ライフサイエンスコーパス: bronchoalveolar

1 s enhanced cell-penetrating ability in human bronchoalveolar carcinoma A549 cells.

2 2287 significantly decreased esophageal and bronchoalveolar eosinophilia but only when given as a th

3 ral generation of cytokines IL-4 and TSG6 in bronchoalveolar fluid (BALF).

4 d anti-inflammatory cytokines (IL-10) in the bronchoalveolar fluid, and IL-2 and IFN-gamma cytokines

5 Lung tissue, bronchoalveolar fluid, and plasma were analyzed, in addi

6 ntral and peripheral airway remodelling, and bronchoalveolar inflammation were assessed.

7 tegrated study of human small airway tissue, bronchoalveolar lavage (BAL) and an experimental murine

8 TTR for bronchoalveolar lavage (BAL) and endotracheal aspirate (

9 bit AHR, increased numbers of eosinophils in bronchoalveolar lavage (BAL) and increased collagen cont

10 a methacholine test, airway inflammation in bronchoalveolar lavage (BAL) and lung tissue, and total

11 e expression and activity were determined in bronchoalveolar lavage (BAL) and lungs of human donors a

12 Conventional methods to identify HBEC in bronchoalveolar lavage (BAL) and wash (BW) have throughp

13 We characterized bronchoalveolar lavage (BAL) cells and fluid (BALF) prot

14 cDNA library derived from mRNA isolated from bronchoalveolar lavage (BAL) cells and leukocytes of sar

15 was to investigate the expression of iNOS in bronchoalveolar lavage (BAL) cells and tissue from centr

16 ring healthy aging, the proportions of blood bronchoalveolar lavage (BAL) classical monocytes peak in

17 increased eosinophilia and interleukin-5 in bronchoalveolar lavage (BAL) compared to sham-OVA mice.

18 We sought to determine the relationship of bronchoalveolar lavage (BAL) cytokine/chemokine expressi

19 perimental model of allergic asthma, matched bronchoalveolar lavage (BAL) fluid and plasma were colle

20 igatus Its performance has been validated on bronchoalveolar lavage (BAL) fluid and serum specimens,

21 ILC2s were isolated from blood and bronchoalveolar lavage (BAL) fluid before and after segm

22 ormed in nasopharyngeal aspirates (NPAs) and bronchoalveolar lavage (BAL) fluid before HCT.

23 ecommendation was made in favor of obtaining bronchoalveolar lavage (BAL) fluid for lymphocyte cellul

24 (log(10) mean value, 4.2), secretory IgA in bronchoalveolar lavage (BAL) fluid from immunized mice,

25 Unlike blood ILC2s, bronchoalveolar lavage (BAL) fluid ILC2s from asthmatic

26 We examined bronchoalveolar lavage (BAL) fluid leukocytes, cytokines

27 70 have been found in the sputum, serum, and bronchoalveolar lavage (BAL) fluid of asthma patients an

28 nsus sequences of viruses collected from the bronchoalveolar lavage (BAL) fluid of the animals.

29 rometry based proteome analysis of acellular bronchoalveolar lavage (BAL) fluid samples on an observa

30 Analysis of lung homogenates and bronchoalveolar lavage (BAL) fluid showed that LPS-induc

31 Inflammatory cells in bronchoalveolar lavage (BAL) fluid were assessed by flow

32 al swabs in the above-named transport media, bronchoalveolar lavage (BAL) fluid, and sputum.

33 red CD4+ T cells in whole blood, spleen, and bronchoalveolar lavage (BAL) fluid, but not in the lung

34 flammatory cytokines KC, IP-10, and IL-33 in bronchoalveolar lavage (BAL) fluid.

35 mediators TNF-alpha, IL-6, and leukocytes in bronchoalveolar lavage (BAL) fluids.

36 ics and function in rhesus macaque blood and bronchoalveolar lavage (BAL) following mucosal adenoviru

37 This analysis included bronchoalveolar lavage (BAL) for cell and fluid analysis

38 igated M. tuberculosis-specific responses in bronchoalveolar lavage (BAL) from persons with latent M.

