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1 nt a critical barrier for a bacterium in the bloodstream.
2 elease of tumor-specific biomarkers into the bloodstream.
3 anslocation from the lamina propria into the bloodstream.
4 s can circulate for at least 48 hours in the bloodstream.
5 urface proteins (e.g., CA-125) shed into the bloodstream.
6 iversal property of HIV-1 circulating in the bloodstream.
7 sure in hydrodynamic situations, such as the bloodstream.
8 levels and the glucagon/insulin ratio in the bloodstream.
9 , where they secrete their products into the bloodstream.
10 mensal bacteria and can be absorbed into the bloodstream.
11 d for full S. marcescens pathogenesis in the bloodstream.
12 ect clonal transmission of probiotics to the bloodstream.
13 ly-labeled circulating cells directly in the bloodstream.
14 ion through the endothelium barrier into the bloodstream.
15 ital tract before virus is detectable in the bloodstream.
16 irst 2 weeks after virus was detected in the bloodstream.
17 e transmissible "stumpy forms" in their host bloodstream.
18  and efferent lymphatic vessels to enter the bloodstream.
19 ion and transmission potential in the mammal bloodstream.
20 ic sites including the respiratory tract and bloodstream.
21  a stochastic translocation pattern into the bloodstream.
22 eractions under dynamic flow that mimics the bloodstream.
23 poietic progenitors that migrate through the bloodstream.
24 ap that can actively recruit cancer cells in bloodstream.
25 e used to deliver intravenous therapy to the bloodstream.
26  microsensors that circulate directly in the bloodstream.
27 arbor complex viral populations within their bloodstreams.
28 inary tract (23%), bone and joint (16%), and bloodstream (11%).
29 tible), opportunistic (21.9% vs. 10.8%), and bloodstream (19.1% vs. 5.4%) (P < .001).
30 movement of interstitial chemokines into the bloodstream, a response that supported abluminal-to-lumi
31 solates and compared these isolates with 293 bloodstream and 83 veterinary surveillance ESBL-E coli i
32 ks protein synthesis, can spread through the bloodstream and affect organs, such as the heart and kid
33  thus enabling Y. pestis to reproduce in the bloodstream and be transmitted to new hosts through flea
34             From the lungs it spreads to the bloodstream and causes organ damage.
35  scales given for butyrate circulates in the bloodstream and colon were incorrect.
36 gen that has been associated with nosocomial bloodstream and deep wound infections causing a high mor
37 tal stimuli that Babesia spp. utilize in the bloodstream and for transmission to the tick, with an em
38                      Platelets circulate the bloodstream and principally maintain hemostasis.
39                                  We examined bloodstream and procyclic life cycle stages, and a knock
40 pe that provides a survival advantage in the bloodstream and promotes their ability to establish over
41 numbers of B. burgdorferi spirochetes in the bloodstream and, ultimately, consistently reduced spiroc
42 zoites navigate through the dermis, into the bloodstream, and eventually invade hepatocytes.
43 e that mainly affects the skin, lymph nodes, bloodstream, and lungs.
44 ith circulating tumor cells (CTC) within the bloodstream, and their involvement in the establishment
45 d motility migrate and are launched into the bloodstream as single circulating tumor cells (CTC) or m
46 arp photoacoustic resonances directly in the bloodstream as the basis for new super-resolution photoa
47 eonates, a surge of AVP is released into the bloodstream at levels exceeding release during life-thre
48 on in the airway and oral application in the bloodstream: BK dysfunction recovered acutely and over t
49 radiation, may promote an IGFBP-4 release in bloodstream both in mice irradiated with 100 mGy X-ray a
50 sitive S. aureus cultures using a hierarchy (bloodstream [BSI], surgical site [SSI], and all other ty
51  only effective to kill microfilariae in the bloodstream, but is often ineffective to kill adult fila
52 ss to the circulation are plucked out of the bloodstream by the intravascular macrophages of the live
53 d sample for enhanced molecular diagnosis of bloodstream Candida infection and especially compared it
54 ignificantly improved molecular diagnosis of bloodstream Candida infection.
55 rt of entry to the lymphatic drainage and/or bloodstream, causing dissemination of life-threatening i
56 tory factors are intensely released into the bloodstream, causing the so-called "cytokine storm".
