ライフサイエンスコーパス: carbamazepine

1 40s who developed DIHS after treatment with carbamazepine.

2 pharmacological rescue by glibenclamide and carbamazepine.

3 er by the prototypical Na(+) channel blocker carbamazepine.

4 he antiepileptic drug topiramate, but not by carbamazepine.

5 accumulated to a greater degree in bile than carbamazepine.

6 alproate and for lower verbal abilities with carbamazepine.

7 s alleviated by the sodium channel inhibitor carbamazepine.

8 with epilepsy or seizure when compared with carbamazepine.

9 nts compared with topiramate and secondarily carbamazepine.

10 agents, and possibly the antiepileptic agent carbamazepine.

11 non-inferiority of lamotrigine compared with carbamazepine.

12 cochaperones glibenclamide, repaglinide, and carbamazepine.

13 rotein-induced EAE treated with phenytoin or carbamazepine.

14 c treatments such as lithium, valproate, and carbamazepine.

15 s the best candidate enzyme for metabolizing carbamazepine.

16 taking placebo to 3015 minutes while taking carbamazepine.

17 taking placebo to 45.3 minutes while taking carbamazepine.

18 ally relevant thermal stimuli was reduced by carbamazepine.

19 intenance period to 136 minutes while taking carbamazepine.

20 enance period and 32 awakenings while taking carbamazepine.

21 ndetectable or significantly lower levels of carbamazepine.

22 d with pain during treatment with placebo or carbamazepine.

23 87, 0.85-0.90), and treatment (gabapentin vs carbamazepine, 0.71, 0.59-0.86; topiramate vs carbamazep

24 5, 1.06-1.47), and treatment (lamotrigine vs carbamazepine, 0.76, 0.61-0.95).

25 arbamazepine, 0.71, 0.59-0.86; topiramate vs carbamazepine, 0.81, 0.68-0.98).

26 The anticonvulsant carbamazepine 1 is associated with adverse drug reaction

27 increase was found among children exposed to carbamazepine (1/50) or lamotrigine (2/30).

28 ure toxicity of four putative OBPs, that is, carbamazepine 10,11-epoxide, 10,11-Dihydrocarbamazepine,

29 ere validated using metabolites (10,11-epoxy carbamazepine, 10,11-dihydroxy carbamazepine, and acridi

30 uding 10,11-dihydro-10-hydroxycarbamazepine, carbamazepine-10,11-epoxide, acridone-N-carbaldehyde, 4-

31 Four TPs (carbamazepine-10,11-epoxide, metoprolol acid, 1-naphthol

32 o valproate (mean 97, 95% CI 94-101) than to carbamazepine (105, 102-108; p=0.0015), lamotrigine (108

33 ed by oxcarbazepine (46%), duloxetine (30%), carbamazepine (26%), topiramate (25%), pregabalin and ga

34 For carbamazepine (~ 27% free at t = 0), the ibuprofen infus

35 e paralleled by pharmacological coupling, as carbamazepine (30 muM) depolarizes S214T activation, as

36 ghted average concentration: 250-4800 ng/L), carbamazepine (33-910 ng/L), diclofenac (78-1000 ng/L),

37 thoxazole (2-33 days), naproxen (6-19 days), carbamazepine (355-1,624 days), and ciprofloxacin were n

38 e taking placebo to 9.1 minutes while taking carbamazepine (400 mg then 200 mg/day), and a reduction

39 taking placebo to 231.9 minutes while taking carbamazepine (400 mg/day), and a reduction in total TIP

40 oxen, 82 +/- 3% for ibuprofen, 66 +/- 2% for carbamazepine, 57 +/- 15% for tylosin, and 88 +/- 1% for

41 95% CI 5.5% to 8.3%) compared with 2.6% with carbamazepine (95% CI 1.9% to 3.5%) and 2.3% with lamotr

42 nts lower than the score of those exposed to carbamazepine (95% CI, 0.6 to 12.0; P=0.04).

43 Carbamazepine, a widely used neuroactive pharmaceutical,

44 for degradation of 1,4-dioxane, benzoate and carbamazepine across pH 5.5-8.3.

45 ates suggest a non-significant advantage for carbamazepine against lamotrigine (0.91 [0.77-1.09]), to

46 , suggesting that lacosamide, lidocaine, and carbamazepine all bind to the same site.