39 We measured mtDNA concentrations in bronchoalveolar lavage (BAL) from subjects with and with

40 ollowing alveolar airspace infiltration, the bronchoalveolar lavage (BAL) neutrophil proteome is furt

41 Bronchoalveolar lavage (BAL) NK cells were immunophenoty

42 xpression in airway immune cells obtained by bronchoalveolar lavage (BAL) of individuals with latent

43 phenotype of NKG2C NK cells in the blood and bronchoalveolar lavage (BAL) of lung transplant recipien

44 We collected endobronchial brush (EB) and bronchoalveolar lavage (BAL) samples from 39 asthmatic p

45 ng injury in CIP, we prospectively collected bronchoalveolar lavage (BAL) samples in ICI-treated pati

46 Twenty consecutive bronchoalveolar lavage (BAL) samples were plated to stan

47 nd increased CD4+ T cell counts in blood and bronchoalveolar lavage (BAL) samples, it did not reduce

48 vation of cell counts and protein content in bronchoalveolar lavage (BAL) samples.

49 ng flow cytometry and a multiplex assay with bronchoalveolar lavage (BAL) specimens (n = 68) from 52

50 eporting for 259 adult inpatients submitting bronchoalveolar lavage (BAL) specimens for laboratory an

51 Thus, the diagnosis of PJP requires bronchoalveolar lavage (BAL) specimens or a decision to

52 However, no CMV DNA threshold exists in bronchoalveolar lavage (BAL) to differentiate pneumonia

53 Airway responsiveness was assessed, bronchoalveolar lavage (BAL) was performed, and lung cel

54 s of CD4(+) T and B cells in the spleens and bronchoalveolar lavage (BAL) were also observed.

55 ergic patients underwent SAC, and cells from bronchoalveolar lavage (BAL) were collected after 24 hou

56 Bronchoscopy and bronchoalveolar lavage (BAL) were performed 7 days post-

57 ased age, a low percentage of lymphocytes in bronchoalveolar lavage (BAL), a decreased transfer facto

58 flux of neutrophils and macrophages into the bronchoalveolar lavage (BAL), and human CD45(+) cells in

59 Subjects underwent bronchoalveolar lavage (BAL), and peripheral whole blood

60 linically indicated fiberoptic bronchoscopy, bronchoalveolar lavage (BAL), endobronchial brushings, a

61 mation as proven by increased cellularity in bronchoalveolar lavage (BAL), pulmonary transcriptomic p

62 ia also reduced inflammatory cell numbers in bronchoalveolar lavage (BAL).

63 t computed tomography, and bronchoscopy with bronchoalveolar lavage (BAL).

64 by an accumulation of CD4(+) T cells in the bronchoalveolar lavage (BAL).

65 f control (ACQ-7) underwent bronchoscopy and bronchoalveolar lavage (BAL).

66 aeruginosa, P. aeruginosa) were analyzed in bronchoalveolar lavage (BAL); and alveolar SGLT1 was ana

67 oneal (p = 0.037), systemic (p = 0.019), and bronchoalveolar lavage (p = 0.011) quantitative bacteria

68 ower respiratory tract specimens (sputum and bronchoalveolar lavage [BAL] fluid).

69 We interrogated the bronchoalveolar lavage and blood viromes during the peri

70 oxic amyloids that can be recovered from the bronchoalveolar lavage and cerebrospinal fluids of criti

71 4 and CD8 T cell responses in blood, spleen, bronchoalveolar lavage and lung lymph nodes.

72 ion model, IL-10-producing CD4(+) T cells in bronchoalveolar lavage and lung were significantly decre

73 log(10) reductions in median viral loads in bronchoalveolar lavage and nasal mucosa compared with af

74 log(10) reductions in median viral loads in bronchoalveolar lavage and nasal mucosa, respectively, a

75 ided complete or near-complete protection in bronchoalveolar lavage and nasal swabs after SARS-CoV-2

76 fic IgG antibodies to the offending antigen, bronchoalveolar lavage and pathological features.

