ライフサイエンスコーパス: bronchoalveolar lavage

1 antibiotic-free days in the 7 days following bronchoalveolar lavage.

2 els of Aspergillus and total fungus in their bronchoalveolar lavage.

3 olating large numbers of cells by whole-lung bronchoalveolar lavage.

4 r infiltration, and E. coli colony counts in bronchoalveolar lavage.

5 ed numbers of lymphocytes and neutrophils in bronchoalveolar lavage.

6 All patients underwent bronchoalveolar lavage.

7 samples, including blood, plasma, urine and bronchoalveolar lavages.

8 We interrogated the bronchoalveolar lavage and blood viromes during the peri

9 oxic amyloids that can be recovered from the bronchoalveolar lavage and cerebrospinal fluids of criti

10 4 and CD8 T cell responses in blood, spleen, bronchoalveolar lavage and lung lymph nodes.

11 ion model, IL-10-producing CD4(+) T cells in bronchoalveolar lavage and lung were significantly decre

12 log(10) reductions in median viral loads in bronchoalveolar lavage and nasal mucosa compared with af

13 log(10) reductions in median viral loads in bronchoalveolar lavage and nasal mucosa, respectively, a

14 ided complete or near-complete protection in bronchoalveolar lavage and nasal swabs after SARS-CoV-2

15 fic IgG antibodies to the offending antigen, bronchoalveolar lavage and pathological features.

16 ues, we performed transcriptomic analyses of bronchoalveolar lavage and peripheral blood and proteomi

17 ition of ADAM10 reduces sEphrin-B2 levels in bronchoalveolar lavage and prevents lung fibrosis in mic

18 Double-stranded RNA in bronchoalveolar lavage and serum samples following lung

19 MIF decreases neutrophil infiltration to the bronchoalveolar lavage and tissue and simultaneously dec

20 Bronchoalveolar lavage and tissues were sampled for myco

21 ermate control animals through evaluation of bronchoalveolar lavage and tissues.

22 ignificantly higher S. maltophilia counts in bronchoalveolar lavages and lung tissue homogenates.

23 identify cross-tissue compartment (blood and bronchoalveolar lavage) and temporal proteomic signature

24 the lung, proinflammatory cytokine levels in bronchoalveolar lavage, and alveolar capillary leakage.

25 lished prescribing practices, reluctance for bronchoalveolar lavage, and dependence on a chain of tri

26 Eosinophils were examined in blood, bronchoalveolar lavage, and endobronchial biopsies 48 ho

27 Silica-induced SPP1 in lung tissue, bronchoalveolar lavage, and serum increased more in male

28 edding measured from nasal and throat swabs, bronchoalveolar lavages, and tissues was not reduced wit

29 ophils in bone marrow (BM), blood, lung, and bronchoalveolar lavage as well as airway hyperresponsive

30 tegrated study of human small airway tissue, bronchoalveolar lavage (BAL) and an experimental murine

31 TTR for bronchoalveolar lavage (BAL) and endotracheal aspirate (

32 bit AHR, increased numbers of eosinophils in bronchoalveolar lavage (BAL) and increased collagen cont

33 a methacholine test, airway inflammation in bronchoalveolar lavage (BAL) and lung tissue, and total

34 e expression and activity were determined in bronchoalveolar lavage (BAL) and lungs of human donors a

35 Conventional methods to identify HBEC in bronchoalveolar lavage (BAL) and wash (BW) have throughp

36 We characterized bronchoalveolar lavage (BAL) cells and fluid (BALF) prot

37 cDNA library derived from mRNA isolated from bronchoalveolar lavage (BAL) cells and leukocytes of sar

38 was to investigate the expression of iNOS in bronchoalveolar lavage (BAL) cells and tissue from centr

39 ring healthy aging, the proportions of blood bronchoalveolar lavage (BAL) classical monocytes peak in

40 increased eosinophilia and interleukin-5 in bronchoalveolar lavage (BAL) compared to sham-OVA mice.