47 Patients who stopped taking carbamazepine also had a 31.2% decline in lipoprotein(a)

48 dition to its effect on channel trafficking, carbamazepine also inhibited KATP channel activity.

49 This study focuses on carbamazepine, an anticonvulsant drug which is ubiquitou

50 We report that carbamazepine, an anticonvulsant, corrects the trafficki

51 e further evaluate the possibility to detect carbamazepine, an antiepileptic massively prescribed and

52 mazepine was only transformed to 10,11-epoxy carbamazepine and 10,11-dihydroxy carbamazepine as a dea

53 ard and feedback inhibition is unaffected by carbamazepine and additional commonly used Na(+) channel

54 by the use-dependent sodium channel blocker carbamazepine and by a blocker of reverse Na-Ca exchange

55 ging preliminary evidence include memantine, carbamazepine and citalopram.

56 Coexposure of plants to carbamazepine and fluoxetine or amitriptyline decreased

57 ies showed complete elimination of (14)C for carbamazepine and fluoxetine treatments and partial elim

58 following short-term laboratory exposures to carbamazepine and fluoxetine.

59 vidence that despite structural differences, carbamazepine and glibenclamide compete for binding to K

60 HLA) associations: abacavir and HLA-B*57:01, carbamazepine and HLA-B*15:02, allo-purinol and HLA-B*58

61 In carbamazepine and HLA-B*15:02, certain T-cell receptor (

62 laimed wastewater-irrigated produce excreted carbamazepine and its metabolites in their urine, while

63 public alphabetaTCR has binding affinity for carbamazepine and its structural analogs, thereby mediat

64 CMs after monotherapy exposure to valproate, carbamazepine and lamotrigine.

65 Aggravation of seizures was reported for carbamazepine and lamotrigine.

66 We conclude that carbamazepine and lidocaine access the sodium channel in

67 endent rapamycin as well as mTOR-independent carbamazepine and minoxidil, markedly attenuated cyst fo

68 uticals acetaminophen, sulfamethoxazole, and carbamazepine and on the pharmaceutical intermediate 5-m

69 Carbamazepine and oxcarbazepine are drugs of first choic

70 Because carbamazepine and oxcarbazepine can enhance the sensitiv

71 Carbamazepine and oxcarbazepine were not immunosuppressi

72 s treatment with the sodium channel blockers carbamazepine and oxcarbazepine, which although effectiv

73 targets to antiepileptic drugs (AEDs; i.e., carbamazepine and phenytoin (CBZ, PHT)).

74 treatments with antiepileptic drugs, such as carbamazepine and phenytoin, which are sodium channel bl

75 f sodium channels, in contrast to drugs like carbamazepine and phenytoin, which bind tightly to fast-

76 Triazolam, oral midazolam, St. John's Wort, carbamazepine and pimozide, are contraindicated with DAA

77 Prescribing of carbamazepine and sodium valproate have declined since 1

78 escribed in pregnancy over older AEDs namely carbamazepine and sodium valproate.

79 mug L(-1)), and several compounds, including carbamazepine and sulfamethoxazole, were detected throug

80 One of the pharmacological treatments is Carbamazepine and the most prevalent surgical treatments

81 For mood stabilizers, whereas carbamazepine and topiramate continued to produce positi

82 Here we examined whether carbamazepine and two other sodium channel inhibitors, r

83 This study investigated the effect of carbamazepine and verapamil (0.005-10 mg/kg) on a range

84 af concentrations of 821.9 and 2.2 mg/kg for carbamazepine and verapamil, respectively.

85 hotics, some anticonvulsants (divalproex and carbamazepine), and lithium salts.

86 chemicals (boron), pharmaceutical compounds (carbamazepine), and steroidal estrogens (estrone, 17-bet

87 xposed to phenytoin, 98 for those exposed to carbamazepine, and 92 for those exposed to valproate.

