ライフサイエンスコーパス: calcium-induced calcium release

1 ux and hence a decrease in the gain of local calcium-induced calcium release).

2 reticulum and is released by the process of calcium-induced calcium release.

3 actions of the ryanodine receptor by way of calcium-induced calcium release.

4 rs leads to mobilization of store calcium by calcium-induced calcium release.

5 eart cells are mediated by diffusion-coupled calcium-induced calcium release.

6 onstant amplitude; the spread was mostly via calcium-induced calcium release.

7 antrolene and ruthenium red, two blockers of calcium-induced calcium release.

8 uce the afterhyperpolarization by regulating calcium-induced calcium release.

9 Simulations based on a model of calcium-induced calcium release and cell-to-cell diffusi

10 This model uses the calcium-induced calcium release and inositol cross-coupl

11 nts and membrane transporters, mechanisms of calcium-induced calcium release and intracellular calciu

12 ent with this possibility, administration of calcium-induced calcium-release blockers, as well as of

13 Abolishing calcium-induced calcium release by blocking ryanodine re

14 The calcium-induced calcium release channel of the cardiac s

15 e in intracellular calcium could be due to a calcium induced calcium release (CICR) process that is i

16 ve Ca2+ waves and oscillations indicative of calcium-induced calcium release (CICR) activity were ind

17 ivity and extracellular calcium, implicating calcium-induced calcium release (CICR) as the novel sour

18 sponsible for the regulation of regenerative calcium-induced calcium release (CICR) during Ca(2+) spa

19 se in the AD strains, suggesting an aberrant calcium-induced calcium release (CICR) effect within spi

20 It has been proposed that calcium-induced calcium release (CICR) from a near-membr

21 MPAR-mediated calcium signal is amplified by calcium-induced calcium release (CICR) from intracellula

22 ent by activation of glutamate receptors and calcium-induced calcium release (CICR) from intracellula

23 ential (V(m)) and calcium current (I(Ca)) of calcium-induced calcium release (CICR) from the junction

24 sion, thereby suggesting that LMO4 regulates calcium-induced calcium release (CICR) in central neuron

25 Calcium-induced calcium release (CICR) is a mechanism by

26 Stable calcium-induced calcium release (CICR) is critical for m

27 l is generated by calcium influx or requires calcium-induced calcium release (CICR) is not yet known.

28 ), which is an extension of the smooth ER, a calcium-induced calcium release (CICR) is triggered at t

29 ment of smaller local events, probably via a calcium-induced calcium release (CICR) mechanism.

30 om the stellate ganglia to establish whether calcium-induced calcium release (CICR) modulated action

31 ce suggests that internal calcium stores and calcium-induced calcium release (CICR) provide an import

32 I(Ca) did not evoke significant calcium-induced calcium release (CICR) since (i)[Ca2+]i

33 The gain of calcium-induced calcium release (CICR) was increased at

34 In calcium-induced calcium release (CICR), calcium ions flo

35 lcium and was diminished by the inhibitor of calcium-induced calcium release (CICR), dantrolene.

36 ats, ryanodine (1-50 microM), a modulator of calcium-induced calcium release (CICR), had no effect on

37 L-type Ca2+ channels, and amplification via calcium-induced calcium release (CICR).

38 of the cochlea with ryanodine, an agonist of calcium-induced calcium release (CICR).

39 ar free calcium concentrations by activating calcium-induced calcium release from intracellular store

40 a calcium signal to the dendritic shaft, the calcium-induced calcium release from this intracellular

41 These findings indicate that calcium-induced calcium released from intraneuronal stor

42 he amplitude and duration of Ca2+ sparks and calcium-induced calcium release gain.

43 llular stores strongly implicates a role for calcium-induced calcium release in activity-dependent BD

44 6 microm resiniferatoxin caused a pronounced calcium-induced calcium release in either vanilloid rece

45 , they identify a physiological role for the calcium-induced calcium release in hippocampus and provi

46 It is concluded that partial inhibition of calcium-induced calcium release increases SR Ca2+ conten

47 of cardiac excitation-contraction coupling ('calcium-induced calcium release') is now reasonably well

48 to other stimuli through a ryanodine-based, calcium-induced calcium release mechanism.

49 mic signaling is supported by a ROS-assisted calcium-induced calcium-release mechanism intimately inv

50 e conclude that a Ca(2+) diffusion-dominated calcium-induced calcium-release mechanism is insufficien

51 e release of intracellular calcium through a calcium-induced calcium-release mechanism.

52 Waves of calcium-induced calcium release occur in a variety of ce

53 se two structures to form dyads within which calcium-induced-calcium-release occurs.

54 latory release of calcium is inherent in the calcium-induced calcium release process.

55 sor") whose intracellular dynamics involve a calcium-induced, calcium release process.

56 During calcium-induced calcium-release, the ryanodine receptor

57 f rapidly triggering neighboring channels by calcium-induced calcium release to evoke a puff, optimal

58 caffeine, which enhances the contribution of calcium-induced calcium release to the afterhyperpolariz