ライフサイエンスコーパス: calcium-induced

1 n of a dominant-negative CaR that eliminated calcium-induced 5-HT secretion but not secretion in resp

2 Increasing extracellular calcium induced a dose-dependent increase in MMP-9 expre

3 yers of PIP2 mixed with zwitterionic lipids, calcium induced a rapid, PIP2-dependent surface pressure

4 Calcium induced a reversible change in the extracellular

5 Moreover, calcium induced a significant change in the conformation

6 YDL206W gene leads to a diminished level of calcium-induced activation of calcineurin and a reduced

7 Calcium-induced activation of calpain has been shown to

8 ults in specific cellular changes, including calcium-induced activation of calpain proteases.

9 We propose that calcium-induced activation of cPKC-alpha hypoxia partial

10 data, have been used to develop a model for calcium-induced activation of NDR kinase by S100B.

11 on of PLC-gamma1 by epidermal growth factor, calcium-induced activation of PLC-gamma1 was not a resul

12 Our data suggest that this calcium-induced activation of TGase protein occurs while

13 es in the MyHC IIa promoter are required for calcium-induced activation of the MyHC IIa promoter.

14 This excludes a calcium-induced additive TRPA1 current after TRPV1 stimu

15 Calcium-induced aggregation has been proposed to play a

16 over, this is the first evidence that low pH/calcium-induced aggregation is necessary for sorting of

17 Thus, calcium-induced aggregation is not a passive process; ra

18 eate aggregation chaperones that enhance the calcium-induced aggregation of secretory granule protein

19 cell types, salivary proteins do not exhibit calcium-induced aggregation.

20 ctionality, with increased stability against calcium-induced aggregation.

21 pore antagonist cyclosporin A also inhibited calcium-induced AIF release from mouse liver mitochondri

22 However, the extent to which calcium-induced alternation of electrical activity in th

23 STIM2 interacts with AMPK and regulates calcium-induced AMPK activation.

24 ults suggest that GPAnt-2a peptide augmented calcium-induced amylase release from permeabilized acini

25 antagonist of Go and Gi, showed no effect on calcium-induced amylase release from permeabilized acini

26 ning monolayers; however, in these mixtures, calcium induced an unexpected, PIP2- and multivalency-de

27 astrocyte activation elevating intracellular calcium induced anxiolytic behaviors in astrocyte-specif

28 The formation of calcium-induced AP-1 binding complexes is regulated by p

29 atinocyte marker genes demonstrated that the calcium-induced AP-1 DNA binding activity does not corre

30 Calcium-induced AP-1 DNA binding complexes consist of Fr

31 results suggested that susceptibility during calcium-induced apoptosis is limited by availability of

32 n the lens, 2) a novel signaling pathway for calcium-induced apoptosis, and 3) a novel antiapoptotic

33 8 kinase and JNK2, which are not involved in calcium-induced apoptosis.

34 disruption of tight junction and accelerates calcium-induced assembly of tight junction in Caco-2 cel

35 In this study, the calcium-induced association of cPLA2-alpha with EA.hy.92

36 in-like protein did not significantly affect calcium-induced biofilm structure.

37 A calcium-induced but Dex-inhibited nuclear complex contai

38 urther show that STIM2 deficiency attenuates calcium-induced but not energy stress-induced AMPK activ

39 how that the sustained rise in intracellular calcium induced by activation of P2X(7) receptors direct

40 rated calcium entry" - the cellular entry of calcium induced by depletion of intracellular calcium st

41 the GABA-elicited current, but increases in calcium induced by depolarization alone did not.

42 nate between the varying levels of cytosolic calcium induced by different stimuli.

43 d apoE3 affect the increase in intracellular calcium induced by either NMDA or KCl.

44 n without altering the rise in intracellular calcium induced by glucose or K+.

45 RCASBP(A) blocks increases in intracellular calcium induced by nicotine through alpha7-nAChRs and pr

46 xpressed SphK2 also increased cytosolic free calcium induced by serum starvation.

47 o specifically disrupt the transient rise in calcium induced by serum stimulation of starved Swiss 3T

48 d more slowly from an elevation in cytosolic calcium induced by the InsP3 agonist carbachol.

49 In contrast, the increase of intracellular calcium induced by treatment with calcium and ionophore

50 ed [Ca(2+)](i), followed by propagation (via calcium-induced Ca(2+) release, CICR) to the cell centre

51 acaine can be accounted for by depression of calcium-induced Ca2+ release (CICR).

