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1 phosphate) on binding of ANP, BNP (atrial or brain natriuretic peptide).
2 ality-of-life, and NT-proBNP (N-terminal pro-brain natriuretic peptide).
3 nction, and elevated baseline N-terminal pro-brain natriuretic peptide.
4 ponin I, creatine kinase, and N-terminal pro-brain natriuretic peptide.
5 sed plasma norepinephrine and n-terminal pro-brain natriuretic peptide.
6 tudinal strain, and N-terminal prohormone of brain natriuretic peptide.
7  adjustment for traditional risk factors and brain natriuretic peptide.
8 several biomarkers, including N-terminal pro-brain natriuretic peptide.
9 aseline levels of hs-cTnT and N-terminal pro-brain natriuretic peptide.
10 e prognostication in conjunction with plasma brain natriuretic peptide.
11 vated high-sensitivity C-reactive protein or brain natriuretic peptide.
12 kely to have elevated C-reactive protein and brain natriuretic peptide.
13  receptor for atrial natriuretic peptide and brain natriuretic peptide.
14 osin heavy chains, skeletal alpha-actin, and brain natriuretic peptide.
15 ch immunoassay protocol for the detection of brain natriuretic peptide.
16 l class, and higher levels of N-terminal pro-brain natriuretic peptide.
17  brain natriuretic peptide or N-terminal pro-brain natriuretic peptide.
18 e, invasive hemodynamics, and N-terminal pro-brain natriuretic peptide.
19 c structure and function, and N-terminal pro brain natriuretic peptide.
20  with lower circulating levels of atrial and brain natriuretic peptides.
21  class, medical therapy escalation, and BNP [brain natriuretic peptide]).
22  pmol/L, P=0.041), and had a trend to higher brain natriuretic peptide (12.3 versus 7.2 pmol/L, P=0.0
23 rathyroid hormone, prolactin, N-terminal pro-brain natriuretic peptide, 25-hydroxyvitamin D) and 2 no
24 3 +/- 0.006 mug/L, P = 0.003; N-terminal pro-brain natriuretic peptide, 38.1 +/- 37.7 vs. 5.9 +/- 12.
25 nt protein 1 (117 pg/mL; 119%; P=0.004), and brain natriuretic peptide (40 pg/mL; 57%; P=0.04).
26 tors of reduced survival were elevated serum brain natriuretic peptide (482 +/- 337 pg/mL) and uric a
27                            Induction of both brain natriuretic peptide (5.8-fold versus control, P:<0
28 ansformed BNP and LV-GLS were 4.04 (absolute brain natriuretic peptide: 60 pg/dL) and -20.7%.
29 racoronary group, P=0.03) and N-terminal pro-brain natriuretic peptide (-628 +/- 211 versus -315 +/-
30  131 m; P = 0.001), and serum N-terminal pro-brain natriuretic peptide (647 +/- 1,127 pg/ml vs. 1,578
31  (mean, 66 years), gender (74% male), plasma brain natriuretic peptide (79 pg/mL), left ventricular e
32 iblings, had higher levels of N-terminal pro-brain natriuretic peptide (81.7 and 69.0 pg/mL, respecti
33  and the NT-proBNP (N-terminal prohormone of brain natriuretic peptide)-a biomarker that is used for
34                  Cardiac hormones atrial and brain natriuretic peptides activate guanylyl cyclase/nat
35 l prognostic information to that provided by brain natriuretic peptide alone.
36  of patients misclassified by N-terminal pro-brain natriuretic peptide and 59% (95% confidence interv
37                           Patients with high brain natriuretic peptide and aminoterminal propeptide o
38 PET coupled with biologic biomarkers such as brain natriuretic peptide and antineutrophilic cytoplasm
39                                              Brain natriuretic peptide and atrial natriuretic peptide
40 ained even in the presence of N-terminal pro-brain natriuretic peptide and high-sensitivity troponin
41  was robustly associated with N-terminal pro-brain natriuretic peptide and incident HF or death.
42  parallel with improvement in N-terminal pro-brain natriuretic peptide and left atrial size suggest t
43 =1) at 1 month is predictive, in addition to brain natriuretic peptide and LV ejection fraction, of d
44 fraction who underwent mitral valve surgery, brain natriuretic peptide and LV-GLS provided synergisti
45 tively with pulmonary hemodynamics and serum brain natriuretic peptide and negatively with 6-minute w
46 ly specific, PlGF, as well as N-terminal pro-brain natriuretic peptide and soluble vascular cell adhe
47 atrial size) with concomitant N-terminal pro-brain natriuretic peptide and subsequent HF hospitalizat
48  as significant-but-subclinical increases in brain natriuretic peptide and troponin I.