77 ues, we performed transcriptomic analyses of bronchoalveolar lavage and peripheral blood and proteomi

78 ition of ADAM10 reduces sEphrin-B2 levels in bronchoalveolar lavage and prevents lung fibrosis in mic

79 MIF decreases neutrophil infiltration to the bronchoalveolar lavage and tissue and simultaneously dec

80 Bronchoalveolar lavage and tissues were sampled for myco

81 ermate control animals through evaluation of bronchoalveolar lavage and tissues.

82 ophils in bone marrow (BM), blood, lung, and bronchoalveolar lavage as well as airway hyperresponsive

83 subjects had symptom scores, spirometry, and bronchoalveolar lavage before and after rhinovirus-induc

84 Bronchoalveolar lavage CD8 T cells showed significantly

85 ere analyzed based on serum antibody levels, bronchoalveolar lavage cell counts, lung histology, lung

86 Bronchoalveolar lavage cell mRNA levels of iNOS or iNOS

87 At two independent study sites, bronchoalveolar lavage cells from donors with latent tub

88 T-cell recruitment and activation within bronchoalveolar lavage cells of ACE2-high subjects was r

89 anscriptional profiling of cells isolated by bronchoalveolar lavage confirmed that influenza infectio

90 .RESULTSActivated CD4+ T cell frequencies in bronchoalveolar lavage correlated strongly with local C-

91 Donor testing was performed with bronchoalveolar lavage culture and PCR intraoperatively.

92 increased, and protein concentration in the bronchoalveolar lavage diminished, showing the impact of

93 GR-1) resulted in a significant decrease in bronchoalveolar lavage eosinophil counts, lung interleuk

94 This activation phenotype indicated by bronchoalveolar lavage eosinophil surface markers, as we

95 niae infection was diagnosed on the basis of bronchoalveolar lavage eosinophilia and blood findings.

96 b induced a rise in circulating eosinophils, bronchoalveolar lavage eosinophilia, and eosinophil pero

97 sham at day 13 showed an increased number of bronchoalveolar lavage eosinophils and increased express

98 l7r(cre) mice showed complete suppression of bronchoalveolar lavage eosinophils and mucous metaplasia

99 Before mepolizumab treatment, bronchoalveolar lavage eosinophils had more surface IL-3

100 im of this study was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicti

101 infected with Streptococcus pneumoniae, and bronchoalveolar lavage fluid (BALF) and lung CFU values

102 d temporal kinetics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumi

103 s the overlap in metabolites between matched bronchoalveolar lavage fluid (BALF) and plasma, identifi

104 tinfection and included cellular profiles in bronchoalveolar lavage fluid (BALF) and serum IgG and Ig

105 BORT reduced bronchoalveolar lavage fluid (BALF) and tissue eosinophi

106 understanding of the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients

107 We studied bronchoalveolar lavage fluid (BALF) from 36 patients wit

108 alysis on gene expression data from cells in bronchoalveolar lavage fluid (BALF) from COVID-19 patien

109 us 6B (HHV-6B) DNA is frequently detected in bronchoalveolar lavage fluid (BALF) from immunocompromis

110 with human MSCs when stimulated with LPS or bronchoalveolar lavage fluid (BALF) from patients with A

111 by using mass spectrometry) were measured in bronchoalveolar lavage fluid (BALF) from patients with N

112 gation & density-gradient fractionation from bronchoalveolar lavage fluid (BALF) in vivo.

113 cytomegalovirus (HCMV) DNA detection in the bronchoalveolar lavage fluid (BALF) indicates HCMV repli

114 Bronchoalveolar lavage fluid (BALF) levels of SOCS3 were

115 emic stroke caused a significant increase in bronchoalveolar lavage fluid (BALF) macrophages and neut

116 induce apolipoprotein E (APOE) expression by bronchoalveolar lavage fluid (BALF) macrophages from ast

117 We measured 13 cytokines in bronchoalveolar lavage fluid (BALF) of 88 children with

118 We measured iron and ferritin levels in the bronchoalveolar lavage fluid (BALF) of participants enro

119 a diffuse lung injury marked by increases in bronchoalveolar lavage fluid (BALF) protein and histoche

120 rformed a retrospective multicenter study on bronchoalveolar lavage fluid (BALF) samples obtained fro

121 All archived bronchoalveolar lavage fluid (BALF) samples that had pre

122 Neutrophils in bronchoalveolar lavage fluid (BALF) served as markers of

123 In parallel, adenosine levels in bronchoalveolar lavage fluid (BALF) were increased by ap

124 Lung function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs we

125 in mice resulted in increased OPN levels in bronchoalveolar lavage fluid (BALF).

