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1  effect on Ca2+ sensitivity of permeabilized bronchial smooth muscle.
2 fic potassium channel gene products in human bronchial smooth muscle.
3 e and phosphorylated myosin light chain 2 in bronchial smooth muscles.
4 pes, did not show an increased activation in bronchial smooth muscle after TNF treatment.
5 veral ADAM33 protein isoforms occur in adult bronchial smooth muscle and in human embryonic bronchi a
6 f endothelin receptors are present on canine bronchial smooth muscle and parenchyma.
7 , there is marked enlargement of the mass of bronchial smooth muscle, and emphysema does not occur in
8 ells, whereas sncRNA expression in asthmatic bronchial smooth muscle (BSM) cells is almost completely
9                                  Increase of bronchial smooth muscle (BSM) mass is a crucial feature
10                                    Increased bronchial smooth muscle (BSM) mass is a key feature of a
11                                    Increased bronchial smooth muscle (BSM) mass, a characteristic fea
12  increase in both exacerbation frequency and bronchial smooth muscle (BSM) mass.
13                                    Moreover, bronchial smooth muscle (BSM) remodeling at preschool ag
14 and more specifically with increased mass of bronchial smooth muscle (BSM).
15 e [RBM] thickness, mucus gland, fibrosis and bronchial smooth muscle [BSM] areas, the density of bloo
16 dothelin-1 (ET-1) is a potent constrictor of bronchial smooth muscle, but there is limited informatio
17 beta activity or expression, increased human bronchial smooth muscle cell size, protein synthesis, an
18 ever, in the OVA-challenged animals only the bronchial smooth muscle cells showed increased C3aR expr
19                           Infection of human bronchial smooth muscle cells with pMSCV GSK-3beta-A9, a
20  expressed in vascular endothelial cells and bronchial smooth muscle cells, leading to lethal vascula
21 ntified their progenies in subpopulations of bronchial smooth muscle cells, vascular smooth muscle ce
22                                  Overzealous bronchial smooth muscle constriction is thought to under
23 nimal models of AHR, human tracheal and main bronchial smooth muscle contractility is not increased i
24 ur necrosis factor-alpha (TNF) on guinea-pig bronchial smooth muscle contractility were investigated.
25 d endothelial permeability; it also triggers bronchial smooth muscle contraction and participates in
26 flux, mediator release, and allergen-induced bronchial smooth muscle contraction by CRACM-channel blo
27   Synta-66 also inhibited allergen-dependent bronchial smooth muscle contraction in ex vivo tissue.
28 e, of a role for M3-mAChR phosphorylation in bronchial smooth muscle contraction in health and in a d
29 ensin (ANG II) from mast cell renin elicited bronchial smooth muscle contraction mediated by ANG II t
30 e, indicating that RGS5 negatively regulates bronchial smooth muscle contraction.
31 ate suppressed ovalbumin-mediated guinea pig bronchial smooth muscle contraction.
32 hypothesis has never been tested directly in bronchial smooth muscle embedded within intraparenchymal
33 ate a novel signalling pathway in guinea-pig bronchial smooth muscle leading to an increase in myosin
34      We developed clonal cell lines of human bronchial smooth muscle origin by retroviral transductio
35 We have developed clonal cell lines of human bronchial smooth muscle origin that may be useful for th
36                                              Bronchial smooth muscle (SM) mesenchymal cell precursors
37 ivated contractile response of permeabilized bronchial smooth muscle strips was significantly increas
38  also obtained in passively sensitized human bronchial smooth muscle tissue.
39 rm epoxyeicosatrienoic acids, which modulate bronchial smooth muscle tone and airway transepithelial
40 frican Americans may underlie differences in bronchial smooth muscle tone and thus pulmonary function
41 cluding modulation of pulmonary arterial and bronchial smooth muscle tone.
42             Thus, PAR2 is expressed by human bronchial smooth muscle where its activation mobilizes i