49 , echocardiography, assays of N-terminal pro-brain natriuretic peptide and Troponin T, and functional
50                                     Elevated brain natriuretic peptide and uric acid, decreased left
51         Significant falls occurred in plasma brain natriuretic peptide and urinary metadrenaline excr
52 -regulation of atrial natriuretic factor and brain natriuretic peptide and with increased expression
53 ctional capacity, biomarkers (N-terminal pro-brain natriuretic peptide and/or high-sensitivity tropon
54 nsumption), biomarker (N-terminal prohormone brain natriuretic peptide), and left ventricular functio
55 ongitudinal strain, N-terminal prohormone of brain natriuretic peptide, and adverse cardiac outcomes
56       These findings were all independent of brain natriuretic peptide, and assessment of NRG-1beta a
57 ded traditional risk factors, N-terminal pro-brain natriuretic peptide, and baseline hs-cTnT level.
58 hic marker genes, atrial natriuretic factor, brain natriuretic peptide, and beta-myosin heavy chain.
59 ), 6-minute walk distance, cardiac function, brain natriuretic peptide, and Borg dyspnea index.
60 llment, randomized treatment, N-terminal pro-brain natriuretic peptide, and clinical risk factors.
61 ment in ventricular function, N-terminal pro-brain natriuretic peptide, and exercise capacity compare
62 ge were measured (troponin T, N-terminal pro-brain natriuretic peptide, and heart-type fatty acid bin
63 es and ejection fraction with N-terminal pro-brain natriuretic peptide, and high-sensitive troponin w
64 rinogen, D-dimer, troponin T, N-terminal pro-brain natriuretic peptide, and high-sensitivity C-reacti
65 tus, cardiac status including measurement of brain natriuretic peptide, and leukocyte antibody testin
66 Embolism Severity Index, cardiac troponin I, brain natriuretic peptide, and lower limb ultrasound tes
67 ease, chronic kidney disease, N-terminal-pro brain natriuretic peptide, and lymphocyte GRK2 protein l
68 , previous cardiopulmonary disease, D-dimer, brain natriuretic peptide, and percentage of pulmonary v
69 aphy, echocardiography, serum N-terminal pro-brain natriuretic peptide, and pulmonary function tests
70 ler-derived E/e' at echocardiography, plasma brain natriuretic peptide, and quality-of-life scores.
71 eased expression of beta-myosin heavy chain, brain natriuretic peptide, and skeletal actin and a decr
72 functional class, serum levels of N-terminal brain natriuretic peptide, and time to clinical worsenin
73  concentrations of vasopressin, aldosterone, brain natriuretic peptide, and total renin were measured
74                  Although neither atrial nor brain natriuretic peptide (ANP, BNP) is amidated, the ma
75                                   Atrial and brain natriuretic peptides (ANP and BNP, respectively) a
76 of beta-myosin heavy chain (betaMHC), atrial/brain natriuretic peptides (ANP/BNP), and skeletal alpha
77 gh-sensitivity troponin I and N-terminal pro-brain natriuretic peptide are also prognostic in patient
78                                   Atrial and brain natriuretic peptides are cleaved into smaller biol
79                    First, data on the use of brain natriuretic peptide as a screening biomarker has s
80                                   The use of brain natriuretic peptide as a screening tool for asympt
81 , 6-minute walk distance, and N-terminal pro-brain natriuretic peptide as secondary endpoints.
