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1 excessive brain tissue oxygenation, elevated brain temperature).
2 eration of normal EEG and the maintenance of brain temperature.
3 thy data we built HEATWAVE-a 4D map of human brain temperature.
4 ennes bioheat equation was used to propagate brain temperature.
5 ons and a force behind associated changes in brain temperature.
6 creased excitability consequent to increased brain temperature.
7 rain temperature since it reflects body, not brain temperature.
8 rature 37 +/- 0.5 degrees C) or hypothermia (brain temperature 32 +/- 0.5 degrees C).
9  immediately by 1 hr of either normothermia (brain temperature 37 +/- 0.5 degrees C) or hypothermia (
10 ure ranged from 36.1 to 40.9 degrees C; mean brain temperature (38.5 +/- 0.4 degrees C) exceeded oral
11  ranged from 32.6 to 42.3 degrees C and mean brain temperature (38.5 +/- 0.8 degrees C) exceeded body
12 mployed in the present study with respect to brain temperatures, a dynamic parameter that reflects me
13  undergo interventions to achieve a 'normal' brain temperature; a parameter that remains undefined fo
14 that imaging through a cranial window lowers brain temperature, an effect capable of affecting cerebr
15 hermocouples were placed to measure core and brain temperature and a composite probe placed in the pa
16 brile seizures are associated with increased brain temperature and are often resistant to treatments
17 thods were used to map pre- to post-COVID-19 brain temperature and free water changes.
18                                Post-COVID-19 brain temperature and free water increases, which are in
19       We tested this hypothesis by measuring brain temperature and lactate concentration with multi-v
20 mia, both of which precede slower changes in brain temperature and metabolic brain activity.
21 R1, FOS, and ARC, and indications of a lower brain temperature and sleep-like state.
22 l stimulation can cause local changes in the brain temperature and subsequent local changes in the ox
23  use does not lead to profound elevations in brain temperature and sustained vasoconstriction, two cr
24 pport the potential importance of monitoring brain temperature and the importance of controlling feve
25 s are remarkable for surviving near-freezing brain temperatures and near cessation of neural activity
26                   During rewarming, regional brain temperatures and neocortical thermal gradients wer
27 tions of the impact of the light-dark cycle, brain temperature, and blood flow on the function of the
28 he profound influence of sleep-wake state on brain temperature, and can be harnessed to differentiate
29 ependent from locomotor activity, changes in brain temperature, and theta rhythm.
30         Significant but smaller increases in brain temperature ( approximately 0.2 degrees C for 4-6
31                         Maximal increases in brain temperature ( approximately 0.8-1.2 degrees C for
32 e brain when experimental conditions such as brain temperature are controlled.
33 e additional support for the hypothesis that brain temperatures are elevated during winter depression
34                                During sleep, brain temperatures are in part determined by the level o
35  they are in the ICU for a week or more, and brain temperatures are likely to be as much as 2 degrees
36             We anticipate the advancement of brain temperature as a marker of health and injury will
37                  In contrast, maintenance of brain temperature at 37 degrees C resulted in a 12-to-40
38 ment between model-predicted and MR-measured brain temperatures at the voxel-level.
39 n tissue PO(2) in the thalamus (PtO(2)), and brain temperature (Bt) simultaneously during acute hyper
40 th cocaine and PRO their ability to increase brain temperature but failed to induce temperature decre
41 ethylone and MDPV dose-dependently increased brain temperature, but even at high doses that induced r
42 S-201 (1.5-2.5 mg/kg, i.p.) reduces body and brain temperature by 2-5 degrees C in 15-30 min in a dos
43        These results suggest that changes in brain temperature can alter the regulation of extracellu
44 ogical parameters, such as body temperature, brain temperature, cerebral blood flow, blood gases, blo
45 en an increase in estimated power output and brain temperature change (P = .9).
46 (POA), with ~30% of its neurons sensitive to brain temperature change.