88 (10,11-epoxy carbamazepine, 10,11-dihydroxy carbamazepine, and acridine) as primary substrates and f

89 nt mood stabilizers, e.g., valproic acid and carbamazepine, and atypical antipsychotic drugs (APDs),

90 Ibuprofen, carbamazepine, and caffeine contributed most to the risk

91 Lamotrigine, 10,11,-dihydro-10,11,-dihydroxy-carbamazepine, and carbamazepine were the most persisten

92 ine, olanzapine, ziprasidone, valproic acid, carbamazepine, and citalopram were associated with highe

93 ions of triclosan, triclocarban, miconazole, carbamazepine, and diphenhydramine in plant tissues that

94 range (0.5 to 200 mug/mL for sulfamethazine, carbamazepine, and flavone), and low noise levels (at 30

95 All the PCs except acesulfame, carbamazepine, and fluconazole were attenuated along the

96 he tricyclic anticonvulsant drugs phenytoin, carbamazepine, and lamotrigine block neuronal voltage-ga

97 orketamine, O-desmethyltramadol, diclofenac, carbamazepine, and methoxetamine were not substantially

98 Diphenhydramine, diltiazem, carbamazepine, and norfluoxetine were detected in 11 of

99 Finally, rapamycin as well as spermidine, carbamazepine, and tamoxifen could also rescue the motor

100 hree other autophagy activators (spermidine, carbamazepine, and tamoxifen) in a FTLD-U mouse model wi

101 odegradation rate constants of atorvastatin, carbamazepine, and venlafaxine, which react primarily wi

102 lsant mood stabilizers (AMS), valproic acid, carbamazepine, and zonisamide, but not lithium, also pre

103 Divalproex and carbamazepine are effective in the treatment of acute ma

104 ely prescribed anticonvulsants phenytoin and carbamazepine are potent inducers of cytochrome P450 enz

105 0,11-epoxy carbamazepine and 10,11-dihydroxy carbamazepine as a dead-end product.

106 Our findings identify carbamazepine as a novel small molecule corrector that m

107 sample volume were evaluated for five drugs: carbamazepine, atenolol, sulfamethazine, diazepam, and a

108 odynamic analysis, and functional profiling, carbamazepine attenuated pain in patients with IEM due t

109 the closely related FZD5 and other FZDs for carbamazepine binding.

110 1.7 angstrom resolution, which reveals that carbamazepine binds at a novel pocket on the FZD8 CRD.

111 onic or uncharged trimethoprim and uncharged carbamazepine, but did not affect sequestration of fast-

112 Advanced oxidation can efficiently degrade carbamazepine, but the toxicity and persistence of the o

113 We followed the decomposition of carbamazepine by the white-rot fungus Pleurotus ostreatu

114 gate transport of phenytoin, lamotrigine and carbamazepine by using seven in-vitro transport models.

115 In both crops, the nonionic PCs (carbamazepine, caffeine, and lamotrigine) were detected

116 hether the products of advanced oxidation of carbamazepine can be biotransformed and ultimately miner

117 Cellular functional assays demonstrated that carbamazepine can suppress FZD8-mediated Wnt/beta-cateni

118 lacosamide monotherapy or controlled-release carbamazepine (carbamazepine-CR) twice daily.

119 95% CI and p values for no difference) were: carbamazepine (CBZ) 0.43 (-0.19 to 1.05) p=0.17; lamotri

120 Carbamazepine (CBZ) and oxcarbazepine (OXC) are widely u

121 re we show that the autophagy-enhancing drug carbamazepine (CBZ) decreased the hepatic load of ATZ an

122 ed the impacts of chronic, low-concentration carbamazepine (CBZ) exposure on a suite of male reproduc

123 Although carbamazepine (CBZ) has a known anti-epileptic mechanism

124 The drug carbamazepine (CBZ) has the opposite effect on sleep; it

125 al sunlight photolysis of the pharmaceutical carbamazepine (CBZ) in the presence of dissolved organic

126 Carbamazepine (CBZ) is a worldwide used antiepileptic dr

127 Carbamazepine (CBZ) is an anticonvulsant medication with

128 Carbamazepine (CBZ) is an antiepileptic drug which is pe

129 The antiepileptic drug carbamazepine (CBZ) is one of the most frequently detect

130 ental emission rates of alprazolam (APZ) and carbamazepine (CBZ) through domestic WWTPs (both sludge

131 rated TPs of the recalcitrant pharmaceutical carbamazepine (CBZ) were used for a target screening for

