ライフサイエンスコーパス: carcinogenic

1 acidification, global warming potential, and carcinogenics).

2 than PAC in two categories (eutrophication, carcinogenics).

3 rotoxic, teratogenic, immunosuppressive, and carcinogenic.

4 s, including carbon nanotubes (CNTs), may be carcinogenic.

5 ave endocrine disrupting effects, or even be carcinogenic.

6 Aristolochic acids (AA) are nephrotoxic and carcinogenic.

7 matic hydrocarbons (PAHs), some of which are carcinogenic.

8 s DNA damage in vivo and is considered to be carcinogenic.

9 elminthes and major human pathogens, are not carcinogenic.

10 mation of bromate, which is considered to be carcinogenic.

11 f-heterozygosity (LOH), which is potentially carcinogenic.

12 demonstrated to be cytotoxic, mutagenic, and carcinogenic.

13 ince dimethylated monothioarsenate is highly carcinogenic.

14 nogenic for humans and red meats as probably carcinogenic.

15 KSHV from latency is important for it to be carcinogenic.

16 d are known or suspected to be mutagenic and carcinogenic.

17 s infection with only a few helminth species carcinogenic?

18 s infection with only a few helminth species carcinogenic?

19 ed nonharmful and mitigate the generation of carcinogenic acridine products known to form when advanc

20 The formation of toxic and potentially carcinogenic acrylamide, alongside volatile aroma compou

21 ious forms of cancer have been linked to the carcinogenic activities of microorganisms(1-3).

22 nd how PTPN14 degradation contributes to the carcinogenic activity of high-risk HPV E7 used variants

23 n the treatment of ulcerative colitis, and a carcinogenic activity under host inflammatory conditions

24 Several PAHs are toxic, and a subset exhibit carcinogenic activity.

25 on of differentiation and contributes to its carcinogenic activity.IMPORTANCE The E7 oncoprotein is a

26 h varying abilities to produce highly toxic, carcinogenic aflatoxins.

27 h of consumers due to its ability to produce carcinogenic aflatoxins.

28 rafish, rainbow trout, hamsters, and mice by carcinogenic agents (methylcholanthrene, thioacetamide),

29 directly targeted, avoidance of exposure to carcinogenic agents is likely to prevent other non-commu

30 s with exposure to environmental noxious and carcinogenic agents.

31 imetric indicator compound for many types of carcinogenic alkylating agents.

32 ed to kill microorganisms for decades but is carcinogenic and cataractogenic, recent evidence has sho

33 tly in the European community because of its carcinogenic and endocrine disrupting characters.

34 ears, because arsenic species are considered carcinogenic and found at high concentrations in the sam

35 ,7,8-tetrachlorodibenzo-p-dioxin, TCDD) is a carcinogenic and highly toxic industrial byproduct that

36 e a class of ON compounds known to be highly carcinogenic and include species formed from nicotine de

37 Formalin is carcinogenic and is detrimental to public health.

38 e been linked to cardiovascular diseases and carcinogenic and mutagenic effects.

39 Acrylamide is a carcinogenic and neurotoxic process contaminant that is

40 the potential for human exposure and adverse carcinogenic and non-cancer health effects.

41 od dye due to non-precarious, non-toxic, non-carcinogenic and non-poisonous nature.

42 s MIC = 5 mg/mL) and no cytotoxicity against carcinogenic and non-tumour primary liver (PLP) cells.

43 re to curcumin, a natural compound with anti-carcinogenic and telomerase activity-reducing properties

44 nd most used DNA intercalators, but presents carcinogenic and teratogenic properties.

45 These compounds present carcinogenic and/or mutagenic properties and offer a ris

46 Some OPFRs are considered carcinogenic and/or neurodevelopmental toxicants, and ch

47 ibutable to antioxidant, antimicrobial, anti-carcinogenic, and anti-inflammatory properties.

48 Regarding teratogenic, carcinogenic, and immunotoxic nature of ochratoxin A (OT

49 Magenta III is regarded as possibly carcinogenic, and rhodamine B was reported to cause loca

50 Melanin may thus be carcinogenic as well as protective against cancer.

51 variants to assist further investigation of carcinogenic association and the development of diagnost

52 Known toxic and carcinogenic azaarenes, such as benzo[c]acridine and dib

53 Although the carcinogenic basis of AFB1 has been ascribed to its muta

54 ethene (50-190 Gg y(-1)) and is s source of carcinogenic benzene (30-280 Gg y(-1)).

