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1 surement of serum and urine telopeptides and bone-specific alkaline phosphatase.
2 markers of skeletal turnover osteocalcin and bone-specific alkaline phosphatase.
3 entrations of intact parathyroid hormone and bone-specific alkaline phosphatase also varied in a sinu
4 Biochemical markers of bone turnover such as bone-specific alkaline phosphatase and N-terminal telope
5                                        Serum bone-specific alkaline phosphatase and osteocalcin conce
6                                        Serum bone-specific alkaline phosphatase and osteocalcin decre
7 ts of biochemical markers of bone formation (bone-specific alkaline phosphatase and osteocalcin) and
8 ing markers, including serum osteocalcin and bone-specific alkaline phosphatase and urine N-telopepti
9 ing protein III, and bone formation markers, bone-specific alkaline phosphatase, and osteocalcin.
10 -23), intact parathyroid hormone (iPTH), and bone-specific alkaline phosphatase (B-SAP).
11 eukin (IL)-1beta, osteoprotegerin (OPG), and bone-specific alkaline phosphatase (BALP) serum levels w
12     Leukogram, liver and kidney enzymes, and bone-specific alkaline phosphatase (BALP) serum levels w
13     Blood samples were collected to evaluate bone-specific alkaline phosphatase (BALP), leukogram, an
14 treatment, urinary N:-telopeptides and serum bone-specific alkaline phosphatase (BAP) were measured.
15 elopeptide type 1 collagen, osteocalcin, and bone-specific alkaline phosphatase (BAP), infant plasma
16 vitamin D, parathyroid hormone, osteocalcin, bone-specific alkaline phosphatase (BAP), procollagen I
17 n, N:-telopeptides of type I collagen (NTx), bone-specific alkaline phosphatase (BSAP), and phylloqui
18 e of type 1 collagen, and 28.1% (P<.001) for bone-specific alkaline phosphatase, but after 5 years wi
19 %ucOC was positively associated with NTx and bone-specific alkaline phosphatase concentrations (P < 0
20  upfront group, mean serum N-telopeptide and bone-specific alkaline phosphatase concentrations decrea
21 T resulted in significant decreases in serum bone-specific alkaline phosphatase levels (mean change,
22                        Serum osteocalcin and bone specific alkaline phosphatase, markers of skeletal
23  affecting parathyroid hormone, osteocalcin, bone-specific alkaline phosphatase, or tartrate-resistan
24 %-15%), 18% (13%-27%), and 14% (10%-21%) for bone-specific alkaline phosphatase, osteocalcin, and uri
25 henytoin had significantly greater levels of bone-specific alkaline phosphatase (p = 0.007).
26 (2); P=0.30) and bone biomarker measurements-bone-specific alkaline phosphatase (soy-fed: 82.3 +/- 4.
27 itive for osteocalcin and cells positive for bone-specific alkaline phosphatase were detected in the
28 1 collagen (NTx) breakdown, osteocalcin, and bone-specific alkaline phosphatase were measured to refl
29 m markers of bone formation (osteocalcin and bone-specific alkaline phosphatase) were lower during re
30 ventions; however, a significant increase in bone-specific alkaline phosphatase, which is a bone-form