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1 ent in comatose survivors of out-of-hospital cardiac arrest.
2 table angina, heart failure, or resuscitated cardiac arrest.
3 on, extracorporeal membrane oxygenation, and cardiac arrest.
4 of hospital patients at risk for developing cardiac arrest.
5 rt addresses the concept of autoimmunity and cardiac arrest.
6 longation, ventricular tachy-arrhythmias and cardiac arrest.
7 tion/ventricular tachycardia out-of-hospital cardiac arrest.
8 ion may improve outcome from out-of-hospital cardiac arrest.
9 xia or ischemia due to low cardiac output or cardiac arrest.
10 parents/caregivers of children who survived cardiac arrest.
11 ry as a consequence of cardiogenic shock and cardiac arrest.
12 etrograde amnesia covering ~5 y prior to the cardiac arrest.
13 e (MAP) in patients with AMI and shock after cardiac arrest.
14 electric EEG, which precedes respiratory and cardiac arrest.
15 od with reduced signal noise after pediatric cardiac arrest.
16 table angina, heart failure, or resuscitated cardiac arrest.
17 tion with hypotension, cardiogenic shock, or cardiac arrest.
18 rporeal cardiopulmonary resuscitation during cardiac arrest.
19 ions, could improve outcomes in survivors of cardiac arrest.
20 rk with which to compare systems of care for cardiac arrest.
21 nterventions delivered to patients following cardiac arrest.
22 m was recorded during the first 3 days after cardiac arrest.
23 tent coma after resuscitated out-of-hospital cardiac arrest.
24 ssure would improve neurologic outcome after cardiac arrest.
25 ients with cardiac diagnoses and in-hospital cardiac arrest.
26 included consecutive, comatose survivors of cardiac arrest.
27 sociated with confidence in resuscitation of cardiac arrest.
28 atures predict neurologic recovery following cardiac arrest.
29 neurological consequences occurring after a cardiac arrest.
30 sequent pulselessness, and initial pulseless cardiac arrest.
31 dysfunction limits neurologic recovery after cardiac arrest.
32 survival with good neurologic outcome after cardiac arrest.
33 imit of autoregulation in patients following cardiac arrest.
34 ximately half of all comatose patients after cardiac arrest.
35 mparison with placebo during out-of-hospital cardiac arrest.
36 opportunity to improve survival from sudden cardiac arrest.
37 al releasing system after resuscitation from cardiac arrest.
38 with known cardiac risk factors) had a fatal cardiac arrest.
39 process of care for patients who have had a cardiac arrest.
40 ent with infectious disease consultants, and cardiac arrest.
41 R delivery and survival from out-of-hospital cardiac arrest.
42 rowning, and harm from CPR to victims not in cardiac arrest.
43 , Medicare beneficiaries, and lower rates of cardiac arrest.
44 ody screening was performed in patients with cardiac arrest.
45 in the chain of survival for out-of-hospital cardiac arrest.
46 eath, disabling stroke, serious bleeding, or cardiac arrest.
47 s are assessed between 6 and 12 months after cardiac arrest.
48 in function and calculation of changes after cardiac arrest.
49 n unexpected and unsuccessfully resuscitated cardiac arrest.
50 evation myocardial infarction, and 43.3% had cardiac arrest.
51 antibody signature specific to patients with cardiac arrest.
52 were cardiovascular (21 of 33), including 15 cardiac arrests.
53 and poor perfusion and those with pulseless cardiac arrests.