132 It is shown here that carbamazepine (CBZ) would undergo direct photolysis and

133 hlighting this difficulty, it is unclear why carbamazepine (CBZ), a frontline AED with a known molecu

134 Here, we show that carbamazepine (CBZ), an autophagy-stimulating drug that

135 nce immunoassay (SAFIA) for the detection of carbamazepine (CBZ), diclofenac (DCF), caffeine (CAF), a

136 Three CECs (carbamazepine (CBZ), flumequine (FLU), and thiabendazole

137 (4)(2-), Fe(VI)) to oxidize pharmaceuticals (carbamazepine (CBZ), naproxen (NAP), trimethoprim (TMP),

138 ed the HLA-B *1502 genetic predisposition to carbamazepine (CBZ)-induced Stevens-Johnson syndrome (SJ

139 rmaceuticals urea: barbituric acid (UBA) and carbamazepine (CBZ).

140 Canadian family in which pain is relieved by carbamazepine (CBZ).

141 sing preliminary evidence include memantine, carbamazepine, citalopram, and prazosin, but none of the

142 ve metabolites of acetaminophen, diclofenac, carbamazepine, clozapine, p-cresol, 4-ethylphenol, and 3

143 esults show that the oxidation byproducts of carbamazepine containing a hydroxyl or carbonyl group ca

144 ically distinct compounds, glibenclamide and carbamazepine, correct biogenesis defects in ATP-sensiti

145 ths was 90% taking lacosamide and 91% taking carbamazepine-CR (absolute treatment-difference: -1.3%,

146 in the lacosamide group and 308 (70%) in the carbamazepine-CR group completed 6 months of treatment w

147 tients taking lacosamide and 43 (10%) taking carbamazepine-CR had serious treatment-emergent adverse

148 44 patients taking lacosamide and 442 taking carbamazepine-CR were included in the full analysis set

149 ay for lacosamide and 800 or 1200 mg/day for carbamazepine-CR) over 2 weeks with a 1-week stabilisati

150 therapy or controlled-release carbamazepine (carbamazepine-CR) twice daily.

151 doses of 100 mg/day lacosamide or 200 mg/day carbamazepine-CR, uptitration to the first target level

152 non-inferiority criteria when compared with carbamazepine-CR.

153 non-inferiority criteria when compared with carbamazepine-CR.

154 receiving lacosamide and 332 (75%) receiving carbamazepine-CR.

155 A detailed study of carbamazepine crystal growth in four different bis(urea)

156 ), anti-anxiety diazepam (DZP), anti-seizure carbamazepine (CZP) drugs and their metabolites in groce

157 ncluding an antiepileptic drug (phenytoin or carbamazepine), dexamethasone, and ranitidine.

158 sensitivity was observed with amitriptyline, carbamazepine, diazepam and gabapentin.

159 tigated the fate and uptake of (14)C-labeled carbamazepine, diclofenac, fluoxetine, and orlistat in s

160 rmaceuticals (acetazolamide, beclomethasone, carbamazepine, diclofenac, gemfibrozil, ibuprofen, ketop

161 r several other protein misfolding diseases, carbamazepine did not correct KATP channel trafficking d

162 Furthermore, although carbamazepine did not selectively target resurgent curre

163 ding caffeine, meprobamate, primidone, DEET, carbamazepine, dilantin, naproxen, and triclosan.

164 n in these soils can retard trimethoprim and carbamazepine dissipation.

165 osition was affected by flow conditions with carbamazepine dominating the base flow and caffeine domi

166 erbal domains and negatively associated with carbamazepine dose for verbal performance.

167 model to assess the removal of phenytoin and carbamazepine during HD with or without binding-competit

168 transformation products of the (14)C-labeled carbamazepine during UV/hydrogen peroxide advanced oxida

169 ons of widely monitored HHCB, triclosan, and carbamazepine explained 71-82% of the variability in the

170 Our results indicate that parental carbamazepine exposure history impacts the unexposed pro

171 armaceuticals found in reclaimed wastewater: carbamazepine, fluoxetine, amitriptyline, and lamotrigin

172 ile fish bioconcentrated both fluoxetine and carbamazepine from exposure water, fluoxetine accumulate