55 tive hydrogenation, a potential approach for carcinogenic benzene removal from gasoline, is probed us

56 wever, the assay does not detect potentially carcinogenic bulky adducts that can arise when metabolic

57 s tend to live longer, have more potentially carcinogenic cells, and undergo more cell divisions.

58 ong-term consequences, but these may include carcinogenic change or granulomatous inflammation.

59 drugs, understanding the mode of actions of carcinogenic chemicals, and monitoring the genotypic tox

60 (including a Sandmeyer halogenation), use of carcinogenic chloromethyl methyl ether to install a prot

61 r example, to control emissions of toxic and carcinogenic combustion by-products, which also greatly

62 ted that DNA adducts of aristolochic acid, a carcinogenic component of Aristolochia herbs used in tra

63 Aflatoxin M1 (AFM1), a potentially carcinogenic compound, is found in milk obtained from an

64 Acetaldehyde is a naturally-occurring carcinogenic compound, present in different food items,

65 refighters have shown increased exposures to carcinogenic compounds and elevated rates of certain can

66 Our methodology entails an exposure to carcinogenic compounds during the animal's regeneration

67 Trihalomethanes (THMs) are conditionally carcinogenic compounds formed during chlorine disinfecti

68 nalysis for very low concentrations of these carcinogenic compounds was developed and compared by app

69 m stem cell dynamics, to detect and classify carcinogenic compounds.

70 allowing the bacteria to persist and induce carcinogenic consequences in the gastric niche.

71 allowing the bacteria to persist and induce carcinogenic consequences in the gastric niche.

72 Arsenic is a carcinogenic contaminant of water and food and a growing

73 umans, animals and crops, a source of potent carcinogenic contaminants of food, and an important gene

74 s other Cr supplements, was also oxidized to carcinogenic Cr(VI) and Cr(V) in living cells.

75 simple act of cooking can convert all of the carcinogenic Cr(VI) to the safer Cr(III).

76 N-Nitrosodimethylamine (NDMA) is a carcinogenic disinfection byproduct from water chloramin

77 c mechanisms that explain the geographic and carcinogenic diversity of HPV58 variants are still poorl

78 N(2)-Furfuryl-deoxyguanosine (fdG) is carcinogenic DNA adduct that originates from furfuryl al

79 ctivity with DNA templates modified with the carcinogenic DNA adducts, 2-aminofluoene (AF) or N-acety

80 BPDE), which then can react with DNA to form carcinogenic DNA adducts.

81 protection against potentially mutagenic and carcinogenic DNA damage.

82 methylbenzidine(TMB) as a new remarkable non-carcinogenic DNA indicator for genosensing purposes, whi

83 Endogenous and carcinogenic DNA lesions can also trap Top1cc.

84 ive immune system; however, it is considered carcinogenic due to its strong association with lymphoid

85 radiation oncology, has potentially greater carcinogenic effect compared with sparsely ionizing radi

86 Further studies to determine carcinogenic effects and impact of HPV vaccinations are

87 nzenes are natural toxins with genotoxic and carcinogenic effects in rodents, which are highly presen

88 a DNA repair enzyme that protects cells from carcinogenic effects of alkylating agents; however, MGMT

89 he exact molecular mechanisms underlying the carcinogenic effects of arsenic remain incompletely unde

90 hanisms involved including withdrawal of the carcinogenic effects of calcineurin inhibitors and/or th

91 These data suggest that carcinogenic effects of densely ionizing radiation are m

92 Carcinogenic effects of hexavalent chromium in waters ar

93 The enhanced carcinogenic effects of hormones on ATF3-deficient prost

94 d is one of the most sensitive organs to the carcinogenic effects of IR, and we have recently highlig

95 omain) family known to mediate the toxic and carcinogenic effects of xenobiotics.

96 We showed significant carcinogenic effects on female animals while revealing a

97 erintuitive conclusion that UVR has anti-BCC carcinogenic effects that can explain, at least in part,

98 on (IR) is a public concern due to its known carcinogenic effects.

99 which early-life iAs exposure elicits latent carcinogenic effects.

100 omatic hydrocarbon with strong mutagenic and carcinogenic effects.

101 ted with supply chain production, except for carcinogenic effects.

102 idespread environmental prevalence and known carcinogenic effects.

103 ry metabolites of fungi that cause toxic and carcinogenic effects.

