ライフサイエンスコーパス: calcium deposit

1 1A1) and calcification (eg, RUNX2, BMP2, and calcium deposits).

2 rements of alkaline phosphatase activity and calcium deposits.

3 munofluorescence, and von Kossa staining for calcium deposits.

4 duction of osteocalcin, and the formation of calcium deposits.

5 association of levels of Lp(a) with coronary calcium deposits.

6 nary atherosclerosis as measured by coronary calcium deposits.

7 equent neovascular hyperemia and microscopic calcium deposits.

8 c features of solid tumors or any associated calcium deposits.

9 Compared with no or one calcium deposit and no or one coronary risk factor other

10 PC treatment led to significant reduction of calcium deposits and arrested OA disease progression.

11 , and myocardial degeneration with extensive calcium deposits and fibrosis.

12 The spatial relationship between calcium deposits and intraleaflet hematomas was analyzed

13 They also developed hypercalciuria and renal calcium deposits and some had deformities of the spine.

14 osclerosis, including inflammatory cells and calcium deposits, and correlates with coronary plaque bu

15 eries frequently become calcified, and these calcium deposits are associated with a high risk of adve

16 Thoracic aortic calcium deposits are frequently detected on tomography o

17 Multivariate analyses identified multiple calcium deposits as a significant predictor for an abnor

18 Initial calcium deposits, associated mainly with elastic fibers,

19 nd in the subjects with significant coronary calcium deposit (calcium score > or =50).

20 Thickened valve leaflets and calcium deposits, characteristic of valve disease, are o

21 t, growth failure, brain dysmyelination with calcium deposits, cutaneous photosensitivity, pigmentary

22 t; growth failure; brain dysmyelination with calcium deposits; cutaneous photosensitivity; pigmentary

23 METHODS AND Macrophages near calcium deposits display an alternative phenotype being

24 Particles that stained with the calcium deposit dye, Alizarin red, were abundant in the

25 lasia, characterized by punctate or dot-like calcium deposits in cartilage observed on neonatal radio

26 tiation even at low doses (2.5%), increasing calcium deposits in HPLSCs.

27 esults indicate that macrophages surrounding calcium deposits in human atherosclerotic plaques are ph

28 macrophages located in areas surrounding the calcium deposits in human atherosclerotic plaques.

29 38% of wild-type levels in Abcc6+/- mice, no calcium deposits in injured cardiac tissue were observed

30 udes both metastatic calcification, in which calcium deposits in previously normal lung or dystrophic

31 into consideration, the presence of multiple calcium deposits in the mitral annulus, aortic valve or

32 sessed at 12 weeks of age by quantitation of calcium deposits in the muzzle skin containing dermal sh

33 s been successfully reported for superficial calcium deposits in the skin from other processes.

34 Five subjects had no calcium deposit on either scan.

35 nty-seven of 33 subjects had coronary artery calcium deposits on both CT scans.

36 One subject had calcium deposits on only one scan.

37 -null and wild-type mice after TBI with more calcium deposits present in the wild-type mice.

38 lloon-based system to induce fracture in the calcium deposits to facilitate luminal gain and stent ex

39 , Osterix, and Notch1 despite no evidence of calcium deposits, which suggests ongoing active processe