54 D implantation, ventricular arrhythmias, and cardiac arrest: 0.96% (95% CI: 0.77% to 1.18%) for sarco
55 Common complications were arrhythmias (19%), cardiac arrest (10%), sepsis (7%), and acute renal failu
56 racorporeal membrane oxygenation (88%) (nine cardiac arrest; 13 cardiogenic shock) and three had veno
57 y complication (3.1-fold, 95% CI 2.94-3.36), cardiac arrest (3.0-fold, 95% CI 2.64-3.46), myocardial
58 rsus 78.7%), acute noncardiac organ failure, cardiac arrest (34.3% versus 35.7%), and received less-f
60 3 were male (63%), 3,019 had out-of-hospital cardiac arrest (48%), and 2,289 had initial shockable he
61 ontaneous circulation) from 39 patients with cardiac arrest, 5 with myocardial infarction, and 10 hea
62 ; 53.9% vs 26.2%; P = .002), postreperfusion cardiac arrest (7.7% vs 0.3%; P < .001), primary nonfunc
63 es (eg, age, initial rhythm, duration of the cardiac arrest), a multifaceted approach considering bot
64 levant ventricular arrhythmias, resuscitated cardiac arrest, acute kidney failure, and corrected QT i
65 hms as well as EMS-witnessed and unwitnessed cardiac arrests all had a significant association betwee
66 ndpoint was MACE, including all-cause death, cardiac arrest, AMI, cardiogenic shock, sustained ventri
67 nal injury in the brain caused by stroke and cardiac arrest among other diseases including pain, infl
68 not evolve to BD, 4 died after an unexpected cardiac arrest and 18 after the withdrawal of life-susta
69 A total of 7433 patients with a pulseless cardiac arrest and 5751 patients with a nonpulseless eve
71 al', which includes immediate recognition of cardiac arrest and activation of the emergency response
72 y (SPK) grafts from donors with a history of cardiac arrest and cardiopulmonary resuscitation (CACPR)
75 supported by international guidelines (adult cardiac arrest and hypoxic-ischemic encephalopathy of ne
76 indicated by international guidelines (adult cardiac arrest and hypoxic-ischemic encephalopathy of ne
77 t outcomes due to organ hypoperfusion during cardiac arrest and mechanical trauma during resuscitatio
79 were performed for patients with a pulseless cardiac arrest and patients with a nonpulseless event (b
81 with prevention and early identification of cardiac arrest and proceeding through resuscitation to p
82 e first opportunity to identify a patient in cardiac arrest and provide initial care by delivering CP
84 ide, Oxygen and Mean Arterial Pressure After Cardiac Arrest and Resuscitation; NCT02698917) trials wh
85 elate with brain injury severity early after cardiac arrest and return of spontaneous circulation.
87 90-day mortality in patients with witnessed cardiac arrest and with greater than or equal to 1 minut
89 on interval, 4400-9900) cases were pulseless cardiac arrests and 8100 (95% prediction interval, 4700-
90 Because cardiac arrhythmias underlie most cardiac arrests and increasing evidence strongly support
91 ulted from two separate factors: the initial cardiac arrest (and respiratory distress) and the recurr
92 t 5-year follow-up: SCD, resuscitated sudden cardiac arrest, and aborted SCD, that is, appropriate sh
93 y limiting use of TRI (renal failure, shock, cardiac arrest, and mechanical circulatory support) were
94 are common among patients who have survived cardiac arrest, and often go unrecognised despite being
95 l cardiopulmonary resuscitation in pediatric cardiac arrest, and pediatric targeted temperature manag
96 d of a composite of cardiac syncope, aborted cardiac arrest, and sudden cardiac death, but a 38.8-fol
97 ioverter-defibrillator therapy, resuscitated cardiac arrest, and sustained ventricular tachycardia).
101 evascularization procedures, or resuscitated cardiac arrest) assessed using measures of model discrim
103 l benefit in select patients with refractory cardiac arrest but there is insufficient data on the fre
108 b plus lenalidomide and dexamethasone group (cardiac arrest, cardiac failure, myocarditis, large inte
109 d metabolism during ventricular fibrillation cardiac arrest, cardiopulmonary resuscitation, and epine
110 s unknown whether certain characteristics of cardiac arrest care and outcomes of IHCAs during the COV
116 re compared with those from 22 patients with cardiac arrest cases of ischemic origin and a group of 2
117 rdiography for intra-arrest prognostication, cardiac arrest caused by pulmonary embolism, postresusci
118 linical debriefing, transport to specialized cardiac arrest centers, organ donation, and performance
119 entation and performance in communities, and cardiac arrest centers; advanced life support training,
120 upon carbon monoxide treatment, whereas post-cardiac arrest cerebral perfusion differences were dimin
123 n traumatic brain injury and anoxo-ischemic (cardiac arrest) coma patients by using an original multi
126 he durvalumab plus platinum-etoposide group (cardiac arrest, dehydration, hepatotoxicity, interstitia
127 ever, their rate of progression to pulseless cardiac arrest despite CPR and the differences in surviv
128 uscitated after experiencing out-of-hospital cardiac arrest, discharge survival was significantly low
129 y of cases, individuals with out-of-hospital cardiac arrest do not receive lay rescuer CPR and wait f
130 By multivariable analysis-adjusting for cardiac arrest duration, Sequential Organ Failure Assess
131 is higher rates of PRS, PNF, postreperfusion cardiac arrest, EAD, and AKI should be anticipated.