57                                However, most bloodstream cells contain GLUT1 and are not directly aff
58 sphorylation (OxPhos) to generate ATP, while bloodstream cells switch to aerobic glycolysis.
59 kinetics and intensity of Ara h 6 passage in bloodstream depend on both individual and food matrix.
60      Moreover, periopathogens can invade the bloodstream, disseminate and promote cardiovascular dise
61 scle, and sustained elevation of MG53 in the bloodstream does not have a deleterious impact on db/db
62 cting part of the hydrodynamic forces of the bloodstream encountered by the classical spherical shape
63  as both a survival niche and conduit to the bloodstream for S. pyogenes, explaining the phenomenon o
64        The transferrin receptor (TfR) of the bloodstream form (BSF) of Trypanosoma brucei is a hetero
65 lar axonemal inner-arm dynein complex in the bloodstream form and investigated its mechanistic role i
66 y for the initiation of cell division of the bloodstream form in the mammalian host.
67  developed permeabilized cell system for the bloodstream form of T. brucei, we show that down-regulat
68 ility-mediated cytokinesis initiation in the bloodstream form of T. brucei.
69 ndrial respiratome and VSG gene silencing in bloodstream form T. brucei.
70 ellum, and are required for viability in the bloodstream form.
71 on of the specialized subtelomeric PTUs, the Bloodstream-form Expression-Sites (BESs), which house th
72 dited CYb mRNA is downregulated in mammalian bloodstream forms (BSF) at the level of editing initiati
73 hat show nanomolar potency against T. brucei bloodstream forms, Leishmania and Trypanosoma cruzi.
74 ivity against Trypanosoma brucei rhodesiense bloodstream forms, which is another important aetiologic
75 r ferroportin to control iron entry into the bloodstream from dietary sources, iron recycling macroph
76 venges toxic hemoglobin (Hb) leaked into the bloodstream from erythrocytes.
77 ent of circulating proteins, which enter the bloodstream from inflamed tissues, also offers insight i
78 ulating tumor cells (CTCs) are shed into the bloodstream from primary tumors, but only a small subset
79 apted clade, of which 23 were represented in bloodstream, hospital sewage, and municipal wastewater i
80                           When it enters the bloodstream, however, this bacterium may cause the serio
81                          In contrast, in the bloodstream, iMPs activated endothelial cells and platel
82  pathogens most frequently isolated from the bloodstream in humans.
83 th increased incidence of and mortality from bloodstream infection (BSI) and sepsis.
84 ital, a retrospective cohort of adult KPC-KP bloodstream infection (BSI) cases (January 2014 to Decem
85 tibiotic treatment for lower UTI and risk of bloodstream infection (BSI) in adults aged >=65 years in
86 icenter prospective study of all episodes of bloodstream infection (BSI) in high-risk FN patients (20
87                                         MRSA bloodstream infection (BSI) incidence per 100 000 popula
88 prediction models have been shown to predict bloodstream infection (BSI) likelihood in this populatio
89 t of vancomycin-resistant Enterococcus (VRE) bloodstream infection (BSI) on outcomes of allogeneic he
90 007-2017) of hospital-based Salmonella Typhi bloodstream infection (BSI) surveillance in the Democrat
91 ed a DOOR endpoint for Staphylococcus aureus bloodstream infection (BSI) through a survey to infectio
92    Little is known of the long-term risks of bloodstream infection (BSI) with extended spectrum beta-
93                  Hospital-associated sepsis, bloodstream infection (BSI), and mortality (>3 days afte
94 fficile infection (CDI) is a risk factor for bloodstream infection (BSI).
95 rtality of patients with liver cirrhosis and bloodstream infection (BSI).