173 Patients were randomly assigned to receive carbamazepine, gabapentin, lamotrigine, oxcarbazepine, o

174 , and they were randomly assigned to receive carbamazepine, gabapentin, lamotrigine, oxcarbazepine, o

175 iprofloxacin, trimethoprim, propranolol, and carbamazepine (>80%) was achieved within 3 h of electrol

176 nnel inhibitors, including sulfonylureas and carbamazepine, have been shown to correct channel traffi

177 luding clopidogrel, pegylated-interferon and carbamazepine, have led to the identification of specifi

178 e, lamotrigine was significantly better than carbamazepine (hazard ratio [HR] 0.78 [95% CI 0.63-0.97]

179 Upon subsequent removal of carbamazepine, however, the function of rescued channels

180 Carbamazepine in a modified cyclodextrin (hydroxypropyl-

181 interaction was observed for lamotrigine or carbamazepine in any of the seven validated in-vitro tra

182 harmacogenetic testing for HLA-B*1502 before carbamazepine in patients of certain Asian ethnicities a

183 e-Carlo based pharmacokinetic simulations of Carbamazepine in treatment of Trigeminal Neuralgia.

184 including human leukocyte antigen loci with carbamazepine-induced dermatologic outcome and MC4R with

185 ated the relationship between HLA-B*1502 and carbamazepine-induced SJS and TEN and that reported suff

186 lationship between the HLA-B*1502 allele and carbamazepine-induced SJS and TEN in Han-Chinese, Thai,

187 for the relationship between HLA-B*1502 and carbamazepine-induced SJS and TEN was 79.84 (95% CI, 28.

188 The primary outcome was carbamazepine-induced SJS and TEN.

189 bacavir hypersensitivity and HLA-B*57:01 and carbamazepine-induced Stevens-Johnson syndrome and HLA-B

190 sgenic mice receiving oral administration of carbamazepine induces multi-organ injuries and symptoms

191 However, carbamazepine inhibited sodium channels effectively and

192 In the whole-cell configuration, carbamazepine inhibited sodium current within seconds wh

193 ntration 70 mg/L, using 2000 mg aspirin, (2) carbamazepine, initial concentration 35 mg/L, using 800

194 ated by other techniques show that, in fact, carbamazepine is not a DNA intercalator.

195 Carbamazepine is one of the most persistent pharmaceutic

196 he widely used anticonvulsant pharmaceutical carbamazepine is recalcitrant in many environmental nich

197 Carbamazepine is widely accepted as a drug of first choi

198 achieving a 12-month remission (lamotrigine-carbamazepine) is 0 (-8 to 7) and 5 (-3 to 12), suggesti

199 ebo were significantly better tolerated than carbamazepine (lamotrigine, RR 5.24, 1.07-26.32; placebo

200 Parent compounds (e.g., buproprion, carbamazepine, lamotrigine) generally were more persiste

201 All active treatments other than carbamazepine, lamotrigine+valproate (no data) and palip

202 eatments, other than aripiprazole+valproate, carbamazepine, lamotrigine, and lamotrigine+valproate, o

203 ntiepileptics approved for bipolar disorder (carbamazepine, lamotrigine, and valproate) are associate

204 otrigine, aripiprazole+valproate, asenapine, carbamazepine, lamotrigine, lamotrigine+valproate, lithi

205 ed to maternal IQs among children exposed to carbamazepine, lamotrigine, or phenytoin but not among t

206 ified as either exposed to an antiepileptic (carbamazepine, lamotrigine, or valproate) or not exposed

207 fects exist across four commonly used drugs (carbamazepine, lamotrigine, phenytoin and valproate).

208 epilepsy on antiepileptic drug monotherapy (carbamazepine, lamotrigine, phenytoin, or valproate) bet

209 ho were taking a single antiepileptic agent (carbamazepine, lamotrigine, phenytoin, or valproate) in

210 men with epilepsy receiving monotherapy (ie, carbamazepine, lamotrigine, phenytoin, or valproate) wer

211 Withdrawal of carbamazepine led to acute worsening of EAE symptoms, in

212 ion of the KATP channel opener diazoxide and carbamazepine led to enhanced mutant channel function wi

213 Four units ceased activity with carbamazepine levels above the therapeutic range (15.4-1

214 ption coefficients increased in the order of carbamazepine < diclofenac < fluoxetine < orlistat.

