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1 osteomyelitis, septic arthritis, or epidural/brain abscess).
2 remature infant with sepsis, meningitis, and brain abscess.
3         Epilepsy is a common complication of brain abscess.
4 y bacteria are the most frequent etiology of brain abscess.
5 ks of mortality and new-onset epilepsy after brain abscess.
6 ecommendations for neurosurgical drainage of brain abscess.
7 e part of the anaerobic community that cause brain abscess.
8 crophorum who developed a right frontal lobe brain abscess.
9 agent of CNS parenchymal infections, such as brain abscess.
10 ested case-control study of risk factors for brain abscess.
11 tion between innate and adaptive immunity in brain abscess.
12 was unsuccessfully treated for a C. bantiana brain abscess.
13 ks of mortality and new-onset epilepsy after brain abscess.
14 plantation, and who later developed multiple brain abscesses.
15 on of infected teeth, and community-acquired brain abscesses.
16 ontogenic bacteremia, and community-acquired brain abscesses.
17 ormed as the method of choice for confirming brain abscesses.
18 n reveal inflammatory changes and developing brain abscesses.
19 ight frontal lobe, which could correspond to brain abscesses.
20  a prospective nationwide study on bacterial brain abscesses.
21 e establishment of spontaneous polymicrobial brain abscesses.
22 cerebrally infected with S. aureus to induce brain abscesses.
23 tis, 60 (4%) had hemorrhages, and 2 (1%) had brain abscesses.
24 munity-acquired pneumonia (CAP) and multiple brain abscesses.
25 uable method to identify bacterial agents of brain abscesses.
26 -resistant mold, was cultured from bilateral brain abscesses.
27 ich often progresses to establish multifocal brain abscesses.
28 delta T cells were also a source of IL-17 in brain abscesses.
29 c central nervous system infection including brain abscesses.
30 filled macrophages within the ventricles and brain abscesses.
31  confirmed CNS melioidosis; 20 (38.5%) had a brain abscess, 18 (34.6%) had encephalomyelitis, 4 (7.7%
32 ; 47%); brain imaging findings were multiple brain abscesses (19/30; 63%).
33 sions, malignancy, or non-Aspergillus fungal brain abscesses accounted for all such lesions.
34 was observed in 21 of 362 (6%) patients with brain abscess and 179 of 3257 (5%) population controls (
35      We included 435 patients diagnosed with brain abscess and 3909 individuals in the comparison coh
36                             Focal cerebritis/brain abscess and corpora amylacea may also establish ch
37  medications between patients diagnosed with brain abscess and individuals from the general populatio
38 nimals consistently demonstrated more severe brain abscesses and higher CNS bacterial burdens compare
39 ation, ciglitazone may effectively sequester brain abscesses and limit bacterial dissemination.
40 g to the anaerobic community responsible for brain abscess, and M. oralis may participate in the path
41                                              Brain abscesses are frequently caused by oral cavity bac
42                                              Brain abscesses are very rarely diagnosed in neonates, b
43                                              Brain abscesses arise following parenchymal infection wi
44 d a well-characterized model of experimental brain abscess as a tool to query effects of the CNS infl
45 of polymicrobial infections, using bacterial brain abscesses as an example.
46 e describe the recovery of M. hominis from a brain abscess associated with a postpartum infection.
47 rred in a left lobe recipient, from a fungal brain abscess at 90 days.
48                                Evaluation of brain abscesses by immunohistochemistry and flow cytomet
49                                 In neonates, brain abscesses can be detected on transfontanelle ultra
50 sting that a population of cells forming the brain abscess capsule originate from a bone marrow precu
51 s and invasive dental procedures and risk of brain abscess caused by oral cavity bacteria from 1989 t
52 ts were not associated with culture-verified brain abscess caused by oral cavity bacteria.
53  identified 362 patients with culture-proven brain abscess caused by oral cavity bacteria.