47 hermometry to provide an estimate of in vivo brain temperature changes during MR-ARFI, and pressure a
48                   A feedback method of local brain temperature control was developed where ICSI flow
49 , real-time data acquisition, and continuous brain temperature control, in this new rat model, provid
50 e technique and laser doppler flowmetry with brain temperature controlled.
51                          When postmortem rat brain temperature cooled rapidly to near room temperatur
52 -4.2 mmHg to 14.8+/-5.2 mmHg (P=0.004) while brain temperature decreased from 36.5+0.3 degrees C to 3
53 ter immersion objective at room temperature, brain temperature decreases by ~2-3 degrees C, causing d
54 d, but that these increases are blunted when brain temperature decreases.
55                                In survivors, brain temperature (degrees C) measured at 2-cm depth in
56 th increasing time after stroke, ipsilateral brain temperature did not change, but contralateral hemi
57                                              Brain temperature drops from waking levels during non-ra
58    Adenosine treatment significantly lowered brain temperature during recovery, and a part of the neu
59       The level of core body, and presumably brain temperature during sleep varies with clinical stat
60     To explore this possibility, we measured brain temperature dynamics during a 10-min forced swim i
61                      Striking differences in brain temperature dynamics seen in the beginning of a se
62                       We conclude that early brain temperature elevation after stroke is not directly
63  studies are required to determine why early brain temperature elevation is highest in potential penu
64                                        Early brain temperature elevation may result from different me
65 jury and worsened functional outcomes if the brain temperature exceeds 39 degrees C.
66 ic brain activation as the primary source of brain temperature fluctuations and a force behind associ
67 med with the same dose/pattern as SA induced brain temperature fluctuations similar in many ways to t
68  mimicked cocaine in its ability to increase brain temperature following the initial injection and to
69                                     Although brain temperature has neurobiological and clinical impor
70 g/kg, s.c.) or MDPV (0.1-1.0 mg/kg, s.c.) on brain temperature homeostasis in rats maintained in a st
71 aimed to determine the clinical relevance of brain temperature in patients by establishing how much i
72 p to the sleep-waking cycle, blood flow, and brain temperature in specific brain areas.
73          In individual patients, the average brain temperature increase over the core body temperatur
74               The largest pre- to post-COVID brain temperature increase was observed in the left olfa
75 G desynchronization, EMG activation, a large brain temperature increase, but weaker hyperlocomotion.
76 ortical blood flow among groups with varying brain temperature, indicating that delayed deterioration
77                                              Brain temperature is an understudied parameter relevant
78                                        Human brain temperature is higher and varies more than previou
79 perficial cortex regions, where the baseline brain temperature is lower than the temperature of incom
80 mic brain temperature variation-not absolute brain temperature-is one way in which human brain physio
81                                              Brain temperatures known to improve neurologic outcome c
82 mary endpoint was the time required to reach brain temperature less than 35 degrees C beginning from
83 on would decrease the time required to reach brain temperature less than 35 degrees C compared to act
84 euronal activity in the absence of a rise in brain temperature (<0.01 degrees C).
85 ever in severely head-injured patients since brain temperature may be higher than expected.
86                    Only patients with direct brain temperature measurements and without targeted temp
87 y after acute ischaemic stroke, elevation of brain temperature might augment tissue metabolic rate an
88 neural cells is accompanied by heat release, brain temperature monitoring provides insight into behav
89 e was no effect of drug treatment on body or brain temperature, nor on the duration or rate of Type I
90 2 degrees C saline aortic flush to achieve a brain temperature of 10 degrees C to 15 degrees C.
91                 The time required to reach a brain temperature of 35 degrees C was decreased with sod
92    Compared with anesthetized controls, core brain temperatures of the saline and slurry groups dropp
93 companied by heat production, measurement of brain temperature offers a method for assessing global a
94 rebral blood flow, changes in blood gases or brain temperature, or rat strain; (3) the neuroprotectiv
95 k was developed to facilitate individualized brain temperature predictions.