41 We sought to determine the relationship of bronchoalveolar lavage (BAL) cytokine/chemokine expressi

42 perimental model of allergic asthma, matched bronchoalveolar lavage (BAL) fluid and plasma were colle

43 igatus Its performance has been validated on bronchoalveolar lavage (BAL) fluid and serum specimens,

44 ILC2s were isolated from blood and bronchoalveolar lavage (BAL) fluid before and after segm

45 ormed in nasopharyngeal aspirates (NPAs) and bronchoalveolar lavage (BAL) fluid before HCT.

46 ecommendation was made in favor of obtaining bronchoalveolar lavage (BAL) fluid for lymphocyte cellul

47 (log(10) mean value, 4.2), secretory IgA in bronchoalveolar lavage (BAL) fluid from immunized mice,

48 Unlike blood ILC2s, bronchoalveolar lavage (BAL) fluid ILC2s from asthmatic

49 We examined bronchoalveolar lavage (BAL) fluid leukocytes, cytokines

50 70 have been found in the sputum, serum, and bronchoalveolar lavage (BAL) fluid of asthma patients an

51 nsus sequences of viruses collected from the bronchoalveolar lavage (BAL) fluid of the animals.

52 rometry based proteome analysis of acellular bronchoalveolar lavage (BAL) fluid samples on an observa

53 Analysis of lung homogenates and bronchoalveolar lavage (BAL) fluid showed that LPS-induc

54 Inflammatory cells in bronchoalveolar lavage (BAL) fluid were assessed by flow

55 al swabs in the above-named transport media, bronchoalveolar lavage (BAL) fluid, and sputum.

56 red CD4+ T cells in whole blood, spleen, and bronchoalveolar lavage (BAL) fluid, but not in the lung

57 flammatory cytokines KC, IP-10, and IL-33 in bronchoalveolar lavage (BAL) fluid.

58 mediators TNF-alpha, IL-6, and leukocytes in bronchoalveolar lavage (BAL) fluids.

59 ics and function in rhesus macaque blood and bronchoalveolar lavage (BAL) following mucosal adenoviru

60 This analysis included bronchoalveolar lavage (BAL) for cell and fluid analysis

61 igated M. tuberculosis-specific responses in bronchoalveolar lavage (BAL) from persons with latent M.

62 We measured mtDNA concentrations in bronchoalveolar lavage (BAL) from subjects with and with

63 ALI was quantified by weight loss, bronchoalveolar lavage (BAL) inflammatory cell number, c

64 ollowing alveolar airspace infiltration, the bronchoalveolar lavage (BAL) neutrophil proteome is furt

65 Bronchoalveolar lavage (BAL) NK cells were immunophenoty

66 xpression in airway immune cells obtained by bronchoalveolar lavage (BAL) of individuals with latent

67 phenotype of NKG2C NK cells in the blood and bronchoalveolar lavage (BAL) of lung transplant recipien

68 We collected endobronchial brush (EB) and bronchoalveolar lavage (BAL) samples from 39 asthmatic p

69 ng injury in CIP, we prospectively collected bronchoalveolar lavage (BAL) samples in ICI-treated pati

70 Twenty consecutive bronchoalveolar lavage (BAL) samples were plated to stan

71 nd increased CD4+ T cell counts in blood and bronchoalveolar lavage (BAL) samples, it did not reduce

72 vation of cell counts and protein content in bronchoalveolar lavage (BAL) samples.

73 ng flow cytometry and a multiplex assay with bronchoalveolar lavage (BAL) specimens (n = 68) from 52

74 eporting for 259 adult inpatients submitting bronchoalveolar lavage (BAL) specimens for laboratory an

75 Thus, the diagnosis of PJP requires bronchoalveolar lavage (BAL) specimens or a decision to

76 However, no CMV DNA threshold exists in bronchoalveolar lavage (BAL) to differentiate pneumonia

77 Airway responsiveness was assessed, bronchoalveolar lavage (BAL) was performed, and lung cel

78 s of CD4(+) T and B cells in the spleens and bronchoalveolar lavage (BAL) were also observed.

