ライフサイエンスコーパス: calcium oscillation

1 exclusively involved in calcium entry during calcium oscillations.

2 egg laying, in part by interfering with HSN calcium oscillations.

3 the active phase) of the various regimes of calcium oscillations.

4 population of cells maintained synchronized calcium oscillations.

5 cells acts on purinergic receptors to induce calcium oscillations.

6 Cx43-null neurospheres displayed spontaneous calcium oscillations.

7 in the IP3 receptor slow or eliminate these calcium oscillations.

8 ns in which sperm extracts elicit pronounced calcium oscillations.

9 lly modulate effects of EFS-induced cellular calcium oscillations.

10 n turn coincided with the termination of the calcium oscillations.

11 ion in the pancreatic islets of mice through calcium oscillations.

12 lethal and cardiomyocytes exhibit arrhythmic calcium oscillations.

13 significant effect on the properties of the calcium oscillations.

14 gher agonist concentration for intracellular calcium oscillations.

15 +)] spikes and exerts a positive feedback on calcium oscillations.

16 not individual cells and monolayers, exhibit calcium oscillations.

17 lso been reported to occur in the absence of calcium oscillations.

18 patiotemporal properties of receptor-induced calcium oscillations.

19 emonstration that it acts upstream of oocyte calcium oscillations.

20 ciated with the rapid onset of intracellular calcium oscillations.

21 ions and is activated via CaM binding during calcium oscillations.

22 MI1 and improved its efficiency in mediating calcium oscillations.

23 easing the frequency and/or amplitude of the calcium oscillations.

24 9593) mimicked the effect of Dyn-A (1-13) on calcium oscillations.

25 oncentration and the frequency of alpha-cell calcium oscillations.

26 erative discharges of stored calcium, termed calcium oscillations [1].

27 M) was appropriate for the interpretation of calcium oscillations (~125-850 nM), while that of EF-han

28 calcium influx as an essential component of calcium oscillations [2].

29 d as a positive regulator of sperm-triggered calcium oscillations, a finding that may apply to other

30 Rapid cellular calcium oscillations activate gene expression hours late

31 Calcium oscillations activate nodule development; we wan

32 d indefinitely with (delta)90 cyclin B1, the calcium oscillations also continue indefinitely.

33 P/Q-, and T-type VGCCs are not required for calcium oscillations, although inhibitors of these chann

34 the diversity in damping characteristics of calcium oscillations among cells.

35 Calcium oscillation amplitude and frequency control gene

36 d uptake NE; and exhibit pacemaker activity, calcium oscillation and chemoreceptor activity in respon

37 n, attenuating SST2 effects on intracellular calcium oscillation and internalization.

38 o different calcium responses: intra-nuclear calcium oscillations and a calcium influx at the root ha

39 olerant B cells, self-antigen stimulated low calcium oscillations and activated NF-AT and ERK/pp90rsk

40 ctrical activity across the islet underlying calcium oscillations and beta-cell synchronization, we u

41 es insights into mechanisms of intracellular calcium oscillations and capacitative calcium entry.

42 TLC-S induced global calcium oscillations and extended calcium transients as

43 calcium channels is critical for alpha-cell calcium oscillations and glucagon secretion at low gluco

44 of dexamethasone were sufficient to inhibit calcium oscillations and interleukin-2 mRNA after weak T

45 Calcium oscillations and maturation were typically lacki

46 hic drug niclosamide, which markedly blunted calcium oscillations and membrane conductance in spike-e

47 opressin only will evoke localized cytosolic calcium oscillations and modest increases in hepatic glu

48 Here, we show that PAWP triggers calcium oscillations and pronuclear formation in human a

49 y connexin-36 coordinates intracellular free calcium oscillations and pulsatile insulin release in is

50 n36 gap junctions coordinate glucose-induced calcium oscillations and pulsatile insulin secretion acr

51 n was necessary to maintain the frequency of calcium oscillations and STIM2 KD reduced spontaneous OP

52 A model for the mechanism of neuronal calcium oscillations and the reason for their synchrony

53 from the endoplasmic reticulum, resulting in calcium oscillations and the secretion of luteinizing ho

54 lating inositol 1,4,5-trisphosphate-mediated calcium oscillations and the up-regulation of the transc

55 timulation induces long-lasting elevation of calcium oscillations and transcriptional reprogramming o

56 Calcium oscillations and traveling calcium waves have be

57 hat includes five genes required to generate calcium oscillations and two genes required for the perc

58 Calcium oscillations and waves induce depolarization in

59 GT+TBX20 demonstrated more frequent beating, calcium oscillation, and higher energy metabolism as evi

60 nsor, as a key link between LPS-induced ROS, calcium oscillations, and endothelial cell (EC) dysfunct

61 l, enhances glucose-stimulated intracellular calcium oscillations, and enhances insulin secretion fro

62 alcium uptake explained our results, causing calcium oscillations, AP amplitude alternans, and TWA th

63 In addition, the calcium oscillations appear to serve as a bidirectional

64 In the absence of TMEM24, calcium oscillations are abolished, leading to a defect

65 d elevated CDK activity, the sperm-triggered calcium oscillations are again prolonged.

