ライフサイエンスコーパス: calcium pyrophosphate

1 f the beta3 subunit resulted in lower acidic calcium, pyrophosphate, and polyphosphate content as wel

2 that hypomagnesaemia may be associated with calcium pyrophosphate crystal inflammatory arthritis (ch

3 um resorption tests as causative for chronic calcium pyrophosphate crystal inflammatory arthritis (ps

4 The coexistence of calcium phosphate and calcium pyrophosphate crystals in osteoarthritis is a we

5 hage responses to monosodium urate crystals, calcium pyrophosphate crystals, aluminum salts, and sili

6 s an autoinflammatory condition triggered by calcium pyrophosphate dehydrate (CPPD) crystal depositio

7 (n = 15), spondyloarthropathy (n = 15), and calcium pyrophosphate deposition disease (CPPD) (n = 15)

8 ANKH mutations are associated with calcium pyrophosphate deposition disease and craniometap

9 age-related pathology recognized as primary calcium pyrophosphate deposition disease indicating its

10 KH overexpression-plasmids containing either calcium pyrophosphate deposition disease or craniometaph

11 on diseases (including gouty arthropathy and calcium pyrophosphate deposition disease), seronegative

12 gs of rheumatoid arthritis, gouty arthritis, calcium pyrophosphate deposition disease, psoriatic arth

13 e patients with sporadic as well as familial calcium pyrophosphate deposition disease.

14 f crystalline arthropathies, including gout, calcium pyrophosphate deposition, and hydroxyapatite dep

15 Calcium pyrophosphate dihydrate (CPPD) and basic calcium

16 Calcium deposition diseases caused by calcium pyrophosphate dihydrate (CPPD) and basic calcium

17 Microcrystals of calcium pyrophosphate dihydrate (CPPD) and monosodium ur

18 Familial autosomal dominant calcium pyrophosphate dihydrate (CPPD) chondrocalcinosis

19 tion, and increased concentrations promoting calcium pyrophosphate dihydrate (CPPD) crystal depositio

20 Calcium pyrophosphate dihydrate (CPPD) crystal depositio

21 characterization of the role of NTPPHase in calcium pyrophosphate dihydrate (CPPD) crystal depositio

22 he pathologic mineralization that results in calcium pyrophosphate dihydrate (CPPD) crystal formation

23 In this study, following our earlier work on calcium pyrophosphate dihydrate (CPPD) crystal-induced m

24 Monosodium urate monohydrate (MSU) and calcium pyrophosphate dihydrate (CPPD) crystals cause ac

25 The formation of calcium pyrophosphate dihydrate (CPPD) crystals in artic

26 luding nanoscale silicon dioxide (NanoSiO2), calcium pyrophosphate dihydrate (CPPD) crystals, and mur

27 Articular calcium-containing crystals cause calcium pyrophosphate dihydrate (CPPD) deposition diseas

28 of fluid containing synthetic or native BCP, calcium pyrophosphate dihydrate (CPPD), or monosodium ur

29 ed spectroscopy revealed crystals resembling calcium pyrophosphate dihydrate (CPPD).

30 of the molecular and cellular mechanisms of calcium pyrophosphate dihydrate and apatite crystal form

31 Calcium pyrophosphate dihydrate and basic calcium phosph

32 concerning clinical and etiologic aspects of calcium pyrophosphate dihydrate and basic calcium phosph

33 the pathologic matrix mineralization seen in calcium pyrophosphate dihydrate and basic calcium phosph

34 the cellular responses to monosodium urate, calcium pyrophosphate dihydrate and basic calcium phosph

35 The pathologic matrix mineralization seen in calcium pyrophosphate dihydrate and basic calcium phosph

36 Calcium crystals, including calcium pyrophosphate dihydrate and basic calcium phosph

37 ystals of calcium oxalate, monosodium urate, calcium pyrophosphate dihydrate and cystine trigger casp

38 ved in the pathogenesis of monosodium urate, calcium pyrophosphate dihydrate and hydroxyapatite cryst

39 of ePPi while excess levels of ePPi leads to calcium pyrophosphate dihydrate crystal deposition (CPPD

40 load is a likely source of pyrophosphate in calcium pyrophosphate dihydrate crystal deposition disea

41 In familial calcium pyrophosphate dihydrate crystal deposition disea

42 hic arthropathy of the atlantoaxial joint in calcium pyrophosphate dihydrate crystal deposition disea

43 c pyrophosphate in cartilage matrix leads to calcium pyrophosphate dihydrate crystal deposits.

44 Current models of calcium pyrophosphate dihydrate crystal formation are le

45 view, the author discusses various models of calcium pyrophosphate dihydrate crystal formation from e

46 l for studying the major factors involved in calcium pyrophosphate dihydrate crystal formation.

47 Calcium pyrophosphate dihydrate crystals are common comp

48 Progress in understanding why and how calcium pyrophosphate dihydrate crystals form in articul

49 young animals might promote the formation of calcium pyrophosphate dihydrate crystals in aged cartila

50 Elimination of calcium pyrophosphate dihydrate crystals may occur extra

51 Tumoral deposition of calcium pyrophosphate dihydrate crystals may occur in si

52 The effect of calcium pyrophosphate dihydrate crystals on the progress

53 Addition of calcium pyrophosphate dihydrate crystals to a lapine men

54 osition of either basic calcium phosphate or calcium pyrophosphate dihydrate crystals, remains unclea

55 sphate (PPi) that promotes the deposition of calcium pyrophosphate dihydrate crystals.

56 A discussion of ANKH as the familial calcium pyrophosphate dihydrate deposition disease gene

57 radiographic techniques to the diagnosis of calcium pyrophosphate dihydrate deposition disease holds

58 nt as a definitive rheumatic disease such as calcium pyrophosphate dihydrate deposition disease or as

59 nts were identified that segregated with the calcium pyrophosphate dihydrate deposition disease pheno

60 t literature reminds us of the propensity of calcium pyrophosphate dihydrate deposition disease to mi

61 In genetic studies of familial calcium pyrophosphate dihydrate deposition disease, a re

62 as a potential positional candidate gene for calcium pyrophosphate dihydrate deposition disease, and

63 rs: craniometaphyseal dysplasia and familial calcium pyrophosphate dihydrate deposition disease.

64 evalence and significance of extra-articular calcium pyrophosphate dihydrate deposits, and demonstrat

65 play radiographically detectable crystals of calcium pyrophosphate dihydrate in their joint spaces.

66 In addition to monosodium urate, calcium pyrophosphate dihydrate, and apatite crystals, a

67 als of calcium oxalate, monosodium urate, or calcium pyrophosphate dihydrate, as well as silica micro

68 ATD5 cells were basic calcium phosphate, not calcium pyrophosphate dihydrate, underlying the signific

69 lysis supports the role of ANKH mutations in calcium pyrophosphate dihydrate-induced arthritis.