82                                              Brain natriuretic peptide as well as biomarkers of myoca
83 onsistent effect on N-terminal prohormone of brain natriuretic peptide at 18 months was observed in t
84   Patisiran lowered N-terminal prohormone of brain natriuretic peptide at 9 and 18 months (at 18 mont
85 ) damage and of decongestion (N-terminal pro-brain natriuretic peptide) at day 2 and worsening heart
86 ptide levels, and cardiac mRNA expression of brain natriuretic peptide, atrial natriuretic factor, an
87       Multivariable regression revealed that brain natriuretic peptide (beta=-0.250; P<0.001) and rec
88 phy gene markers atrial natriuretic peptide, brain natriuretic peptide, beta-myosin heavy chain and m
89                               N-terminal pro-brain natriuretic peptide blood levels decreased by 620
90 ort-term administration of subcutaneous (SQ) brain natriuretic peptide (BNP) administration during th
91                                   We sampled brain natriuretic peptide (BNP) and amino-terminal (NT)-
92                                     Baseline brain natriuretic peptide (BNP) and C-reactive protein (
93 sed less atrial natriuretic factor (ANF) and brain natriuretic peptide (BNP) and more glucose transpo
94                                              Brain natriuretic peptide (BNP) and N-terminal pro-brain
95                                              Brain natriuretic peptide (BNP) and N-terminal proBNP (N
96               We examined whether changes in brain natriuretic peptide (BNP) and norepinephrine (NE)
97         Atrial natriuretic peptide (ANP) and brain natriuretic peptide (BNP) are sensitive biomarkers
98 orts have suggested the usefulness of plasma brain natriuretic peptide (BNP) as a screening test for
99 mited data on the prognostic implications of brain natriuretic peptide (BNP) assessment in patients w
100 actor of activated T cells -c3 (NFATc3), and brain natriuretic peptide (BNP) compared to controls.
101 re, assessed by history of heart failure and brain natriuretic peptide (BNP) concentration at baselin
102 mine the effects of age and gender on plasma brain natriuretic peptide (BNP) concentration in a popul
103                                              Brain natriuretic peptide (BNP) gene expression is a wel
104 xpression profile analysis revealed that the brain natriuretic peptide (BNP) gene was significantly u
105                                              Brain natriuretic peptide (BNP) has an established role
106                         The renal actions of brain natriuretic peptide (BNP) in congestive heart fail
107  chronic subcutaneous (SQ) administration of brain natriuretic peptide (BNP) in experimental CHF impr
108                                              Brain natriuretic peptide (BNP) is a cardiac peptide wit
109                                              Brain natriuretic peptide (BNP) is a marker of diagnosis
110                             It is unknown if brain natriuretic peptide (BNP) is synthesized by CFs an
111 tional class (HR 1.50; 95% CI 1.02-2.2), log brain natriuretic peptide (BNP) levels (HR 1.45; 95% CI
112                                              Brain natriuretic peptide (BNP) levels are used in the e
113 ctive study to evaluate whether preoperative brain natriuretic peptide (BNP) levels predict postopera
114 o cardiac volume loading and increased serum brain natriuretic peptide (BNP) levels.
115     We sought to determine the usefulness of brain natriuretic peptide (BNP) measurements to differen
116                    We postulated that plasma brain natriuretic peptide (BNP) might serve as a biomark
117  not innervated by pruritoceptors expressing brain natriuretic peptide (BNP) most of them contained m
118  vs. 443 ms; P = 0.05), an elevated pre-TIPS brain natriuretic peptide (BNP) or N-terminal pro-brain
119 a, congestion on chest radiograph, increased brain natriuretic peptide (BNP) or N-terminal prohormone
120 ongestion on chest radiograph, and increased brain natriuretic peptide (BNP) or N-terminal prohormone
121                                              Brain natriuretic peptide (BNP) serum concentration has
122  sought to derive a new staging system using brain natriuretic peptide (BNP) that would correlate wit
123 time of admission the plasma level of plasma brain natriuretic peptide (BNP) to determine its utility
124                                              Brain natriuretic peptide (BNP) treatment increases hear
125 crosis factor-alpha receptor-1 (sTNFR-1) and brain natriuretic peptide (BNP) were highly predictive o
126 cid for atrial natriuretic peptide (ANP) and brain natriuretic peptide (BNP) were increased 7- and 3-
127  the sodium calcium exchanger (Ncx1) and the brain natriuretic peptide (Bnp) whose hypertrophic upreg
128 nstant of the interaction of a 32-amino acid brain natriuretic peptide (BNP) with a mAbs and demonstr
129 agenesis to characterize the interactions of brain natriuretic peptide (BNP) with two monoclonal anti
130 tified angiotensin II as a potent inducer of brain natriuretic peptide (BNP), a hormone that recently
131 diac hormones atrial natriuretic peptide and brain natriuretic peptide (BNP), but little is known abo
132 rations of atrial natriuretic peptide (ANP), brain natriuretic peptide (BNP), endothelin-1 (ET-1), re
133 rial natriuretic peptide (ANP) and B-type or brain natriuretic peptide (BNP), in the general communit
134 cluding atrial natriuretic peptide (ANP) and brain natriuretic peptide (BNP), is a hallmark for malad
135 r levels of atrial natriuretic factor (ANF), brain natriuretic peptide (BNP), the RNA helicase Csm, a
136 xpression for the natriuretic peptides [ANP, brain natriuretic peptide (BNP), type-C natriuretic pept
137 te (cGMP), nitrosative/oxidative stress, and brain natriuretic peptide (BNP).