52 from patients with CKD accumulated calcium; calcium induced calcification more potently than phospha

53 e in intracellular calcium could be due to a calcium induced calcium release (CICR) process that is i

54 ium concentration in stores, and Orai-1, the calcium-induced calcium entry channel, are colocalized w

55 ve Ca2+ waves and oscillations indicative of calcium-induced calcium release (CICR) activity were ind

56 ivity and extracellular calcium, implicating calcium-induced calcium release (CICR) as the novel sour

57 sponsible for the regulation of regenerative calcium-induced calcium release (CICR) during Ca(2+) spa

58 se in the AD strains, suggesting an aberrant calcium-induced calcium release (CICR) effect within spi

59 It has been proposed that calcium-induced calcium release (CICR) from a near-membr

60 MPAR-mediated calcium signal is amplified by calcium-induced calcium release (CICR) from intracellula

61 ent by activation of glutamate receptors and calcium-induced calcium release (CICR) from intracellula

62 ential (V(m)) and calcium current (I(Ca)) of calcium-induced calcium release (CICR) from the junction

63 sion, thereby suggesting that LMO4 regulates calcium-induced calcium release (CICR) in central neuron

64 Calcium-induced calcium release (CICR) is a mechanism by

65 Stable calcium-induced calcium release (CICR) is critical for m

66 l is generated by calcium influx or requires calcium-induced calcium release (CICR) is not yet known.

67 ), which is an extension of the smooth ER, a calcium-induced calcium release (CICR) is triggered at t

68 ment of smaller local events, probably via a calcium-induced calcium release (CICR) mechanism.

69 om the stellate ganglia to establish whether calcium-induced calcium release (CICR) modulated action

70 ce suggests that internal calcium stores and calcium-induced calcium release (CICR) provide an import

71 I(Ca) did not evoke significant calcium-induced calcium release (CICR) since (i)[Ca2+]i

72 The gain of calcium-induced calcium release (CICR) was increased at

73 In calcium-induced calcium release (CICR), calcium ions flo

74 lcium and was diminished by the inhibitor of calcium-induced calcium release (CICR), dantrolene.

75 ats, ryanodine (1-50 microM), a modulator of calcium-induced calcium release (CICR), had no effect on

76 L-type Ca2+ channels, and amplification via calcium-induced calcium release (CICR).

77 of the cochlea with ryanodine, an agonist of calcium-induced calcium release (CICR).

78 Simulations based on a model of calcium-induced calcium release and cell-to-cell diffusi

79 This model uses the calcium-induced calcium release and inositol cross-coupl

80 nts and membrane transporters, mechanisms of calcium-induced calcium release and intracellular calciu

81 Abolishing calcium-induced calcium release by blocking ryanodine re

82 The calcium-induced calcium release channel of the cardiac s

83 ar free calcium concentrations by activating calcium-induced calcium release from intracellular store

84 he amplitude and duration of Ca2+ sparks and calcium-induced calcium release gain.

85 llular stores strongly implicates a role for calcium-induced calcium release in activity-dependent BD

86 6 microm resiniferatoxin caused a pronounced calcium-induced calcium release in either vanilloid rece

87 , they identify a physiological role for the calcium-induced calcium release in hippocampus and provi

88 It is concluded that partial inhibition of calcium-induced calcium release increases SR Ca2+ conten

89 to other stimuli through a ryanodine-based, calcium-induced calcium release mechanism.

90 Waves of calcium-induced calcium release occur in a variety of ce

91 latory release of calcium is inherent in the calcium-induced calcium release process.

92 f rapidly triggering neighboring channels by calcium-induced calcium release to evoke a puff, optimal

93 caffeine, which enhances the contribution of calcium-induced calcium release to the afterhyperpolariz

94 of cardiac excitation-contraction coupling ('calcium-induced calcium release') is now reasonably well

95 ux and hence a decrease in the gain of local calcium-induced calcium release).

96 reticulum and is released by the process of calcium-induced calcium release.

97 rs leads to mobilization of store calcium by calcium-induced calcium release.

98 eart cells are mediated by diffusion-coupled calcium-induced calcium release.

99 onstant amplitude; the spread was mostly via calcium-induced calcium release.

