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1 py vs no therapy with risk of hepatocellular carcinoma.
2 with platinum-refractory advanced urothelial carcinoma.
3 nd possibly also of pharyngeal squamous cell carcinoma.
4 clinical surveillance for early detection of carcinoma.
5 argeting c-Myc for degradation in human lung carcinoma.
6 inal (GI)-related cancers, including gastric carcinoma.
7 premetastatic niche) in the models of breast carcinoma.
8 epithelial cell-derived nasopharyngeal cell carcinoma.
9 th metastatic platinum-refractory urothelial carcinoma.
10 or the treatment of VHL-deficient renal cell carcinoma.
11 angitis and a newly diagnosed hepatocellular carcinoma.
12 tocyte growth and progress to hepatocellular carcinoma.
13 causes Burkitt's lymphoma and nasopharyngeal carcinoma.
14 It is called squamous cell prostate carcinoma.
15 mab for the treatment of advanced renal cell carcinoma.
16 oma, urothelial carcinoma, and squamous cell carcinoma.
17 ng adenocarcinoma to melanoma and basal cell carcinoma.
18 fenib in patients with metastatic renal cell carcinoma.
19 in platinum-refractory, advanced urothelial carcinoma.
20 7 with cirrhosis and 113 with hepatocellular carcinoma.
21 patient with extremely aggressive colorectal carcinoma.
22 n locally advanced and metastatic urothelial carcinoma.
23 e 6-29) were enrolled, 26 had hepatocellular carcinoma.
24 FDG PET than the more common invasive ductal carcinoma.
25 s of mouse and human cutaneous squamous cell carcinoma.
26 s, hepatic decompensation, or hepatocellular carcinoma.
27 in patients with unresectable hepatocellular carcinoma.
28 of ~30 cancer cells from a recurrent ovarian carcinoma.
29 th locally advanced or metastatic urothelial carcinoma.
30 (lncRNA) which is induced in hepatocellular carcinoma.
31 vival in patients with metastatic urothelial carcinoma.
32 for preoperative demarcation of keratinocyte carcinomas.
33 uctal adenocarcinomas and 40% of acinar cell carcinomas.
34 rgeted agents used to treat other renal cell carcinomas.
35 c and genetic features of human adenomas and carcinomas.
36 se resemblance to human uterine endometrioid carcinomas.
37 plastic lesions into invasive and metastatic carcinomas.
38 for the treatment of cutaneous squamous cell carcinomas.
40 ormation from 12 human colorectal tumors (11 carcinomas, 1 adenoma) obtained through saturation micro
42 tridium difficile(1), promote hepatocellular carcinoma(2) and modulate host metabolism via the G-prot
43 0.40 [95% CI, 0.28-0.56) and hepatocellular carcinoma (20 studies, n = 84 491; pooled HR, 0.29 [95%
46 rs, a papillary transitional cell renal cell carcinoma, a duodenal carcinoma, two metachronous colore
50 Pre-treatment determination of renal cell carcinoma aggressiveness may help guide clinical decisio
51 eived radioiodine for differentiated thyroid carcinoma also showed interstitial pneumonia on SPECT/CT
52 exceptions were observed for nasopharyngeal carcinoma, an EBV-associated cancer, and CLL/SLL forms o
53 o test this hypothesis, HUH-7 hepatocellular carcinoma and AML12 normal hepatocytes were incubated wi
54 (encompassing MYC) in clear cell renal cell carcinoma and chromosome 11q13.3 (encompassing CCND1) in
55 co-primary outcomes (incident primary liver carcinoma and death from any cause) were ascertained fro
57 ing, we estimated the risk of hepatocellular carcinoma and liver-related mortality, accounting for co
58 a significantly lower risk of hepatocellular carcinoma and lower liver-related mortality than no use
61 decrease the risk of laryngeal squamous cell carcinoma and possibly also of pharyngeal squamous cell
62 had metastatic castration-resistant prostate carcinoma and who had exhausted available therapy option
65 e transition from in situ to invasive breast carcinoma, and, accordingly, high EPN3 levels are detect
66 mesenchymal transition in a subset of common carcinomas, and in chordoma, a rare cancer showing notoc
67 denal carcinoma, two metachronous colorectal carcinomas, and multi-regional sampling in a triple-nega
69 al characteristics divergent from urothelial carcinoma are extreme examples of tumour heterogeneity.
