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1 ne a signaling pathway from chloride channel calcium-activated 1 (CLCA1) to MAPK13 that is responsibl
2 ctrometry identified murine chloride channel calcium-activated 1 (mCLCA1) as the 10.1.1 Ag, a 90-kDa
3 s, Western blot analysis of chloride channel calcium activated 3 (CLCA3) expression in lung homogenat
4   Lowering the activity of Gelsolin, a known calcium-activated actin filament-severing protein, also
5 chment), which regulates voltage-independent calcium-activated action potentials at the neuronal syna
6 w that the functional expression of the slow calcium-activated afterhyperpolarizing current in pyrami
7 ensor proteins in the activation of the slow calcium-activated afterhyperpolarizing current.
8                                         Both calcium-activated and stretch-activated tensions increas
9 significantly from caspases because they are calcium-activated, arginine-specific peptidases that do
10 tress axis-regulated exon (STREX)-containing calcium-activated big potassium (BKCa) channel splice va
11                                              Calcium-activated binding of S100A4 to CD16A, promoted b
12 an-neurexin deletion reduced the function of calcium-activated BK potassium channels, whose activatio
13                            Large conductance calcium-activated (BK) channels are broadly expressed in
14                   This increase in cytosolic calcium activated by postsynaptic Gs-coupled CRF recepto
15 emonstrated that the increase in cytoplasmic calcium activated Ca(2+) dependent potassium currents an
16 amine receptor (amitraz metabolite), and the calcium-activated calcium channel (diamides).
17 shots of ryanodine receptor type 1 (RyR1), a calcium-activated calcium channel engaged in the binding
18 ha-mediated Ser648 phosphorylation abrogated calcium-activated calmodulin (CaM) binding to the regula
19 cing it into a peptide that is recognized by calcium-activated calmodulin.
20        Proteolytic processing of PKCalpha by calcium-activated calpain activates pathological cardiac
21            Moreover, these findings identify calcium-activated calpains as powerful modulators of cel
22 norganic phosphate [Pi] were investigated in calcium-activated cardiac myofibrils.
23    One candidate subpopulation expresses the calcium activated cation channel TRPM5 (transient recept
24                                              Calcium-activated CCTalpha nuclear import is mediated by
25 mily of membrane proteins, which encompasses calcium-activated channels for ions and lipids.
26 ial cells (PDECs) have been shown to express calcium activated chloride channels (CaCCs) and there is
27                          TMEM16A (ANO1) is a calcium-activated chloride channel (CaCC) expressed in s
28 eins with unknown function 16 (TMEM16A) is a calcium-activated chloride channel (CaCC) important for
29 ver, the molecular identity of the olfactory calcium-activated chloride channel (CaCC) is unknown.
30   Here, we show that the recently identified calcium-activated chloride channel (CaCC) TMEM16A is exp
31                                          The calcium-activated chloride channel (CaCC) TMEM16A plays
32 te that TMEM16A, an evolutionarily conserved calcium-activated chloride channel (CaCC), regulates cyt
33  inhibit self-cleavage prevent activation of calcium-activated chloride channel (CaCC)-mediated chlor
34                TMEM16A (ANO1) functions as a calcium-activated chloride channel (CaCC).
35 ugmentation in iPSC-RPE fully restored BEST1 calcium-activated chloride channel activity and improved
36         BEST1 is presumed to assemble into a calcium-activated chloride channel and be involved in ch
37                                          The calcium-activated chloride channel ANO1 regulates multip
38 To date, only five mutations in the proposed calcium-activated chloride channel ANO5/TMEM16E gene hav
39 sought to investigate the involvement of the calcium-activated chloride channel anoctamin 1 (ANO1) in
40                                          The calcium-activated chloride channel anoctamin 1 (ANO1) is
41 s in the ANO5 gene, which encodes a putative calcium-activated chloride channel belonging to the Anoc
42 EM16A), also called anoctamin 1 (ANO1), is a calcium-activated chloride channel expressed widely mamm
43             Human bestrophin-1 (hBest1) is a calcium-activated chloride channel from the retinal pigm
44 16A is the major constituent of the vascular calcium-activated chloride channel in rat pulmonary arte
45  characterized the expression of the TMEM16B calcium-activated chloride channel in the LS and showed
46 inhibited the expression of Gob5, a putative calcium-activated chloride channel involved in the regul
47                                              Calcium-activated chloride channel regulator 1 (CLCA1) a
48                                              Calcium-activated chloride channel regulator 1 (CLCA1) i
49 nes, including IL13, IL5, periostin (POSTN), calcium-activated chloride channel regulator 1 (CLCA1),
50 ), which revealed an increased expression of calcium-activated chloride channel regulator 1 (CLCA1).
