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1  age who have clinical evidence of a primary cardiac disorder.
2 nts with existing hypertension or history of cardiac disorder.
3 fatal arrhythmias when harboring a quiescent cardiac disorder.
4 es causes a new autosomal dominant Mendelian cardiac disorder.
5 rdiac events in patients with this inherited cardiac disorder.
6 y involved in an, as yet, unidentified human cardiac disorder.
7 chitis, diarrhea, headache, inflammation and cardiac disorders.
8 t development, ischemic cardiac disease, and cardiac disorders.
9 elta) serves as a central driver of multiple cardiac disorders.
10 tin has emerged as a major disease locus for cardiac disorders.
11 cardiac cirrhosis, a serious complication of cardiac disorders.
12 receptor B) signaling accounts for brain and cardiac disorders.
13 al myocarditis, and other vaccine-associated cardiac disorders.
14 nality and the pathophysiology of associated cardiac disorders.
15  evidence as to their actions in early-stage cardiac disorders.
16 and reveal potential therapeutic targets for cardiac disorders.
17  odds of diabetes, hyperlipidemia, gout, and cardiac disorders.
18 reased prevalence of physical disability and cardiac disorders.
19 ariety of chronic neurologic, endocrine, and cardiac disorders.
20 trophic cardiomyopathy, and other hereditary cardiac disorders.
21 iple neurodegenerative disorders, aging, and cardiac disorders.
22 old hearts with implications for age-related cardiac disorders.
23 el a promising novel target for treatment of cardiac disorders.
24 tial of physical activity in obesity-related cardiac disorders.
25 otential of GPR55 as a therapeutic target in cardiac disorders.
26 mong the most frequently occurring inherited cardiac disorders.
27  ECM is observed in both pediatric and adult cardiac disorders.
28  new mechanisms in the pathogenesis of these cardiac disorders.
29 on can be a potential therapeutic target for cardiac disorders.
30 y on subcellular scales and life-threatening cardiac disorders.
31 ic and local iron metabolism correlates with cardiac disorders.
32  a role in the pathogenesis of lipid-related cardiac disorders.
33 ffer from a severe complication of different cardiac disorders.
34  as a valuable therapy for various end-stage cardiac disorders.
35  increase contractile force in patients with cardiac disorders.
36 t disease, accounting for 1-3% of congenital cardiac disorders.
37 derlie arrhythmias and sudden death in human cardiac disorders.
38 e apoptosis is potentially important in many cardiac disorders.
39 r than 1 month were excluded unless they had cardiac disorders.
40  3-4 adverse events of special interest were cardiac disorders (41 [8%] of 542 patients in the abirat
41 ebrile neutropenia (10% v 3%; P = .003), and cardiac disorders (5% v 5%; P = .759); transfusion rates
42 toxicity (188 [8%]), hypertension (99 [4%]), cardiac disorders (52 [2%]), osteoporosis (31 [1%]), hyp
43      Although SCN3B has been associated with cardiac disorders, a link with neurodevelopmental disord
44 ardiomyopathy (HCM) is a naturally occurring cardiac disorder afflicting humans, cats, rhesus macaque
45 r (aHR = 2.05[2.00, 2.11], p < 0.001), other cardiac disorders (aHR = 2.31[2.26, 2.35], p < 0.001), t
46 1.2%) patients had 12 serious AEs; most were cardiac disorders; all were unrelated to DEX treatment.
47 linked to judged to be related to rucaparib (cardiac disorder and myelodysplastic syndrome), and one
48 ssion correlates with human diseases such as cardiac disorders and cancer.
49       Fibrosis is a common pathology in many cardiac disorders and is driven by the activation of res
50 ss in the development of drugs for epilepsy, cardiac disorders and pain.
51    Sera were obtained from 100 patients with cardiac disorders and periodontal disease and 73 patient
52  and duplications in 101 genes implicated in cardiac disorders and sudden death using a postmortem bl
53 ega-3 polyunsaturated fatty acids on various cardiac disorders and the risk factors for cardiac disea
54 brillation and heart failure are very common cardiac disorders, and both are associated with symptoms
55 olic disorders, neurodegenerative disorders, cardiac disorders, and cancer.