79 ergic patients underwent SAC, and cells from bronchoalveolar lavage (BAL) were collected after 24 hou

80 Bronchoscopy and bronchoalveolar lavage (BAL) were performed 7 days post-

81 ased age, a low percentage of lymphocytes in bronchoalveolar lavage (BAL), a decreased transfer facto

82 flux of neutrophils and macrophages into the bronchoalveolar lavage (BAL), and human CD45(+) cells in

83 Subjects underwent bronchoalveolar lavage (BAL), and peripheral whole blood

84 linically indicated fiberoptic bronchoscopy, bronchoalveolar lavage (BAL), endobronchial brushings, a

85 mation as proven by increased cellularity in bronchoalveolar lavage (BAL), pulmonary transcriptomic p

86 t computed tomography, and bronchoscopy with bronchoalveolar lavage (BAL).

87 by an accumulation of CD4(+) T cells in the bronchoalveolar lavage (BAL).

88 f control (ACQ-7) underwent bronchoscopy and bronchoalveolar lavage (BAL).

89 ia also reduced inflammatory cell numbers in bronchoalveolar lavage (BAL).

90 aeruginosa, P. aeruginosa) were analyzed in bronchoalveolar lavage (BAL); and alveolar SGLT1 was ana

91 a higher number of lung cells obtained from bronchoalveolar lavages (BAL) than TAC pre-treatment alo

92 ctive viral replication and viral genomes in bronchoalveolar-lavage (BAL) fluid and nasal swab specim

93 Detection of toxicants in bronchoalveolar-lavage (BAL) fluid from patients with EV

94 ower respiratory tract specimens (sputum and bronchoalveolar lavage [BAL] fluid).

95 r advanced glycation end products (sRAGE) in bronchoalveolar lavage(BAL) and plasma were performed.

96 Bronchoalveolar lavages (BALs) from coinfected mice show

97 subjects had symptom scores, spirometry, and bronchoalveolar lavage before and after rhinovirus-induc

98 Bronchoalveolar lavage CD8 T cells showed significantly

99 ere analyzed based on serum antibody levels, bronchoalveolar lavage cell counts, lung histology, lung

100 Bronchoalveolar lavage cell mRNA levels of iNOS or iNOS

101 At two independent study sites, bronchoalveolar lavage cells from donors with latent tub

102 T-cell recruitment and activation within bronchoalveolar lavage cells of ACE2-high subjects was r

103 anscriptional profiling of cells isolated by bronchoalveolar lavage confirmed that influenza infectio

104 .RESULTSActivated CD4+ T cell frequencies in bronchoalveolar lavage correlated strongly with local C-

105 Donor testing was performed with bronchoalveolar lavage culture and PCR intraoperatively.

106 increased, and protein concentration in the bronchoalveolar lavage diminished, showing the impact of

107 GR-1) resulted in a significant decrease in bronchoalveolar lavage eosinophil counts, lung interleuk

108 This activation phenotype indicated by bronchoalveolar lavage eosinophil surface markers, as we

109 niae infection was diagnosed on the basis of bronchoalveolar lavage eosinophilia and blood findings.

110 b induced a rise in circulating eosinophils, bronchoalveolar lavage eosinophilia, and eosinophil pero

111 sham at day 13 showed an increased number of bronchoalveolar lavage eosinophils and increased express

112 l7r(cre) mice showed complete suppression of bronchoalveolar lavage eosinophils and mucous metaplasia

113 Before mepolizumab treatment, bronchoalveolar lavage eosinophils had more surface IL-3

114 im of this study was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicti

115 infected with Streptococcus pneumoniae, and bronchoalveolar lavage fluid (BALF) and lung CFU values

116 d temporal kinetics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumi

117 s the overlap in metabolites between matched bronchoalveolar lavage fluid (BALF) and plasma, identifi

118 tinfection and included cellular profiles in bronchoalveolar lavage fluid (BALF) and serum IgG and Ig

119 BORT reduced bronchoalveolar lavage fluid (BALF) and tissue eosinophi

120 understanding of the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients

121 We studied bronchoalveolar lavage fluid (BALF) from 36 patients wit

122 alysis on gene expression data from cells in bronchoalveolar lavage fluid (BALF) from COVID-19 patien

123 us 6B (HHV-6B) DNA is frequently detected in bronchoalveolar lavage fluid (BALF) from immunocompromis

124 with human MSCs when stimulated with LPS or bronchoalveolar lavage fluid (BALF) from patients with A

125 by using mass spectrometry) were measured in bronchoalveolar lavage fluid (BALF) from patients with N

126 gation & density-gradient fractionation from bronchoalveolar lavage fluid (BALF) in vivo.