138 nction, exercise capacity, heart rhythm, and brain natriuretic peptide (BNP).
139 ctivity (44+/-15%, P<0.05) in processing pro-brain natriuretic peptide (BNP).
140 h atrial natriuretic peptide (ANP), and with brain natriuretic peptide (BNP).
141 transcription factor activates expression of brain natriuretic peptide (BNP).
142 al proatrial natriuretic peptide (N-ANP) and brain natriuretic peptide (BNP).
143        Monoclonal antibody (mAb) 106.3 binds brain natriuretic peptide (BNP)5-13(C10A) and full-lengt
144 omodulin (sTM)]) and raised plasma levels of brain natriuretic peptide (BNP, a peptide hormone associ
145  40% or less, elevated natriuretic peptides (brain natriuretic peptide [BNP] >/= 400 pg/mL or N -term
146  the fiber direction increased expression of brain natriuretic peptide, but off-axis stretches (causi
147                                              Brain natriuretic peptide can be useful in distinguishin
148 and blood samples were evaluated for cardiac brain natriuretic peptide, choline, and TMAO levels.
149                  Independent predictors were brain natriuretic peptide concentration > or =300 pg/ml
150                                              Brain natriuretic peptide concentration ranged from <4 t
151 Of those without LVSD, plasma N-terminal pro-brain natriuretic peptide concentration was greater than
152 t-for-age z score, Ross heart failure class, brain natriuretic peptide concentration, Bayley scores o
153 r volume, including central venous pressure, brain-natriuretic-peptide concentration, and fractional
154 alked in 6 min, and N-terminal prohormone of brain natriuretic peptide concentrations between the 2 g
155 lecules, and plasma N-terminal prohormone of brain natriuretic peptide concentrations were also measu
156 ss, Minnesota quality-of-life questionnaire, brain natriuretic peptide concentrations, and 6-minute w
157 and baseline and increases in N-terminal pro-brain natriuretic peptide concentrations.
158  anti-mitogenic activities of the atrial and brain natriuretic peptides depend upon their binding to
159  or persistently elevated amino terminal pro-brain natriuretic peptide despite treatment for heart fa
160 ome clinical information (eg, N terminal pro-brain natriuretic peptide, distinctive chest x-ray findi
161 ependent after adjustment for N-terminal pro-brain natriuretic peptide, ejection fraction, stroke vol
162 Perfusion of isolated guinea pig hearts with brain natriuretic peptide elicited a 3-fold increase in
163 tivity cardiac troponin T and N-terminal pro-brain natriuretic peptide) emerged as the most powerful
164 vity, right ventricular function, as well as brain natriuretic peptide, endothelin-1, uric acid, and
165                                Only 4 drugs (brain natriuretic peptide, exenatide, metoprolol, and es
166 dent protein kinase IIdelta phosphorylation, brain natriuretic peptide expression, and sustained capi
167 r activity and suppressed ET-induced ANP and brain natriuretic peptide gene expression.
168 6 [1.02-3.39]; P=0.043) 1 month after MI and brain natriuretic peptide &gt;100 pg/mL (2.35 [1.28-4.31];
169                     N-terminal prohormone of brain natriuretic peptide &gt;8500 ng/L could not be overco
170 re and for improved clinical outcomes with a brain natriuretic peptide-guided approach to heart failu
171                                Additionally, brain natriuretic peptide has garnered significant inter
172 ntricular ejection fraction, creatinine, and brain natriuretic peptide (hazard ratio, 0.58 [95% CI, 0
173 2.133; p = 0.040), as well as N-terminal pro-brain natriuretic peptide (hazard ratio: 1.655; p < 0.00
174 ogical activity of orally administered human brain natriuretic peptide (hBNP).
