ライフサイエンスコーパス: cable

1 luding a resistive strain gauge and an ionic cable.

2 d by constricting a supracellular actomyosin cable.

3 s not change the regions of alternans in the cable.

4 ough corrosion-free, lightweight fiber optic cables.

5 associated with cortical longitudinal actin cables.

6 and this decrease is dependent on the actin cables.

7 tions by continual polymerization of F-actin cables.

8 locations using optical fibres or microwave cables.

9 hat the junctional regions act like inverted cables.

10 cargos along F-actin bundles known as actin cables.

11 clumps, meshworks or double rings, and stars/cables.

12 cribed state on selectively stabilized actin cables.

13 s-linking parameters generates thicker actin cables.

14 he spatial and dynamical properties of actin cables.

15 risome protein in the stabilization of actin cables.

16 egates most organelles along polarized actin cables.

17 filaments forming long and flexible filament cables.

18 (i) It produced thicker, more pronounced HA cables.

19 o the dissociation of Gic1 from the filament cables.

20 rol unit via a bundle of flexible tubing and cables.

21 pulses and/or the formation of supracellular cables.

22 ber cells accumulated disoriented transverse cables.

23 e of the platform for constructing molecular cables, 1,3,5-trifluorenylcyclohexane (TFC) and its difl

24 the formation of the leading edge actomyosin cable, a structure that is essential for wound closure.

25 esults in long, bent, and hyper-stable actin cables, accompanied by defects in secretory vesicle traf

26 earing, MII-contractility-dependent node-and-cable actin network in the cell body cortex.

27 istinct regulatory positions for tropomyosin cables along thin filaments on actin dominated by stereo

28 lays misoriented and architecturally altered cables, along with impaired secretory vesicle traffic.

29 Cable analysis of the neurons indicated that the axonal

30 he influence of fiberoptic/electrophysiology cables, analyzing specified portions of time, and defini

31 an independent contractile unit, with actin cables anchored end-on to cadherin complexes at tricellu

32 configuration, aligned with the apical actin cable and adherens-junctions within chick and mouse neur

33 from the newly attached cells into the actin cable and defusion from the previously lined cells, ther

34 ic percussive test values (PTVs) measured by cabled and wireless electronic percussive testing (EPT)

35 lates Bnr1 activity, but also binds to actin cables and aligns them along the mother-bud axis.

36 myces pombe, the formin For3 nucleates actin cables and also co-operates for CAR assembly during cyto

37 o an increased number and stability of actin cables and causes misplacement of the division site in c

38 nnect to follower cells via peripheral actin cables and discontinuous adherens junctions, and lead mi

39 ions between the cortical longitudinal actin cables and plasma membrane in the shank region of growin

40 support were evaluated while walking without cables and reacting to the perturbations.

41 ed both the coupling of the nucleus to actin cables and the oriented flow of the cables necessary for

42 of electric cables (polyvinyl chloride, PVC-cables) and plastic garbage bag (polyethylene, PE-bags),

43 protrusions and/or a contractile actomyosin cable, and these actin structures drive wound closure.

44 e industry (electronic, aerospace, wires and cables, and textiles) has been built around them.

45 s localizations to cytoplasmic puncta, actin cables, and the contractile ring.

46 ent density J(E), especially in high-current cables, and the danger of quenches.

47 d into large bundles of fibrils, or collagen cables, and the number of these cables (but not their si

48 tial for Bud14 functions in regulating actin cable architecture and function in vivo.

49 Actin cables are dynamic structures regulated by assembly, sta

50 In a yeast cell undergoing budding, cables are in constant dynamic turnover yet some cables

51 Actin cables are linear cytoskeletal structures that serve as

52 ost from cell extremities, actin patches and cables are reorganized into actin bodies, which are stab

53 romyces cerevisiae, formin-polymerized actin cables are spatially organized and aligned along the mot

54 er in a single assembly, termed as molecular cable, are promising next-generation materials for effec

55 A supracellular actomyosin cable around the wound coordinates cellular movements an

56 ating the assembly of a supracellular myosin cable around the wound that coordinates cell migration.