215 elimination of (14)C, increased in the order carbamazepine < diclofenac < fluoxetine and orlistat.

216 Carbamazepine may reduce the dose-limiting neurotoxicity

217 These findings suggest that phenytoin and carbamazepine may substantially increase the risk for ca

218 Furthermore, the observed carbamazepine-mediated Wnt signaling inhibition may help

219 During solid-state fermentation, carbamazepine metabolism resulted in the generation of a

220 iptyline decreased accumulation of the toxic carbamazepine metabolite 10,11-epoxycarbamazepine.

221 We also report that the carbamazepine metabolite pattern at this low exposure le

222 Carbamazepine metabolites were found mainly in the leave

223 Carbamazepine metabolites were identified using liquid c

224 re exposed to valproate monotherapy, 1718 to carbamazepine monotherapy and 2198 to lamotrigine monoth

225 mal fields; Group III, 14 patients receiving carbamazepine monotherapy; Group IV, 20 normal individua

226 cated agents were lamotrigine, azithromycin, carbamazepine, moxifloxacin, cephalexin, diclofenac, and

227 e review reporting genetic associations with carbamazepine (n = 31), other aromatic antiepileptic dru

228 similar irrespective of the system, and with carbamazepine no significant degradation was obtained.

229 s further evident at a cellular level, where carbamazepine normalized the hyperexcitability of dorsal

230 is usually resistant to pharmacotherapy, but carbamazepine normalizes activation of Na(V)1.7-V400M mu

231 Some TrOCs, such as dihydroxy-carbamazepine, O-desmethylvenlafaxine, and venlafaxine,

232 how that between 95% (4-iodophenol) and 67% (carbamazepine) of the total internal amount in 26 h post

233 technology was used to assess the effect of carbamazepine on firing of DRG neurons carrying S241T mu

234 The corrector effect of carbamazepine on mutant KATP channels was also demonstra

235 xture exposure to propranolol, diazepam, and carbamazepine on the crustacean Daphnia magna at environ

236 of refractory epilepsy through transport of carbamazepine or lamotrigine.

237 We recruited 34 epilepsy patients taking carbamazepine or phenytoin in monotherapy whose physicia

238 g epilepsy patients from the enzyme-inducers carbamazepine or phenytoin to the noninducing drugs leve

239 functions compared with lamotrigine (but not carbamazepine or phenytoin).

240 nic use of many antiepileptic drugs, such as carbamazepine, oxcarbazepine, and topiramate, enhances h

241 tments of BD such as valproate, lamotrigine, carbamazepine, oxcarbazepine, antipsychotics, electrocon

242 her MCM risk than lamotrigine (p=0.0001) and carbamazepine (p=0.0001) monotherapy.

243 ffect was seen with valproate (p=0.0006) and carbamazepine (p=0.03) exposed pregnancies.

244 metabolized via acridine and 10,11-dihydroxy carbamazepine pathways.

245 Addition of carbamazepine permitted dose escalation to 160 mg/m2 wit

246 ere significantly less in subjects receiving carbamazepine, phenytoin, and valproate than in those re

247 esults of the one-step CEDIA for three AEDs (carbamazepine, phenytoin, and valproic acid), in the pre

248 f bone mass in women, and both phenytoin and carbamazepine produce increases in serum lipids and C-re

249 sy patients, switch from either phenytoin or carbamazepine produced significant declines in total cho

250 sing measured photolysis rates for atenolol, carbamazepine, propranolol, and sulfamethoxazole in wetl

251 actory solid tumors and to determine whether carbamazepine reduces oxaliplatin-induced neurotoxicity.

252 this study, a mechanism is proposed by which carbamazepine resists biodegradation, and a previously u

253 levels for diclofenac, sulfamethoxazole and carbamazepine, respectively.

254 1.7-V400M mutant channels from a family with carbamazepine-responsive inherited erythromelalgia.

255 sensitivity was observed with amitriptyline, carbamazepine, rofecoxib, and diazepam.