175 ogate markers were evaluated: N-terminal-pro brain natriuretic peptide, high-sensitivity C-reactive p
176                  Inclusion of N-terminal pro-brain natriuretic peptide improved the overall classific
177                          However, studies of brain natriuretic peptide in patients with heart failure
178            Strong evidence exists for use of brain natriuretic peptide in the diagnosis of acute hear
179                    In HF-CON, n-terminal pro-brain natriuretic peptide increased by 88 +/- 120 pg/mL
180                     Importantly, proBNP (pro-brain natriuretic peptide) increased at 12 months in 20
181 nt and dysfunction and higher N-terminal pro brain natriuretic peptide independent of RA size and pre
182                                              Brain natriuretic peptide is the most widely studied, wi
183                  Nesiritide (synthetic human brain natriuretic peptide) is approved for the treatment
184 tic peptide, and assessment of NRG-1beta and brain natriuretic peptide jointly provided better risk s
185 on problem list, inpatient loop diuretic, or brain natriuretic peptide level of 500 pg/mL or higher;
186 ; mean EF was 56%; and median N-terminal pro-brain natriuretic peptide level was 1403 pg/mL; 761 (96.
187 f patients were women, median N-terminal pro-brain natriuretic peptide level was 918 pg/ml (interquar
188 logy and to the subgroup with baseline serum brain natriuretic peptide levels >/=100 pg/mL.
189 [CI], -2, -16; n = 13, p = 0.015) and plasma brain natriuretic peptide levels (-19.4 fmol x ml(-1); 9
190                 Secondary endpoints included brain natriuretic peptide levels and catecholamine excre
191 ment was also associated with a reduction of brain natriuretic peptide levels and no increase in the
192 igh-sensitivity troponin, and N-terminal pro-brain natriuretic peptide levels are predictive of death
193                                              Brain natriuretic peptide levels correlated moderately w
194                        N-terminal prohormone brain natriuretic peptide levels did not increase signif
195 eased lung/body weight ratios, and increased brain natriuretic peptide levels in cMyBP-C(t3SA) mice d
196                                              Brain natriuretic peptide levels on admission were lower
197                                At follow-up, brain natriuretic peptide levels remained stable in grou
198 r reductions from baseline in N-terminal pro-brain natriuretic peptide levels than did the pooled-mon
199 nd control groups; only serum creatinine and brain natriuretic peptide levels varied in laboratory ev
200 ysaccharide binding protein, troponin T, and brain natriuretic peptide levels were measured.
201 put, decreased wedge pressure, and decreased brain natriuretic peptide levels were observed in animal
202           New diagnostic tests (troponin and brain natriuretic peptide levels) are being used to aid
203                                       Plasma brain natriuretic peptide levels, and cardiac mRNA expre
204 um oxygen consumption, N-terminal prohormone brain natriuretic peptide levels, and left ventricular e
205 tion, plasma norepinephrine, angiotensin II, brain natriuretic peptide levels, and medications.
206 rioperative changes in left atrial pressure, brain natriuretic peptide levels, lactic acid, or renal
207 , quality of life, incidence of arrhythmias, brain natriuretic peptide levels, left ventricular eject
208 is characterized by increased N-terminal pro-brain natriuretic peptide levels, troponin release, elev
209 ricular ejection fraction, or N-terminal pro-brain natriuretic peptide levels.
210 truction, such as exercise testing and serum brain natriuretic peptide levels.
211 as assessed by measurement of N-terminal pro-brain natriuretic peptide levels; the rate of recurrent
212                    NT-proBNP (N-terminal pro brain natriuretic peptide) levels are included in the mu
213  of diabetes mellitus, higher N-terminal pro-brain natriuretic peptide, lower estimated glomerular fi
214  homocysteine <10 micromol/L, N-terminal pro-brain natriuretic peptide &lt;100 pg/mL, no microalbuminuri
215              The inclusion of N-terminal pro-brain natriuretic peptide markedly improves HF risk pred
216 y echocardiography and plasma N-terminal pro-brain natriuretic peptide measurement and were followed
217 iopulmonary exercise testing, N-terminal pro-brain natriuretic peptide measurement, cardiac magnetic
218 alizations) hospitalizations in which either brain natriuretic peptide measures or ejection fraction
219 to compare urinary and plasma N-terminal pro-brain natriuretic peptide (N-BNP) in left ventricular sy
220 on) in comparison with plasma N-terminal pro-brain natriuretic peptide (N-BNP) levels.