126 2 ligands was significantly increased in the bronchoalveolar lavage fluid 48 hours after segmental al

127 o DC subsets (DC2/3 and DC5) are expanded in bronchoalveolar lavage fluid 8 h after lipopolysaccharid

128 urbiprofen augmented the release of IL-33 in bronchoalveolar lavage fluid after Alternaria challenge,

129 ased airway hyperreactivity to methacholine, bronchoalveolar lavage fluid albumin, and serum IgE leve

130 healthy volunteers and examine the impact on bronchoalveolar lavage fluid and blood MP repertoire.

131 ar lavage fluid was evaluated by microscopy; bronchoalveolar lavage fluid and blood were assessed by

132 PM2.5 increased neutrophil numbers and KC in bronchoalveolar lavage fluid and caused slight peribronc

133 recruitment of monocytes and neutrophils in bronchoalveolar lavage fluid and increased neutrophils i

134 fspring had lower white blood cell counts in bronchoalveolar lavage fluid and less pronounced peribro

135 ikingly reduced numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inf

136 ed a significantly reduced viral load in the bronchoalveolar lavage fluid and lower respiratory tract

137 ine expression in ILC2s and TH2 cells in the bronchoalveolar lavage fluid and lung tissue were assess

138 Flow cytometry and cytokine measurements in bronchoalveolar lavage fluid and lung tissue were follow

139 antioxidant responses were assessed in both bronchoalveolar lavage fluid and lung tissue.

140 Bronchoalveolar lavage fluid and matched blood were obta

141 We detected the presence of PRELP in human bronchoalveolar lavage fluid and showed that PRELP can b

142 bronchitis, perivasculitis, and increases in bronchoalveolar lavage fluid cell numbers were detected

143 Analysis of bronchoalveolar lavage fluid collected from human patien

144 eduction of eosinophil and T cell numbers in bronchoalveolar lavage fluid compared with those in dilu

145 2 223K/K mice had persistent eosinophilia in bronchoalveolar lavage fluid compared with wild-type and

146 a, accompanied with significant reduction in bronchoalveolar lavage fluid concentration of IL-5, a cy

147 Bronchoalveolar lavage fluid demonstrates greater than 2

148 , IL-33(KO)/Tg+ mice had complete absence of bronchoalveolar lavage fluid eosinophilia, accompanied w

149 Bronchoalveolar lavage fluid from 23 lean, 12 overweight

150 Microbiology was done on bronchoalveolar lavage fluid from all patients and venti

151 ein levels were significantly upregulated in bronchoalveolar lavage fluid from HIV-infected smokers,

152 In bronchoalveolar lavage fluid from humans with ARDS, gut-

153 patients with severe asthma compared with in bronchoalveolar lavage fluid from individuals without as

154 s of IL-1beta and IL-8 rapidly determined in bronchoalveolar lavage fluid from patients randomised to

155 vo, histone-C1INH complexes were detected in bronchoalveolar lavage fluid from patients with acute re

156 he protein expression patterns in plasma and bronchoalveolar lavage fluid from patients with ARDS.

157 We use our method to characterize bronchoalveolar lavage fluid from patients with asthma,

158 nase activities were quantified in serum and bronchoalveolar lavage fluid from patients with CF, asth

159 anges in the miRNA composition of EVs in the bronchoalveolar lavage fluid from patients with IAV-indu

160 pulmonary eosinophilia were measured in the bronchoalveolar lavage fluid from patients with mild ast

161 r, specific ceramide species were altered in bronchoalveolar lavage fluid from patients with severe a

162 Low concentrations of IL-1beta and IL-8 in bronchoalveolar lavage fluid have been validated as effe

163 al aimed to determine whether measurement of bronchoalveolar lavage fluid IL-1beta and IL-8 could eff