54   The case of a patient who presented with a brain abscess caused by Streptomyces infection following
55 rt the case of a previously healthy boy with brain abscesses caused by M/emm type 12 GAS and review t
56        Murine Staphylococcus aureus-mediated brain abscess comprises 2 major phases, an initial phase
57 sion of select inflammatory mediators during brain abscess development including inducible NO synthas
58                                              Brain abscess development is orchestrated by IL-12 at di
59 d for its ability to influence the course of brain abscess development when treatment was initiated 3
60 majority of the cellular infiltrate in early brain abscess development, subsequent analysis focused o
61 he contribution of virulence determinants in brain abscess development, the abilities of S. aureus st
62 CNS host response during the early stages of brain abscess development, whereas MyD88-independent pat
63 of acquiring fibrotic characteristics during brain abscess development.
64 l for limiting pathogen dissemination during brain abscess development.
65 -toxin is a central virulence determinant in brain abscess development.
66                                      Date of brain abscess diagnosis was considered the index date.
67 comprising all adults (aged >=18 years) with brain abscess due to oral cavity bacteria in Denmark fro
68                                              Brain abscess due to oral cavity bacteria often occurred
69       We report the first documented case of brain abscess due to the dematiaceous fungus Microascus
70 ly restricted, since all previous reports of brain abscesses due to this organism have been for patie
71 is the extremely high propensity to initiate brain abscesses during neonatal meningitis.
72   Unexpectedly, ciglitazone also accelerated brain abscess encapsulation, which was typified by the h
73                                              Brain abscesses form in response to a parenchymal infect
74 e of central nervous system involvement with brain abscess formation in a patient with chronic granul
75 ammatory cytokine or chemokine expression or brain abscess formation in vivo.
76 continued innate responses during late-stage brain abscess formation is not known but is important, b
77 re unique in their frequent association with brain abscess formation.
78  and elicits chronic infection, resulting in brain abscess formation.
79 ic resonance imaging technology to visualize brain abscess formation.
80 iminated 2 distinct bacterial populations in brain abscess from dental and sinusal origin.
81  hundred thirteen of 362 (59%) patients with brain abscess had visited their dentist within 1 year be
82 nervous system bacterial infections, such as brain abscess, has not yet been investigated.
83 t is unknown whether patients diagnosed with brain abscess have an increased risk of psychiatric diso
84 ion, the influx of fibrocyte-like cells into brain abscesses immediately preceded the onset of fibrot
85 dy of all adults (>=18 years) diagnosed with brain abscess in Denmark from 2007 until 2023.
86 es, we examined all patients with first-time brain abscess in Denmark, 1982-2016.
87 thogenesis of S. aureus-induced experimental brain abscess in TLR2 knockout (KO) and wild-type (WT) m
88 reus is one of the major etiologic agents of brain abscesses in humans, occasionally leading to focal
89  aureus, one of the main etiologic agents of brain abscesses in humans.
90                           Empiric therapy of brain abscesses in liver transplant recipients should in
91  showed that C. koseri causes meningitis and brain abscesses in the neonatal rat model, and we utiliz
92 Streptococcus intermedius, a common cause of brain abscesses, in both CSF samples as well as in the f
93 ld in 253 of 280 (90%), and risk factors for brain abscess included immunocompromise in 95 of 287 (33
94 cribe a case of polymicrobial infection in a brain abscess including two rapidly growing Mycobacteriu
95                                              Brain abscess is associated with an increased long-term
96                Knowledge on risk factors for brain abscess is limited and relies on single-center coh
97 s of Citrobacter spp. causing meningitis and brain abscess is not well characterized; however, as wit
98              The bacterial flora involved in brain abscess is often complex.
99                M. cinereus was isolated from brain abscess material from a bone marrow transplant rec
100 enesis of C. freundii causing meningitis and brain abscess may relate to invasion of and intracellula
101 omised persons.Among the 30-day survivors of brain abscess (median follow up 7.6 years [IQR 2.2-15.5]
102 omised persons.Among the 30-day survivors of brain abscess (median follow-up 7.6 years [IQR 2.2-15.5]
103                    We have developed a mouse brain abscess model by using Staphylococcus aureus, one
104  and chemokine expression in an experimental brain abscess model in the rat during the acute stage of
105 e modulatory effects in a mouse experimental brain abscess model, we found that minocycline significa
106 sis of S. aureus in the brain using a murine brain abscess model.
