ライフサイエンスコーパス: brain white matter

1 ical grey matter, deep grey matter and whole brain white matter.

2 sclerosis, a disease affecting primarily the brain white matter.

3 ioblastomas was compared with that of normal brain white matter.

4 cement of OX-42 microglial immunostaining in brain white matter.

5 ggest a genetic basis for the involvement of brain white matter aberrations in the pathophysiology of

6 well documented, but we know much less about brain white matter abnormalities in people who have both

7 while 39%, the "cerebral AMN" group, showed brain white matter abnormalities.

8 nt T cells in post-mortem multiple sclerosis brain white matter active lesions confirmed their involv

9 Emerging evidence suggests brain white matter alterations in adolescents with early

10 These findings were supported by whole-brain white matter analyses that revealed significantly

11 microvascular disease predominantly affects brain white matter and deep grey matter, resulting in is

12 el between-group FA differences across whole-brain white matter and fiber tracts at each age; fiber t

13 -sectional area, brain T2 lesion volume, and brain white matter and grey matter volume fractions.

14 ociated with levels of CSF biomarkers across brain white matter and in areas preferentially affected

15 olecular weights of 60- and 65-kDa in normal brain white matter and in astrocytic tumors, with an add

16 ociated with reduced diffusion anisotropy in brain white matter and lower cognition.

17 uses "spongiform" vacuolation of superficial brain white matter and neighboring gray matter.

18 eIF2B mutations predominantly affect the brain white matter, and the characteristic features of V

19 ; golden algae (Schizochytrium sp.), primate brain white matter, and transgenic mouse liver.

20 hat uses diffusion MRI to characterize whole-brain white matter architecture as a single local connec

21 Additional FAME identified in primate brain white matter are 20:1n-7, 20:1n-9, 20:2n-7, 20:2n-

22 ut the effects of these medications on human brain white matter are not well understood.

23 called MUQUBIA (Multimodal Quantification of Brain whIte matter biomArkers).

24 Brain white matter bulk consists predominantly of axons

25 NA was absent or barely detectable in normal brain white matter but was easily detectable in malignan

26 vel cationic lipid was separated from bovine brain white matter by a series of chromatographies on ca

27 MRI) reveals microstructural features of the brain white matter by quantifying the anisotropic diffus

28 rian insufficiency, developmental delay, and brain white matter changes.

29 ther the NTRK2 and BDNF polymorphisms impact brain white matter connections in major depressive disor

30 The integrity of brain white matter connections is central to a patient's

31 obabilistic tractography analyses calculated brain white matter connectivity (streamlines) as an esti

32 used as early and sensitive markers of human brain white matter connectivity, development, natural ag

33 on-weighted imaging (DWI) to construct whole-brain white-matter connectomes.

34 es correlated with executive function; whole brain white matter correlated with episodic memory, proc

35 bromatosis type 1 (NF1), a disorder in which brain white matter deficits identified by neuroimaging a

36 Brain white matter diffusion tensor imaging metrics were

37 Brain white matter disruptions have been implicated in c

38 -galactosylsphingosine (psychosine) found in brain white matter, enhances p140trk (Trk A) phosphoryla

39 l FAD mutation carriers (n = 12), mean whole brain white-matter FA (P = 0.045), FA of the columns of

40 In whole-brain white matter, FA was significantly lower across th

41 The relationship between brain white matter fiber-tract properties and immersion

42 (i) to characterize the relationship between brain white matter fiber-tract properties and second-lan

43 ted with subtle structural properties of the brain white matter found in multiple sclerosis (P = 0.02

44 We evaluated in brain white matter how NTRK1-T and NTRK1 rs4661063 allel

45 eased brain volume (B = -0.25) and increased brain white matter hyperintensities (B = 0.17).

46 ed brain volume (beta = -0.25) and increased brain white matter hyperintensities (beta = 0.17).

47 Underlying pathomechanisms of brain white matter hyperintensities (WMHs), commonly obs

48 hological deficits with the volumes of whole brain white matter hyperintensities and gray and white m

49 ns correlated with executive function; whole brain white matter hyperintensities correlated with exec

50 n mobility, cognition, and mood; the role of brain white matter hyperintensities in mediating this as

51 vascular disease was quantified by measuring brain white matter hyperintensities on fluid attenuation

52 Segmented brain white matter hyperintensities were compared betwee

53 Candidate MRI biomarkers included whole brain white matter hyperintensity (WMH) and cerebellar,

54 f CA and its relationship to the presence of brain white matter hyperintensity (WMH) in older adults,

55 on (CA) and determined its associations with brain white matter hyperintensity (WMH) in older adults.

56 ortical microvascular disease represented by brain white matter hyperintensity on magnetic resonance

57 of 24-hour systolic BP in the progression of brain white matter hyperintensity volume burden associat

58 hy and neurobehavioural, neurocognitive, and brain white matter imaging outcomes in long-term survivo

59 h methylphenidate affects specific tracts in brain white matter in boys with attention-deficit/hypera

60 eyond the frontal lobes, occupy 66% of total brain white matter in humans and 48% in three monkey spe

61 of age-related FW changes by assessing whole brain white matter in individuals with schizophrenia and

62 d xenon combined with hypothermia attenuates brain white matter injury in comatose survivors of out-o

63 iations between periodontal disease (Pd) and brain white matter integrity (WMI).