57 for the efficient assembly of an actomyosin cable around the wound, and constitutively active myosin

58 junctions, where filamentous actin (F-actin) cables assemble.

59 uided by the genetic requirements for proper cable assembly and dynamics, we show that seven evolutio

60 the basis for comprehensive understanding of cable assembly and regulation.

61 tes actin filament binding to regulate actin cable assembly and stability in cells.

62 ey target for cell cycle regulation of actin cable assembly in budding yeast, and suggests an underly

63 kinase 1 activity also was found to regulate cable assembly in vivo.

64 fimbrin is a critical Cdk1 target for actin cable assembly regulation in budding yeast.

65 metaphase cells preferentially promote actin cable assembly through cyclin-dependent kinase 1 (Cdk1)

66 nsights into mechanisms for regulating actin cable assembly, we reconstituted the assembly process in

67 unction is required for proper Bni1-mediated cable assembly.

68 from the wound edge and defective actomyosin cable assembly.

69 iderm, proliferation, or supracellular actin cable assembly.

70 When nurse cells contract, actin cables associate laterally with the nuclei, in some case

71 ikely driven by contraction of an actomyosin cable at the boundary between the amnion and serosa.

72 ng contraction of a pluricellular actomyosin cable at the free edge.

73 ion or by forces generated by the actomyosin cable at the leading edge.

74 ite the presence of a contractile actomyosin cable at the periphery of the wound.

75 ng wound closure, a supracellular actomyosin cable at the wound edge coordinates cells, while actin-b

76 AJs promotes the assembly of an actin-myosin cable at the wound margin; contraction of the actin cabl

77 ia rapidly assemble a contractile actomyosin cable at their leading edge, as well as dynamic filopodi

78 central element is composed of two parallel cables at a distance of approximately 100 nm, which are

79 ates planar-polarized assembly of actomyosin cables at tissue boundaries by affecting dynamics of mem

80 , oxygen, and pH indicated low or no in situ cable bacteria activity at all sites.

81 As cable bacteria are present in many seasonally hypoxic sy

82 mmer, when bottom water hypoxia develops and cable bacteria are undetectable, the phosphorus associat

83 ance, activity, and biogeochemical impact of cable bacteria at 12 Baltic Sea sites.

84 Cable bacteria can limit sulfide release by promoting ir

85 ow that cells within individual filaments of cable bacteria display a remarkable dichotomy in biosynt

86 However, filamentous cable bacteria display a unique metabolism in which redo

87 Cable bacteria dominate the sediment geochemistry in win

88 The specific electrogenic metabolism of cable bacteria generates a large buffer of sedimentary i

89 at the long-range electrogenic metabolism of cable bacteria leads to a dissolution of iron sulfides i

90 Cable bacteria of the family Desulfobulbaceae form centi

91 tronema enrichment, the genomes suggest that cable bacteria oxidize sulfide by reversing the canonica

92 Our geochemical data suggest that cable bacteria promote conversion of iron monosulfides t

93 energy conservation, and filament growth in cable bacteria remains enigmatic.

94 Cable bacteria show limited organotrophic potential, may

95 Cable bacteria were mostly absent in sediments overlain

96 erlands), which suggest that the activity of cable bacteria, a recently discovered group of sulfur-ox

97 ion of iron-oxide-bound phosphorus driven by cable bacteria, as observed in this study, contributes t

98 and specifically the population dynamics of cable bacteria, can also induce strong seasonality in se

99 nes (from 197 gene families) conserved among cable bacteria.

100 We found no relationship between cable bacterial densities and macrofaunal abundances, sa

101 At seasonally hypoxic sites, cable bacterial densities correlated linearly with the s

102 At sites that were temporarily reoxygenated, cable bacterial densities were low.

103 This model proposes that an active cable bacterium gains energy in the anodic, sulfide-oxid

104 cases inducing nuclear turning so that actin cables become partially wound around the nuclei.