71 The permanent activation of hepatocellular carcinoma-associated proto-oncogenes such as c-Jun and a
74 om liver disease (cirrhosis + hepatocellular carcinoma), but data are lacking at the local level (eg,
75 -line treatment for some types of colorectal carcinoma, causes peripheral neuropathic pain in patient
77 r features that define clear cell renal cell carcinoma (ccRCC) initiation and progression are being i
78 e most prevalent type, clear cell renal cell carcinoma (ccRCC), is characterized by genetic mutations
79 a tumor-suppressor in clear cell renal cell carcinoma (ccRCC), primarily based on functional prolife
80 is highly expressed in clear cell renal cell carcinoma (ccRCC), where SLC2A3 expression is associated
84 ISPR-Cas9-mediated screen using a human lung carcinoma cell line and identify semaphorin (SEMA) 6A an
85 21 enhances the proliferation of MCF7 breast carcinoma cells and counteracts the decrease in cell pro
89 uppressor using a xenograft mouse model with carcinoma cells harboring defined ARHGAP35 alterations.
90 ng has key roles in cancer progression: most carcinoma cells have inactivated their epithelial antipr
91 re-cultured with TMPs from irradiated breast carcinoma cells increases tumor growth rates in mice rec
92 In vitro, restoration of p190A expression in carcinoma cells promotes contact inhibition of prolifera
93 ally, our data from NCI-H295R adrenocortical carcinoma cells suggest that adrenocortical tumors may e
94 acts as a metabolism modulator in pancreatic carcinoma cells through the regulation of argininosuccin
95 s increased the sensitivity of mouse mammary carcinoma cells to radiation therapy in vitro and in viv
99 enriched in a subset of human hepatocellular carcinomas characterized by comparatively poor prognosis
100 V, BKV and EBV in patient-derived colorectal carcinoma (CRC) cells typifying all molecular subtypes o
102 ancer (NMSC) such as cutaneous squamous cell carcinoma (cSCC) is caused by solar ultraviolet (SUV) ex
103 skin cancer such as cutaneous squamous cell carcinoma (cSCC) is the most common form of cancer and c
107 ified by International Metastatic Renal Cell Carcinoma Database Consortium risk status and geographic
108 tients had higher incidence of squamous cell carcinoma despite lower prevalence of behavioral risk fa
111 sts (CAFs) in the microenvironment of murine carcinomas, each endowed with unique phenotypic features
116 on of patients with esophageal squamous cell carcinoma (ESCC) who do not benefit from standard chemor
118 patients with mUC and metastatic renal cell carcinoma, even in tumors that were classically CD8(+) T
119 reas, lung adenocarcinoma, and squamous cell carcinoma) for the frequency of codon mutations of 10 Rh
120 ational Cancer Database with invasive breast carcinoma from 2012-2016 that underwent upfront lumpecto
121 ng the diagnosis of undifferentiated gastric carcinoma from the biopsy taken from the ulcerated lesio
122 for patients with an advanced hepatocellular carcinoma from the German statutory health insurance per
123 th locally advanced or metastatic urothelial carcinoma, from 221 sites in 35 countries, were randomly
125 nd MRI-guided cryoablation of cT1 renal cell carcinoma had similar excellent intermediate- and long-t
127 ith locally advanced cutaneous squamous cell carcinoma have poor prognosis with conventional systemic
128 For instance, ALK-rearranged renal cell carcinomas have shown responses to alectinib and crizoti
129 " (MTM) histologic subtype of hepatocellular carcinoma (HCC) (MTM-HCC) represents an aggressive form
130 tologic type of liver cancer, hepatocellular carcinoma (HCC) accounts for the great majority of liver
131 s frequently overexpressed in hepatocellular carcinoma (HCC) and is an independent diagnostic marker
132 r disease have lower rates of hepatocellular carcinoma (HCC) as compared to men; it is unknown if the
133 l chemoembolization (TACE) in hepatocellular carcinoma (HCC) because of the potential for profound ad
134 w that activated AKT in human hepatocellular carcinoma (HCC) cells phosphorylates cytosolic phosphoen
135 iver function and facilitates hepatocellular carcinoma (HCC) development, representing a major threat
136 There are limited data on hepatocellular carcinoma (HCC) growth patterns, particularly in Western
137 ion on the risk of developing hepatocellular carcinoma (HCC) in HCV-infected patients who achieve sus
142 h hepatitis C virus (HCV) and hepatocellular carcinoma (HCC) listed for liver transplantation (LT).