96  of Community acquired Staphylococcus aureus bloodstream infection (CA-SABSI) with myocardial infarct
97 luate differences in central line-associated bloodstream infection (CLABSI) rates by how central line
98                      Central line-associated bloodstream infection (CLABSI) remains prevalent in hosp
99  hospital-onset multidrug-resistant organism bloodstream infection (MDRO-BSI) and Clostridium diffici
100 sed association with central-line associated bloodstream infection (odds ratio, 0.505; 95% CI, 0.336-
101 1,000 line days) and central-line associated bloodstream infection (peripherally inserted central cat
102 esistant Enterococcus (VRE) and ICU-acquired bloodstream infection (UABSIs) were analysed from 1,189,
103    Salmonella Typhi was the leading cause of bloodstream infection among infants and young children <
104 from 16 sites in ten countries: 174 with CSE bloodstream infection and 123 with CRE bloodstream infec
105 um samples from 30 children with a bacterial bloodstream infection and 35 children with Plasmodium fa
106 % (35 of 174 patients) for patients with CSE bloodstream infection and 35% (43 of 123 patients) for p
107 ous infections usually arise from an initial bloodstream infection and are frequently recalcitrant to
108 roorganisms associated with catheter-related bloodstream infection and colonization was significantly
109  Salmonella (NTS) isolated from persons with bloodstream infection and diarrheal disease from 2007 th
110 d Salmonella Typhimurium are major causes of bloodstream infection and diarrheal disease in East Afri
111 ne prophylaxis was associated with decreased bloodstream infection and intestinal colonization by gra
112 nomic analysis of E. faecium associated with bloodstream infection and isolated from wastewater.
113  epidemic, using temporal trends in S. Typhi bloodstream infection and perforated abdominal viscus at
114 with higher rates of central-line associated bloodstream infection and venous thromboembolism than ce
115 o be associated with central-line associated bloodstream infection and venous thromboembolism were in
116 wastewater with 187 isolates associated with bloodstream infection at five hospitals in the East of E
117 ics, previous antibiotic exposure, and index bloodstream infection caused by either rGNB or Candida s
118 biotics, previous antibiotic exposure, index bloodstream infection caused by either rGNB or Candida s
119 re independent, thus saving precious time in bloodstream infection diagnostics.
120 d its impact on the outcome of patients with bloodstream infection due to Enterobacteriaceae (BSI-E).
121 spital stay among inpatients in LMICs with a bloodstream infection due to Enterobacteriaceae.
122  with an increased risk for catheter-related bloodstream infection due to nonfermenting Gram-negative
123  swabs of their environment, together with 1 bloodstream infection during the study and 4 others over
124                        Patients with Candida bloodstream infection experienced a prior marked intesti
125  centre, aged 18 years or older with candida bloodstream infection from 2002 to 2015.
126  with daptomycin plus a beta-lactam for MRSA bloodstream infection had lower odds of composite clinic
127 ous catheters (CVCs) reduce catheter-related bloodstream infection in adults and children receiving i
128  of staphylococci and for protection against bloodstream infection in animals.
129 uring intestinal colonization and subsequent bloodstream infection in immunocompromised pediatric pat
130                                       During bloodstream infection in mice, DARC targeting by S. aure
131 ial burden and inflammation during S. aureus bloodstream infection in mice.
132 ontributing to the lethality observed during bloodstream infection in mice.
133 ith two or more second-degree relatives with bloodstream infection in the follow-up period.
134                                              Bloodstream infection is an important cause of death wor
135                                              Bloodstream infection is associated with high mortality
136                                      Candida bloodstream infection is associated with high mortality.
137                            Mortality in MSSA bloodstream infection is declining, associated with a de
138  be considered if a femoral catheter-related bloodstream infection is suspected.
139                                              Bloodstream infection is the most severe, with mortality
140 am isolate showed that the subject's E. coli bloodstream infection likely originated from the intesti
141 ted in a strain that was acutely virulent in bloodstream infection models in mice and in ex vivo mode
142  treated with curative intent at the time of bloodstream infection onset.
143                         Neonatal sepsis is a bloodstream infection primarily caused by Escherichia co
144 n the post-Directive central line-associated bloodstream infection rates associated with a unit incre
145                        Patients with candida bloodstream infection receiving an infectious disease co
146 ano-mupirocin in a murine model of S. aureus bloodstream infection resulted in improved antibiotic di
147 teria was used as predictor of gram-negative bloodstream infection using Cox proportional hazards mod
148 Candidemia is a common healthcare-associated bloodstream infection with high morbidity and mortality.
149 eviated oxidative stress in our rat model of bloodstream infection with MRSA.
150 to how VREfm adapted during colonization and bloodstream infection within each patient.