221 erived cardiomyocytes and CFs via the atrial/brain natriuretic peptide-natriuretic peptide receptor-1
222 inical heart failure trials with recombinant brain natriuretic peptide (nesiritide) failed to prove i
223 diography, an increased serum N-terminal pro-brain natriuretic peptide (NT-pro-BNP) level, and pulmon
224 he predictive capabilities of N-terminal pro-brain natriuretic peptide (NT-proBNP) and C-reactive pro
225   Current staging systems use N-terminal pro-brain natriuretic peptide (NT-proBNP) and cardiac tropon
226                               N-terminal pro-brain natriuretic peptide (NT-proBNP) and high-sensitivi
227 ded whether the N-terminal of the prohormone brain natriuretic peptide (NT-proBNP) and high-sensitivi
228    The Hokusai-VTE study used N-terminal pro-brain natriuretic peptide (NT-proBNP) and right to left
229 ating plasma levels of N-terminal prohormone brain natriuretic peptide (NT-proBNP) and soluble vascul
230            Combining abnormal N-terminal pro-brain natriuretic peptide (NT-proBNP) and troponin T wit
231            Elevated levels of N-terminal pro-brain natriuretic peptide (NT-proBNP) are associated wit
232 troponins (cTnT and cTnI) and N-terminal pro-brain natriuretic peptide (NT-proBNP) are prognostic in
233 natriuretic peptide (BNP) and N-terminal pro-brain natriuretic peptide (NT-proBNP) are well-establish
234 distance (6MWD) and change in N-terminal pro-brain natriuretic peptide (NT-proBNP) by after therapy.
235 dary efficacy measure was the N-terminal pro-brain natriuretic peptide (NT-proBNP) concentration afte
236 ized as biosensors to measure N-terminal pro-brain natriuretic peptide (NT-proBNP) in human serum wit
237               The ventricular N-terminal pro-brain natriuretic peptide (NT-proBNP) is a marker of CHF
238     Others have reported that N-terminal pro-brain natriuretic peptide (NT-proBNP) is a valuable prog
239  natriuretic peptide (BNP) or N-terminal pro-brain natriuretic peptide (NT-proBNP) level, an elevated
240 ulmonary vascular resistance, N-terminal pro-brain natriuretic peptide (NT-proBNP) level, World Healt
241 n renal insufficiency and amino-terminal pro-brain natriuretic peptide (NT-proBNP) levels remains unc
242 ery in patients with elevated N-terminal pro-brain natriuretic peptide (NT-proBNP) levels, reduces th
243 ulmonary vascular resistance, N-terminal pro-brain natriuretic peptide (NT-proBNP) levels, World Heal
244 etween renal function and amino-terminal pro-brain natriuretic peptide (NT-proBNP) levels.
245  but is associated with lower N-terminal pro-brain natriuretic peptide (NT-proBNP) levels.
246 ured troponin T and N-terminal prohormone of brain natriuretic peptide (NT-proBNP) on postoperative d
247                     Levels of N-terminal pro-brain natriuretic peptide (NT-proBNP) provide such infor
248                 The biomarker N-terminal pro-brain natriuretic peptide (NT-proBNP) was assessed.
249                                   N-terminal brain natriuretic peptide (NT-proBNP) was significantly
250 /ml; p < 0.001), although amino-terminal pro-brain natriuretic peptide (NT-proBNP) was superior to ST
251 biomarkers (such as N-terminal prohormone of brain natriuretic peptide (NT-proBNP)) and a measure of
252 vestigated whether plasma amino-terminal pro-brain natriuretic peptide (NT-proBNP), a marker of cardi
253 sitivity troponin T (hs-TnT), N-terminal pro-brain natriuretic peptide (NT-proBNP), and growth differ
254 cardiac troponin T (cTnT) and N-terminal pro-brain natriuretic peptide (NT-proBNP).
255  walk distance, and levels of N-terminal pro-brain natriuretic peptide (NT-proBNP).