51                                    ANO1 is a calcium-activated chloride channel that is frequently ov
52             Human Bestrophin 1 (hBest1) is a calcium-activated chloride channel that regulates neuron
53   Inducible expression of either CFTR or the calcium-activated chloride channel TMEM16A attenuated th
54                     Here, we report that the calcium-activated chloride channel TMEM16A is a biomarke
55                                          The calcium-activated chloride channel TMEM16A is a member o
56                                          The calcium-activated chloride channel TMEM16A is overexpres
57                              KEY POINTS: The calcium-activated chloride channel TMEM16A provides a pa
58                   Targeted disruption of the calcium-activated chloride channel TMEM16A, also known a
59                 A selective inhibitor of the calcium-activated chloride channel TMEM16A, N-((4-methox
60 produced by a nanomolar-potency inhibitor of calcium-activated chloride channel TMEM16A.
61 igated the physiological contribution of the calcium-activated chloride channel to LS neuronal signal
62 terstitial cells of Cajal, which express the calcium-activated chloride channel transmembrane member
63                                      ANO1, a calcium-activated chloride channel, is highly expressed
64  we determined the expression pattern of the calcium-activated chloride channel, TMEM16B, in the LS o
65  propose that ANO2 constitutes the olfactory calcium-activated chloride channel.
66 y was performed to detect ANO4 activity as a calcium-activated chloride channel.
67                                              Calcium-activated chloride channels (CaCC) with similar
68                                              Calcium-activated chloride channels (CaCCs) are key play
69                                              Calcium-activated chloride channels (CaCCs) are major re
70                                              Calcium-activated chloride channels (CaCCs) encoded by T
71                                              Calcium-activated chloride channels (CaCCs) play importa
72 r components of the physiologically relevant calcium-activated chloride channels (CaCCs) present in m
73                                   Bestrophin calcium-activated chloride channels (CaCCs) regulate the
74                                TMEM16A forms calcium-activated chloride channels (CaCCs) that regulat
75                      TMEM16A and TMEM16B are calcium-activated chloride channels (CaCCs) with importa
76 enuated inward currents carried by TRPC2 and calcium-activated chloride channels (CACCs).
77 hippocampal neuronal signaling that involves calcium-activated chloride channels (CaCCs).
78 n absence of expression/activity of reported calcium-activated chloride channels (TMEM16A, Bestrophin
79 an intrinsic property observed in endogenous calcium-activated chloride channels and could be relevan
80 ng pathway in the VNO and the requirement of calcium-activated chloride channels currents to mediate
81                                    Recently, calcium-activated chloride channels have been shown to c
82          Evidence is provided for a role for calcium-activated chloride channels such as TMEM16a in G
83 r the analyses of physiological functions of calcium-activated chloride channels that contain TMEM16A
84                                              Calcium-activated chloride channels TMEM16A and TMEM16B
85   In contrast to its paralogs, the TMEM16A/B calcium-activated chloride channels, mouse TMEM16F has b
86 gative effect through interaction with other calcium-activated chloride channels, such as hBest2, 3,
87 ffects of calcium signaling on CFTR or other calcium-activated chloride channels; here, we investigat
88                                          The calcium-activated chloride conductance anoctamin-1 (Ano1
89  vertebrate olfactory signal transduction, a calcium-activated chloride conductance serves as a major
90 ide secretion, which consists, in part, of a calcium-activated chloride conductance.
91 re, our analysis suggests that activation of calcium-activated chloride conductances by intracellular
92 ient for HIF-1alpha almost completely lacked calcium-activated chloride secretion.
93 rowth, primarily due to HIF-1alpha-dependent calcium-activated chloride secretion.