151 -resistant urinary tract infection (UTI) and bloodstream infection worldwide.
152 ound three loci with a suggestive linkage to bloodstream infection, all on chromosome 4, at 46.6 cent
153 estinal colonization is highly predictive of bloodstream infection, in the setting of allo-HCT.
154 mia, one of the most common causes of fungal bloodstream infection, leads to mortality rates up to 40
155 ong central venous catheter catheter-related bloodstream infection, nonfermenting Gram-negative bacil
156  the early host response to S. aureus during bloodstream infection, promoting enhanced responses by b
157 al domination was associated with subsequent bloodstream infection, which was observed overall and in
158            Brucella spp. was the most common bloodstream infection, with B. melitensis isolated from
159 d genetic intermixing between wastewater and bloodstream infection, with highly related isolates shar
160  to be linked with the risk of contracting a bloodstream infection.
161 , enabling replication that can seed intense bloodstream infection.
162 ciated with protection against gram-negative bloodstream infection.
163 tant exhibits a severe fitness defect during bloodstream infection.
164 in flora-related, or central line-associated bloodstream infection.
165 rt of clinical care of patients with candida bloodstream infection.
166 % (43 of 123 patients) for patients with CRE bloodstream infection.
167 in-flora-related, or central-line-associated bloodstream infection.
168 phi and 9 (1.3%) with Salmonella Paratyphi A bloodstream infection.
169  and black patients, and in the setting of a bloodstream infection.
170 h CSE bloodstream infection and 123 with CRE bloodstream infection.
171 genetic loci linked to risk of contracting a bloodstream infection.
172 te UMH9, which was recovered from a clinical bloodstream infection.
173 s thromboembolism or central-line associated bloodstream infection.
174 an promote host protection against S. aureus bloodstream infection.
175 lity, we analysed 1691 patients with candida bloodstream infection; 776 (45.9%) who had an infectious
176 ital, a retrospective cohort of adult KPC-KP bloodstream infections (BSI) cases (January 2014 to Dece
177 in etiologies and susceptibility patterns of bloodstream infections (BSI) in hospitalized children in
178 ith K. aerogenes versus Enterobacter species bloodstream infections (BSI).
179 eatment with DAP alone in patients with MRSA bloodstream infections (BSI).
180                                              Bloodstream infections (BSI-SA) and noninvasive coagulas
181                                              Bloodstream infections (BSIs) cause significant morbidit
182 ), yet the prevalence of IE in patients with bloodstream infections (BSIs) caused by different strept
183            Rapid diagnostic tests (RDTs) for bloodstream infections (BSIs) decrease the time to organ
184 as notified by hospital A of 3 patients with bloodstream infections (BSIs) with a rapidly growing non
185 d cultures, the gold standard for diagnosing bloodstream infections (BSIs), are insensitive and limit
186  critically needed for Staphylococcus aureus bloodstream infections (BSIs), particularly for methicil
187 eatment with DAP alone in patients with MRSA bloodstream infections (BSIs).
188 tor of early mortality risk in patients with bloodstream infections (BSIs).
189 robes are an important but uncommon cause of bloodstream infections (BSIs).
190 fections (CAUTI) and central line-associated bloodstream infections (CLABSI) per 1000 device days.
191                      Central line-associated bloodstream infections (CLABSIs) often result from intra
192 he incidence of VTE, central line-associated bloodstream infections (CLABSIs), and catheter malfuncti
193       Little is known about catheter-related bloodstream infections (CRBSIs) in this population.
194                    Among patients with CRGNB bloodstream infections (n=319) overall 30- and 90-day mo
195 or equal to median was associated with fewer bloodstream infections (OR, 0.67 [95% CI, 0.45-0.98).
196    Clinical trials for Staphylococcus aureus bloodstream infections (SAB) are broadly grouped into 2
197  events were febrile neutropenia (22 [66%]), bloodstream infections (six [16%]), and invasive fungal
198 sistant Enterococcus (VRE), and ICU-acquired bloodstream infections (UABSIs) for 1 189 142 patients f
199                       An outbreak of 162 Bcc bloodstream infections across 59 nursing facilities in 5
200 of antimicrobial therapy targeted toward CPE bloodstream infections and assist infection control and
201 s, with hypercapsule mutants associated with bloodstream infections and capsule-deficient mutants ass
202  anemia, intermittent proteinuria, recurrent bloodstream infections and chronic pulmonary disease.
