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1 spread to the peritoneal cavity (peritoneal carcinomatosis).
2 cks metastatic disease, including peritoneal carcinomatosis.
3 s may be applied in patients with peritoneal carcinomatosis.
4 life expectancy of patients with peritoneal carcinomatosis.
5 safety of methylnaltrexone in patients with carcinomatosis.
6 l in a syngeneic model of ovarian peritoneal carcinomatosis.
7 highly effective way to suppress peritoneal carcinomatosis.
8 the therapeutic index of IPC for peritoneal carcinomatosis.
9 d complement the diagnosis of leptomeningeal carcinomatosis.
10 esophageal cancer reduced LRR and peritoneal carcinomatosis.
11 tation, which invariably involves peritoneal carcinomatosis.
12 mmunotherapy" in a mouse model of peritoneal carcinomatosis.
13 to benefit selected patients with peritoneal carcinomatosis.
14 Cs) and their capacity to initiate an active carcinomatosis.
15 l tumor excision in patients with peritoneal carcinomatosis.
16 al cavity in a condition known as peritoneal carcinomatosis.
17 results in selected patients with peritoneal carcinomatosis.
18 e (median age 55, range 4-88) had peritoneal carcinomatosis.
19 operable bowel obstruction due to peritoneal carcinomatosis.
20 d has shown clinical promise against ovarian carcinomatosis.
21 eports of high rates of local recurrence and carcinomatosis.
22 aggressive mammary cancer-induced peritoneal carcinomatosis.
23 setting of hepatic metastases and peritoneal carcinomatosis.
24 ntional imaging in the staging of peritoneal carcinomatosis.
25 and the subsequent development of peritoneal carcinomatosis.
26 (SW620) were used as a model for peritoneal carcinomatosis.
27 and treating human patients with peritoneal carcinomatosis.
28 for the diagnosis or treatment of peritoneal carcinomatosis.
29 ents with portal hypertension and peritoneal carcinomatosis.
30 calcified benign pleural lesions and pleural carcinomatosis.
31 pproach for patients with ovarian peritoneal carcinomatosis.
32 e model of aggressive GAC-derived peritoneal carcinomatosis.
33 patients with cancer with confirmed pleural carcinomatosis and 40 patients with benign pleural lesio
34 mechanisms that drive HER2(+) leptomeningeal carcinomatosis and demonstrates the efficacy of anti-GMC
42 trast MR imaging enabled better detection of carcinomatosis and tumors less than 1 cm in diameter (75
43 strating the presence of mucinous peritoneal carcinomatosis and were not considered candidates for co
47 xenograft model of ovarian cancer peritoneal carcinomatosis are provided: silencing the Rev3l subunit
49 tients, one patient was found with extensive carcinomatosis at the time of laparoscopy and had no sur
51 an cancer frequently present with peritoneal carcinomatosis, but the mechanisms that induce naive per
55 breast cancer diagnosed with leptomeningeal carcinomatosis, CSF samples were subjected to a two-step
57 ely contraindicated in those with peritoneal carcinomatosis due to theoretical risk and reported case
58 after complete CRS of colorectal peritoneal carcinomatosis, followed by IPC, in selected patients is
60 malignant bowel obstruction from peritoneal carcinomatosis from any primary malignant neoplasm and e
61 motherapy alone, the treatment of peritoneal carcinomatosis from colorectal cancer (CRPC) is still a
63 al chemotherapy for patients with peritoneal carcinomatosis from colorectal carcinoma remains to be e
67 rosarcoma, and in a model of intraperitoneal carcinomatosis, imaging of CJ215 with ambient light allo
69 ole of (18)F-FDG PET in detecting peritoneal carcinomatosis in patients with stomach, ovarian, and ad
71 nder, age, elevated tumor makers, peritoneal carcinomatosis index, and tumor grade influenced OS.
73 ods of quantifying tumour burden (peritoneal carcinomatosis index, PCI), and criteria for unresectabi
82 rn of metastasis corresponds to lymphangitic carcinomatosis metastatic phenotype in human cancer pati
84 e (LMD), encompassing entities of 'meningeal carcinomatosis', neoplastic meningitis' and 'leukaemic/l
86 urface malignancies and no gross evidence of carcinomatosis on the computed tomographic scan were enr
89 e-cell transcriptome profiling of peritoneal carcinomatosis (PC) from 15 patients with gastric adenoc
94 of the radiological extent of the peritoneal carcinomatosis (PC) or the clinical examination; and sec
95 Ascites fluid consistent with peritoneal carcinomatosis (PC) was most common, 150/241 (62%), foll
96 (LM) in patients with colorectal peritoneal carcinomatosis (PC) who underwent complete cytoreductive
104 py treatment (P < .001), peritoneal mucinous carcinomatosis (PMCA) histopathologic subtype (P < .001)
106 dy to evaluate the outcomes of patients with carcinomatosis receiving methylnaltrexone and the first
107 ients, 18.4%) was associated with peritoneal carcinomatosis (RR: 5.9; 95% CI: 3.8, 9.2; P = .03).
108 h which SPARC ameliorates peritoneal ovarian carcinomatosis seems to be multifaceted and has yet to b
109 tients which were classified as lymphangitic carcinomatosis showed high levels of VEGF-C expression i
110 uzumab treatment of patients with peritoneal carcinomatosis showed little agent-related toxicity, con
111 local recurrence, port site recurrence, and carcinomatosis that can occur with these aggressive tumo
112 st that SPARC ameliorates ovarian peritoneal carcinomatosis through abrogation of the initial steps o
114 ive and qualitative determination of pleural carcinomatosis versus noncalcified benign pleural lesion
116 to mouse ovaries and establishes peritoneal carcinomatosis, was used to evaluate the cooperative eff
117 ice), ascites production and intraperitoneal carcinomatosis were detected 3 to 7 weeks after SKOV-3 i
119 ame from the National Registry of Peritoneal Carcinomatosis, which includes 27 Spanish specialized pe