are affected under these metabolic condition
26 e present study was to determine whether low blood sugar augments glucagon's ability to increase gluc
29 in supplementation also led to lower fasting blood sugar, body mass index, and total cholesterol.
30 r type 2 diabetes, metformin, not only lower blood sugar, but also promote longevity in preclinical m
31 when diabetic patients try to control their blood sugar, but the molecular mechanism of this 'metabo
33 ese findings support the hypothesis that low blood sugar causes greater participant cooperation - whi
34 vascular biomarkers, including lipoproteins, blood sugar, circulatory pressure, proinflammatory marke
36 e for individuals with diabetes by improving blood sugar control and limiting the risk of hypoglycemi
37 cose and lipid metabolism, leading to better blood-sugar control and decreased insulin resistance.
41 ted with an increased risk of higher fasting blood sugar (FBS) (OR = 6.07; 95% CI: 1.33-27.74, P valu
42 ver craniocaudal length (P = 0.012), fasting blood sugar (FBS) (P = 0.047), aspartate aminotransferas
43 garding diabetes-related traits like fasting blood sugar (FBS) and type 2 diabetes (T2D) and glaucoma
44 oglobin (HbA1c), and mean changes in fasting blood sugar (FBS) level, body weight (BW) and homeostasi
46 albumin, complete blood count (CBC), fasting blood sugar (FBS), Alanine aminotransferase (ALT), and a
48 andial glycemic response, or the increase in blood sugar following a meal, is a crucial component to
49 strain should provide utility in separating blood sugar from blood pressure-related treatment effect
50 drink causes some participants who have low blood sugar from fasting to give lower slant estimates.
51 that can reveal a patient's average level of blood sugar from the past 2-3 months instead of just mea
53 DAN score (0 to 6 points) comprises baseline blood Sugar (glucose; 8.1-12.0 mmol/l [145-216 mg/dl] =
57 d metabolic dysregulation (obesity, elevated blood sugar, high blood pressure, high levels of triglyc
58 Buruli ulcer pathogenesis is that changes in blood sugar homeostasis in infected patients are mirrore
59 ptor (GCGR), a Family B GPCR responsible for blood sugar homeostasis, and broadly inhibits receptor-i
60 h as lipid levels, circulating sex hormones, blood sugar homeostasis, inflammation and immune functio
66 ation significantly reduced the elevation in blood sugar in mice following an oral but not intraperit
70 days exhibited similar or better control of blood sugar level (20% reduction in HbA1c) and weight co
71 ound that when APR systemically quenched the blood sugar level in diet-induced obesity (DIO) diabetic
73 n premedication, Wistar male rats presenting blood sugar levels >20 mmol/L were selected for investig
74 serum antibody titers to these bacteria, and blood sugar levels (glycosylated hemoglobin, HbAlc) were
75 significant changes in creatinine, urate, or blood sugar levels after conversion, but the mean plasma
82 del with increased blood pressure und higher blood sugar levels had a reduced survival compared to co
87 d 5 days after the start of treatment, lower blood sugar levels were observed in the particle treatme
95 Biochemical parameters such as FBS (fasting blood sugar), lipid profile, and liver and kidney functi
96 ted of poisoning with illicit drugs and high blood sugar, methadone poisoning should be considered in
97 A significant decrease in BMI (Kg/m(2)) and blood sugar (mg/dL) levels, while an increase in systoli
101 cept for people with diabetes and those with blood sugar problems), desirable flavor, and taste,maple
102 , periodontal index, body mass index, random blood sugar (RBS), and glycated hemoglobin (HbA1c).
103 ally reduce the GI, contributing to improved blood sugar regulation and overall metabolic health.
105 o produce insulin Abs that may lower fasting blood sugars similar to an insulin autoimmune syndrome.
106 le to be able to predict what will happen to blood sugar so that they can do something to prevent hyp
111 gher body mass index, transaminases, fasting blood sugar, triglyceride, low density lipoprotein level
117 ressure, low-density lipoprotein, and random blood sugar were significantly higher in NP+C and P+C, w
119 uous measures for blood pressure and fasting blood sugar were used instead of hypertension and diabet
121 d consistently more often when their owner's blood sugars were reported to be outside, than within, t
122 he enhanced hyperammonemia and lower fasting blood sugar, which are both seen in the DKO mice, indica