ライフサイエンスコーパス: calcium oxalate

1 e of indinavir and other substances, such as calcium oxalate.

2 re, and carbon sequestration via crystalline calcium oxalate.

3 rinary stones, which largely are composed of calcium oxalate.

4 Most kidney stones are composed primarily of calcium oxalate.

5 nd powder forms, precipitating manganese and calcium oxalates.

6 ned in 11 patients: calcium phosphate (55%), calcium oxalate (18%), mixed calcium phosphate and oxala

7 n measurements show that, after consumption, calcium oxalate also interferes with the conversion of p

8 ividuals with kidney stones containing >=50% calcium oxalate and 44 controls matched for age, sex, an

9 (70-71 U(Slope)), struvite (56-60 U(Slope)), calcium oxalate and calcium phosphate (17-59 U(Slope)),

10 Polycrystalline calcium oxalate and calcium phosphate calculi were found

11 done therapy, the supersaturation ratios for calcium oxalate and calcium phosphate fell by 25% and 35

12 us, the effect of potassium citrate on urine calcium oxalate and calcium phosphate supersaturation an

13 Furthermore, calcium oxalate and calcium phosphate supersaturation we

14 late, and phosphate levels lead to increased calcium oxalate and calcium phosphate supersaturation.

15 stals in renal stones, which are composed of calcium oxalate and calcium phosphate.

16 lower urine oxalate and supersaturation for calcium oxalate and uric acid.

17 cant genetic influence are the excretions of calcium, oxalate and citrate.

18 Most kidney stones consist of calcium oxalate, and higher urinary oxalate increases th

19 ves: amorphous calcium carbonate cystoliths, calcium oxalates, and silica phytoliths.

20 r handling of lithogenic substrates, such as calcium, oxalate, and phosphate, and of inhibitors of cr

21 uding gender, diet, and urinary excretion of calcium, oxalate, and uric acid.

22 um phosphate (BCP)-associated syndromes, and calcium oxalate arthritis.

23 nt correlated positively with mRNA levels of calcium/oxalate/ascorbate-related and stress-responsive

24 Because aluminum citrate blocks calcium oxalate binding and toxicity in human kidney cel

25 treatment strategies target the formation of calcium oxalate but not its interaction with kidney tiss

26 dium, resulting in lower supersaturation for calcium oxalate, calcium phosphate, and uric acid.

27 e composition (uric acid, cystine, struvite, calcium oxalate, calcium phosphate, brushite), and 20 we

28 hemical maps at <= 1 um scales collected for calcium oxalate, calcium phosphate, uric acid, and struv

29 Approximately 80% of stones are composed of calcium oxalate (CaOx) and calcium phosphate (CaP); 10%

30 ost common form of renal stone disease, with calcium oxalate (CaOx) being the predominant constituent

31 The study investigated calcium oxalate (CaOx) crystal composition, accumulation

32 We report that calcium oxalate (CaOx) crystal deposition led to rapid t

33 inhibitors and appear capable of inhibiting calcium oxalate (CaOx) crystallization in vitro.

34 associated E. coli differentially influenced calcium oxalate (CaOx) crystallization through the incor

35 onolayers and exposed to oxalate (Ox) and/or calcium oxalate (CaOx) crystals to investigate cellular

36 nce kidney transplant biopsies for recurrent calcium oxalate (CaOx) deposits in 37 kidney transplants

37 Calcium oxalate (CaOx) is one of the most common bio-min

38 Calcium oxalate (CaOx) kidney stones may be associated w

39 e diet of the GHS rats leads to formation of calcium oxalate (CaOx) kidney stones.

40 1 and TNFR2 in human and murine kidneys with calcium oxalate (CaOx) nephrocalcinosis-related CKD comp

41 Nephrocalcinosis, acute calcium oxalate (CaOx) nephropathy, and renal stone dise

42 of the human renal papilla in patients with calcium oxalate (CaOx) stone disease and healthy subject

43 effect can stimulate calcium phosphate (CaP)/calcium oxalate (CaOx) stone formation.

44 Idiopathic calcium oxalate (CaOx) stones often develop attached to

45 Multiple calcium oxalate (CaOx) stones were removed from a Mayo C

46 nephron of the kidney is supersaturated with calcium oxalate (CaOx), which crystallizes in the tubule

47 n culture (cIMCD) and selectively adsorbs to calcium oxalate (CaOx).