127 cytomegalovirus (HCMV) DNA detection in the bronchoalveolar lavage fluid (BALF) indicates HCMV repli

128 Bronchoalveolar lavage fluid (BALF) levels of SOCS3 were

129 emic stroke caused a significant increase in bronchoalveolar lavage fluid (BALF) macrophages and neut

130 induce apolipoprotein E (APOE) expression by bronchoalveolar lavage fluid (BALF) macrophages from ast

131 We measured 13 cytokines in bronchoalveolar lavage fluid (BALF) of 88 children with

132 We measured iron and ferritin levels in the bronchoalveolar lavage fluid (BALF) of participants enro

133 a diffuse lung injury marked by increases in bronchoalveolar lavage fluid (BALF) protein and histoche

134 rformed a retrospective multicenter study on bronchoalveolar lavage fluid (BALF) samples obtained fro

135 All archived bronchoalveolar lavage fluid (BALF) samples that had pre

136 Neutrophils in bronchoalveolar lavage fluid (BALF) served as markers of

137 In parallel, adenosine levels in bronchoalveolar lavage fluid (BALF) were increased by ap

138 Lung function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs we

139 in mice resulted in increased OPN levels in bronchoalveolar lavage fluid (BALF).

140 2 ligands was significantly increased in the bronchoalveolar lavage fluid 48 hours after segmental al

141 o DC subsets (DC2/3 and DC5) are expanded in bronchoalveolar lavage fluid 8 h after lipopolysaccharid

142 urbiprofen augmented the release of IL-33 in bronchoalveolar lavage fluid after Alternaria challenge,

143 ased airway hyperreactivity to methacholine, bronchoalveolar lavage fluid albumin, and serum IgE leve

144 healthy volunteers and examine the impact on bronchoalveolar lavage fluid and blood MP repertoire.

145 ar lavage fluid was evaluated by microscopy; bronchoalveolar lavage fluid and blood were assessed by

146 PM2.5 increased neutrophil numbers and KC in bronchoalveolar lavage fluid and caused slight peribronc

147 recruitment of monocytes and neutrophils in bronchoalveolar lavage fluid and increased neutrophils i

148 fspring had lower white blood cell counts in bronchoalveolar lavage fluid and less pronounced peribro

149 ikingly reduced numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inf

150 ed a significantly reduced viral load in the bronchoalveolar lavage fluid and lower respiratory tract

151 ine expression in ILC2s and TH2 cells in the bronchoalveolar lavage fluid and lung tissue were assess

152 Flow cytometry and cytokine measurements in bronchoalveolar lavage fluid and lung tissue were follow

153 antioxidant responses were assessed in both bronchoalveolar lavage fluid and lung tissue.

154 nd primary lung monocytes/macrophages, mouse bronchoalveolar lavage fluid and lung tissues, and AHR i

155 Bronchoalveolar lavage fluid and matched blood were obta

156 We detected the presence of PRELP in human bronchoalveolar lavage fluid and showed that PRELP can b

157 bronchitis, perivasculitis, and increases in bronchoalveolar lavage fluid cell numbers were detected

158 Analysis of bronchoalveolar lavage fluid collected from human patien

159 eduction of eosinophil and T cell numbers in bronchoalveolar lavage fluid compared with those in dilu

160 2 223K/K mice had persistent eosinophilia in bronchoalveolar lavage fluid compared with wild-type and

161 a, accompanied with significant reduction in bronchoalveolar lavage fluid concentration of IL-5, a cy

162 Bronchoalveolar lavage fluid demonstrates greater than 2

163 , IL-33(KO)/Tg+ mice had complete absence of bronchoalveolar lavage fluid eosinophilia, accompanied w

164 Bronchoalveolar lavage fluid from 23 lean, 12 overweight

165 Microbiology was done on bronchoalveolar lavage fluid from all patients and venti

166 ein levels were significantly upregulated in bronchoalveolar lavage fluid from HIV-infected smokers,

167 In bronchoalveolar lavage fluid from humans with ARDS, gut-

168 patients with severe asthma compared with in bronchoalveolar lavage fluid from individuals without as

169 was consistently inhibited by treatment with bronchoalveolar lavage fluid from inhibitory kappaB kina

170 s of IL-1beta and IL-8 rapidly determined in bronchoalveolar lavage fluid from patients randomised to

171 vo, histone-C1INH complexes were detected in bronchoalveolar lavage fluid from patients with acute re

172 he protein expression patterns in plasma and bronchoalveolar lavage fluid from patients with ARDS.