164 ry inflammation, eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adop

165 eficient mice had fewer DEP exposure-induced bronchoalveolar lavage fluid immune cells and proinflamm

166 s, angiotensin-converting enzyme activity in bronchoalveolar lavage fluid increased 3.2-fold in elder

167 Following repetitive O3 exposure, higher bronchoalveolar lavage fluid inflammatory cells were obs

168 Bronchoalveolar lavage fluid LT levels were increased in

169 I are seen in HAECs, in association with low bronchoalveolar lavage fluid mitochondrial DNA and more

170 cells in lymph nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques

171 we report that SOCS3 protein was elevated in bronchoalveolar lavage fluid of both virus- and bacteria

172 med that leukotriene levels are increased in bronchoalveolar lavage fluid of HIV-infected patients.

173 -promoting cytokines and chemokines into the bronchoalveolar lavage fluid of Lipa(-/-) mice.

174 oxidase were more frequently detected in the bronchoalveolar lavage fluid of lung transplant patients

175 f RAGE was determined in protein, serum, and bronchoalveolar lavage fluid of mice and lungs and serum

176 shown to reduce the total number of cells in bronchoalveolar lavage fluid of mice challenged with hou

177 rply increased the levels of UDP detected in bronchoalveolar lavage fluid of mice.

178 NF-alpha, IL-9, CXCL1, CCL2, and CCL5 in the bronchoalveolar lavage fluid of RSV-infected mice, witho

179 y/severity; (2) RSV-specific CD8+ T cells in bronchoalveolar lavage fluid preinfection (subjects with

180 e exposure of vehicle-treated rats increased bronchoalveolar lavage fluid protein, albumin, neutrophi

181 Higher numbers of Asm(-/-) T cells in bronchoalveolar lavage fluid released lower levels of IL

182 ynthase similarly attenuated the increase in bronchoalveolar lavage fluid SOCS3 noted in lungs of mic

183 ption factor GATA3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of

184 ation and analysis of Aspergillus conidia in bronchoalveolar lavage fluid using the combination of tr

185 determined by whole-body plethysmography and bronchoalveolar lavage fluid was analyzed for cellular c

186 Bronchoalveolar lavage fluid was collected from 23 stero

187 Bronchoalveolar lavage fluid was collected from patients

188 Bronchoalveolar lavage fluid was evaluated by microscopy

189 had a separate donor; however, pretransplant bronchoalveolar lavage fluid was only available from the

190 Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower

191 ity in lymphoid tissues and Th2 responses in bronchoalveolar lavage fluid), they also accumulate func

192 rol and PNE rat pups: 1) the 5-HT content in bronchoalveolar lavage fluid, 2) the apneic response to

193 chronic rhinosinusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen c

194 HIV has been recovered from cell-free bronchoalveolar lavage fluid, alveolar macrophages, and

195 concentration was also induced in the serum, bronchoalveolar lavage fluid, alveolar type II epithelia

196 d lower total cell counts and neutrophils in bronchoalveolar lavage fluid, and had earlier influx of

197 nd disaturated PC in lung tissue homogenate, bronchoalveolar lavage fluid, and lung LB was increased

198 were sensitized and challenged with OVA and bronchoalveolar lavage fluid, and the lungs were collect

199 d tumor-promoting cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expressio

200 fferential cell counts were performed on the bronchoalveolar lavage fluid, followed by histological a

201 und significantly elevated total proteins in bronchoalveolar lavage fluid, higher parasitemia and tis

202 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasa

203 ay and analyses of the injury markers in the bronchoalveolar lavage fluid, respectively.

204 tained pulmonary or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflam

205 In serum and bronchoalveolar lavage fluid, total anti-IAV IgG and IgA

206 levels of iron-carrier proteins in cells or bronchoalveolar lavage fluid.

207 t least 10(4) colony forming units per mL of bronchoalveolar lavage fluid.

208 r protein leak and inflammatory cells in the bronchoalveolar lavage fluid.

209 Recruited eosinophils were enumerated in bronchoalveolar lavage fluid.

210 lung while also reducing LPA 18:2 content in bronchoalveolar lavage fluid.

211 rase chain reaction using RNA extracted from bronchoalveolar lavage fluid.