105 ed transport than the yeast form, with actin cables being essential for the transition.

106 se matrices do not resemble the rope-like HA cables but occur in distinct sheets or rafts that can ca

107 of inhibition does not alter actin levels in cables but, instead, cable shape and functionality.

108 or collagen cables, and the number of these cables (but not their size) increases in desmin knockout

109 cables enhanced leukocyte adhesion to these cables, but it also had several unexpected effects.

110 d the total amount of PAEs released from PVC-cables by a factor of up to 5, whereas they had no influ

111 hibition of F-actin or laser ablation of the cable causes neural fold separation.

112 ery different from optical and/or electrical cable communications, acoustic waves can be simply and e

113 Actin cables, composed of actin filament bundles nucleated by

114 Loss of these cables compromises orderly apical targeting of vesicles.

115 h the fish surface, and an extended parylene cable connected the underwater chest electrodes with the

116 and parallel phases with straight or curved cables, consistent with observations of cells overexpres

117 ical surface, finding that apical actomyosin cables contract against the apoptotic nucleus, which its

118 , the mechanical coupling between actomyosin cable contraction and cell crawling acts as a large-scal

119 ing force generated by a tissue-level myosin cable contributes to SG invagination.

120 ults suggest that a heterogeneous actomyosin cable contributes to the fluidization of the leading edg

121 ses the question: how is the length of these cables controlled?

122 lled out with dendrites and axons optimizing cable costs and conduction time while keeping the connec

123 ensing technology coupled to existing subsea cables (dark fiber) allows observation of ocean and soli

124 mple in vitro reconstitution system leads to cable defects that closely approximate in vivo cable phe

125 In contrast, control cable deletions caused kinase-on output, whereas inserti

126 es of sensitive animals when planning future cable deployments and predicting their environmental eff

127 Cable deployments in coastal waters are increasing world

128 usly unappreciated ATP requirement for actin cable disassembly.

129 nism of wound healing, we propose actomyosin cable-driven local tissue fluidization as a conserved mo

130 A cable-driven robot was used to train nine patients with

131 ibit aberrant equatorial clustering of actin cables during ring assembly and are particularly suscept

132 Axons act like cables, electrically wiring the nervous system.

133 The two parallel cable elements form twisted helical structures that are

134 al pillars (~200 nm apart), from which actin cables emerge and grow into the mother cell.

135 on of printed circuit boards and copper-core cables emitted large amounts of OM with Br-rich inclusio

136 ches we unveil the presence of an actomyosin cable encircling TEMs.

137 Here, we first derive a full cable energy function for cortical axons based on classi

138 y (HH) neuronal equations and then apply the cable energy function to precisely estimate the energy c

139 redicted, the addition of heavy chains to HA cables enhanced leukocyte adhesion to these cables, but

140 PT device were significantly higher than the cabled EPT device (P <0.05), indicating lower implant st

141 reless EPT device gives PTVs higher than the cabled EPT device, indicating lower implant stability, a

142 as they are actively transported along actin cables, Exo70p displays actin-independent localization t

143 Cable externalization was found to be more common in the

144 edictors of electrical lead failure included cable externalization, higher left ventricular ejection

145 2212 racetrack coils wound with a Rutherford cable fabricated from wires made with a new precursor po

146 rs away if engagement of MyoE(MYO5) to actin cables fails.

147 ure (30%), battery failure (19%), or patient cable failure (14%), whereas only 13% were because of pu

148 ermanent networks of seabed and water-column-cabled (fixed) and docked mobile platforms is presently

149 This movement, retrograde actin cable flow (RACF), is similar to retrograde actin flow i

150 these nuclear movement defects, dorsal actin cable flow was nondirectional in cells lacking emerin.

151 h voltage direct current (HVDC) transmission cable for domestic electricity supply.

152 ase the lifespan and sustainability of power cables for electronics and energy applications.

153 ing the system with micro-drives or flex-PCB cables for recording from multiple brain regions, as wel

154 age pre-existing submarine telecommunication cables for seismic monitoring.