100 antrolene and ruthenium red, two blockers of calcium-induced calcium release.

101 uce the afterhyperpolarization by regulating calcium-induced calcium release.

102 actions of the ryanodine receptor by way of calcium-induced calcium release.

103 These findings indicate that calcium-induced calcium released from intraneuronal stor

104 ent with this possibility, administration of calcium-induced calcium-release blockers, as well as of

105 mic signaling is supported by a ROS-assisted calcium-induced calcium-release mechanism intimately inv

106 e conclude that a Ca(2+) diffusion-dominated calcium-induced calcium-release mechanism is insufficien

107 e release of intracellular calcium through a calcium-induced calcium-release mechanism.

108 During calcium-induced calcium-release, the ryanodine receptor

109 sor") whose intracellular dynamics involve a calcium-induced, calcium release process.

110 se two structures to form dyads within which calcium-induced-calcium-release occurs.

111 Activation is mediated by calcium-induced calmodulin binding to an IQ domain near

112 Calcium-induced calmodulin-mediated inhibition of myocar

113 Q motif binds apo-calmodulin (CaM), and that calcium-induced CaM release triggers a reversible confor

114 ork, we model the spatiotemporal dynamics of calcium-induced cAMP signaling pathway in dendritic spin

115 esent a well-mixed model for the dynamics of calcium-induced cAMP/PKA dynamics in dendritic spines.

116 mics of calcium are reasonably well-studied, calcium-induced cAMP/PKA dynamics, particularly with res

117 e role of tamoxifen in calcium signaling and calcium-induced cell death was studied in both malignant

118 lve the C-terminus and linker regions, these calcium-induced changes have implications for the role o

119 ry protein calmodulin is a major mediator of calcium-induced changes in cellular activity.

120 ion of native disulfide bonds and detectable calcium-induced changes in structure when the two C-term

121 in but depended on propagation of effects of calcium-induced changes in the C-terminal domain.

122 ermeation chromatography was used to resolve calcium-induced changes in the hydrated shape of CaM at

123 The other region that undergoes calcium-induced changes is at the receptor region, where

124 of calcium activation and the basis for the calcium-induced changes remain unclear.

125 vironment, using a biosensor that visualizes calcium-induced chloride ion flux in the cell.

126 t cysteine protease, effectively inhibit the calcium-induced cleavage of p35.

127 Calcium-induced conformation change of TN-XXL results in

128 ed rate of dissociation is limited by a slow calcium-induced conformational change (3 s-1).

129 Helix 3, which undergoes a large, calcium-induced conformational change necessary for targ

130 t sequence in the hybrid protein undergoes a calcium-induced conformational change to bind to the cal

131 Q), and complete (E151Q,E188Q) disruption of calcium-induced conformational changes determined by NMR

132 Calcium-induced conformational changes expose a hydropho

133 toward describing the molecular dynamics of calcium-induced conformational changes in proteins using

134 ference in the A93G mutant that prevents the calcium-induced conformational changes in the S1 domain

135 148) were interpreted as directly reflecting calcium-induced conformational changes in whole calmodul

136 pray mass spectrometry investigations of the calcium-induced conformational changes of calbindin D(28

137 A plasmonic switch based on the calcium-induced conformational changes of calmodulin is

138 Biophysical studies of the calcium-induced conformational changes of CaM disagree o

139 Possible implications of this calcium-induced conformational switch for the membrane a

140 ccurs because of the antagonistic effects of calcium-induced contractility and stretch-activated calc

141 Similar to binary mixtures, subphase calcium induced contraction of ternary cholesterol-conta

142 NP diffusive behaviors change as a result of calcium-induced cross-linking of the alginate matrix.

143 t the same level as CaMKII in the pathway of calcium-induced CSF release by cooperating with CaMKII t

144 econd photolytic step originates from a slow calcium-induced dark rearrangement of the first intermed

145 ession of IFN-gamma, and of IL-2, depends on calcium-induced de novo transcription and PKC-dependent

146 V and CREB play a critical role in mediating calcium-induced dendritic growth in cortical neurons.

147 These experiments indicate that calcium-induced dendritic growth is regulated by activat

148 ate, CNS mitochondria maintained a sustained calcium-induced depolarization without appreciable swell

149 This calcium-induced depression of the kainate receptor curre

150 During calcium-induced desmosome assembly, treatment of primary

151 Calcium-induced differentially expressed genes (DEGs) we

152 ail to exit S and G2/M phases in response to calcium-induced differentiation and also resist exhausti

153 up-regulated and correlated temporally with calcium-induced differentiation and expression of the la

154 ion of TIP39 in keratinocytes changed during calcium-induced differentiation and shifted to colocaliz

155 mary human keratinocytes that are undergoing calcium-induced differentiation can rapidly activate mat

156 required for mediating calcium signaling and calcium-induced differentiation in keratinocytes.