66 Most important, sperm-induced calcium oscillations are blocked by coinjection of a com

67 Secondly, although calcium oscillations are caused by the stochastic openin

68 tmem33-mediated endothelial calcium oscillations are critical for formation of endot

69 In animal systems, comparable calcium oscillations are decoded by calmodulin (CaM)-dep

70 ET technology have been used to describe how calcium oscillations are decoded through phase-locked os

71 We have found that these calcium oscillations are dependent on an influx of extra

72 uscular mycorrhizal fungi, nuclear localized calcium oscillations are essential to transduce the micr

73 re fertilized by sperm, a distinct series of calcium oscillations are generated which serve as the es

74 vide genetic evidence that stimulus-specific calcium oscillations are necessary for stomatal closure.

75 Specifically, we show that synchronized calcium oscillations are present in vivo , which are los

76 Here we demonstrate that these calcium oscillations are regulated by kappa opioid recep

77 lic calcium concentration (calcium spikes or calcium oscillations) are a common mode of signal transd

78 onse to ABA is unlinked to the generation of calcium oscillations, as the pp2aB'1 mutant displays a n

79 Baseline spontaneous calcium oscillations assessed by fluorescent confocal mi

80 While the calcium oscillations associated with cells directly expo

81 The generation of calcium oscillations at fertilisation and during mitosis

82 o other oocytes that display sperm-triggered calcium oscillations at fertilisation.

83 wering SERCA level will enable intracellular calcium oscillations at low agonist concentrations where

84 Furthermore, the calcium oscillation attenuates the histamine-induced cyt

85 In the presence of 4-AP, synchronized calcium oscillations become independent of NMDA receptor

86 imulation, for example, not only altered the calcium oscillations but also facilitated osteodifferent

87 , were also able to produce local and global calcium oscillations (but at higher concentrations than

88 re-operated calcium channels is critical for calcium oscillations, but calcium entry through voltage-

89 Regulation of calcium oscillation by external physical stimulation cou

90 e report the manipulation of sperm-triggered calcium oscillations by cyclin-dependent kinase (CDK) ac

91 ubsystem can account for these complex islet calcium oscillations by modifying the relative contribut

92 Information embedded in calcium oscillation can provide molecular cues for cell

93 was also able to prevent the suppression of calcium oscillations caused by Dyn-A (1-13).

94 otif regulates damping of G-protein-mediated calcium oscillations consistent with experimental data.

95 n culture likely results from the decline in calcium oscillations/contractile activity and mitofusin

96 The data show that the calcium oscillations contribute a significant fraction o

97 Calcium oscillations control pollen tube growth and fert

98 Cytosolic calcium oscillations control signaling in animal cells,

99 , and computational modeling reveal that the calcium oscillation depends on the number of neighboring

100 he ATP stimulus increases the propensity for calcium oscillations, despite large cell-to-cell variabi

101 These findings suggest that intracellular calcium oscillations directly regulate hOPC fate and tha

102 es an auto-regulatory feedback mechanism for calcium oscillations during pollen tube growth.

103 antly larger calcium responses and displayed calcium oscillations during prolonged agonist applicatio

104 ve of mitochondria to cause dysregulation of calcium oscillations during prolonged stimulation.

105 including mechanical stress, and involved in calcium oscillations during the early stages of osteocla

106 Calcium oscillations evoked by physiological concentrati

107 This architecture explains why calcium oscillations evoked by synchronized periodic act

108 Calcium oscillations exert physiological control on mito

109 channel-mediated augmentation of spike-like calcium oscillations first promoted stable expression of

110 is initiated by sperm-mediated intracellular calcium oscillations, followed by activation of metaphas

111 olonies, both communication channels lead to calcium oscillations following the stimulation by extern

112 Action potential and calcium oscillation frequencies are increased in Insr kn

113 s expressed in astrocytes tripled astrocytic calcium oscillation frequency in both the preBotzinger c

114 Further, we measured the calcium oscillation frequency in dendritic spines of cul

115 ly regulate hOPC fate and that modulation of calcium oscillation frequency may overcome inhibitory Ga

116 n is important for setting the physiological calcium oscillation frequency.