155 the in vivo regulatory mechanisms for actin cable formation are less clear.

156 t be favorable enough to promote tropomyosin cable formation but not so tenacious that polymerization

157 Formins drive cable formation by promoting actin nucleation and by acc

158 ), both E-cadherin down-regulation and actin-cable formation fail, thus resulting in open epidermal g

159 w tropomyosins contribute mechanistically to cable formation has been unclear, but genetic studies de

160 gnaling, whereas the reduction in hyaluronan cable formation induced by V3 expression is mediated by

161 Actomyosin cable formation is accompanied by junctional rearrangeme

162 by explosive tropomyosin polymerization once cable formation is initiated on actin filaments.

163 R1, but not BNI1, leads to severe defects in cable formation, polarized secretion, and cell growth, s

164 ering of formins at cell tips promotes actin cable formation.

165 y as Bud14 in regulating Bnr1-mediated actin cable formation.

166 metry, we identified components of the actin cables formed in yeast extracts, providing the basis for

167 ompletion of spinal closure is imminent, the cable forms a continuous ring around the neuropore, and

168 nd regulate the formation of radiating actin cables from this site.

169 sophila embryo is mediated by apical F-actin cables generated by the formin-family protein Diaphanous

170 es are in constant dynamic turnover yet some cables grow from the bud neck toward the back of the mot

171 nurse cells, continuous filopodia-like actin cables, growing from the plasma membrane and extending t

172 Yet linking quantum-enabled devices with cables has proved difficult because most cavity or circu

173 crease in number in a model of fibrosis, and cables have unique interactions with collagen-producing

174 membrane helix (TM2), a five-residue control cable helix at the membrane-cytoplasm interface, and a f

175 up interface, causing a break in the control cable helix to attenuate the register mismatch and enhan

176 rs and belowground buried resistance heating cables in each of 72-7.0 m(2) plots.

177 es are delivered by myosin-V on linear actin cables in fission yeast cytokinesis.

178 le actin structures with features similar to cables in living cells.

179 t the activity of MyoVc to specialized actin cables in order to co-ordinate and target the final stag

180 We identify oriented actomyosin cables in the medial SpM-SHF as a potential Wnt5a-mediat

181 binds to actin filaments and organizes actin cables in vivo.

182 intain steady-state lengths similar to actin cables in vivo.

183 ndergo processive movement along actin-Cdc8p cables in vivo.

184 t the wound margin; contraction of the actin cable, in turn, closes the wound.

185 ire, multifilament form that can be wound or cabled into arbitrary geometries and will be especially

186 d to transform telecommunication fiber-optic cables into dense seismic arrays that are cost effective

187 utput, whereas insertions at the TM2-control cable junction caused kinase-off output.

188 ommunity Atmosphere Biosphere Land Exchange (CABLE) land surface model.

189 scale material geometries including anchors, cables, lattices and webs, as well as functional materia

190 having thousands of branch points and total cable length >10 mm (Otopalik et al., 2017a; 2017b).

191 we describe a novel molecular mechanism for cable length control inspired by recent experimental obs

192 redictions of the antenna mechanism of actin-cable length control.

193 activity is crucial in vivo for proper actin cable length, shape, and velocity and, in turn, efficien

194 We quantify animal-to-animal variability in cable lengths (CV = 0.4) and branching patterns in the G

195 We compute the probability distribution of cable lengths as a function of several experimentally tu

196 Cable-like copper oxide/carbon-nitride core-shell nanost

197 MSC comparatively, through the formation of cable-like hyaluronic acid structures.

198 Axons are cable-like neuronal processes wiring the nervous system.

199 form of HA (HC-HA) leads to the formation of cable-like structures that promote adhesion of leukocyte

200 observed and characterized two distinct Cdc8 cables loading and spreading cooperatively on individual

201 n V, Myo2, responds by relocalizing to actin cables, making it the fastest response documented.

202 as roads, rail tracks, pipelines, fences and cables, many of which divide the landscape and limit ani

203 d severely underestimates energy cost in the cable model by 20-70%.