104 Arsenic (As) is a carcinogenic element threatening the health of millions

105 Arsenic is a ubiquitous and carcinogenic environmental element that enters the biosp

106 sample preparation procedure for analysis of carcinogenic ethyl carbamate (EC) in wine by GC-MS-SIM i

107 l DNA damage and repair processes as well as carcinogenic exposures or cancer related aberrations of

108 tures can be readily assigned to known human carcinogenic exposures or endogenous mechanisms of mutag

109 en under 50 years, reflect recent changes in carcinogenic exposures, which could foreshadow the futur

110 cancer risk persists following cessation of carcinogenic exposures.

111 ur findings suggest that minimal exposure to carcinogenic fibers may significantly increase the risk

112 on Cancer recently classified cured meats as carcinogenic for humans and red meats as probably carcin

113 The latter is known for being carcinogenic for vertebrates, potentially causing enviro

114 ge of these polymers is the reduction of the carcinogenic formaldehyde.

115 icro RNA (miRNA), are able to knock down the carcinogenic genes by targeting the mRNA expression, whi

116 an algorithm for identifying combinations of carcinogenic genes with mutations (multi-hit combination

117 HPV16 was the most common carcinogenic genotype detectable in 25% of urine, 33.8%

118 door air pollution and particulate matter as carcinogenic (Group 1).

119 ), but higher reactivity in forming the more carcinogenic haloacetonitrile after chlorination (244%-o

120 elevance of the in vitro positive results to carcinogenic hazard.

121 ydrocarbon (PAH)-contaminated media, wherein carcinogenic hazards are evaluated using a dose-addition

122 While potential carcinogenic health risks present for both adult and chi

123 le heavy metal exposure, no potential of non carcinogenic health risks was found, while carcinogenic

124 n carcinogenic health risks was found, while carcinogenic health risks were found only for As.

125 4,5-f]quinoline (IQ), a highly mutagenic and carcinogenic heterocyclic amine found in cooked meats, w

126 an informed choice of antioxidants to reduce carcinogenic heterocyclic aromatic amine (HAA) formation

127 -Amino-9H-pyrido[2,3-b]indole (AalphaC) is a carcinogenic heterocyclic aromatic amine formed during t

128 l-6-phenylimidazo[4,5-b]pyridine (PhIP) is a carcinogenic heterocyclic aromatic amine formed in cooke

129 Carcinogenic hexavalent chromium [Cr(VI)] requires cellu

130 alignant lesions, to define the effects of a carcinogenic host environment on the virulence phenotype

131 Urine-based carcinogenic HPV detection had a clinical sensitivity of

132 Although urine-based HPV carcinogenic HPV detection was lower (58.3%) than cervic

133 real-time PCR assay for the detection of 13 carcinogenic HPV types (the H13 assay; Hybribio, Hong Ko

134 of naturally occurring antibodies against 8 carcinogenic HPV types with subsequent infections.

135 risk of HSIL in women infected with multiple carcinogenic HPV types.

136 ultivalent vaccines that include most of the carcinogenic HPV types; continued surveillance postvacci

137 Carcinogenic impacts range over 4 orders of magnitude, f

138 of ARBs prompted by discovery of potentially carcinogenic impurities shifted utilization of ARBs indi

139 ilms comprise microbial communities that are carcinogenic in CRC mouse models.

140 chloromethane was classified as likely to be carcinogenic in humans based primarily on evidence of ca

141 e considered to be potentially genotoxic and carcinogenic in humans.

142 Most HAAs are carcinogenic in long-term animal studies.

143 he keratin-14 promoter and showed that E7 is carcinogenic in mice.

144 iduals undergoing screening colonoscopy, are carcinogenic in murine models of CRC.

145 llomaviruses (HPV) have been suspected to be carcinogenic in nonmelanoma skin cancers (NMSC), but the

146 Sudan I is a carcinogenic industrial azo-dye, forbidden for use in fo

147 ia Helicobacter pylori is the single leading carcinogenic infectious agent and the main cause of stom

148 Interleukin-6 (IL-6) is a pro-carcinogenic inflammatory cytokine that plays a pivotal

149 methylarsenite [MAs(III)] to less toxic and carcinogenic inorganic arsenite [As(III)] by C-As bond c

150 indings leading first to the hypothesis that carcinogenic insults leave characteristic imprints on th

151 sociated altered expression of apoptotic and carcinogenic key genes.