56 ate cardiac function have been implicated in cardiac disorders, and efforts to develop therapeutic an
57 ividuals is commonly attributed to inherited cardiac disorders, and familial evaluation is advocated.
58  events were infections, vascular disorders, cardiac disorders, and neoplasms.
59 noic acid have beneficial effects in various cardiac disorders, and their use is recommended in guide
60 mostly hereditary, structural, or electrical cardiac disorders are associated with SCD in young athle
61                          Concomitant chronic cardiac disorders are frequent in patients with COPD, li
62                                              Cardiac disorders are increasingly recognised as an impo
63  it has become evident that most inheritable cardiac disorders are not monogenic but, rather, have a
64                                              Cardiac disorders are the main cause of mortality in aut
65 ental gene associated with various important cardiac disorders, as a healthy adult cardiac fibroblast
66 action cardiomyopathy is a common congenital cardiac disorder associated with abnormal ventricular ca
67 luminated the genetic basis of the inherited cardiac disorders associated with sudden cardiac death.
68 da syndrome (BrS) is a highly arrhythmogenic cardiac disorder, associated with an increased incidence
69  disorders, ischemic heart disease and other cardiac disorders between 30 days and (up to) 3.5 years
70          Cardiac MRI (CMR) has been used for cardiac disorders, but its use in cryptogenic stroke is
71 ulators of inflammation and are activated in cardiac disorders, but their precise contributions and t
72                  Long QT syndrome (LQT) is a cardiac disorder causing syncope and sudden death from a
73 ondria dysfunctions are typical hallmarks of cardiac disorders (CDs).
74 rdiomyopathy is a common but underrecognized cardiac disorder characterized by a heterogenous phenoty
75 ure contractions syndrome presents as a rare cardiac disorder characterized by frequent multifocal ec
76 pertrophic cardiomyopathy (HCM) is a genetic cardiac disorder characterized by left ventricular hyper
77 icular cardiomyopathy (ARVC) is an inherited cardiac disorder characterized by loss of cardiomyocytes
78 with Wolff-Parkinson-White (WPW) syndrome, a cardiac disorder characterized by ventricular pre-excita
79                                              Cardiac disorders, coagulation abnormalities, and infect
80                               This inherited cardiac disorder constitutes an important cause of malig
81 diomyopathy (HCM), the most common inherited cardiac disorder, exhibits remarkable genetic and clinic
82 srhythmia, ischemic heart disease, and other cardiac disorders following infection.
83 y ), the most common genetically transmitted cardiac disorder, has been the focus of extensive resear
84 ysis of the CFC1 gene in patients with these cardiac disorders identified two disease-related mutatio
85 hereas the clinical course of this inherited cardiac disorder in the older population has not been st
86 arify the etiology of neurodevelopmental and cardiac disorders in children; however, the contribution
87  the hiPSC technology for modeling inherited cardiac disorders in general and ARVC specifically.
88 ere fewer serious adverse events and serious cardiac disorders in participants treated with semagluti
89 cular systolic dysfunction, a higher rate of cardiac disorders in the control groups than in the stud
90                 The genetic bases of several cardiac disorders, including cardiomyopathies that cause
91 nies a wide spectrum of clinically important cardiac disorders, including ischemia/reperfusion, diabe
92 tress is common in many clinically important cardiac disorders, including ischemia/reperfusion, diabe
93                                 The onset of cardiac disorders indicated that subjects with truncatio
94         In the United Kingdom, screening for cardiac disorders is confined to symptomatic individuals
95 Adverse cardiac remodeling in RIHD and other cardiac disorders is frequently accompanied by mast cell
96 eciphering the genetic architecture of human cardiac disorders is of fundamental importance but their
97 diomyopathy (HCM), the most common inherited cardiac disorder, is characterized by increased ventricu
98 rdiomyopathy (HCM) is a prevalent hereditary cardiac disorder linked to arrhythmia and sudden cardiac
99                                     Like the cardiac disorders (long QT syndrome type 3 or Brugada sy
100 icular cardiomyopathy (ARVC) is an inherited cardiac disorder mainly caused by dominant mutations in
101 ses including epilepsy, pain conditions, and cardiac disorders, making Nav channels a significant pha
102 re enriched in genes involved in metabolism, cardiac disorders, muscle contraction, reproduction, beh
103  (12% belatacept versus 8% CsA), and serious cardiac disorders occurred more frequently with CsA (2%
104 n of Na(+) channels and Na(+) homeostasis in cardiac disorders of excitability and mechanics emphasiz
105 potentially related to rucaparib (one due to cardiac disorder, one due to myelodysplastic syndrome, a
106 e in this homeostasis is likely to influence cardiac disorders or cardiomyopathy.