212 ction and asthma-associated cytokines in the bronchoalveolar lavage fluid.

213 n, and surfactant protein A concentration in bronchoalveolar lavage fluid.

214 release from lung epithelium as detected in bronchoalveolar lavage fluid.

215 in (TSLP), IL-9, and IL-13, but not IL-5, in bronchoalveolar lavage fluid.

216 the macrophage chemoattractant MCP-1 in lung bronchoalveolar lavage fluid.

217 and was highly correlated with IFN levels in bronchoalveolar lavage fluid.

218 ed the influx of inflammatory cells into the bronchoalveolar lavage fluid.

219 d resulted in IL-1beta accumulation into the bronchoalveolar lavage fluid.

220 Quantitative bacterial culture of bronchoalveolar lavage fluids (BALF) is labor-intensive,

221 ncreased ATP concentrations were reported in bronchoalveolar lavage fluids of asthmatic patients.

222 ocessed 25-kDa IL-33 protein was detected in bronchoalveolar lavage fluids without any exogenous stim

223 Positive bronchoalveolar lavage for GM or Aspergillus cultures wa

224 AMs were obtained from bronchoalveolar lavage from healthy donors or patients w

225 leukocytes, macrophages, and neutrophils in bronchoalveolar lavage from O3-exposed mice.

226 ts antifibrotic properties and is reduced in bronchoalveolar lavage from patients with IPF.

227 ol for invasive pulmonary aspergillosis with bronchoalveolar lavage galactomannan and cultures perfor

228 ent in anastomotic plaques with reduction in bronchoalveolar lavage galactomannan values (7.48-2.15 n

229 L-1alpha positively correlated with elevated bronchoalveolar lavage IL-8 levels (r(2) = 0.6095, p < 0

230 were most frequently cultured from blood and bronchoalveolar lavage in humans and lungs in animals.

231 antibiotic-free days in the 7 days following bronchoalveolar lavage in the intention-to-treat analysi

232 Bronchoscopy with bronchoalveolar lavage is very useful in its diagnosing.

233 as adjuvants resulted in elevated serum and bronchoalveolar lavage levels of anti-P6-specific IgG an

234 OVA-induced increases in bronchoalveolar lavage lymphocytes, eosinophils, IL-13,

235 rtension, low peripheral blood, and elevated bronchoalveolar lavage lymphocytes.

236 Bronchoalveolar lavage macrophages were stimulated in vi

237 cell therapy groups, despite a reduction in bronchoalveolar lavage neutrophils.

238 ll as 16S ribosomal RNA sequencing data from bronchoalveolar lavage obtained as part of the COMET-IPF

239 Lung fibroblasts (LFs) were derived from bronchoalveolar lavage of BOS patients.

240 highly autofluorescent cells present in the bronchoalveolar lavage of cigarette smokers.

241 IV-1-infected airway macrophages obtained by bronchoalveolar lavage of HIV-1-infected individuals.

242 used to characterize macrophages from human bronchoalveolar lavage of HIV-infected and -uninfected s

243 rleukin-1 alpha (IL-1alpha) was increased in bronchoalveolar lavage of lung transplant recipients gro

244 des of the biculture and was also present in bronchoalveolar lavage of lung transplantation patients.

245 Uninfected AMs were isolated from bronchoalveolar lavage of young (3 mo) and old (18 mo) C

246 marker of PARP activation) and IL-6, in the bronchoalveolar lavage or the lung tissue, and histology

247 phenotype and function of T lymphocytes from bronchoalveolar lavage or the peripheral blood.

248 lature was supported by normal histology and bronchoalveolar lavage protein levels.

249 Incubating control cells with disease bronchoalveolar lavage recapitulated the aberrant functi

250 of interferon and interleukin-6 pathways in bronchoalveolar lavage samples and repression of natural

251 y evaluating both upper airway and acellular bronchoalveolar lavage samples from 49 subjects from thr

252 have previously demonstrated that acellular bronchoalveolar lavage samples from half of the healthy

253 rst identified two redondovirus genomes from bronchoalveolar lavage samples from human lung donors.