173 We use our method to characterize bronchoalveolar lavage fluid from patients with asthma,

174 nase activities were quantified in serum and bronchoalveolar lavage fluid from patients with CF, asth

175 anges in the miRNA composition of EVs in the bronchoalveolar lavage fluid from patients with IAV-indu

176 pulmonary eosinophilia were measured in the bronchoalveolar lavage fluid from patients with mild ast

177 r, specific ceramide species were altered in bronchoalveolar lavage fluid from patients with severe a

178 Low concentrations of IL-1beta and IL-8 in bronchoalveolar lavage fluid have been validated as effe

179 al aimed to determine whether measurement of bronchoalveolar lavage fluid IL-1beta and IL-8 could eff

180 ry inflammation, eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adop

181 eficient mice had fewer DEP exposure-induced bronchoalveolar lavage fluid immune cells and proinflamm

182 s, angiotensin-converting enzyme activity in bronchoalveolar lavage fluid increased 3.2-fold in elder

183 Following repetitive O3 exposure, higher bronchoalveolar lavage fluid inflammatory cells were obs

184 Bronchoalveolar lavage fluid LT levels were increased in

185 I are seen in HAECs, in association with low bronchoalveolar lavage fluid mitochondrial DNA and more

186 cells in lymph nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques

187 we report that SOCS3 protein was elevated in bronchoalveolar lavage fluid of both virus- and bacteria

188 med that leukotriene levels are increased in bronchoalveolar lavage fluid of HIV-infected patients.

189 -promoting cytokines and chemokines into the bronchoalveolar lavage fluid of Lipa(-/-) mice.

190 oxidase were more frequently detected in the bronchoalveolar lavage fluid of lung transplant patients

191 f RAGE was determined in protein, serum, and bronchoalveolar lavage fluid of mice and lungs and serum

192 shown to reduce the total number of cells in bronchoalveolar lavage fluid of mice challenged with hou

193 rply increased the levels of UDP detected in bronchoalveolar lavage fluid of mice.

194 NF-alpha, IL-9, CXCL1, CCL2, and CCL5 in the bronchoalveolar lavage fluid of RSV-infected mice, witho

195 HDM, elevated the levels of chemerin in the bronchoalveolar lavage fluid of WT mice.

196 y/severity; (2) RSV-specific CD8+ T cells in bronchoalveolar lavage fluid preinfection (subjects with

197 e exposure of vehicle-treated rats increased bronchoalveolar lavage fluid protein, albumin, neutrophi

198 Higher numbers of Asm(-/-) T cells in bronchoalveolar lavage fluid released lower levels of IL

199 resulted in hypovirulence, while analysis of bronchoalveolar lavage fluid revealed that tumor necrosi

200 istress syndrome 1, we used paired serum and bronchoalveolar lavage fluid samples obtained within 48

201 ynthase similarly attenuated the increase in bronchoalveolar lavage fluid SOCS3 noted in lungs of mic

202 ption factor GATA3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of

203 ation and analysis of Aspergillus conidia in bronchoalveolar lavage fluid using the combination of tr

204 determined by whole-body plethysmography and bronchoalveolar lavage fluid was analyzed for cellular c

205 Bronchoalveolar lavage fluid was collected from 23 stero

206 Bronchoalveolar lavage fluid was collected from patients

207 Bronchoalveolar lavage fluid was evaluated by microscopy

208 had a separate donor; however, pretransplant bronchoalveolar lavage fluid was only available from the

209 Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower

210 ity in lymphoid tissues and Th2 responses in bronchoalveolar lavage fluid), they also accumulate func