107 flozin reduced the rate of admissions due to cardiac disorders, renal and urinary disorders, metaboli
108 es are associated, at least indirectly, with cardiac disorders, some of which have severe consequence
109 nt disorders (SR, 1.54 [95% CI, 1.19-1.93]), cardiac disorders (SR, 1.18 [95% CI, 1.05-1.32]), endocr
110 tio, 2.2; 95% CI, 1.6-3.1) versus those with cardiac disorders (subhazard ratio, 1.5; 95% CI, 0.9-2.5
111 s such as Alzheimer disease and epilepsy and cardiac disorders such as coronary artery disease and my
112 he development of optogenetic treatments for cardiac disorders such as the long QT syndrome.
113 ase might promote the development of various cardiac disorders (such as arrhythmias and cardiomyopath
114                  Long-QT (LQT) syndrome is a cardiac disorder that causes syncope, seizures, and sudd
115 ms underlying long QT syndrome, an inherited cardiac disorder that causes syncope, seizures, and sudd
116       Long QT syndrome (LQT) is an inherited cardiac disorder that results in syncope, seizures, and
117 emic cardiomyopathies are a diverse group of cardiac disorders that frequently cause heart failure an
118 es account for more than 50% of an inherited cardiac disorder, the Long QT syndrome.
119            By serving as models of inherited cardiac disorders, these systems have the potential to f
120 ere nervous system disorders (four [3%]) and cardiac disorders (three [2%]).
121 C-CMs) from patients with various hereditary cardiac disorders to model differences in cardiac drug t
122 nt group discontinued due to adverse events (cardiac disorders unrelated to 4F-PCC).
123 of discontinuation of trastuzumab because of cardiac disorders was low (5.1%).
124  AI-ECG models for structural and functional cardiac disorders was more likely to be associated with
125                       Neonates with possible cardiac disorders were identified by the general pediatr
126  77 LMNA mutation carriers from 45 families; cardiac disorders were retrospectively analyzed.
127  people with chronic morbidity; diabetes and cardiac disorders were the main susceptibility factors.
128 ent adverse events, including infections and cardiac disorders, were similar in the amiselimod treatm
129 ng therapeutic strategy for treating various cardiac disorders where Cx43 is remodelled.
130 ssible therapeutic potential of rapamycin in cardiac disorders where pathologic mTORC1 activation occ
131 her mutations that may underlie many genetic cardiac disorders, whereas RNA sequencing can be used to
132 an autosomal-recessive neurodegenerative and cardiac disorder which occurs when transcription of the
133 MKIIdelta) is a prominent inducer of various cardiac disorders, which is mediated by 2 oxidation-sens
134                                  Inheritable cardiac disorders, which may be associated with cardiomy
135 r data suggest a novel pathogenic process in cardiac disorders with abnormal protein aggregation.
136 an important pathogenic mechanism underlying cardiac disorders with abnormal protein aggregation.
137 he significance of sudden death in heritable cardiac disorders with delayed expression is incompletel
138 ces the clinical management and treatment of cardiac disorders with highly variable outcome.
139                                     Modeling cardiac disorders with human induced pluripotent stem ce
140 omyopathy (HCM) is the most common inherited cardiac disorder, with an overall prevalence of at least

 
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