204 is model with an anatomically realistic axon-cable model of motoneurons, interneurons, and myelinated

205 Finally, a cable model of the OHC, which can match our data, indica

206 ptotic analysis of a two-compartment passive cable model, given a pair of time-dependent synaptic con

207 for a patch of membrane within a distributed cable model.

208 We also show, using simulations of 720 cable models spanning a broad range of geometries and pa

209 These actin cables move nuclei away from ring canals.

210 to actin cables and the oriented flow of the cables necessary for nuclear movement and centrosome ori

211 Thus Hof1 tunes formins to sculpt the actin cable network.

212 n "superhighways" composed of parallel actin cables nucleated by formins from the plasma membrane [4]

213 The increase in cable number is accompanied by increased muscle stiffnes

214 A new cabled observatory atop Axial Seamount on the Juan de Fu

215 ucted at a coastal artificial reef through a cabled observatory system, which allowed gathering under

216 were captured in real time by a new seafloor cabled observatory, revealing the timing, location, and

217 Actin cables of budding yeast are bundles of F-actin that exte

218 ectrical wave propagation in one dimensional cables of healthy and failing cells.

219 h are oriented perpendicular to two parallel cables of the lateral element arranged at a distance of

220 ng F-actin, the structure of the tropomyosin cable on actin is uncertain.

221 and then its polymerization into continuous cables on the filament surface must be precisely tuned t

222 t-of-things (IoT) sensors, freeing them from cables or batteries and thus making them especially usef

223 nct spatial architecture of the apical actin cables (or actin cap) facilitates rapid biophysical sign

224 r" domain of Hof1, and its deletion leads to cable organization defects in vivo.

225 odels of seamless and continuous tropomyosin cables over the F-actin substrate, which were optimized

226 ique that enables determinations of multiple cable parameters in action potential-firing fibres inclu

227 ble defects that closely approximate in vivo cable phenotypes caused by disrupting the corresponding

228 c materials, an insulation layer of electric cables (polyvinyl chloride, PVC-cables) and plastic garb

229 In neither drought did CABLE predict that trees would have reached critical PLC

230 cells, would enhance leukocyte binding to HA cables produced in response to poly(I:C).

231 arising at least partly from differences in cable properties and the nonlinear behaviour of the resp

232 Macroscopic branching patterns and fine cable properties are variable within and across neuron t

233 at source estimation methods, as well as the cable properties of neurons, which all assume ohmic extr

234 of local field potentials, as well as on the cable properties of neurons.

235 Our findings suggest that cable properties play a central role in determining wher

236 tensively reflect the influence of dendritic cable properties.

237 ed formation of the perinuclear apical actin cables protects the nuclear structural integrity from ex

238 The use of the cable-reconstitution system to test roles for the key ac

239 idue in the Tsr N-terminal linker or control cable reduces conformational heterogeneity at the N-term

240 We hypothesized that actin cables regulate the processive properties of MyoVc.

241 Myo2 immobilized on cables releases its secretory cargo, defining a new rigo

242 t alter the sidechain environment of control cable residues at the membrane core-headgroup interface,

243 lar F-actin network, which includes an actin cable running along the neural fold tips.

244 ide a comprehensive model of the tropomyosin cable running continuously on F-actin.

245 35%-45% reduction in root mean square error) CABLE's previous predictions of latent heat fluxes durin

246 e insertions or deletions in the TM2-control cable segment of Tsr.

247 Such actin-cable segment switching occurs favorably at high curvatu

248 rd traction force signifies successful actin-cable segment switching.

249 t alter actin levels in cables but, instead, cable shape and functionality.

250 Prominent actin cables, spanning several cells, are abundant both in cul

251 As with a cable, spine neck resistance (R(neck)) increases with in

252 mmatory matrix leads to dissolution of HC-HA cable structures and abolishes leukocyte adhesion.