256 .62, 95% credible interval [CrI] 0.38-1.03), carbamazepine (RR 0.68, 0.44-1.06), imipramine (RR 0.95,

257 Caffeine and carbamazepine showed correlations with florescence index

258 Both phenytoin and carbamazepine significantly improved the clinical course

259 n identity test for monoclonal antibodies to carbamazepine, sirolimus, tacrolimus, cyclosporine, cort

260 the cytotoxic T lymphocytes of patients with carbamazepine-SJS/TEN, with its expression showing drug/

261 Specifically, carbamazepine (SMD, -1.57 [CrI, -2.83 to -0.31]), venlaf

262 peroning mechanism wherein glibenclamide and carbamazepine stabilize the heteromeric subunit interfac

263 xaliplatin); regimen B (to determine whether carbamazepine starting 24 hours before and ending 48 hou

264 ment with 300 microM lidocaine or 300 microM carbamazepine, suggesting that lacosamide, lidocaine, an

265 nd that the wastewater borne MPs diclofenac, carbamazepine, sulfamethoxazole, acesulfame, sucralose,

266 ponent (bisphenol A), three pharmaceuticals (carbamazepine, sulfamethoxazole, and meprobamate), and t

267 Lamotrigine is clinically better than carbamazepine, the standard drug treatment, for time to

268 r the HLA-B*1502 allele before initiation of carbamazepine therapy in patients of Asian ancestry, but

269 HLA-B*1502 screening in patients requiring carbamazepine therapy is warranted.

270 he lake system ranged from 780-5700 days for carbamazepine to <1-2 days for ketoprofen.

271 all molecule screens, we observed binding of carbamazepine to the cysteine-rich domain (CRD) of the W

272 ipogenesis in some patients during long-term carbamazepine treatment.

273 at the observed uptake in the fluoxetine and carbamazepine treatments was due to the parent compounds

274 representative OMPs (i.e., sulfamethoxazole, carbamazepine, tylosin, atrazine, naproxen, and ibuprofe

275 Increased carbamazepine uptake by zucchini resulted in a decrease

276 ) and toxic epidermal necrolysis (TEN) among carbamazepine users, especially in some racial/ethnic po

277 ineffective, while valproate, gabapentin and carbamazepine varied in their potencies, with only the l

278 Neither lamotrigine nor carbamazepine was a substrate for P-gp in any of the mod

279 on treatment with low doses of rapamycin and carbamazepine was able to attenuate cyst formation as ef

280 sformation of trimethoprim, propranolol, and carbamazepine was attributed to direct electron transfer

281 Arm A included patients for whom carbamazepine was considered the first-line treatment, m

282 Arm A recruited 1721 patients for whom carbamazepine was deemed to be standard treatment, and t

283 icking rescue by reversible sulfonylureas or carbamazepine was facilitated by the KATP channel opener

284 In contrast, the antiepileptic drug carbamazepine was found to inhibit voltage-gated sodium

285 Pharmacoresponsiveness of S241T to carbamazepine was further evident at a cellular level, w

286 d-state-fermentation conditions, 10,11-epoxy carbamazepine was further metabolized via acridine and 1

287 In liquid culture, carbamazepine was only transformed to 10,11-epoxy carbam

288 ore slowly (t1/2 approximately 1.5-2 d), and carbamazepine was recalcitrant.

289 For time to 12-month remission carbamazepine was significantly better than gabapentin (

290 When (14)C-carbonyl-labeled carbamazepine was used as the substrate, (14)C-CO2 relea

291 and functional analysis to be responsive to carbamazepine, was assessed in a double-blind, placebo-c

292 to enhanced mutant channel function without carbamazepine washout.

293 hium, quetiapine, olanzapine, bupropion, and carbamazepine were associated with high morbidity indice

294 -dihydro-10,11,-dihydroxy-carbamazepine, and carbamazepine were the most persistent (t1/2 = 12 +/- 2.

295 ants such as sucralose, sulfamethoxazole and carbamazepine, which are typical wastewater markers in r

296 taking placebo to 274.1 minutes while taking carbamazepine, while patient 2 reported a reduction of t

297 dwater and revealed persistence for the drug carbamazepine, while the herbicide MCPA (2-methyl-4-chlo

298 ds ranged from 0.5 (fluoxetine) to 306 days (carbamazepine), with 20 substances responding significan

299 attenuation of all compounds studied except carbamazepine within an area similar to that typical of

300 As with valproic acid and carbamazepine, zonisamide (12.5 and 25 mg/kg) increased