133 ccessfully resuscitated from out-of-hospital cardiac arrest enrolled in the CCC trial (Trial of Conti
134 evere tracheal intubation-associated events (cardiac arrest, esophageal intubation with delayed recog
136 ted in less than or equal to six in-hospital cardiac arrest events per year, and 41% of respondents w
137 patients with refractory cardiogenic shock (cardiac arrest excluded) who required venoarterial extra
138 for advanced airway management in pediatric cardiac arrest, extracorporeal cardiopulmonary resuscita
139 12, or 16-minute potassium chloride-induced cardiac arrest followed by 90 seconds of cardiopulmonary
140 om 84 089 adult patients with an in-hospital cardiac arrest from 166 hospitals with continuous partic
141 prove overall survival after out-of-hospital cardiac arrest from shock-refractory ventricular fibrill
142 we compared a primary composite end point of cardiac arrest from ventricular tachycardia/fibrillation
143 ardized survival rate (RSSR) for in-hospital cardiac arrest has emerged as an important metric to ben
145 mes of patients experiencing out-of-hospital cardiac arrest have demonstrated that, despite a higher
146 myocardial infarction, stroke, resuscitated cardiac arrest, heart failure, or revascularization.
153 a prospective registry enrolling all sudden cardiac arrest in Paris and suburbs (6.7 million inhabit
154 in a multivariable analysis, out-of-hospital cardiac arrest in predominantly Hispanic neighborhoods h
156 for patients presenting with out-of-hospital cardiac arrest in states with public reporting versus wi
157 us adverse event occurred in each group (one cardiac arrest in the control group and one episode of a
159 The average annual incidence of in-hospital cardiac arrest in the United States was estimated at 292
160 ood functional outcome after out-of-hospital cardiac arrest in this cohort, this association is prima
162 en translated to clinical care for pediatric cardiac arrest, in part because signal noise causes high
164 d patients, 36 (31.6%) screened positive for cardiac arrest-induced posttraumatic stress symptomatolo
167 Among patients experiencing out-of-hospital cardiac arrest, intra-arrest transport to hospital compa
171 ing cardiopulmonary resuscitation for sudden cardiac arrest is common, occurring even in the absence
172 ity of cardiogenic shock following asystolic cardiac arrest is dependent on the length of cardiac arr
174 lin G purified from patients with idiopathic cardiac arrest is proarrhythmogenic by reducing the acti
176 phy after resuscitation from out-of-hospital cardiac arrest is uncertain for patients without ST-segm
178 rdinal severity score (worst to best: death, cardiac arrest, mechanical ventilation with mechanical c
183 artile range, 55-80), 37% were women, 86% of cardiac arrests occurred in a private location, 49% were
187 ber of DALY because of adult out-of-hospital cardiac arrest (OHCA) in the United States is unknown.
189 ms of care for patients with out-of-hospital cardiac arrest (OHCA), as little evidence exists to guid
193 n complete remission from sepsis (two [3%]), cardiac arrest (one [1%]), therapy-related acute myeloid
195 can assess myocardial physiology and predict cardiac arrest outcomes, making these measures a candida
197 c support did not increase the risk of a new cardiac arrest (p = 0.88) or atrial fibrillation (p = 0.
198 ntly identified as a biomarker of idiopathic cardiac arrest (P=0.002; false discovery rate, 0.007; cl
199 , mechanical ventilation, mental status, and cardiac arrest parameters of the PBS were independent ri
203 ous coronary intervention (PCI) after sudden cardiac arrest, particularly in the absence of ST-segmen
204 t on the survival rate among out-of-hospital cardiac arrest patients (OHCA) is not well studied.