203 enous catheter and arterial catheter-related bloodstream infections and colonization according to the
204                                              Bloodstream infections and invasive nontyphoidal Salmone
205 r severe and often deadly infections such as bloodstream infections and pneumonia.
206  have tremendous impact on the management of bloodstream infections and sepsis.
207 lications, including central-line associated bloodstream infections and venous thromboembolism.
208 us infections account for 15 to 50% of fatal bloodstream infections annually.
209                          Salmonella enterica bloodstream infections are an important cause of childho
210                                              Bloodstream infections are very rare for COVID-19 patien
211 exidine (CHG) bathing decreases incidence of bloodstream infections at intensive care units, but its
212 al center in New York City who had S. aureus bloodstream infections between 1 January 2007 and 31 Dec
213  and could help to optimize therapy early in bloodstream infections by CPE.
214                                              Bloodstream infections by Salmonella enterica serovar Ty
215                           The possibility of bloodstream infections caused by 3rd-generation cephalos
216                                              Bloodstream infections caused by nontyphoidal Salmonella
217 can identify patients at risk for subsequent bloodstream infections caused by resistant bacteria.
218  better understanding of the microbiology of bloodstream infections could improve outcomes.
219 aches to identify patients at risk of fungal bloodstream infections for pre-emptive therapeutic inter
220 eillance of Salmonella Typhi and Paratyphi A bloodstream infections from 5 October 2015 through 4 Oct
221 es transitioning patients with gram-negative bloodstream infections from intravenous to oral therapy
222                            Hospital-acquired bloodstream infections have a definite impact on patient
223 nd 2018, there was an associated rise in VRE bloodstream infections in hospitals where contact precau
224 ine (CHG) bathing decreases the incidence of bloodstream infections in intensive care units, but its
225 on reduces multidrug-resistant pathogens and bloodstream infections in intensive care units.
226 hospital stay and mortality in patients with bloodstream infections in LMICs.
227                  The identification of 4 Bcc bloodstream infections in patients residing at a single
228  the predominant cause of community-acquired bloodstream infections in sub-Saharan Africa (sSA).
229                      In order to prevent the bloodstream infections in the neonates, it is indispensa
230 lso been developed detecting the presence of bloodstream infections including electrochemical, potent
231    We hypothesized that septic patients with bloodstream infections may transition across states char
232 on profiles of 9,215 P. vivax parasites from bloodstream infections of Aotus and Saimiri monkeys.
233                No significant differences in bloodstream infections or laboratory alterations were re
234 next-generation sequencing-based analyses of bloodstream infections provide a valuable diagnostic pla
235 l fungi (that is, the mycobiota) with fungal bloodstream infections remains undefined(9).
236                                Gram-negative bloodstream infections represent a significant complicat
237                                  The NPV for bloodstream infections was 96.5%, for intra-abdominal cu
238                                  The NPV for bloodstream infections was 96.5%, for intraabdominal cul
239 cal blood culture samples from patients with bloodstream infections were incubated for 1 h with the "
240  spp. causing illness, describe non-Brucella bloodstream infections, and identify risk factors for br
241 dida albicans is a leading cause of systemic bloodstream infections, and synthesis of the phospholipi
242             We sought to describe Salmonella bloodstream infections, antimicrobial resistance, and ag
243 both in terms of intestinal colonization and bloodstream infections, compared with non-prophylaxed pa
244 lently for 30 isolates derived from clinical bloodstream infections, confirming system optimization f
245 nesis has primarily focused on pneumonia and bloodstream infections, even though one in five A. bauma
246 obiota is connected to risk of gram-negative bloodstream infections, expanding on our prior work in t
247 iated with male sex, central line-associated bloodstream infections, long-term acute care hospitals,
248 ate earlier optimization of the treatment of bloodstream infections, particularly in conjunction with
249 lmonella isolates from African patients with bloodstream infections, spanning 1966 to 2018.