254 Bronchoalveolar lavage samples from Ugandan patients wit

255 Although sputum and bronchoalveolar lavage samples provide the highest diagn

256 navirus testing on pooled nasopharyngeal and bronchoalveolar lavage samples taken from patients who h

257 tes for the galactomannan assay in serum and bronchoalveolar lavage samples were 61.3% and 57.1%, res

258 Bronchoalveolar lavage sampling may provide useful diagn

259 Plasma and bronchoalveolar lavage soluble receptor for advanced gly

260 ubset of 61 patients, respiratory specimens (bronchoalveolar lavage specimens, tracheal aspirates, an

261 ch on-ECMO experimental step, but the lowest bronchoalveolar lavage sRAGE levels were obtained at min

262 Bronchoalveolar lavage was associated with a small and t

263 Bronchoalveolar lavage was done in all patients, with co

264 Inflammation in bronchoalveolar lavage was increased in old versus young

265 Three days after instillation, bronchoalveolar lavage was performed and plastic-adheren

266 E. coli colony-forming units in bronchoalveolar lavage were reduced in both cell therapy

267 r advanced glycation end products (sRAGE) in bronchoalveolar lavage(BAL) and plasma were performed.

268 identify cross-tissue compartment (blood and bronchoalveolar lavage) and temporal proteomic signature

269 the lung, proinflammatory cytokine levels in bronchoalveolar lavage, and alveolar capillary leakage.

270 lished prescribing practices, reluctance for bronchoalveolar lavage, and dependence on a chain of tri

271 Eosinophils were examined in blood, bronchoalveolar lavage, and endobronchial biopsies 48 ho

272 Silica-induced SPP1 in lung tissue, bronchoalveolar lavage, and serum increased more in male

273 Blood, bronchoalveolar lavage, large proximal, and small distal

274 antibiotic-free days in the 7 days following bronchoalveolar lavage.

275 els of Aspergillus and total fungus in their bronchoalveolar lavage.

276 olating large numbers of cells by whole-lung bronchoalveolar lavage.

277 r infiltration, and E. coli colony counts in bronchoalveolar lavage.

278 ed numbers of lymphocytes and neutrophils in bronchoalveolar lavage.

279 All patients underwent bronchoalveolar lavage.

280 ctive viral replication and viral genomes in bronchoalveolar-lavage (BAL) fluid and nasal swab specim

281 Detection of toxicants in bronchoalveolar-lavage (BAL) fluid from patients with EV

282 a higher number of lung cells obtained from bronchoalveolar lavages (BAL) than TAC pre-treatment alo

283 Bronchoalveolar lavages (BALs) from coinfected mice show

284 ignificantly higher S. maltophilia counts in bronchoalveolar lavages and lung tissue homogenates.

285 s are reticent to perform aerosol-generating bronchoalveolar lavages for galactomannan testing and mi

286 ial alarmins were measured longitudinally in bronchoalveolar lavages from lung transplant recipients

287 sed eosinophil and neutrophil populations in bronchoalveolar lavages from mice with asthma.

288 NA and host total RNA were isolated from 203 bronchoalveolar lavages obtained from 112 patients post-

289 NA and host total RNA were isolated from 189 bronchoalveolar lavages obtained from 116 patients post

290 s on radiographs and reduced virus titres in bronchoalveolar lavages twelve hours after the first dos

291 edding measured from nasal and throat swabs, bronchoalveolar lavages, and tissues was not reduced wit

292 After bronchoalveolar lavages, piglets developed respiratory d

293 samples, including blood, plasma, urine and bronchoalveolar lavages.

294 throat of all of the macaques, as well as in bronchoalveolar lavages; in one macaque, we observed pro

295 Only fluticasone/salmeterol decreased bronchoalveolar neutrophilia (p = 0.03) to the same exte

296 cs of human DCs and monocytes recruited into bronchoalveolar space immediately following localised ac

297 mmune cells in whole lung parenchyma and the bronchoalveolar space of mice, exposed to 48 hours of hy

298 platelet-leukocyte complex formation in the bronchoalveolar space.

299 involves PKCiota-dependent transformation of bronchoalveolar stem cells (BASCs).

300 Bronchoalveolar wash and lavage were obtained 24 hours a