211 rol and PNE rat pups: 1) the 5-HT content in bronchoalveolar lavage fluid, 2) the apneic response to

212 chronic rhinosinusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen c

213 HIV has been recovered from cell-free bronchoalveolar lavage fluid, alveolar macrophages, and

214 concentration was also induced in the serum, bronchoalveolar lavage fluid, alveolar type II epithelia

215 d lower total cell counts and neutrophils in bronchoalveolar lavage fluid, and had earlier influx of

216 nd disaturated PC in lung tissue homogenate, bronchoalveolar lavage fluid, and lung LB was increased

217 were sensitized and challenged with OVA and bronchoalveolar lavage fluid, and the lungs were collect

218 d tumor-promoting cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expressio

219 fferential cell counts were performed on the bronchoalveolar lavage fluid, followed by histological a

220 und significantly elevated total proteins in bronchoalveolar lavage fluid, higher parasitemia and tis

221 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasa

222 ay and analyses of the injury markers in the bronchoalveolar lavage fluid, respectively.

223 tained pulmonary or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflam

224 In serum and bronchoalveolar lavage fluid, total anti-IAV IgG and IgA

225 levels of iron-carrier proteins in cells or bronchoalveolar lavage fluid.

226 t least 10(4) colony forming units per mL of bronchoalveolar lavage fluid.

227 r protein leak and inflammatory cells in the bronchoalveolar lavage fluid.

228 Recruited eosinophils were enumerated in bronchoalveolar lavage fluid.

229 lung while also reducing LPA 18:2 content in bronchoalveolar lavage fluid.

230 rase chain reaction using RNA extracted from bronchoalveolar lavage fluid.

231 ction and asthma-associated cytokines in the bronchoalveolar lavage fluid.

232 n, and surfactant protein A concentration in bronchoalveolar lavage fluid.

233 release from lung epithelium as detected in bronchoalveolar lavage fluid.

234 in (TSLP), IL-9, and IL-13, but not IL-5, in bronchoalveolar lavage fluid.

235 the macrophage chemoattractant MCP-1 in lung bronchoalveolar lavage fluid.

236 llikrein (KLK) 5 and KLK14, were assessed in bronchoalveolar lavage fluid.

237 and was highly correlated with IFN levels in bronchoalveolar lavage fluid.

238 ed the influx of inflammatory cells into the bronchoalveolar lavage fluid.

239 d resulted in IL-1beta accumulation into the bronchoalveolar lavage fluid.

240 Quantitative bacterial culture of bronchoalveolar lavage fluids (BALF) is labor-intensive,

241 ncreased ATP concentrations were reported in bronchoalveolar lavage fluids of asthmatic patients.

242 ocessed 25-kDa IL-33 protein was detected in bronchoalveolar lavage fluids without any exogenous stim

243 Positive bronchoalveolar lavage for GM or Aspergillus cultures wa

244 s are reticent to perform aerosol-generating bronchoalveolar lavages for galactomannan testing and mi

245 AMs were obtained from bronchoalveolar lavage from healthy donors or patients w

246 AMs were recovered from bronchoalveolar lavage from healthy subjects and patient

247 leukocytes, macrophages, and neutrophils in bronchoalveolar lavage from O3-exposed mice.

248 ts antifibrotic properties and is reduced in bronchoalveolar lavage from patients with IPF.

249 ial alarmins were measured longitudinally in bronchoalveolar lavages from lung transplant recipients

250 sed eosinophil and neutrophil populations in bronchoalveolar lavages from mice with asthma.

251 ol for invasive pulmonary aspergillosis with bronchoalveolar lavage galactomannan and cultures perfor

252 ent in anastomotic plaques with reduction in bronchoalveolar lavage galactomannan values (7.48-2.15 n

253 L-1alpha positively correlated with elevated bronchoalveolar lavage IL-8 levels (r(2) = 0.6095, p < 0

254 were most frequently cultured from blood and bronchoalveolar lavage in humans and lungs in animals.