206 ARTICIPANTS (SUBJECTS): We enrolled 41 adult cardiac arrest patients in whom blood could be obtained
210 y used patient-level pooled analysis of post-cardiac arrest patients with shock after AMI randomized
213 and neurologic injury in intensive care and cardiac arrest patients; however, few studies have inves
214 al Directed Hemodynamic Optimization in Post-cardiac Arrest Patients; NCT02541591) and COMACARE (Carb
216 osseous route for drug administration during cardiac arrest, point-of-care echocardiography for intra
219 round appropriate use of mechanical support, cardiac arrest prevention, and optimal heart transplanta
220 cardiac arrest is dependent on the length of cardiac arrest prior to cardiopulmonary resuscitation an
221 rdiac arrest, we discovered that duration of cardiac arrest prior to cardiopulmonary resuscitation de
222 the public health impact of out-of-hospital cardiac arrest, provides guidance and resources to const
223 time series analysis was used to compare the cardiac arrest rate in the 18 months before and 4.5 year
229 tions about community initiatives to promote cardiac arrest recognition, cardiopulmonary resuscitatio
235 both confident participating in in-hospital cardiac arrest resuscitation and did not worry about mak
236 re most likely to participate in in-hospital cardiac arrest resuscitation at a community, rural, or c
237 transthoracic echocardiography (TTE) during cardiac arrest resuscitation can enable the characteriza
239 luating clinician perceptions of in-hospital cardiac arrest resuscitation participation was developed
241 luate and optimize the use of epinephrine in cardiac arrest resuscitation, particularly the dose, tim
243 linicians' sentiments about participating in cardiac arrest resuscitations and identify factors assoc
244 ion analysis identified initial nonshockable cardiac arrest rhythm (odds ratio, 12.2; 95% CI, 2.83-52
245 d survival after adjustment for age, gender, cardiac arrest rhythm, witnessed status, bystander cardi
247 n 2, and our previously developed electronic Cardiac Arrest Risk Triage score were compared for predi
248 ed disease (CID) causing resuscitated sudden cardiac arrest (RSCA) on a population basis is unknown.
249 serious arrhythmogenic endpoints like sudden cardiac arrest (SCA) or ventricular arrhythmia (VA).
252 nrolled, interventions that were part of the cardiac arrest structured care pathway, and outcomes.
256 eported outcomes of clinical trials of adult cardiac arrest survivors and supported this P-COSCA init
257 Most of the poor outcomes and deaths of cardiac arrest survivors have been attributed to widespr
258 ologic outcome prediction in out-of-hospital cardiac arrest survivors is highly limited due to the la
259 atients with acute cardiovascular events and cardiac arrest survivors, testing for primary and second
263 lmonary resuscitation with a Lund University Cardiac Arrest System, and estimated transfer time short
265 ould consider methods to provide in-hospital cardiac arrest teams additional "effective experience,"
267 n of spontaneous circulation for in-hospital cardiac arrest than nonaward hospitals (median [interqua
268 versus 54.9%), and had a lower proportion of cardiac arrests that were witnessed (55.1% versus 64.5%)
269 rlying characteristics, steroid use prior to cardiac arrest, the vasopressors, and shockable rhythm d
271 m of the study was to evaluate the effect of cardiac arrest time (CAT) in donors after brain death (D
272 7 hours (15-88 hr), and median duration from cardiac arrest to inclusion was 15 hours (6-44 hr).
273 the COSCA initiative (Core Outcome Set After Cardiac Arrest) to improve consistency in reported outco
274 tion/ventricular tachycardia out-of-hospital cardiac arrest treated with the University of Minnesota
277 the following topics: dispatch diagnosis of cardiac arrest, use of a firm surface for CPR, sequence
278 neurologic outcome at 12 and 24 hours after cardiac arrest using electroencephalogram epochs and out
279 delivery and survival after out-of-hospital cardiac arrest vary at the neighborhood level, with lowe
283 the initial 6 hours after resuscitation from cardiac arrest was independently associated with good ne
285 ompared with patients receiving neither drug cardiac arrest was significantly more likely in patients
287 k of hypoxic ischemic brain injury following cardiac arrest, we sought to: 1) characterize brain oxyg
288 ma samples from 23 patients with unexplained cardiac arrest were compared with those from 22 patients
289 ion after resuscitation from out-of-hospital cardiac arrest were prospectively randomized in a 1:1 fa
291 e to haemophagocytic lymphohistiocytosis and cardiac arrest) were previously reported, but no new tre
292 Innovative digital tools for recognizing cardiac arrest where and when it occurs, notifying poten
293 less ventricular tachycardia out-of-hospital cardiac arrest who were randomly assigned by emergency m
294 re management strategy after out-of-hospital cardiac arrest with initial shockable rhythm resulted in
295 nts with coma who had been resuscitated from cardiac arrest with nonshockable rhythm, moderate therap
297 esearch agenda for the use of focused TEE in cardiac arrest with the goal to improve resuscitation ou
298 increased vasopressor 32 [19%] vs 31 [18%], cardiac arrest within 1 h seven [4%] vs two [1%], death
299 l, we randomly assigned 552 patients who had cardiac arrest without signs of STEMI to undergo immedia