157 ent in cultured human keratinocytes and that calcium-induced differentiation markedly decreases SR-BI

158 Calcium-induced differentiation of these cells has been

159 keratinocytes and suggested that it signaled calcium-induced differentiation of these cells.

160 n keratinocytes and determined the effect of calcium-induced differentiation on its mRNA levels.

161 In isolated primary mouse keratinocytes, calcium-induced differentiation was accompanied by speci

162 s show alterations in plating efficiency and calcium-induced differentiation, but proliferate normall

163 During calcium-induced differentiation, H3K27me3 was erased at

164 examined its impact on calcium signaling and calcium-induced differentiation.

165 alcium response to extracellular calcium and calcium-induced differentiation.

166 hway integral to or overlapping with that of calcium-induced differentiation.

167 Enzyme activation requires calcium-induced dimerisation plus bilayer perturbation.

168 results suggest that passive clustering, via calcium-induced dimerization or membrane ordering, may c

169 This may arise from calcium-induced disruption of interactions between the a

170 Our results suggest that calcium-induced electrical instability may increase arrh

171 The calcium-induced endocytosis rate increase was a result o

172 the role of the calcium-sensing receptor in calcium induced epidermal differentiation, we investigat

173 In Parkinson's disease oxidative stress and calcium-induced excitotoxicity have been considered impo

174 but not kinase-dead Aurora B INCENP, blocked calcium-induced exit from metaphase arrest in egg extrac

175 sms of platelet secretion, we focused on the calcium-induced exocytosis of dense core granules.

176 Upon calcium-induced exocytosis, XlMyo1c is recruited to exoc

177 es cell proliferation and temporally affects calcium-induced expression of differentiation markers.

178 In this study, we examined the calcium-induced expression of the cytoprotective beta ce

179 The calcium-induced extracellular matrix of mucoid P. aerugi

180 Calcium-induced F-actin depolymerization was attenuated

181 The impact of calcium-induced fat droplet aggregation on the microstru

182 een identified as the PLA(2) responsible for calcium-induced fatty acid release and prostaglandin E(2

183 in fusion at the C-terminus likewise enabled calcium-induced folding but fusions solely at the N-term

184 This demonstrates that calcium-induced folding is an inherent property of the b

185 he involvement of individual residues in the calcium-induced folding reactions.

186 natural C-terminal flank was used to enable calcium-induced folding, pointing to its cooperative par

187 al muscle troponin activator, CK-2066260, on calcium-induced force development was studied in skinned

188 sesses critical determinants necessary for a calcium-induced functionally required conformation.

189 acking SV2 have fewer vesicles competent for calcium-induced fusion.

190 excitability, neurotransmitter release, and calcium-induced gene regulation.

191 ry keratinocytes were partially resistant to calcium-induced growth arrest.

192 We have previously demonstrated that calcium-induced human keratinocyte differentiation requi

193 phospholipase C (PLC)-gamma1 is required for calcium-induced human keratinocyte differentiation.

194 This activation is required for calcium-induced human keratinocyte differentiation.

195 mutation dramatically reduced the extent of calcium-induced hydrophobic exposure by the C-domain.

196 ix of CyaA makes extensive contacts with the calcium-induced, hydrophobic pocket of calmodulin.

197 Calcium-induced in vitro proteolysis was retarded in the

198 of EADs caused by a reduction in the rate of calcium-induced inactivation of the L-type calcium chann

199 he dependence on diastolic calcium is due to calcium-induced inactivation of the L-type calcium curre

200 ke components (pf14 and pf17) do not exhibit calcium-induced increase in dynein activity in pCa4 buff

201 The calcium-induced increase in dynein activity in pf18 axon

202 is, increased p53 levels are necessary for a calcium-induced increase in neurons.