143 rterial embolization (TAE) of hepatocellular carcinoma (HCC) provides a compelling clinical correlate
144 b (SOR) is currently used for hepatocellular carcinoma (HCC) recurring after liver transplantation (L
147 s aerobic glycolysis in human hepatocellular carcinoma (HCC) through nuclear relocalization of pyruva
148 ulting cell debris stimulates hepatocellular carcinoma (HCC) tumor growth via an "eicosanoid and cyto
149 ey roles in tumors, including hepatocellular carcinoma (HCC), a malignancy with no effective treatmen
150 ational landscape of advanced hepatocellular carcinoma (HCC), and predictive biomarkers of response t
151 e marked sexual dimorphism of hepatocellular carcinoma (HCC), sex hormone receptor signaling has been
152 t progression in the study of hepatocellular carcinoma (HCC), the role of the proteasome in regulatin
153 ce is predominantly driven by hepatocellular carcinoma (HCC), which accounts for 75% of LC cases.
154 ter liver transplantation for hepatocellular carcinoma (HCC), with and without hypothermic oxygenated
164 vise a strategy for targeting hepatocellular carcinoma (HCC, one of the deadliest malignancies).
165 r liver-related events (i.e., hepatocellular carcinoma [HCC], hepatic decompensation, or liver-relate
166 Background Intermediate stage hepatocellular carcinomas (HCCs) are treated by inducing ischemic cell
173 Interrogation of head and neck squamous cell carcinoma (HNSCC) cell lines and patient tumors showed t
175 nant features of head and neck squamous cell carcinoma (HNSCC) may be derived from the presence of st
180 ionally advanced head and neck squamous cell carcinoma (HNSCC; stage III-IV according to the Union fo
181 ate incidence of head and neck squamous cell carcinomas (HNSCC), including oral cavity and pharynx (O
183 lood vasculature from human invasive bladder carcinoma (I-BLCA) and normal bladder tissue vasculature
184 ence of cases of invasive anal squamous-cell carcinoma (IASCC) in persons with HIV-1, we performed a
187 rescent PARP1 inhibitor can also detect oral carcinoma in a patient when applied as a mouthwash, and
191 expression was lost in microdissected ductal carcinoma in situ (DCIS) from patients with luminal and
195 with completely excised non-low-risk ductal carcinoma in situ were randomly assigned, by use of a mi
197 rams induced by cMYC, a response that blunts carcinoma initiation, but does not perturb the normal pr
200 ted with other malignancies (e.g. renal cell carcinoma), little is known about the role of this varia
203 ly integrated in 80% of cases of Merkel cell carcinoma (MCC), a rare but aggressive form of human ski
204 t the etiologic role of MCPyV in Merkel cell carcinoma (MCC), an extremely lethal form of skin cancer
207 itumorally in vivo in the EMT6 mouse mammary carcinoma model, OxLys-SNAs significantly increase the p
209 In B16-F10 melanoma and MC38 colorectal carcinoma mouse models, reprogramming nanoparticles in c
210 oIpi) in patients with metastatic urothelial carcinoma (mUC) and other genitourinary (GU) malignances
211 noma (EA; n = 855), esophageal squamous cell carcinoma (n = 267), and gastric cancer (cardia: n = 603
212 with stage IV lung carcinomas (n=24), renal carcinomas (n=11), or melanoma (n=44) treated with immun
213 vely included 79 patients with stage IV lung carcinomas (n=24), renal carcinomas (n=11), or melanoma
216 from patients with oral cavity squamous cell carcinoma (OCSCC) in order to map metabologenomic events
217 ed independently associated with the risk of carcinoma (odds ratio, 1.97; 95% confidence interval, 1.