152 The recognition that tobacco smoke is carcinogenic led to the most significant and successful

153 beta) inserts, albeit slowly, T opposite the carcinogenic lesion O6-methylguanine (O6MeG) approximate

154 The carcinogenic mechanism depends mainly on the expression

155 tinct cancers has the potential to elucidate carcinogenic mechanisms and inform broadly applicable ri

156 wn to be involved in a number of potentially carcinogenic mechanisms such as altering gene transcript

157 ey have been implicated in a variety of anti-carcinogenic mechanisms.

158 r metabolite levels for TSNAs (including the carcinogenic metabolite 4-(methylnitrosamino)-1-(3-pyrid

159 iliary and fecal elimination of PhIP and its carcinogenic metabolites and may affect PhIP-induced car

160 -), but not Bcrp1;Mdr1a/b;Mrp(-/-) mice, the carcinogenic metabolites N2-OH-PhIP (2-hydroxyamino-1-me

161 organic arsenite (As(III)) to more toxic and carcinogenic methylarsenite (MAs(III)).

162 However, it is banned due to its carcinogenic, mutagenic and genotoxic properties, which

163 own to cause negative health effects through carcinogenic, mutagenic, teratogenic, or other toxic mec

164 er: we search for combinations of genes with carcinogenic mutations (multi-hit combinations) instead

165 DNA damage can lead to carcinogenic mutations and toxicity that promotes diseas

166 ite for identifying specific combinations of carcinogenic mutations and understanding the etiology of

167 a >2 fold inhibition of biosynthesis of the carcinogenic mycotoxin and secondary metabolite, aflatox

168 Omicronchratoxin A (OTA) is a carcinogenic mycotoxin produced by A. carbonarius that c

169 ght hapten, Ochratoxin A (OTA), is a natural carcinogenic mycotoxin produced by Aspergillus and Penic

170 monitoring of Ochratoxin A (OTA), a natural carcinogenic mycotoxin produced by Aspergillus and Penic

171 Fumonisin B1 (FB1) is a carcinogenic mycotoxin produced by Fusarium species cont

172 The fungus also produces carcinogenic mycotoxins, fumonisins on infested maize.

173 Aflatoxins are the most potent genotoxic and carcinogenic mycotoxins.

174 e concerns over the formation of potentially carcinogenic N-nitrosamine byproducts from reactions bet

175 However, the formation of potentially carcinogenic N-nitrosamines and N-nitramines from reacti

176 The occurrence of eight carcinogenic N-nitrosamines in biosolids from 74 wastewa

177 risk potentially via endogenous formation of carcinogenic N-nitroso compounds or increased lipid and

178 the precursors and reactions leading to the carcinogenic N-nitrosodimethylamine (NDMA) during drinki

179 ammonium polymers leads to the formation of carcinogenic N-nitrosodimethylamine (NDMA).

180 sistent organic pollutants, whose documented carcinogenic, neurological, and respiratory toxicities a

181 e threshold value of 10(-6), suggesting that carcinogenic Ni still was a concern to the residents.

182 mines present in all amine solvents can form carcinogenic nitrosamines.

183 trosate in the desorber, forming potentially carcinogenic nitrosamines.

184 5-b]pyridine (PhIP) is considered as a human carcinogenic or mutagenic compound that is produced from

185 hymine dimer which is potentially mutagenic, carcinogenic, or lethal to the organism.

186 The efficient removal of trace carcinogenic organic pollutants, such as polycyclic arom

187 integrated network that combats potentially carcinogenic oxidative damage yet also protects cancer c

188 The formation of bromate, a potentially carcinogenic ozonation byproduct, could be significantly

189 ich 5-methylchrysene (5-MC), a mutagenic and carcinogenic PAH, had the highest individual observed OR

190 ic approach that assumes dose additivity for carcinogenic PAHs and (ii) a bioassay-based approach tha

191 reviewed, were unable to successfully remove carcinogenic PAHs from contaminated soils to concentrati

192 five-ring PAHs, including five of the seven carcinogenic PAHs, with removals up to 80%.

193 ency equivalency factors (PEFs) for targeted carcinogenic PAHs.

194 We explored an alternative carcinogenic pathway characterized by episomal E2, E4, a

195 er, it remains unclear at which stage in the carcinogenic pathway fiber may act or which food sources

196 the miR-183 family regulates zinc levels and carcinogenic pathways in prostate cells.