94           TMEM16A was found recently to be a calcium-activated Cl(-) channel (CaCC).
95 lso found to strongly inhibit the intestinal calcium-activated Cl(-) channel TMEM16A by a voltage-ind
96 ost two decades, it has been postulated that calcium-activated Cl(-) channels (CaCCs) play a role in
97                         The chloride channel calcium-activated (CLCA) family are secreted proteins th
98       Although a weaker activator than zinc, calcium activated CPE in vitro.
99  a potential feed-forward mechanism in which calcium-activated CPK32 activates CNGC18, further promot
100 the mean intracellular activity of calpains, calcium-activated cysteine proteases that are known to c
101                                    Calpains, calcium-activated cysteine proteases, have been shown to
102   The neuronal signal is transmitted through calcium-activated dense core vesicle neurosecretion.
103                   A new study reveals that a calcium-activated endoribonuclease of the EndoU protein
104  force of diaphragm strips, absolute maximal calcium activated force, and maximal specific calcium-ac
105               Compared with control, maximal calcium-activated force and calcium sensitivity declined
106 alcium activated force, and maximal specific calcium-activated force of permeabilized diaphragm fiber
107 ss spectrometric analysis of the products of calcium-activated hydrolysis of endogenous mitochondrial
108 , as a result of the excessive activation of calcium-activated hyperpolarizing conductances.
109 ium influx accompanying such signaling opens calcium-activated ion channels for feedback regulation.
110                By aligning distantly related calcium-activated ion channels in the TMEM16 family and
111                                          The calcium activated K(+) channel KCa3.1 plays an important
112 est in the pharmacology of large conductance calcium-activated K (BK) channels.
113 ses of ion channels, including examples from calcium-activated K(+) (BK(Ca)), voltage-activated K(+)
114 idal neurons revealed that large-conductance calcium-activated K(+) (BK) channel open probability was
115                           Large conductance, calcium-activated K(+) (BK) channels are important regul
116               Large-conductance voltage- and calcium-activated K(+) (BK) channels are key physiologic
117 ons, FMRP interaction with large-conductance calcium-activated K(+) (BK) channels, specifically their
118                            Large-conductance calcium-activated K(+) (BK-type) channels, abundantly di
119                        Voltage-dependent and calcium-activated K(+) (MaxiK, BK) channels are widely e
120                                          The calcium-activated K(+) channel KCa3.1 plays an important
121 tigated two candidate ethanol effectors, the calcium-activated K(+) channel SLO-1 and gap junctions,
122  excitotoxicity: anti-apoptotic Bcl-2, and a calcium-activated K(+) channel, SK2.
123 1-2.3) and intermediate-conductance (KCa3.1) calcium-activated K(+) channels are critically involved
124 ctance (KCa3.1) and small-conductance (KCa2) calcium-activated K(+) channels are gated by calcium bin
125 (KCa2) and intermediate-conductance (KCa3.1) calcium-activated K(+) channels are voltage-independent
126  mediated by activation of small conductance calcium-activated K(+) channels in PDGFRalpha(+) cells,
127  Purkinje-cell-specific knockout (KO) of the calcium-activated K+ channel SK2 (L7-SK2) show intact ve
128 assium efflux through voltage gated (Kv) and calcium activated (K(Ca)) potassium channels.
129  goal of this study was to determine whether calcium-activated kinases such as calcium/calmodulin-dep
130 l increase in phosphorylation likely tied to calcium-activated kinases.
131 n synaptic transmission properties, LTD, and calcium-activated membrane channels of hippocampal CA1 p
132 id, an antagonist of a previously identified calcium activated non-selective cation channel (I(CAN)).
133 while flufenamic acid, an antagonist for the calcium-activated non-selective cation conductance (ICAN
134 ity, while flufenamic acid, a blocker of the calcium-activated non-selective cation conductance, abol
135 ion further excites the cell by recruiting a calcium-activated non-selective cation current (ICAN) ca
136 t sodium current ([Formula: see text]) and a calcium-activated non-selective cationic current ([Formu
137                                              Calcium-activated non-specific cation (CAN) channels con
138 d synaptic efficacy, and a small-conductance calcium-activated nonselective cation channel, TMEM16F,
139 carinic afterdepolarization is mediated by a calcium-activated nonselective cation current, suggestin
140 ained responses were found to be mediated by calcium-activated nonselective cationic current induced
141                            Here we show that calcium activated NOX-independent NETosis is fast and me
142 tinguished DBQD type 1 and identified CANT1 (calcium activated nucleotidase 1) mutations as responsib
143                                Human soluble calcium-activated nucleotidase 1 (hSCAN-1) represents a
144 sponses to herbivory can be separated into a calcium-activated oxidative response and a K(+) -depende
145 ignaling through the small GTPase Rho and by calcium-activated pathways.