250  rapid identification of Escherichia coli in bloodstream infections, we employed an existing colorime
251 f 4967 unique patients with Enterobacterales bloodstream infections, we sought to answer the question
252 onset (CO) and hospital-onset (HO) suspected bloodstream infections.
253 esponses, which collectively protect against bloodstream infections.
254 rbapenem-resistant Entero-bacteriaceae (CRE) bloodstream infections.
255 nces in management with mortality in candida bloodstream infections.
256 the 2 most prevalent Candida species causing bloodstream infections.
257 tles in pediatric patients with a concern of bloodstream infections.
258 hich is critical for successful treatment of bloodstream infections.
259            Radiolabeled NT3 crossed from the bloodstream into the brain and spinal cord in rodents wi
260 e intestinal lumen, predisposing patients to bloodstream invasion and sepsis.
261 n the colon, markedly increasing the risk of bloodstream invasion, sepsis, and death.
262                Fungal dissemination into the bloodstream is a critical step leading to invasive funga
263 li strains across time points as well as the bloodstream isolate showed that the subject's E. coli bl
264 he virulence of 100 individual P. aeruginosa bloodstream isolates and performed whole-genome sequenci
265  strains comprised of both oral isolates and bloodstream isolates from patients diagnosed with IE.
266  be absorbed through the urothelium into the bloodstream, leading to systemic adverse effects.
267 to be used with whole blood for detection of bloodstream microorganisms in sepsis.
268     Blood culture (BC) often fails to detect bloodstream microorganisms in sepsis.
269 ubset of non-classical (NC) monocytes in the bloodstream, namely the slan(+) -monocytes.
270 se risk associated with the full spectrum of bloodstream non-HDL cholesterol concentrations.
271                                       In the bloodstream, NSAIDs are mostly bound to serum albumin (S
272 es through the liver stage and back into the bloodstream of mice.
273 rom respiratory pathogens are present in the bloodstream of most CF patients, which could potentially
274 rge from the intestinal lumen and invade the bloodstream of vulnerable patients, causing disseminated
275 ocation to first microbiologically confirmed bloodstream or cerebrospinal fluid (CSF) infection betwe
276              Why some pneumococci invade the bloodstream or CSF (so-called invasive pneumococcal dise
277     The time from random allocation to first bloodstream or CSF infection was similar between the two
278                                       In the bloodstream, podoplanin induces platelet activation by b
279 d blood volume and the rarity of CTCs in the bloodstream preclude longitudinal, in-depth studies of t
280  conclude that EPS protects hosts from acute bloodstream S. aureus infection not only by inducing mac
281                      TbGPR89 is expressed on bloodstream "slender form" trypanosomes, which receive t
282 s more common among respiratory tract versus bloodstream specimens.
283  that depending on the environment, local or bloodstream, the consequences of the interactions betwee
284 aemoparasites that survive in the vertebrate bloodstream through antigenic variation of their Variant
285 cule drugs are not readily absorbed into the bloodstream through the gastrointestinal tract(8).
286        When an immune cell migrates from the bloodstream to a site of chronic inflammation, it experi
287 it is essential to maintain contact with the bloodstream to exert protective effects.
288 ntracellular content, DNA included, into the bloodstream to form neutrophil extracellular traps (NETs
289 that these bacteria are disseminated via the bloodstream to other organs.
290 agocyte, they are rapidly recruited from the bloodstream to sites of infection or injury to internali
291 crucial for spread of P. aeruginosa from the bloodstream to the feces during bacteremia, a process th
292  show that P. aeruginosa can spread from the bloodstream to the gallbladder, where it replicates to e
293 showed that these cells migrated through the bloodstream to the remodelled bone after injury.
294          However the virus journey, from the bloodstream to tissues through a mature endothelium, rem
295 mice with sustained elevation of MG53 in the bloodstream (tPA-MG53) have a healthier and longer life-
296 chronic osteomyelitis was passed through the bloodstream using a bacteriemia mouse model and derivati
297              Median time to clearance of the bloodstream was comparable between groups.
298 of key virulence factors for survival in the bloodstream was investigated.
299 ntially deliver the drugs efficiently to the bloodstream, where the microfilariae reside, while also
300 able therapeutic daptomycin treatment in the bloodstream, while preventing transmissible resistance e

 
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