107 ed pneumonitis (n = 7), myocarditis (n = 5), brain abscesses (n = 5), chorioretinitis (n = 3), lymph
108 ubstantial contributors to the occurrence of brain abscess neurosurgery (12%); solid cancer (11%); ea
109 oprotein receptor-1 (LOX-1) was increased in brain abscesses of both TLR2 KO and WT mice compared to
110 n neutrophil and macrophage recruitment into brain abscesses of MyD88 KO animals.
111 crophages, and neutrophils isolated from the brain abscesses of MyD88 KO mice produced significantly
112                  We previously reported that brain abscesses of TLR2 knockout (KO) mice exhibited ele
113 d CD8(+) T cell infiltrates were elevated in brain abscesses of TLR2 KO mice at days 3, 7, and 14 pos
114 y protein-2, was significantly attenuated in brain abscesses of TLR2 KO mice compared to WT mice duri
115                  However, LOX-1 induction in brain abscesses of TLR2 KO mice was significantly attenu
116 unctions in a compensatory manner to control brain abscess pathogenesis, with cells other than glia a
117 unctional significance of these mediators in brain abscess pathogenesis.
118                           We identified 1384 brain abscess patients (37% females) with a median follo
119                          We identified 1,384 brain abscess patients (37% females) with a median follo
120                           We identified 1384 brain abscess patients in Denmark from 1982 through 2016
121                             The mortality in brain abscess patients was significantly increased regar
122                             The mortality in brain abscess patients was significantly increased regar
123 e response leading to the establishment of a brain abscess remains poorly defined.
124                                              Brain abscess represents the infectious disease sequelae
125                                              Brain abscesses result from a pyogenic parenchymal infec
126 sed to detect specific archaeal sequences in brain abscess samples and controls.
127 ielded archaea, mostly methanogens, in 28/32 brain abscess samples, and no archaea in 71 negative con
128                   Despite these differences, brain abscess severity in TLR2 KO and WT animals was sim
129                      Antibiotic treatment of brain abscess should contain anti-archaeal compounds suc
130 ch correlated with a significant decrease in brain abscess size.
131 rchaea-specific qPCR yielded archaea in 8/18 brain abscess specimens and 1/27 controls (P < .003), an
132                      All bacteria present in brain abscess specimens were identified, in view of the
133 ied 44 bacteria that had never been found in brain abscess specimens, including 22 uncultured bacteri
134 medications (ASMs) in preventing epilepsy in brain abscess survivors is unknown.
135                      In this cohort study of brain abscess survivors, initiation of ASMs was not asso
136 ncertainty about the role of neurosurgery in brain abscess treatment is reflected in surveys and obse
137  of tissue injury, with significantly larger brain abscesses typified by exaggerated edema and necros
138  was restricted to those with a diagnosis of brain abscess using International Statistical Classifica
139 r, and 6-30 year mortality of patients after brain abscess was 21%, 16% and 27% as compared to 1%, 6%
140 r, and 6-30 year mortality of patients after brain abscess was 21%, 16%, and 27% as compared to 1%, 6
141 the importance of MyD88-dependent signals in brain abscesses, we compared disease pathogenesis using
142 vents related to procedures, endocarditis or brain abscess were excluded.
143                      Patients diagnosed with brain abscess were more likely to suffer from comorbidit
144                                          All brain abscesses were fungal; 73% (8/11) of these patient
145                       To address this issue, brain abscesses were induced in TCR alphabeta knockout (
146                         Cells recovered from brain abscesses were shown to harbor S. aureus intracell
147 al hematoma, acute infarcts, and Aspergillus brain abscesses were the predominant etiologies during t
148 ts aged 18 years or older at the time of the brain abscess with at least 1 year of prior enrollment w
149                                Patients with brain abscess without prior psychiatric disorders or rec

 
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