146 ibits AT1R phosphorylation by activating the calcium-activated phosphatase calcineurin in a Ca(2+)/ca
147 cium channels but not on the activity of the calcium-activated phosphatase calcineurin, and was oppos
148             The calcium pathway involved the calcium-activated phosphatase calcineurin, which stabili
149                                   TRPM4 is a calcium-activated, phosphatidylinositol-4,5-bisphosphate
150                                              Calcium-activated phospholipid scramblase mediates the e
151 tructure-function relationship of this novel calcium-activated phosphoryl transfer enzyme.
152 rylation was phenocopied only by deletion of calcium-activated PKC-2.
153 ulting from a reduction in large-conductance calcium-activated potassium (BK) and subthreshold-activa
154 lcium channel CaV1.3 and the big conductance calcium-activated potassium (BK) channel are preferentia
155                        The large-conductance calcium-activated potassium (BK) channel lacks a classic
156           The large conductance, voltage and calcium-activated potassium (BK) channel negatively regu
157 ract with the auxiliary beta4 subunit of the calcium-activated potassium (BK) channel; this interacti
158 ction in the expression of large-conductance calcium-activated potassium (BK) channels and Kv3.3 volt
159 ncreases in cytoplasmic Ca(2+), voltage- and calcium-activated potassium (BK) channels and their modu
160               Large conductance voltage- and calcium-activated potassium (BK) channels are highly exp
161                       The large-conductance, calcium-activated potassium (BK) channels help eliminate
162 xon domain of large conductance voltage- and calcium-activated potassium (BK) channels inhibiting cha
163 MRP's interaction with the large-conductance calcium-activated potassium (BK) channels that modulate
164 h nonmammals and mammals, large-conductance, calcium-activated potassium (BK) channels underlie a pri
165  spines act by suppressing large-conductance calcium-activated potassium (BK) channels, and this effe
166 of ion channels, including large conductance calcium-activated potassium (BK) channels.
167         The appearance of large-conductance, calcium-activated potassium (BK) current is a hallmark o
168              Large-conductance, voltage- and calcium-activated potassium (BK, or K(Ca)1.1) channels a
169                 ABSTRACT: Large conductance, calcium-activated potassium (BKCa ) channels have numero
170 larization potential, a voltage signature of calcium-activated potassium (Kca) channel activity.
171 ting through nitric oxide synthase (NOS) and calcium-activated potassium (KCa) channels in young adul
172 ctivation of nitric oxide synthase (NOS) and calcium-activated potassium (KCa) channels.
173  firing through their close association with calcium-activated potassium (KCa) channels.
174                            Small-conductance calcium-activated potassium (KCa2) channels have also be
175                             High-conductance calcium-activated potassium (Maxi-K) channels are presen
176                            Large conductance calcium-activated potassium (MaxiK) channels play a pivo
177  are occluded by apamin, a small-conductance calcium-activated potassium (SK(Ca)) channel blocker.
178 vivo, and were mediated by small-conductance calcium-activated potassium (SK) channel and CB1 cannabi
179                            Small conductance calcium-activated potassium (SK) channels are required f
180 um conductance mediated by small-conductance calcium-activated potassium (SK) channels in rat MNTB pr
181 +) conductance mediated by small-conductance calcium-activated potassium (SK) channels in the MNTB ne
182                Pharmacological inhibition of calcium-activated potassium (SK) channels increases the
183                            Small-conductance calcium-activated potassium (SK) channels mediate a pota
184                            Small-conductance calcium-activated potassium (SK) channels mediate medium
185                            Small-conductance calcium-activated potassium (SK) channels play an import
186                            Small-conductance calcium-activated potassium (SK) channels regulate actio
187 nucleotide-gated (HCN) and small conductance calcium-activated potassium (SK) channels.
188  increased contribution of small-conductance calcium-activated potassium (SK) channels.
189 t that voltage-independent small conductance calcium-activated potassium (SK)-channel dysfunction cau
190                            Small conductance calcium-activated potassium (SK2/K(Ca)2.2) channels are
191                            Large-conductance calcium-activated potassium BK channels are widely expre
192 ET-cGMP-S (50 muM), and the high-conductance calcium-activated potassium channel (BK(Ca) channel) inh
193             The Drosophila large-conductance calcium-activated potassium channel (dSlo) binds to and
194 e gene encoding the intermediate conductance calcium-activated potassium channel (IKCa) was termed SK
195                            Large-conductance calcium-activated potassium channel (KCa1.1; BK, Slo1, M
196 is associated with reduced small-conductance calcium-activated potassium channel (SK) currents and de
197                      Three small conductance calcium-activated potassium channel (SK) subunits have b
198          KCNN2 encodes the small conductance calcium-activated potassium channel 2 (SK2).
199 inding and also normalized large-conductance calcium-activated potassium channel activity.