117 cord neurons in culture exhibit spontaneous calcium oscillations from day in vitro (DIV) 6 through D

118 The role of cytosolic calcium oscillation has long been recognized in the regu

119 s required to generate the nuclear localized calcium oscillations have been identified, their mechani

120 Intracellular calcium oscillations have fascinated scientists for deca

121 These intracellular calcium oscillations have long fascinated biologists as

122 tions in membrane potential accompanying the calcium oscillations have no significant effect on the p

123 Here we report novel intraciliary calcium oscillations (ICOs) at the LRO that connect cili

124 lular reactive oxygen species production and calcium oscillation in rice roots.

125 agonist dynorphin (Dyn)-A (1-13) suppressed calcium oscillations in a concentration-dependent manner

126 s a statistical description of heterogeneous calcium oscillations in a dynamic environment.

127 e study the conditions for the appearance of calcium oscillations in both a detailed subcellular mode

128 scle had decreased AHR, and the frequency of calcium oscillations in CD148-deficient ASM was substant

129 factor, found in sperm extracts, that causes calcium oscillations in cells; thus, the protein was nam

130 Conversely, cold and abscisic acid elicited calcium oscillations in det3, and stomatal closure occur

131 sed calcium imaging to monitor intracellular calcium oscillations in GnRH-1 neurons maintained in nas

132 hways underlying muscarinic receptor-induced calcium oscillations in human embryonic kidney (HEK293)

133 acute GABA(A) receptor antagonism decreased calcium oscillations in individual GNRH-1 cells as well

134 Here we report that insulin induces calcium oscillations in isolated rat hepatocytes, and th

135 asuring the [ATP]/[ADP] responses to imposed calcium oscillations in mouse beta cells, we found that

136 revealed that PHE caused dramatic cytosolic calcium oscillations in NST neurones.

137 For example, the calcium oscillations in oocytes and embryos occur during

138 CCaMK) is essential in the interpretation of calcium oscillations in plant root cells for the establi

139 t developing murine cortical neurons exhibit calcium oscillations in response to direct activation of

140 zebrafish ECs and is required for cytosolic calcium oscillations in response to Vegfa.

141 whereas a mix of CO4 and Myc-LCOs activated calcium oscillations in rice trichoblasts.

142 symbiosis signaling pathway, with resultant calcium oscillations in root epidermal cells of Medicago

143 Our data suggest that calcium oscillations in the cell cycle may be linked to

144 ased during hypercapnic challenge, increases calcium oscillations in the chemosensitive parafacial re

145 ly multiple factors control the frequency of calcium oscillations in the egg after fertilization and

146 the defecation motor program in C. elegans, calcium oscillations in the pacemaker (intestine), which

147 l a hitherto unsuspected role for tmem33 and calcium oscillations in the regulation of vascular devel

148 ced synchronous glucose-stimulated beta cell calcium oscillations in their islets in vivo, likely dis

149 tes, and (iii) ryanodine receptor-2-mediated calcium oscillations increased fusion activity in HEK293

150 J suppressed Plcg2 expression and downstream calcium oscillations indirectly by a TGF-beta/PLCgamma2/

151 the Gbetagamma translocation rate regulates calcium oscillations induced by G-protein-coupled recept

152 During the higher-frequency sinusoidal calcium oscillations induced by higher doses of ACh, NAD

153 During the transient cytosolic calcium oscillations induced by intermediate doses of AC

154 s termed oscillin because it correlated with calcium oscillation-inducing activity in mammalian eggs.

155 r sperm 33-kDa protein that co-migrated with calcium oscillation-inducing activity was recently descr

156 Previous studies have shown that these calcium oscillations involve the activation of NMDA rece

157 Mg(2+) also suppressed thimerosal-induced calcium oscillations (IP(3)R-dependent).

158 cium-EGTA Krebs solution suggesting that the calcium oscillation is mediated principally by intracell

159 ee eggs show that the general pattern of the calcium oscillations is identical in monospermic and pol

160 e early Nod factor signaling associated with calcium oscillations is limited to the root surface, the

161 ling systems, the frequency of intracellular calcium oscillations is physiologically important.

162 and the number of cells showing synchronous calcium oscillations is reduced.

