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1 nd physical properties of human cortical and cancellous bone.
2 matory process that demineralizes trabecular cancellous bone.
3 m in cortical bone, but tempers bone gain in cancellous bone.
4 c stem and progenitor cells (HSPCs) in human cancellous bone.
5 localized throughout the marrow cavities of cancellous bone.
6 in mouse femur is also present within human cancellous bone.
7 tion of a threaded titanium cage packed with cancellous bone.
8 d unique metabolic programs for cortical and cancellous bone.
9 osteum but not in cells from endocortical or cancellous bone.
13 orphometric analysis of tetracycline-labeled cancellous bone and dual-energy x-ray absorptiometry, re
14 effects of microstructure on fatigue life in cancellous bone and lattice structures are described emp
18 We show that the more ductile surfaces of cancellous bone are a result of reduced accumulation of
19 creased bone density, serum osteocalcin, and cancellous bone area along with trabecular narrowing.
21 palpable difference between the cortical and cancellous bone, both of which have different material p
22 ts osteoclastogenesis and bone resorption in cancellous bone but increases intracortical remodeling a
23 Y, 32:68, wt/wt; DBM mixed with cortical and cancellous bone chips 1:4 (DBMC) (11 mg total, of which
24 ociated with enhanced bone resorption in the cancellous bone compartment and with suppressed endocort
31 ls in postnatal mice dramatically stimulated cancellous bone formation via marked expansion of the os
33 is study, data suggests GTR using allogeneic cancellous bone graft and absorbable collagen membrane t
34 defects treated with GTR using an allogeneic cancellous bone graft and covered by an absorbable membr
38 Histomorphometric parameters characterizing cancellous bone in the distal radius can be derived from
39 anatomical structures including cortical and cancellous bone, intervertebral discs, ligaments, and ca
41 The ability to create mechanically strong 'cancellous bone-like' printable implants for tissue repa
45 ontribute to the increase in osteoclasts and cancellous bone loss that occurs after loss of estrogen.
46 ion in mediating estrogen deficiency-induced cancellous bone loss was investigated in ovariectomized
50 s suggest that CMA plays a role in vertebral cancellous bone mass accrual in young adult mice and tha
51 age female L2ACgKO mice had lower vertebral cancellous bone mass compared to wild-type (WT) controls
53 e, but not in adult mice, whereas epiphyseal cancellous bone mass decreased with loading in both youn
54 likely to accumulate in strut centers making cancellous bone more tolerant of stress concentrations a
56 tructed and registered, and the cortical and cancellous bones of the mandible and the maxilla were se
58 creased prevalence of apoptosis in vertebral cancellous bone osteocytes and osteoblasts that follows
59 n conclusion, Notch2(tm1.1Ecan) mice exhibit cancellous bone osteopenia that can be ameliorated by sy
65 evised a method for obtaining information on cancellous bone structure from iliac bone histomorphomet
70 tes is essential for osteoclast formation in cancellous bone under physiological conditions, and RANK
72 Notch in the skeleton causes an increase in cancellous bone volume and enhanced osteoblastic differe
73 reatment was associated with preservation of cancellous bone volume and inhibition of osteoclast form
75 microstructural abnormalities such as lower cancellous bone volume and reduced trabecular thickness.
76 mined by dual-energy densitometry; decreased cancellous bone volume and trabecular width and increase
77 ob/ob mice pair-fed to WT mice had normal cancellous bone volume fraction (BV/TV) in distal femur,
78 content (BMC) and density (BMD), and higher cancellous bone volume fraction in lumbar vertebra (LV).
82 e characterized by a significant decrease in cancellous bone volume in the tibial and femoral metaphy
84 usly over the calvaria of mice and increased cancellous bone volume when orally administered to rats.
85 7(-/-)) exhibit higher bone mineral density, cancellous bone volume, and mechanical strength compared
86 iblings, demonstrated a striking decrease in cancellous bone volume, connectivity, and trabecular num
87 m, and normalization of bone markers such as cancellous bone volume, trabecular number, osteoblast su
91 ature osteocytes in mineralized cortical and cancellous bone was positive for sclerostin with diffuse
95 fiber reinforcement reached the strength of cancellous bone, which was much stronger than previous i