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1  congenital malformations (eg, spina bifida, cardiac anomalies).
2  patients in whom this defect is the primary cardiac anomaly.
3                The patient had no additional cardiac anomaly.
4 tion of the great arteries (ccTGA) is a rare cardiac anomaly.
5 epsin K protects against diabetes-associated cardiac anomalies.
6  tumours, aortic arch disease and other rare cardiac anomalies.
7 e demonstrated highly penetrant, pleiotropic cardiac anomalies.
8 etermine the diagnostic value of DSCT in the cardiac anomalies.
9 heart disease, particularly those with extra-cardiac anomalies.
10 rget for the treatment to obesity-associated cardiac anomalies.
11 viving cardiac surgery, and many do not have cardiac anomalies.
12 coma, as well as craniofacial, skeletal, and cardiac anomalies.
13 ues results in murine aortic arch artery and cardiac anomalies.
14 ement of neural crest perturbations in these cardiac anomalies.
15 varying degrees of alopecia, but no apparent cardiac anomalies.
16 us, right pulmonary isomerism and a range of cardiac anomalies.
17 alves, ventricular septal defects, and other cardiac anomalies.
18 evelopment and in the etiology of congenital cardiac anomalies.
19 ed in the heart and has been associated with cardiac anomalies.
20 HD phenotypes, surgical complexity and extra-cardiac anomalies.
21 excluding persons with structural congenital cardiac anomalies.
22 phism and skeletal anomalies with or without cardiac anomalies 1 (SSFSC1) syndrome, respectively.
23 itional mutations in groups of probands with cardiac anomalies and first-degree AV block, idiopathic
24 Mutations in human NKX2.5 cause a variety of cardiac anomalies and may account for a clinically signi
25  mild craniofacial dysmorphology, congenital cardiac anomalies and seizures.
26 arizes the prenatal and postnatal outcome of cardiac anomalies and sustained arrhythmias diagnosed du
27 to evaluate the role of MIF in aging-induced cardiac anomalies and the underlying mechanism involved.
28 hibit randomization of left-right asymmetry, cardiac anomalies, and glomerular kidney cysts.
29 al anomaly), nine (11.1%) of 81 fetuses with cardiac anomalies, and ten (15.4%) of 65 fetuses with sk
30 95% confidence interval [CI] 1.61-25.91) and cardiac anomalies (aOR 10.73, 95% CI 1.20-95.55).
31  ARS diagnosed at birth, and the presence of cardiac anomalies as major risk factors.
32 polydactyly, cognitive impairment, renal and cardiac anomalies as well as hypertension and diabetes.
33 polydactyly, cognitive impairment, renal and cardiac anomalies, as well as hypertension and diabetes.
34 e minimally overlapping region implicated in cardiac anomalies associated with 7p22.3 microdeletions.
35                   Here, we show that a human cardiac anomaly can be caused by a combination of rare,
36 nts had a significant interaction with extra-cardiac anomalies for transplant-free survival (P=0.003)
37 iduals with neurodevelopmental disorders and cardiac anomalies, harboring pathogenic variants in CAMK
38 polydactyly, cognitive impairment, renal and cardiac anomalies, hypertension, and diabetes.
39              The more restricted spectrum of cardiac anomalies in Bmpr2(deltaE2) mutants makes this s
40                           Left outflow tract cardiac anomalies in children present as a varied spectr
41 osage to modulate the severity of cranial or cardiac anomalies in DiGeorge/22q1 deletion syndrome (22
42 tems, encompassing severe failure to thrive, cardiac anomalies including hypertrophic cardiomyopathy
43  so called noncompaction, along with diverse cardiac anomalies, including atrioventricular septal def
44 xia may be beneficial and can rescue genetic cardiac anomalies induced by an Nkx2-5 mutation.
45             Emerging evidence indicates that cardiac anomalies lead to a reduction in cerebral oxygen
46 palate and hypodontia, as well as congenital cardiac anomalies, limb dysmorphologies and neurodevelop
47 eeded to harm (NNH)=33, 95% CI=22-77) and of cardiac anomalies (N=1,348,475, k=12; prevalence=1.2%, k
48  deregulated Lbh may account in part for the cardiac anomalies observed in these mice.
49 nts were 2.4 times more likely to have extra-cardiac anomalies (P=5.63x10(-12)).
50                                        These cardiac anomalies present as persistent truncus arterios
51 ar grafts are used to reconstruct congenital cardiac anomalies, redirect flow, and offer vascular acc
52 he second heart field revealed a spectrum of cardiac anomalies resembling forms of human congenital h
53 luding Tbx5, Tbx20 and Nkx2-5, causes severe cardiac anomalies, revealing an essential allelic balanc
54 dverse outcomes among patients without extra-cardiac anomalies, suggesting a benefit for preoperative
55       When disturbed, it leads to congenital cardiac anomalies that affect approximately 1% of live b
56 ncodes a Pbx DNA-binding partner, results in cardiac anomalies that resemble those caused by Pbx muta
57 , de novo variants in patients without extra-cardiac anomalies were associated with worse transplant-
58                               A total of 105 cardiac anomalies were diagnosed at surgery.
59               In addition, a number of other cardiac anomalies were observed after pitx2c mRNA attenu
60 canning of 46 270 fetuses revealed 1722 with cardiac anomalies, with 27.9% dying prenatally.
61 racterized by short stature and skeletal and cardiac anomalies without neurological deficits.
62 s Consortium based on presence of structural cardiac anomaly without genetic diagnosis at the time of