200 ized protein levels of the large conductance calcium-activated potassium channel and the water channe
201 fication of a role for the large conductance calcium-activated potassium channel brings new thinking
202 nergic neuron burst firing by decreasing the calcium-activated potassium channel current (SK), as wel
203 dent activation of MthK, a two transmembrane calcium-activated potassium channel from thermophilic ar
204 rther demonstrate that the activation of the calcium-activated potassium channel is sufficient to ind
205                 The intermediate conductance calcium-activated potassium channel KCa3.1 contributes t
206                                          The calcium-activated potassium channel KCa3.1 controls diff
207                                          The calcium-activated potassium channel KCa3.1 is critically
208                 The intermediate-conductance calcium-activated potassium channel KCa3.1 is expressed
209  Among the altered genes, an increase of the calcium-activated potassium channel Kcnn2 in the motor c
210 staining to determine small conductance (SK) calcium-activated potassium channel protein levels.
211  members of the small-intermediate family of calcium-activated potassium channel proteins.
212                            We found that the calcium-activated potassium channel SK3 and the G protei
213 eterminant of its anthelmintic effect is the calcium-activated potassium channel SLO-1.
214 e found that Magi-1 bound to sequence like a calcium-activated potassium channel sodium-activated (Sl
215 ing peptide (GRP) and the small conductance, calcium-activated potassium channel, SK2.
216 releasing peptide and the small conductance, calcium-activated potassium channel, SK2.
217 g protein) modulates the Drosophila SLOWPOKE calcium-activated potassium channel.
218 um efflux from RBCs is the Gardos channel, a calcium-activated potassium channel.
219                      These findings identify calcium-activated potassium channelopathy as a cause of
220 he presence of functional large-conductance, calcium-activated potassium channels (BK channels) on th
221                 RATIONALE: Large-conductance calcium-activated potassium channels (BK) are composed o
222                                 Voltage- and calcium-activated potassium channels (BK) are important
223                            Large-conductance calcium-activated potassium channels (BK) are potent neg
224    Mammalian large-conductance, voltage- and calcium-activated potassium channels (BK, K(Ca)1.1) are
225 howed several significant results, including calcium-activated potassium channels (GO:0016286; P=2.30
226 ctifier channels (I(KV)) and noninactivating calcium-activated potassium channels (I(BK,steady)), and
227  co-localization of intermediate-conductance calcium-activated potassium channels (IKCa) and IP3 rece
228 e colocalization of intermediate-conductance calcium-activated potassium channels (IKCa) and TRPV4 ch
229  voltage-gated potassium channels (K(v)) and calcium-activated potassium channels (K(Ca)).
230               Large conductance voltage- and calcium-activated potassium channels (MaxiK, BK(Ca)) are
231                            Small conductance calcium-activated potassium channels (SK channels) are p
232  investigate the impact of small conductance calcium-activated potassium channels (SK channels) on de
233 d after acute dissociation, we found that BK calcium-activated potassium channels and Kv2 channels bo
234                      This work shows that BK calcium-activated potassium channels and Kv2 voltage-act
235 o identify the molecular gating mechanism in calcium-activated potassium channels by obtaining struct
236 f N-type voltage-gated calcium channels with calcium-activated potassium channels in DCN neurons.
237 f calcium-calmodulin-dependent kinase II and calcium-activated potassium channels in mediating these
238 d is abolished by blocking small conductance calcium-activated potassium channels with apamin.
239 ent is reduced by blocking large conductance calcium-activated potassium channels with iberiotoxin, a
240                        For large-conductance calcium-activated potassium channels, data are satisfact
241            Administration of an activator of calcium-activated potassium channels, SKA-31, partially
242 ropic acetylcholine receptors and associated calcium-activated potassium channels.
243 enced by the activation of small-conductance calcium-activated potassium channels.
244 ue to an increase in the open probability of calcium-activated potassium channels.
245 ation of small- and intermediate-conductance calcium-activated potassium channels.
246 cellular calcium that gated surface-membrane calcium-activated potassium channels.
247 ncreases the expression of small-conductance calcium-activated potassium channels.
248 ated by calcium-dependent processes, such as calcium-activated potassium channels.