197 Accumulating evidence indicates that common carcinogenic pathways may underlie digestive system canc

198 Considering that flutriafol is a toxic and carcinogenic pesticide, as well as the increase in the n

199 These results indicate that host carcinogenic phenotypes modify cagT4SS function via alte

200 mine dimers is one of the most important DNA carcinogenic photolesions induced by ultraviolet irradia

201 Studies on metabolomes of carcinogenic pollutants among children and adolescents a

202 aromatic hydrocarbons (PAHs) are potentially carcinogenic pollutants emitted by diesel engines, both

203 Bolstering the immune response to carcinogenic polyaromatic hydrocarbons may be an effecti

204 n the induction of cell-mediated immunity to carcinogenic polyaromatic hydrocarbons.

205 ceedances for PM and benzo[a]pyrene B[a]P, a carcinogenic polycyclic aromatic hydrocarbon (PAH) of MW

206 , bioaccessibility, and dermal absorption of carcinogenic polycyclic aromatic hydrocarbons (cPAHs) in

207 DNA adducts derived from carcinogenic polycyclic aromatic hydrocarbons like benzo

208 Risk-based analysis of several carcinogenic POPs indicated that the fish with the highe

209 are generated at concentrations below their carcinogenic potencies; it is unclear how exposure leads

210 (a)pyrene-equivalents (a combined measure of carcinogenic potency across 7 different PAHs), which wer

211 Levels of benzo[a]pyrene, phenanthrene, and carcinogenic potency of PAH mixtures were highest when s

212 dicated MPFs with orders of magnitude higher carcinogenic potency than predicted by established in vi

213 a serious public health risk because of its carcinogenic potential and as climate pollutant.

214 codons of HPV58 variants representing higher carcinogenic potential and/or that are under positive se

215 nvestigated whether microbial effectors with carcinogenic potential influence Lrig1 progenitor cells

216 The carcinogenic potential of CagA is linked to its polymorp

217 Because of the carcinogenic potential of DMA, C. pulverulentus should n

218 Data on the relative carcinogenic potential of human papillomavirus (HPV) typ

219 Although the carcinogenic potential of iron has been shown, evidence

220 this study was to investigate the long-term carcinogenic potential of MTM particulate matter (PMMTM)

221 n be used as a food ingredient to reduce the carcinogenic potential of thermally induced HAAs.

222 ection to studies aimed at defining the full carcinogenic potential of this organism.

223 r ecotoxicity potential, 0-370 kg benzene-eq carcinogenic potential, and 2800-71,000 MT toluene-eq no

224 CNTs), a widely used nanomaterial with known carcinogenic potential, can affect cancer-associated fib

225 nd in precancerous lesions, evidencing their carcinogenic potential, so it is necessary to increase t

226 hydrocarbons (PAHs) which are known to have carcinogenic potential.

227 ctive mutational signatures that explain its carcinogenic potential.

228 omoting events are required to unleash their carcinogenic potential.

229 n commerce have not been evaluated for their carcinogenic potential.

230 One way to interrogate the complex carcinogenic process and interactions between aberrant e

231 by arresting, slowing down, or reversing the carcinogenic process before invasion into surrounding ti

232 standing of racial disparities in cancer and carcinogenic process.

233 rations occur from the very beginning of the carcinogenic process.

234 tation landscape of melanoma reveals diverse carcinogenic processes across its subtypes, some unrelat

235 the involvement of CLIP2 in the fundamental carcinogenic processes including apoptosis, mitogen-acti

236 hat it is a tumor suppressor in more complex carcinogenic processes.

237 Carcinogenic properties are linked to the ability of the

238 Ms) have provided valuable insights into the carcinogenic properties of various human tumor viruses,

239 Several metals have carcinogenic properties, but their associations with bre

240 ng but also exert anti-inflammatory and anti-carcinogenic properties.

241 are environmental pollutants with toxic and carcinogenic properties.

242 We then used the model to investigate the carcinogenic property of Bisphenol A (BPA), an environme

243 d SCID mice models also revalidated the anti-carcinogenic property of nifetepimine.

244 The outcome of the carcinogenic risk assessment revealed that 88.67% (for a

245 lth risk assessment identified potential non-carcinogenic risk due to Pb via ingestion route.

246 gs showed that levels of both non-cancer and carcinogenic risk for PAE congeners in wheat were higher

247 a result of the widely reported potential of carcinogenic risk from x-ray based examinations, many st

248 Importantly, elevated carcinogenic risk is found at doses that yield undetecta

249 dence of imaging-related low-dose (<100 mGy) carcinogenic risk is nonexistent; it is a hypothetical r

250 Also, we found that the carcinogenic risk of arsenic in all three rice types was

251 rd quotient, selenium health benefit values, carcinogenic risk of arsenic, maximum safe consumption a

252 to arsenite and antimonite indicate the skin carcinogenic risk of exposure to antimonite merits close

253 a)pyrene, the marker used for evaluating the carcinogenic risk of PAHs in food, was not detected in t

254 Ni was found to have high carcinogenic risk on adult and children, having exceeded

255 tual risks far greater than the hypothetical carcinogenic risk purportedly avoided.