253 egoing mechanisms, the model generates actin cable structures and dynamics similar to those observed

254 r simulations reproduce the particular actin cable structures in myoVDelta cells and predict the effe

255 ated from control mice generated HA-enriched cable structures in the ECM, providing a substrate for m

256 s and organizes actin filaments into ordered cable structures.

257 smooth muscle cells produced spontaneous HA "cable" structures, without additional stimuli, that were

258 posed of discontinuous struts and continuous cables, such systems are only structurally stable when s

259 ormation of long microtubule-based cytosolic cables suggesting a role in microtubule formation and st

260 We used an optical fiber from the cable supporting the Monterey Accelerated Research Syste

261 retrograde flow of dorsal perinuclear actin cables, supporting the recently proposed function for th

262 emission into the MRI scanner and prevented cable/surface pad heating during imaging, while preservi

263 he spider web applies the concept of elastic cables taking only axial deformation into account.

264 perstructures are composed of dimeric double-cable tape-like structures that, in turn, are supercoile

265 . (2015) identifies an apicobasal actomyosin cable that characterizes apoptotic cells and contributes

266 to build an atomic model of the tropomyosin cable that fits onto the actin filament between the tip

267 tail overlapping domain to form a continuous cable that wraps around the F-actin helix.

268 O2-induced ROS were found to decompose actin cables that are driving meiotic chromosome mobility, an

269 he assembly of an organized network of actin cables that direct polarized secretion.

270 anizes into fungal-like cortical patches and cables that extend into hyphal-like structures.

271 quired for the assembly of an array of actin cables that facilitate polarized vesicle delivery and da

272 les: a relatively stable internal network of cables that moves in concert with and appears to be link

273 thesizing leukocyte-adhesive hyaluronan (HA) cables that remain attached to their cell surfaces.

274 These actin filaments bundle to form actin cables that span the cell and guide the movement of vesi

275 sufficient to reconstitute the formation of cables that undergo polarized turnover and maintain stea

276 x of F-actin to form continuous superhelical cables that wrap around the actin filaments.

277 ingle cell and spatially coupled homogeneous cable, the interplay between alpha and tau affects the d

278 home cells contained long longitudinal actin cables, the short Li1 fiber cells accumulated disoriente

279 In contrast with classical cable theory predictions, the persistent sodium current

280 Based on cable theory, the unique 30 nm width compartment (the ex

281 ndocytosis and strong stabilization of actin cables, thereby revealing a selective and previously una

282 promote formin-dependent nucleation of actin cables, thus expanding our understanding of how specific

283 redress this problem by using a fiber-optic cable to couple an infrared (IR) laser to a mass spectro

284 motions might be relayed through the control cable to reach the input AS1 helix of HAMP by constructi

285 a perinuclear actin meshwork connects actin cables to nuclei via actin-crosslinking proteins such as

286 ene delivery and implantation of fiber-optic cables to produce light-dependent activation of a small

287 Instead, we suggest that the Tsr control cable transmits input signals to HAMP by modulating the

288 uple the nucleus to dorsal perinuclear actin cables undergoing retrograde flow.

289 study reinforces the efficacy of underwater cabled video-observatories as a reliable tool for long-t

290 field components of the EMF emitted by HVDC cables we found that there were DC and unexpectedly AC c

291 interactions between ECM cells and collagen cables were also observed and reconstructed by serial bl

292 mages of the end facets of these fiber optic cables were captured using the smart phone and processed

293 A pulse generator and two extension cables were implanted in a second surgery 3-4 weeks late

294 effects of increased gait stability once the cables were removed.

295 s that collagen is organized into perimysial cables which increase in number in a model of fibrosis,

296 formation of linear multicellular actomyosin cables, which depend on Diaphanous for their nucleation.

297 we focused on Saccharomyces cerevisiae actin cables, which provide polarized tracks for intracellular

298 sion results in externalization of conductor cables, with a higher risk of electrical failure.

299 e (DEP) were the main PAEs released from PVC-cables, with mass fractions as high as 9.5 +/- 1.4 and 6

300 For example, tropomyosin cable wrapping around actin of thin filaments features b