203 However, the calcium-induced increase in tau phosphorylation was inhi

204 of PP2A-Calpha and PP1alpha accelerated the calcium-induced increase in transepithelial electrical r

205 of calcium on energy or fat balance, despite calcium-induced increases (P <0.01) in postprandial seru

206 The calcium-induced increases in tau phosphorylation are not

207 Calcium-induced inhibition of cell proliferation and cal

208 decreased release probability is caused by a calcium-induced inhibition of presynaptic calcium channe

209 s causes calpain-mediated death at levels of calcium-induced injury that are sublethal to cells norma

210 Pore dilation relied on calcium-induced insertion of the tandem C2 domain hydrop

211 CD26 activity was stimulated by calcium-induced intercellular adhesion in keratinocytes,

212 Yeast CcO also displayed a calcium-induced IR and UV/visible binding spectra, thoug

213 Priming is triggered via calcium-induced JNK signaling, which leads to upregulati

214 Here, we show that upon calcium-induced junction biogenesis in Madin-Darby canin

215 on 5, which were suggested to be involved in calcium induced keratinocyte differentiation.

216 MED21 expression also resulted in defects in calcium-induced keratinocyte differentiation, as indicat

217 ium channel-alpha and -beta increased during calcium-induced keratinocyte differentiation.

218 ation of PI3K and phospholipase C-gamma1 and calcium-induced keratinocyte differentiation.

219 Increased extracellular calcium-induced KLK5 and KLK7 mRNA expression and protei

220 Calcium-induced light-scattering, measured in vitro for

221 The calcium-induced loss in fluorescence intensity is only a

222 calcium to the lysosomal lumen and mediates calcium-induced lysosomal proton leakage.

223 signal initiated by RyR1, perhaps through a calcium-induced mechanism.

224 oxygen species, and earlier formation of the calcium-induced membrane permeability transition pore.

225 uced uptake of calcium, but partly inhibited calcium-induced membrane scrambling.

226 all, these findings establish a mechanism of calcium-induced mitochondrial dysfunction and the impact

227 ial respiration and increased sensitivity to calcium-induced mitochondrial permeability transition po

228 The threshold for calcium-induced mitochondrial permeability transition wa

229 Both the extent and rate of calcium-induced mitochondrial swelling and depolarizatio

230 values, 0.2-16.2 mumol/L) and, as a result, calcium-induced mitochondrial swelling, by preventing mP

231 am kinase MAPK kinase 3 (MKK3) increased the calcium induced MMP-9 gene expression, demonstrating tha

232 Together these data support a model wherein calcium-induced MMP-9 expression is differentially regul

233 These findings support a model whereby calcium-induced modification of PICK1 structure is criti

234 NFAT confirmed that the BTP compounds block calcium-induced movement of NFAT from the cytosol to the

235 ull mutants, and (3) raise the threshold for calcium-induced mPT in acutely prepared mitochondria fro

236 nd a similar increased susceptibility to the calcium-induced MPT in liver mitochondria isolated from

237 tochondria showed elevated susceptibility to calcium-induced MPTP opening, whereas mitochondrial oxid

238 e to fatty acyl-CoA-mediated augmentation of calcium-induced mPTP opening.

239 the palmitoyl-CoA-mediated amplification of calcium-induced mPTP opening.

240 lus ionomycin, TPA, and raised extracellular calcium, induced nuclear translocation of NFAT1 and calc

241 ion of CCTalpha with 14-3-3 zeta to initiate calcium-induced nuclear entry.

242 Blood, Bucher et al investigate the role of calcium-induced nuclear factor of activated T cells (NFA

243 ndscape to transcriptionally up-regulate the calcium-induced nuclear factor of activated T cells (NFA

244 However, each isoform undergoes calcium-induced nuclear translocation from the cytoplasm

245 This human lens calcium-induced opacification (HLCO) model enables inves

246 is a critical mechanistic participant in the calcium-induced opening of the mitochondrial permeabilit

247 to longer distances by 8 to 11 A indicate a calcium-induced opening of the N-terminal domain conform

248 se in CaR mRNA in these cells as well as the calcium-induced opening of the nonspecific cation channe

249 ABA-induced, calcium-induced or spontaneous [Ca2+]cyt increases were

250 uard cells, experimentally imposing external calcium-induced oscillations rescued stomatal closure.

251 asome inhibitor, although only CSA inhibited calcium-induced permeability transition in liver-derived

252 sensitivity of mdx diaphragm mitochondria to calcium-induced permeability transition pore opening was

253 ced IMM permeabilization by the mechanism of calcium-induced permeability transition.