219 a, squamous cell carcinoma, or adenosquamous carcinoma of the cervix, with an Eastern Cooperative Onc
220 e timing of surgery after completed nCRT for carcinoma of the esophagus or esophagogastric junction,
221 mary cutaneous signet-ring cell/histiocytoid carcinoma of the eyelid is an extremely rare but aggress
222 oma (RCC), endometrial cancer, squamous cell carcinoma of the head and neck (SCCHN), melanoma, non-sm
223 larly to confirm the presence of intraductal carcinoma of the prostate (IDC-P), an aggressive histopa
224 high risks for second in situ squamous cell carcinoma of the skin were found after Kaposi sarcoma (6
225 1 was on pembrolizumab for his squamous cell carcinoma of the skin, whereas patient 2 received RLT se
234 n, IA2, or IB1 adenocarcinoma, squamous cell carcinoma, or adenosquamous carcinoma of the cervix, wit
238 he heterogeneity of human oral squamous cell carcinoma (OSCC), we have performed genomic analysis of
241 (8/8) in detecting high-grade dysplasias and carcinomas over white-light detection alone with 75% (6/
244 inic keratosis, a precursor of squamous cell carcinoma, p16(INK4a)-expressing cells are found adjacen
247 nical presentation and workup of parathyroid carcinoma (PC) and determine its clinical prognostic par
251 multifocal leukoencephalopathy, Merkel cell carcinoma, pruritic rash or trichodysplasia spinulosa.
254 our initiation and progression in renal cell carcinoma (RCC) and three oncometabolites - fumarate, su
255 treatment landscape of metastatic renal cell carcinoma (RCC) has been revolutionized over the past tw
258 Eligible patients had metastatic renal cell carcinoma (RCC), endometrial cancer, squamous cell carci
263 in most histological subtypes of renal cell carcinomas (RCCs) and carries a decidedly poor prognosis
264 r a patient cohort with advanced endometrial carcinoma receiving lenvatinib plus pembrolizumab in an
270 and mortality from cirrhosis, hepatocellular carcinoma, solid organ malignancies, diabetes mellitus,
271 ese progression-related signatures, and that carcinoma-specific signatures are predictive of survival
273 ions, including serous tubal intraepithelial carcinoma (STIC), with genetic heterogeneity providing a
274 irectly induce cell death in CD40-expressing carcinomas, subsequently releasing tumour-specific antig
275 ho underwent potentially curative esophageal carcinoma surgery in 2011 to 2018, were included in this
276 level, the proliferation of invasive ductal carcinoma through breast tissue is beyond the range of s
279 iew included 53 patients with hepatocellular carcinoma treated with radioembolization at our center.
281 rvival and disease control in hepatocellular carcinoma treated with transarterial radioembolization w
284 tional cell renal cell carcinoma, a duodenal carcinoma, two metachronous colorectal carcinomas, and m
286 re of tumor phenotypes, including small cell carcinoma, urothelial carcinoma, and squamous cell carci
288 r cumulative probabilities of hepatocellular carcinoma, vascular events, and nonhepatic cancers were
289 ellular proliferation.IMPORTANCE Merkel cell carcinoma was first described in 1972 as a neuroendocrin
290 le, the risk of cirrhosis and hepatocellular carcinoma was reduced by 15% and 28%, respectively.
291 ma (83%); however, the rate of squamous cell carcinoma was significantly higher in females (35% vs 11
292 lid tumors, patients with anaplastic thyroid carcinoma were treated with spartalizumab, a humanized m
294 h previously untreated metastatic urothelial carcinoma who benefit from treatment with immune checkpo
295 poor for patients with metastatic urothelial carcinoma who receive standard, first-line, platinum-bas
296 d, platinum-refractory metastatic urothelial carcinoma with measurable disease and bone metastases an
298 MT mice, which spontaneously develop mammary carcinomas, with MC-deficient C57BL/6-Kit(W-sh/W-sh) (Ws
299 different from patients with hepatocellular carcinoma within Milan receiving exception point priorit
300 on remains the major cause of hepatocellular carcinoma worldwide, with more than half of HCC patients