256 nt was conducted to assess the potential non-carcinogenic risk resulting from wine consumption.

257 Current paradigms of carcinogenic risk suggest that genetic, hormonal, and en

258 arsenic, these reductions did not lower the carcinogenic risk to an acceptable level.

259 In addition, the non-carcinogenic risk value only for the Indian rice type, w

260 toxic metals reduction was such that the non-carcinogenic risk was regarded as acceptable level after

261 the highest impact on the total value of non-carcinogenic risk was related to As.

262 While, for non-carcinogenic risk, Hazard Index (HI) values in adult wer

263 fic DNA hypomethylation of genes that confer carcinogenic risk, with greatest hypomethylation at the

264 The hazard quotient (HQ) for non-carcinogenic risks from heavy metal exposure was below t

265 ancer (IARC) Programme for the Evaluation of Carcinogenic Risks to Humans has been criticized for sev

266 tor (IARC Working Group on the Evaluation of Carcinogenic Risks to Humans, 2012) for cholangiocarcino

267 Experimental and clinical studies support a carcinogenic role for PTGS2 (cyclooxygenase-2), which is

268 sses in vertebrates and invertebrates, and a carcinogenic role has been observed in mammals.

269 The pro-carcinogenic role of Epsins suggests that they are poten

270 H2 histone methyltransferase (EZH2), but the carcinogenic role of these mutations is unclear.

271 ase that develops via one of the two primary carcinogenic routes: chemical carcinogenesis through exp

272 osteoporosis while minimizing or eliminating carcinogenic side effects.In this study, we sought to de

273 However, the molecular mechanisms of the carcinogenic signaling pathway remain largely unclear.

274 Condensate and stormwater contained numerous carcinogenic solvents used in resin synthesis, endocrine

275 ases concentration of NMVOCs (including some carcinogenic species) and peak ozone levels by 20-30% an

276 uces the blank level and avoids the use of a carcinogenic species.

277 s the formation of potentially mutagenic and carcinogenic substances.

278 however, there is limited surveillance about carcinogenic subtypes in different states of the country

279 -nitrosodipropylamine, which is 10-fold less carcinogenic than NDMA, at 20-fold lower yields.

280 (60.7%; 37/61), which further supported the carcinogenic theory of field cancerization.

281 Although radiation exposure is potentially carcinogenic, there are limited data on cumulative expos

282 mesenchymal stem/stromal cells (MSCs) in non-carcinogenic thyroids and papillary thyroid cancer (PTC)

283 ironmental toxicants, such as TCDD, that are carcinogenic to dietary indoles that are anti-inflammato

284 pollution and airborne particulate matter as carcinogenic to humans (Group 1) for lung cancer.

285 s of the > 100 agents classified as Group 1, carcinogenic to humans (IARC Monographs Volume 100, part

286 clic aromatic hydrocarbon, is classified as "carcinogenic to humans" and is ubiquitous in the environ

287 sment classifying red and processed meat as "carcinogenic to humans" on the basis of the positive ass

288 iate classification of a number of agents as carcinogenic to humans.

289 ancer (IARC) recently declared air pollution carcinogenic to humans.

290 pollution and airborne particulate matter as carcinogenic to humans.

291 the correlational structures of 26 probable carcinogenic toxics, and estimated odds ratios by brain

292 We conclude that Ate1-KO results in carcinogenic transformation of cultured fibroblasts, sug

293 potential to be used as an effector in colon carcinogenic transformation.

294 ant correlations exist between conditionally carcinogenic trihalomethanes (THMs) levels, water temper

295 < 0.001), HPV18 (P(trend) = 0.07), and other carcinogenic types (P(trend) < 0.001).

296 cious in preventing infections with the most carcinogenic types of HPV (HPV 16 and HPV 18) at the cer

297 positive results for HPV16, HPV18, and other carcinogenic types.

298 e sole mechanism in humans for the repair of carcinogenic UV irradiation-induced photoproducts in the

299 gents that may be allergenic and potentially carcinogenic, which represents a potentially hazardous c

300 emoval and preconcentration of two important carcinogenic xanthine dyes named rhodamine-B (RB) and rh