48 mplanted rats had a 43 to 88% rate of kidney calcium oxalate crystal deposition.

49 on, plasma oxalate concentration, and kidney calcium oxalate crystal deposition.

50 for 2 wk to induce hyperoxaluria and kidney calcium oxalate crystal deposition.

51 Stiripentol protected kidneys against calcium oxalate crystal deposits in acute ethylene glyco

52 ormally high oxalate production resulting in calcium oxalate crystal formation and deposition in the

53 on were visually screened for alterations in calcium oxalate crystal formation.

54 showed no evidence for reduced inhibition of calcium oxalate crystal growth, so low inhibition of gro

55 Oxalate overproduction may cause calcium-oxalate crystal formation leading to kidney ston

56 UAP) is a urinary glycoprotein that inhibits calcium oxalate crystallization in vitro.

57 Their calcium oxalate crystallization inhibitory activity was

58 UAP exhibited a strong calcium oxalate crystallization inhibitory activity.

59 Oxalate-producing plants accumulate calcium oxalate crystals (CaOx(c)) in the range of 3-80%

60 Carbon-calcium inclusions (CCaI) either as calcium oxalate crystals (CaOx) or amorphous calcium car

61 ate in the urine results in the formation of calcium oxalate crystals and subsequent kidney stone for

62 Calcium oxalate crystals are widespread among animals an

63 Calcium oxalate crystals can also dissolve after renal c

64 eir detrimental effects on a chewing insect, calcium oxalate crystals do not negatively affect the pe

65 OH-stretching signals reveal that calcium oxalate crystals have a mixed hydration state.

66 In combination, calcium oxalate crystals in leaves can act as a biochemi

67 sors give rise to oxalic acid (OxA) found in calcium oxalate crystals in specialized crystal idioblas

68 control rats, and resisted the formation of calcium oxalate crystals in their nephrons.

69 Mice with acute oxalosis displayed calcium oxalate crystals inside distal tubular epithelia

70 ae feeding on wild-type plants with abundant calcium oxalate crystals suffer significantly reduced gr

71 Tree different form of calcium oxalate crystals were raported in Dienia ophrydi

72 ts of M. truncatula with decreased levels of calcium oxalate crystals were used to assess the defensi

73 uric acid stones and increased frequency of calcium oxalate crystals within regions of elongated cry

74 ins, starch) and depicts the accumulation of calcium oxalate crystals, flavonols, and anthocyanins in

75 oradiography for incorporation of (14)C into calcium oxalate crystals.

76 hat these cells may be more resistant to the calcium oxalate crystals.

77 s the nucleation, growth, and aggregation of calcium oxalate crystals.

78 on (hyperoxaluria) promotes the formation of calcium oxalate crystals.

79 Most kidney stones are made of calcium oxalate crystals.

80 al blood mononucleated cells stimulated with calcium-oxalate crystals, monosodium urate crystals, or

81 nderstanding of excess urinary excretions of calcium, oxalate, cystine, and uric acid.

82 Seven different classes of calcium oxalate defective mutants were identified that e

83 w a clear feeding preference for tissue from calcium oxalate-defective (cod) mutant lines cod5 and co

84 The calcium oxalate deficient 5 (cod5) mutant of Medicago tr

85 Oxalosis, or calcium oxalate deposition in the tissues, may develop i

86 All species examined have abundant calcium oxalate deposits around the veins.

87 the substrate minerals, and the formation of calcium oxalate deposits at the interface between some s

88 either calcium oxalate monohydrate (COM) or calcium oxalate dihydrate (COD).

89 anganese oxalate dihydrate (lindbergite) and calcium oxalate dihydrate (weddellite).

90 In addition, calcium oxalate dihydrate and calcium oxalate monohydrat

91 Crystals of calcium oxalate dihydrate can also nucleate directly on

92 e cell surface, calcium oxalate monohydrate, calcium oxalate dihydrate, and hydroxyapatite crystals a

93 ories based on their Raman spectrum: type I, calcium oxalate dihydrate, and type II, calcium hydroxya

94 ings into question the hypothesized roles of calcium oxalate formation in supporting tissue structure

95 When the renal secretion capacity exceeds, calcium oxalate forms stones that accumulate in the kidn

96 Plants accumulate crystals of calcium oxalate in a variety of shapes, sizes, amounts,

97 alate leading to the deposition of insoluble calcium oxalate in the kidney.