64 onsumption-related genes are associated with brain white matter integrity and risk of digestive and n

65 erences, indicating widespread alteration in brain white matter integrity but not necessarily white m

66 utation carriers demonstrated a reduction of brain white matter integrity compared to unaffected sibl

67 onance imaging-based measurements of reduced brain white matter integrity in the 1-cm radius white ma

68 Whole brain white matter integrity of HD-monkeys was examined

69 lationships between 1) delirium duration and brain white matter integrity, and 2) white matter integr

70 n and alcohol use disorder (AUD) can disrupt brain white matter integrity, we questioned whether HIV

71 ), the most frequent monogenetic disorder of brain white matter, is highly variable, ranging from slo

72 d docosahexaenoic acid (DHA; 22:6) have more brain white matter lesions (WMLs), an association sugges

73 ctional anisotropy) were measured from whole brain white matter lesions and from both lesions and nor

74 Similarly, brain white matter lesions were mapped voxel-wise as a f

75 ated with subjective cognitive complaints or brain white-matter lesions 5 to 10 years after the hyper

76 mental delay, hypotonia, spastic paraplegia, brain white matter loss, and dysmorphic features overlap

77 Here we examine brain white matter magnetic resonance imaging (MRI) data

78 Current investigations suggest that brain white matter may be qualitatively altered in schiz

79 chitecture between psychiatric disorders and brain white matter may provide mechanistic insights for

80 of cognitive abilities and aspects of whole-brain white matter measures in children with and without

81 ed healthy controls (HCs) and compared their brain white matter microstructural properties using Diff

82 causal effect of liability to depression on brain white matter microstructure (beta: 0.125 to 0.868,

83 ally studying the effects of hemodialysis on brain white matter microstructure and further examine if

84 tested whether there were coupled changes in brain white matter microstructure indexed by fractional

85 Brain white matter microstructure was studied by diffusi

86 s chemotherapy is associated with changes in brain white matter microstructure, we investigated wheth

87 Alcohol dependence exacts a toll on brain white matter microstructure, which has the potenti

88 Disruptions in the brain white matter network, possibly secondary to vascul

89 We show that T2 in brain white matter of nondemented volunteers follows a U

90 not present in control brain gray matter, AD brain white matter, or cerebellum.

91 f mild traumatic brain injury (mTBI) impacts brain white-matter pathways with potentially long-term c

92 n, relative to comparison subjects, in whole-brain white matter, prefrontal and temporal white matter

93 y in the human brain, suggest that the adult brain white matter preserves dynamic characteristics and

94 al analyses conducted across the rest of the brain white matter revealed lower FA bilaterally in the

95 major depression based on an advanced whole brain white matter segmentation framework.

96 Voxelwise analysis of the brain white matter skeleton showed an average decrease o

97 In these experiments, brain white matter slices from 20-day-old rats were incu

98 It is well established that human brain white matter structure changes with aging, but the

99 The impact of the variant on brain white matter structure, however, is controversial

100 Brain white matter T2 lesions, T1-hypointense lesions, c

101 diffusivity and radial diffusivity values in brain white matter tracts.

102 Voxelwise comparison of the whole brain white matter using tract-based spatial statistics

103 Mean whole brain white matter volume was significantly smaller in p

104 ng revealed significant differences in whole brain white matter volume, regional gray and white matte

105 in humans correlated with age, CSF tau, and brain white matter volume.

106 pared with monkeys, when normalized by total brain white matter volume.

107 ved hemodynamic tolerability, and changes in brain white matter were associated with hemodynamic inst

108 Extracts of normal brain white matter were capable of promoting haptotactic

109 ypernatraemic rats there was myelinolysis of brain white matter, with karyorrhexis and necrosis of ne

110 usion tensor MR measures of global damage to brain, white matter ( WM white matter ), and GM gray mat

111 Brain white matter (WM) abnormalities have been hypothes

112 r-income households showed signs of advanced brain white matter (WM) aging with greater mean diffusiv

113 Abnormalities in brain white matter (WM) are reported in youth at-risk fo

114 We investigated focal brain white matter (WM) changes and fluid shifts during

115 genetic and molecular pathways driving human brain white matter (WM) development are only beginning t

116 However, the effect of chronic MMT on brain white matter (WM) is not fully known.

117 rize the association of delivery method with brain white matter (WM) microstructural integrity in pre

118 ensive relations between healthy adult human brain white matter (WM) microstructure and gray matter (

119 difficult bimanual conditions, less optimal brain white matter (WM) microstructure, and a decreased

120 Structural asymmetry of whole brain white matter (WM) pathways, i.e., the connectome,

121 ittle is known about the association between brain white matter (WM) structure and motor function in

122 transfer ratio (MTR(REX)) averaged in whole brain white matter (WM), enhancing tumor and necrosis.

123 tions typical of multiple sclerosis in whole brain white matter (WM), T2-weighted WM hyperintensities

124 about its effect on the development of human brain white matter (WM).PurposeTo determine whether MPH