163 ted to determine whether the second phase of calcium oscillations is required to reactivate the MPF a

164 The nature of the calcium oscillations is similar for LCOs produced by rhi

165 In between, calcium oscillations may appear.

166 The KOR-mediated inhibition of spontaneous calcium oscillations may therefore be a consequence of p

167 ch is linked to an increase in intracellular calcium oscillation mediated by ryanodine receptor (RyR)

168 e ADP level falls during the silent phase of calcium oscillations, mitochondria can still produce eno

169 le model for intercellular communication via calcium oscillations, motivated in part by a recent expe

170 organizes ATP1A1 of fibroblasts that induces calcium oscillations, NF-kappaB activation, and activin

171 By contrast, there was neither spike-like calcium oscillations nor responsive P2Y2 receptors in HD

172 to the normal growth, size, and cytoplasmic calcium oscillations of caulonemal cells.

173 old, and external calcium elicited cytosolic calcium oscillations of differing amplitudes and frequen

174 As one varies the frequency of calcium oscillations of the donor cell, the sensor cell

175 lacking the phosphatase CD45 did not exhibit calcium oscillations or ERK/pp90rsk activation, nor did

176 oscillations may be in phase with cytosolic calcium oscillations or out of phase.

177 While an altered calcium oscillation pattern may be an indicator for hMSC

178 In the present study we characterized the calcium oscillation profiles in hMSCs before and after s

179 malian cortex, where low-frequency (0.01 Hz) calcium oscillations refine topographic maps.

180 The appearance of spontaneous calcium oscillations, reflecting synchronous neuronal ac

181 rrelated with the onset and cessation of the calcium oscillations required for subsequent cleavage, a

182 izocilpine maleate (MK-801), also suppressed calcium oscillations, revealing a dependence on glutamat

183 2-myristate 13-acetate induced low amplitude calcium oscillations, slower translocation of cPLA(2)alp

184 erlying global plasma membrane PI(4,5)P2 and calcium oscillations spatially regulates actin dynamics,

185 ABA that impairs mycorrhizal factor-induced calcium oscillations, suggesting different modes of acti

186 Calcium oscillations suppress mitochondrial movements al

187 n the presence of full-length cyclin B1, the calcium oscillations terminate when cyclin B1 levels fal

188 the eupnoea rhythm, whereas an intracellular calcium oscillation that is slower by orders of magnitud

189 ocytes or eggs to generate multiple wavelike calcium oscillations that arise at least in part from th

190 ar factor kappaB (NF-kappaB) proteins and of calcium oscillations that became apparent 1-3 d after TN

191 of symbiotic associations in plants requires calcium oscillations that must be decoded to invoke down

192 This work provides a model for decoding calcium oscillations that uses differential calcium bind

193 channel blocker nifedipine did not suppress calcium oscillations, the N-type calcium channel blocker

194 nt to restrict the duty cycle of ATP/ADP and calcium oscillations, the parameter that dynamically enc

195 s are not required for fertilization-induced calcium oscillations, they do play a critical role in de

196 exhibit numerous oscillatory behaviors from calcium oscillations to circadian rhythms that recur dai

197 ate with rhizobia, showed Nod factor-induced calcium oscillations to S. fredii NGR234 Nod factors, bu

198 Intracellular calcium oscillations tracked via real-time fluorescence

199 m channels (VGCCs), since diltiazem inhibits calcium oscillations under all conditions.

200 The activity occurred in the form of calcium oscillations, was synchronized across different

201 Calcium oscillations were activated in rice atrichoblast

202 These spontaneous calcium oscillations were blocked by 1 muM tetrodotoxin,

203 In RyR1-expressing cells, regenerative calcium oscillations were observed in response to caffei

204 ricted to central regions, compartmentalized calcium oscillations were sometimes observed.

205 spiking but also suppresses the frequency of calcium oscillations when applied at lower concentration

206 The former causes asynchronous calcium oscillations, whereas the latter leads to a sing

207 zation, are involved in both events, whereas calcium oscillations, which correlate with global fluctu

208 modulin and CCaMK would substantially mirror calcium oscillations, which typically have a 90 s period

209 tration-dependent reduction of intracellular calcium oscillations, while other caged inositol pyropho

210 riods match closely with those of intraislet calcium oscillations, while the slower oscillations are

211 ation in substrate phosphorylation follows a calcium oscillation with a lag of approximately 10 s.

212 Mathematical modeling links intracellular calcium oscillations with actomyosin turnover and force

213 d with CaMKII inhibition contributes both to calcium oscillations within astrocytes and ultimately co

214 l flagella motors, growing microtubules, and calcium oscillations within human embryonic kidney cells