256 O functional class, N-terminal prohormone of brain natriuretic peptide (NT-proBNP)concentrations, Bor
257 s 0.37 +/- 0.04, P = .01) and N-terminal pro-brain natriuretic peptide (NT-proBNP; 191 +/- 261 vs 33
258 T; cardiac injury biomarker), N-terminal pro-brain natriuretic peptide (NT-proBNP; cardiomyopathy bio
259 ng distance [6MWD], N-terminal prohormone of brain natriuretic peptide [NT-proBNP] concentration, and
260 ] symptom classification, and N-terminal pro-brain natriuretic peptide [NT-proBNP] level) at 6 months
261 (troponins I and T, N-terminal prohormone of brain natriuretic peptide [NT-proBNP]) to predict baseli
262  C-reactive protein [hs-CRP], N-terminal pro-brain natriuretic peptide [NT-proBNP], white blood cell
263  IIIB (that is stage III with N-terminal pro-brain natriuretic peptide [NTproBNP] >8500 pg/mL) diseas
264  action of nitric oxide-CAPON signalling and brain natriuretic peptide on cGMP and cAMP regulation of
265 action, and an elevated circulating level of brain natriuretic peptide or N-terminal pro-brain natriu
266  body mass index (p = 0.002), N-terminal pro-brain natriuretic peptide (p < 0.001), early mitral infl
267 P = .703), TRV (P = .503), or N-terminal pro-brain natriuretic peptide (P = .410).
268 04), and had higher levels of N-terminal pro-brain natriuretic peptide (p = 0.001) compared with less
269 erobic exercise capacity, and N-terminal pro-brain natriuretic peptide (P>0.30 for all).
270 tly correlated with N-terminal prohormone of brain natriuretic peptide (P<0.001), 6-minute walk dista
271  (IL)-6 (P=0.019), and N-terminal prohormone brain natriuretic peptide (P=0.028) concentrations were
272 nal pediatric definition; P=0.087) and lower brain natriuretic peptide (P=0.060).
273  [P = .03]), and markers of cardiac stretch (brain natriuretic peptide [P < .001] and N-terminal frag
274 1] and N-terminal fragment of the prohormone brain natriuretic peptide [P = .001]) in HIV.
275 sure and heart rate, 6-minute walk distance, brain natriuretic peptide, percent predicted carbon mono
276 patient's age, race, sex, and N-terminal pro-brain natriuretic peptide performs comparably to the ful
277 tumor necrosis factor-alpha receptor II, pro-brain natriuretic peptide (pro-BNP), and cardiac troponi
278 e LV global longitudinal strain (LV-GLS) and brain natriuretic peptide provided incremental prognosti
279 significant correlations with N-terminal pro-brain natriuretic peptide (R=0.69, P<0.001) and Troponin
280 clinically used biomarkers of heart failure: brain natriuretic peptide (r=0.691; P=0.0003), QRS (r=0.
281 oponin T (cTnT) and N-terminal prohormone of brain natriuretic peptide) related to hemodynamic activi
282  hematologic response and the N-terminal pro-brain natriuretic peptide response.
283 rography (MRN) with hsTNT and N-terminal pro-brain natriuretic peptide serum levels in patients with
284 a ratios, fetal gene expressions (atrial and brain natriuretic peptides, skeletal actin, beta-myosin
285 lified Pulmonary Embolism Severity Index and brain natriuretic peptide testing showed a negative pred
286 ood pressure and in levels of N-terminal pro-brain natriuretic peptide than the placebo group.
287  as well as a higher level of N-terminal pro-brain natriuretic peptide, than noncarriers (1194 partic
288                On addition of N-terminal pro-brain natriuretic peptide, the optimism-corrected area u
289          miR-150 outperformed N-terminal pro-brain natriuretic peptide to predict remodeling (area un
290 left atrial volume, N-terminal propeptide of brain natriuretic peptide, total, collagen-dependent, an
291  LV ejection fraction, plasma n-terminal pro-brain natriuretic peptide, tumor necrosis factor-alpha,
292 , plasma concentration of amino terminal pro-brain natriuretic peptide (univariate and multivariable
293 n, LV volumes, scar size, and N-terminal pro-brain natriuretic peptide values comparing the 2 treatme
294 eft ventricular ejection fraction, and lower brain natriuretic peptide values on admission.
295 er adjusting for serum interleukin-6 levels, brain natriuretic peptide values, and glucocorticoid use
296 25% (SD, 8) and median plasma N-terminal pro-brain natriuretic peptide was 3,070 pg/ml (interquartile
297 y decrease and no decrease in N-terminal-pro-brain natriuretic peptide was not reached and 26 months,
298                             Median levels of brain natriuretic peptide were 550 pg/mL in patients wit
299 iotensin II, atrial natriuretic peptide, and brain natriuretic peptide were measured at baseline.
300 n, plasma norepinephrine, and n-terminal pro-brain natriuretic peptide were measured before (pre) and

 
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