255 antibiotic-free days in the 7 days following bronchoalveolar lavage in the intention-to-treat analysi

256 throat of all of the macaques, as well as in bronchoalveolar lavages; in one macaque, we observed pro

257 Bronchoscopy with bronchoalveolar lavage is very useful in its diagnosing.

258 Blood, bronchoalveolar lavage, large proximal, and small distal

259 as adjuvants resulted in elevated serum and bronchoalveolar lavage levels of anti-P6-specific IgG an

260 OVA-induced increases in bronchoalveolar lavage lymphocytes, eosinophils, IL-13,

261 rtension, low peripheral blood, and elevated bronchoalveolar lavage lymphocytes.

262 Bronchoalveolar lavage macrophages were stimulated in vi

263 cell therapy groups, despite a reduction in bronchoalveolar lavage neutrophils.

264 ll as 16S ribosomal RNA sequencing data from bronchoalveolar lavage obtained as part of the COMET-IPF

265 NA and host total RNA were isolated from 203 bronchoalveolar lavages obtained from 112 patients post-

266 NA and host total RNA were isolated from 189 bronchoalveolar lavages obtained from 116 patients post

267 Lung fibroblasts (LFs) were derived from bronchoalveolar lavage of BOS patients.

268 highly autofluorescent cells present in the bronchoalveolar lavage of cigarette smokers.

269 IV-1-infected airway macrophages obtained by bronchoalveolar lavage of HIV-1-infected individuals.

270 used to characterize macrophages from human bronchoalveolar lavage of HIV-infected and -uninfected s

271 rleukin-1 alpha (IL-1alpha) was increased in bronchoalveolar lavage of lung transplant recipients gro

272 des of the biculture and was also present in bronchoalveolar lavage of lung transplantation patients.

273 was also demonstrated in vivo by challenging bronchoalveolar lavage of SET-M33-treated mice with LPS,

274 Uninfected AMs were isolated from bronchoalveolar lavage of young (3 mo) and old (18 mo) C

275 marker of PARP activation) and IL-6, in the bronchoalveolar lavage or the lung tissue, and histology

276 phenotype and function of T lymphocytes from bronchoalveolar lavage or the peripheral blood.

277 oneal (p = 0.037), systemic (p = 0.019), and bronchoalveolar lavage (p = 0.011) quantitative bacteria

278 After bronchoalveolar lavages, piglets developed respiratory d

279 lature was supported by normal histology and bronchoalveolar lavage protein levels.

280 Incubating control cells with disease bronchoalveolar lavage recapitulated the aberrant functi

281 ximal and distal airways (bronchial wash and bronchoalveolar lavage, respectively), as well as mucosa

282 of interferon and interleukin-6 pathways in bronchoalveolar lavage samples and repression of natural

283 y evaluating both upper airway and acellular bronchoalveolar lavage samples from 49 subjects from thr

284 have previously demonstrated that acellular bronchoalveolar lavage samples from half of the healthy

285 rst identified two redondovirus genomes from bronchoalveolar lavage samples from human lung donors.

286 Bronchoalveolar lavage samples from Ugandan patients wit

287 Although sputum and bronchoalveolar lavage samples provide the highest diagn

288 navirus testing on pooled nasopharyngeal and bronchoalveolar lavage samples taken from patients who h

289 tes for the galactomannan assay in serum and bronchoalveolar lavage samples were 61.3% and 57.1%, res

290 Bronchoalveolar lavage sampling may provide useful diagn

291 Plasma and bronchoalveolar lavage soluble receptor for advanced gly

292 ubset of 61 patients, respiratory specimens (bronchoalveolar lavage specimens, tracheal aspirates, an

293 ch on-ECMO experimental step, but the lowest bronchoalveolar lavage sRAGE levels were obtained at min

294 s on radiographs and reduced virus titres in bronchoalveolar lavages twelve hours after the first dos

295 Bronchoalveolar lavage was associated with a small and t

296 Bronchoalveolar lavage was done in all patients, with co

297 Inflammation in bronchoalveolar lavage was increased in old versus young

298 Three days after instillation, bronchoalveolar lavage was performed and plastic-adheren

299 asis of chest CT findings, bronchoscopy with bronchoalveolar lavage was performed.

300 E. coli colony-forming units in bronchoalveolar lavage were reduced in both cell therapy