96 alysis, 25 displayed a daily rhythm, and the brain temperature range decreased in older patients (P =
97                       In patients (n = 114), brain temperature ranged from 32.6 to 42.3 degrees C and
98                     In healthy participants, brain temperature ranged from 36.1 to 40.9 degrees C; me
99 eous vasodilation; (2) drastic drops in deep brain temperature (reaching a nadir of 22.44 +/- 0.74 de
100 -skull surface, the postsurgical decrease of brain temperature recovers within a few days.
101 ody temperatures, because cooler and varying brain temperatures reduce brain performance and thus fit
102 ole of regional cerebral blood flow in local brain temperature regulation has received scant attentio
103 pplying oxygen for neuromodulator synthesis, brain temperature regulation, signaling to neurons, stab
104                                  In group 1, brain temperature remained constant except for a decreas
105 led the gradients and regional variations in brain temperatures reported in the literature for awake
106 ular tone, two critical factors that control brain temperature responses.
107 ia skin surfaces) that underlie MDMA-induced brain temperature responses.
108                 Of 100 patients eligible for brain temperature rhythm analysis, 25 displayed a daily
109 E in patients, we found that lack of a daily brain temperature rhythm increased the odds of death in
110 rature monitoring and management, with daily brain temperature rhythmicity emerging as one of the str
111                        Local manipulation of brain temperature should be broadly applicable to the id
112 evealing the existence of novel periphery-to-brain temperature signalling channels.
113 els during the hibernation season keep their brain temperature significantly elevated above ambient t
114 ure measurement is not a good measurement of brain temperature since it reflects body, not brain temp
115 e primary triggering force behind changes in brain temperature that are sufficient to affect body tem
116 schemia (HI) is neuroprotective; the precise brain temperature that provides optimal protection is un
117        Drug- and behavior-related changes in brain temperature thus appear to reflect some form of ne
118 observed during warm water forced swim, when brain temperature transiently increased (0.5 degrees C)
119 imals, passive animals had the same pattern; brain temperatures transiently decreased after cocaine i
120                              Model-predicted brain temperatures using our fully conserved model were
121 oach, we previously demonstrated that hourly brain temperature values co-varied strongly with time sp
122              We conclude that daily rhythmic brain temperature variation-not absolute brain temperatu
123                                              Brain temperature varied by time of day, especially in d
124                                A warmer mean brain temperature was associated with survival (P = 0.03
125                                          The brain temperature was controlled for 4 hrs after TBI and
126                                              Brain temperature was decreased from day 3.
127                           After hypothermia, brain temperature was either rapidly (n = 6) or slowly (
128                                              Brain temperature was increased an average of 2.0 degree
129                                              Brain temperature was maintained at 32-34 degrees C (mil
130 e treatment groups: (1) normothermic (Normo)-brain temperature was maintained at 37 degrees C; (2) in
131 an that observed in rats in which postmortem brain temperature was not maintained.
132          In seven control animals (group 1), brain temperature was not manipulated.
133                                              Brain temperature was our primary focus, but we also sim
134                         In hypothermic rats, brain temperature was reduced immediately after the 30-m
135 brupt hypodynamia after drug infusion), mean brain temperature was very stable at an elevated plateau
136 perature than dorsal striatum, each of these brain temperatures was higher than that in deep temporal
137 ns in rapid eye movement sleep (REMS) and in brain temperature were not found.
138 , end tidal Pco2, arterial Po2 and Pco2, and brain temperature were observed before inducing cardiac
139                     EEG, motor activity, and brain temperature were recorded for 23 h on the first, t
140                         Superficial and deep brain temperatures were further lowered to 27.8 +/- 0.8
141             In group 2, superficial and deep brain temperatures were lowered to 32.8 +/- 0.7 (SEM) de
142 ve drug administration of a session elevated brain temperature, while subsequent repeated injections
143 and warm (37 degrees C) water and correlated brain temperatures with behavioral changes.
144 ed that fentanyl induces biphasic changes in brain temperature, with an initial decrease that results
145  negatively correlated with average body and brain temperature, with the largest amount of REM sleep

 
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