249  I(AHP) was occluded by previous blockade of calcium-activated potassium channels.
250 d by a nonselective cation conductance and a calcium-activated potassium conductance (SK), respective
251 , we show that the excessive activation of a calcium-activated potassium conductance disrupts the ace
252  The slow afterhyperpolarization (sAHP) is a calcium-activated potassium conductance with critical ro
253     Elevation of [K(+) ](c) occurs through a calcium-activated potassium conductance, G(BK) , and a l
254  to calcium influx and activation of a large calcium-activated potassium conductance, G(BK) , that ca
255 endritic activation of the small conductance calcium-activated potassium current, SK.
256 hyperpolarization that is mediated by a slow calcium-activated potassium current.
257 perpolarizations that are mediated by a slow calcium-activated potassium current.
258 -clamp) that changes in the leak, sodium and calcium-activated potassium currents are central to thes
259 plasticity mediated by reductions in BK-type calcium-activated potassium currents in spontaneously fi
260 and a reduction in the fast-inactivating and calcium-activated potassium currents.
261 c outward currents through small-conductance calcium-activated potassium SK2 channels.
262 he contribution of the SK (small-conductance calcium-activated potassium) channel to neuronal functio
263 r firing range, and of SK (small-conductance calcium-activated potassium) currents, which were essent
264  ovary cells' plasma membrane gave rise to a calcium-activated, potassium-selective activity in patch
265    Length-tension relationships in maximally calcium activated preparations are relatively shallow an
266 tic cleavage of p35 to a p25 fragment by the calcium-activated protease calpain or by phosphorylation
267 y calpeptin and MDL-28170, inhibitors of the calcium-activated protease calpain.
268 ins220 was found to occur through calpain, a calcium-activated protease.
269  We report that calpastatin, an inhibitor of calcium-activated proteases of the calpain family, funct
270  animal models, the dysregulated activity of calcium-activated proteases, calpains, contributes direc
271 could cleave filamin A through activation of calcium-activated proteases, such as calpains.
272                                          The calcium activated protein, calcium/calmodulin-dependent
273 ction of the carboxypeptidase EGL-21 and the calcium-activated protein for secretion (CAPS) UNC-31, s
274   Additionally, the synaptic protein UNC-31 [calcium-activated protein for secretion (CAPS)] acts thr
275 s and SLO BK potassium channels to repress a calcium-activated protein kinase pathway.
276                    Finally, we show that the calcium-activated protein phosphatase calcineurin dephos
277              We investigated the role of the calcium-activated protein phosphatase calcineurin in sev
278 cataracts, and opacification may result from calcium-activated proteolysis.
279 suggest CXCL12 regulates restitution through calcium-activated Pyk2 localized to active focal adhesio
280     Together, these results demonstrate that calcium-activated removal of RNA from membranes by Xendo
281 we identified type 5 NADPH oxidase (NOX5), a calcium-activated, ROS-forming enzyme, as the missing li
282            The signaling axis comprising the calcium-activated Ser/Thr phosphatase calcineurin (CN),
283                        Calcineurin (Cn) is a calcium-activated serine/threonine protein phosphatase t
284 entify a unique mechanism of virus-mediated, calcium-activated signaling that initiates autophagy and
285 blocked by apamin, a selective antagonist of calcium-activated SK channels.
286 yed rectifier, A-type, and small-conductance calcium-activated (SK) potassium and HCN) and two recept
287 ntaining ionotropic receptors and associated calcium-activated (SK2) potassium channels, providing th
288 ce was mediated via intermediate conductance calcium-activated (SK4) potassium channels.
289 eartbeat, cardiac contractility results from calcium-activated sliding of actin thin filaments toward
290                                          The calcium-activated slow afterhyperpolarization (sAHP) is
291 t KCNQ channels might partially underlie the calcium-activated slow afterhyperpolarization (sAHP), a
292                                  We focus on calcium-activated small conductance (SK) potassium chann
293 ndependent NETosis is fast and mediated by a calcium-activated small conductance potassium (SK) chann
294                                          The calcium-activated small conductance potassium channel SK
295 holinergic receptors functionally coupled to calcium-activated, small conductance (SK2) potassium cha
296 nge likely used during flight), stretch- and calcium-activated tension contributed 80% and 20%, respe
297 V domain-caged protease cleavage site, and a calcium-activated TEV protease that we designed through
298          We found that EGF and extracellular calcium activated the C-terminus of ERalpha and the acti
299                Muscle cell preparations were calcium activated to yield 50% maximal force, after whic
300 Our study links calpain and dysferlin in the calcium-activated vesicle fusion of membrane repair, pla

 
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