254 tenin at the plasma membrane is required for calcium-induced phospholipase C-gamma1 activation and, u

255 Moreover, we found that protein kinase C and calcium-induced phosphorylation of proteins thought to i

256 Calcium-induced PI3K recruitment to E-cadherin stabilize

257 6Q, E78Q were predicted to cause loss of the calcium-induced positive face in calprotectin, reducing

258 hat is reduced to 54% by calcium, permeating calcium-induced potentiation followed by closure, and re

259 Our work thus reveals a novel calcium-induced PP1 activation pathway critical for home

260 cal domain disulfides, are necessary for the calcium-induced progression from the molten globule towa

261 e formation induced by either alamethicin or calcium-induced PTP opening.

262 h aminophospholipid translocase activity and calcium-induced randomization of membrane phospholipids.

263 calcium transients with photolysis of caged calcium induced rapid outgrowth of axonal processes.

264 into the intercellular junctions during the calcium-induced reassembly of tight junction were much g

265 Calcium-induced reassembly of Y398D/Y402D mutant occludi

266 st that such interactions may be involved in calcium-induced reduction in the open probability of NMD

267 t depolarizes the hair cell and triggers the calcium-induced release of the neurotransmitter glutamat

268 Extracellular calcium induced repetitive [Ca2+]cyt transients with pea

269 ntisense phospholipase C-gamma1 to block the calcium-induced rise in intracellular calcium and found

270 Calcium-induced rotation of the helices in the EF-hands,

271 lcium, is, in many cell types, the result of calcium-induced secretion of ATP and activation of purin

272 This calcium-induced secretion relies on the SNARE proteins b

273 The calcium-induced secretion was inhibited by a dominant-ne

274 However, only 1.2 mM calcium induced significant amounts of TGase activity.

275 specifically with nanomolar affinity to the calcium-induced SRCR conformation in an immobilized stat

276 tream of cytosolic calcium and extracellular calcium-induced stomatal closure were unaffected in era1

277 Mutations that interfered with the calcium-induced structural changes in PICK1 precluded LT

278 in is independent of ionic strength, and the calcium-induced structural transition is slightly inhibi

279 he role of gamma-carboxyglutamic acid in the calcium-induced structural transition of conantokin G, w

280 ed small-molecule inhibitors of CypD reduced calcium-induced swelling in mitochondria from mouse and

281 2)gamma(-/-) mice were markedly resistant to calcium-induced swelling in the presence or absence of p

282 Measurements of tissue NAD+ levels and calcium-induced swelling of mitochondria isolated at 3 m

283 ent anion channel and were more resistant to calcium-induced swelling than cardiac mitochondria from

284 ecretion signal, they are believed to act as calcium-induced switches that prevent folding before sec

285 Both constitutive and calcium-induced T-plastin expression was down-regulated

286 levels of alpha(6)p increased 3-fold during calcium-induced terminal differentiation in a normal mou

287 vel of AMPK phosphorylation increases during calcium-induced tight junction assembly and cell polariz

288 We found that AMPK is activated during calcium-induced tight junction assembly.

289 TF family, have been implicated in cAMP- and calcium-induced transcriptional activation.

290 N' region, do not play a direct role in the calcium-induced transition in the cardiac regulatory or

291 2)alpha) is regulated by phosphorylation and calcium-induced translocation to membranes.

292 cells expressing wild type cPLA(2)alpha, but calcium-induced translocation was not impaired.

293 amounts of Yops and display a defect in low-calcium-induced type III secretion of Yop proteins.

294 ion of dominant negative src and fyn blocked calcium-induced tyrosine phosphorylation of the regulato

295 (SLC25A27) and UCP5 (SLC25A14)), compromised calcium-induced uncoupling and increased oxidation of ma

296 Moreover, cGMP, not calcium, induced Unit I/-46GUS activity.

297 This peptide (PP-5) promoted calcium-induced vesicle aggregation of phosphatidylethan

298 pH (pH 4.0) of the BBMV suspension abolished calcium-induced vesicle aggregation, whereas treatment w

299 helium of M. sexta, caused a 50% decrease in calcium-induced vesicle aggregation.

300 rbimycin inhibited the rapid mobilization of calcium induced via CD40, suggesting that calcium mobili