98 provide a different therapeutic approach to calcium oxalate-induced injury.

99 Calcium oxalate is the most abundant insoluble mineral f

100 Calcium oxalate is the predominant component in 70-80% o

101 of gut microbial communities and early-onset calcium oxalate kidney stone disease is unknown.

102 unmasking of which occurs in the hereditary calcium oxalate kidney stone disease primary hyperoxalur

103 evelopment of hyperoxaluria and/or recurrent calcium oxalate kidney stone disease.

104 may be upstream determinants of early-onset calcium oxalate kidney stone disease.

105 inary oxalate is an important determinant of calcium oxalate kidney stone formation.

106 c oxalate production and higher incidence of calcium oxalate kidney stones and potentially kidney fai

107 actors for inhibiting the crystallisation of calcium oxalate kidney stones in susceptible individuals

108 ariety of factors influence the formation of calcium oxalate kidney stones, including gender, diet, a

109 In the ectopic biomineralization of calcium oxalate kidney stones, the competition between c

110 ts potential contribution to protection from calcium oxalate kidney stones.

111 member 1) in two unrelated individuals with calcium oxalate kidney stones.

112 ates of microcrystals, most commonly contain calcium oxalate monohydrate (COM) as the primary constit

113 be used to switch the surface morphology of calcium oxalate monohydrate (COM) back and forth, result

114 Effects of oxalate exposure on calcium oxalate monohydrate (COM) crystal adherence to t

115 t study was undertaken to identify potential calcium oxalate monohydrate (COM) crystal-binding protei

116 Here we examine this phenomenon in calcium oxalate monohydrate (COM) crystallization, a mod

117 a monomer, is known to inhibit the growth of calcium oxalate monohydrate (COM) crystals in renal tubu

118 The attachment of calcium oxalate monohydrate (COM) crystals to renal tubu

119 The nucleation and growth of calcium oxalate monohydrate (COM) crystals were studied

120 Interaction of calcium oxalate monohydrate (COM) crystals with renal ce

121 ury during exposure to oxalate ions (Ox) and calcium oxalate monohydrate (COM) crystals.

122 uman kidney stones are composed primarily of calcium oxalate monohydrate (COM) crystals.

123 alate kidney stones, the competition between calcium oxalate monohydrate (COM) formation and its inhi

124 which crystallizes in the tubules as either calcium oxalate monohydrate (COM) or calcium oxalate dih

125 Studies of calcium oxalate monohydrate (COM) precipitation at Langm

126 of these proteins in the crystallization of calcium oxalate monohydrate (COM), the most prominent co

127 Calcium oxalate monohydrate (COM), which plays a functio

128 Some calcium oxalate monohydrate (whewellite) was formed with

129 Calcium oxalate monohydrate and hydroxyapatite (calcium

130 In addition, calcium oxalate dihydrate and calcium oxalate monohydrate crystal aggregates exhibit h

131 correlated with a corresponding increase in calcium oxalate monohydrate crystal attachment to the ce

132 zed in vitro inhibitor of hydroxyapatite and calcium oxalate monohydrate crystal formation, but it is

133 Calcium oxalate monohydrate crystals are responsible for

134 T techniques were able to help differentiate calcium oxalate monohydrate stones with moderate accurac

135 se features include thin concentric rings of calcium oxalate monohydrate within uric acid stones and

136 Once present on the cell surface, calcium oxalate monohydrate, calcium oxalate dihydrate,

137 is regularly supersaturated with respect to calcium oxalate monohydrate, the most common solid phase

138 atography, and fractions were incubated with calcium oxalate monohydrate.

139 eraction with the positively charged face of calcium-oxalate monohydrate leads to formation of a pept

140 By imaging the growth of calcium-oxalate monohydrate under the influence of aspar

141 Here we report that crystals of calcium oxalate, monosodium urate, calcium pyrophosphate

142 Crystals of calcium oxalate, monosodium urate, or calcium pyrophosph

143 s unclear why men have a higher incidence of calcium oxalate nephrolithiasis than women.

144 ance in primary hyperoxaluria and idiopathic calcium oxalate nephrolithiasis.

145 uffer from disrupted oxalate homeostasis and calcium oxalate nephrolithiasis.

146 an increased incidence of hyperoxaluria and calcium oxalate nephrolithiasis.

147 and enteric hyperoxaluria or even idiopathic calcium oxalate nephrolithiasis.

148 igher urinary oxalate increases the risk for calcium oxalate nephrolithiasis.

149 ute ethylene glycol intoxication and chronic calcium oxalate nephropathy models.

150 hat polyphosphates and phosphonates suppress calcium oxalate nucleation, tailor solvate crystal struc

151 Precipitation of insoluble manganese and calcium oxalate occurred under colonies growing on agar

152 complex and driven by high concentrations of calcium-oxalate or calcium-phosphate in the urine.

153 The urinary relative supersaturations of calcium oxalate (P = 0.03) and brushite (P = 0.002) were

154 re discussed with respect to fine control of calcium oxalate precipitation and the concept of crystal

155 gal cells, possible reproductive structures, calcium oxalate pseudomorphs, abundant nitrogenous compo

156 How and why many plants form crystals of calcium oxalate remain largely unknown.

157 ne glycol toxicity, apparently by inhibiting calcium oxalate's interaction with, and retention by, th

158 The equilibrium parameters for calcium oxalate solubility in tissue culture media were

159 COM crystals dispersed in saturated aqueous calcium oxalate solutions attached preferentially to the

160 time during immersion in the supersaturated calcium oxalate solutions.

161 and potassium citrate combined would reduce calcium oxalate stone formation and improve bone quality

162 Idiopathic calcium oxalate stone formation is a multifactorial dise

163 hanisms contribute to the increased risk for calcium oxalate stone formation observed in patients wit

164 Idiopathic calcium oxalate stone formation results from an interact

165 ading intestinal flora with a higher risk of calcium oxalate stone formation, possibly opening the do

166 c stone-forming rats fed a diet resulting in calcium oxalate stone formation, potassium citrate plus

167 RBIT that affects BP and biochemical risk of calcium oxalate stone formation, thus providing a potent

168 absorption resulting in an increased risk of calcium oxalate stone formation.

169 ody size may be an important risk factor for calcium oxalate stone formation.

170 surgeries have further increased the risk of calcium oxalate stone formation.

171 reduction in the risk for being a recurrent calcium oxalate stone former.

172 The relationship of gut microbiota and calcium oxalate stone has been limited investigated, esp

173 NS) controls, patients with occasional renal calcium oxalate stones (OS) and patients with recurrent

174 involving 247 adult patients with recurrent calcium oxalate stones and 259 age-, gender-, and region

175 Calcium oxalate stones are most prevalent, commonly driv

176 authors randomly assigned 99 persons who had calcium oxalate stones for the first time to a low anima

177 Thirty-six calcium oxalate stones were scanned in an anthropomorphi

178 We provided Sprague Dawley rats of renal calcium oxalate stones with antibiotics and examined the

179 n to have low urinary citrate and to develop calcium oxalate stones) had a 40% decrease in urinary ex

180 oxyproline, every rat in this model develops calcium oxalate stones.

181 A6 develop hyperoxalemia, hyperoxaluria, and calcium-oxalate stones as a result of a defect in intest

182 h as otters, dolphins and ferrets, that form calcium oxalate, struvite, uric acid, cystine and other

183 There were no significant differences in calcium oxalate supersaturation in any group.

184 to increased risk include increased urinary calcium oxalate supersaturation, while urinary citrate,

185 logic studies with patients at high risk for calcium oxalate urolithiasis showed a direct correlation

186 ted with hyperoxaluria, especially recurrent calcium oxalate urolithiasis.

187 xalate, thereby preventing hyperoxaluria and calcium oxalate urolithiasis.

188 lacking Slc26a6 develop a high incidence of calcium oxalate urolithiasis.

189 with an increased risk of hyperoxaluria and calcium-oxalate urolithiasis.

190 uria, and of subsequent nephrocalcinosis and calcium-oxalate urolithiasis.

191 In addition, calcium oxalate was formed from the calcium present in t

192 ey stone formation, as it produces insoluble calcium oxalate, which crystallizes and accumulates in t