ライフサイエンスコーパス: cardiac index

1 P), pulmonary vascular resistance (PVR), and cardiac index.

2 e and negatively with 6-minute walk test and cardiac index.

3 ed with mean pulmonary arterial pressure and cardiac index.

4 with higher right atrial pressure and lower cardiac index.

5 d systemic vascular resistance, and elevated cardiac index.

6 duced systemic vasodilation and elevated the cardiac index.

7 CHF patients and are inversely dependent on cardiac index.

8 eGFR had more women, nonsmokers, and a lower cardiac index.

9 d gas exchange but at the expense of a lower cardiac index.

10 ompared with controls, which correlated with cardiac index.

11 ndex >/=35 mL/m(2) (P<0.01), despite similar cardiac index.

12 0.016) compared with individuals with normal cardiac index.

13 significant association between BMI and both cardiac index (0.003 L/min/m(2); 95% CI, -0.008-0.014; p

14 stance (114 m; 95% CI, 67, 160; p = 0.0002), cardiac index (0.3 L x min(-1) x m(-2); 95% CI, 0.1, 0.4

15 59 m; 95% CI, 29, 89; n = 12, p = 0.001) and cardiac index (0.3; 95% CI, 0.1, 0.4; p = 0.008).

16 gnificantly different from placebo (P=0.10), cardiac index (0.4 L.min(-1).m(-2); 95% confidence inter

17 TTM33 was associated with decreased cardiac index (-0.4 L/min per m(2); P(group) <0.0001), d

18 had more severe myocardial dysfunction (mean cardiac index 1.5 L/min per m(2)vs 2.2 L/min per m(2), L

19 In the study cohort (age 57 +/- 14 years, cardiac index 1.9 +/- 0.6 l/min/m(2), left ventricular e

20 s [WU] vs. 2.1 +/- 1.1 WU; p = 0.017), lower cardiac index (1.6 +/- 0.4 l/min/m(2) vs. 2.7 +/- 0.7 l/

21 e patients had dilated ventricles with a low cardiac index (1.9 +/- 0.6 L/min/m) and high pulmonary a

22 ciated with greater RV systolic dysfunction (cardiac index, 1.9 vs. 2.7 L/min/m2; RV % area change, 2

23 in the hearts perfused in WM (median 11-hour cardiac index/1-hour cardiac index: WM=27% versus non-WM

24 L/min/m (-0.1 to 0.1 L/min/m) (p = 0.86) for cardiac index; -1.8 beats/min (-3.7 to 0.1 beats/min) (p

25 stable contractility after transplantation (cardiac index: 113.0+/-43% of NRP function) and improved

26 spite similar levels of cardiac dysfunction (cardiac index 2.2 and 2.1 liters/minute/m(2), respective

27 tion: 37.6+/-12.8% versus 29.0+/-9%: P<0.05; cardiac index 2.7+/-0.9 versus 2.2+/-0.4 L.min(-1).m(-2)

28 ressive, systolic function mainly preserved (cardiac index 2.8+/-0.6 [1.9-3.9] L/min per m(2)), and d

29 6 [8.3] vs 12.9 [8.3] Wood units), and lower cardiac index (2.11 [0.69] vs 2.51 [0.92] L/min per m(2)

30 Hg vs. 45.7 +/- 9.4 mm Hg, respectively) and cardiac index (2.3 +/- 0.8 l/min/m(2) vs. 2.2 +/- 0.8 l/

31 , 42 +/- 6 mm; ejection fraction, 65 +/- 8%; cardiac index, 2.6 +/- 0.8 L/min per m(2)), patients had

32 - 8 mm Hg vs. 20 +/- 7 mm Hg, p = 0.02, Fick cardiac index: 2.2 l/min/m(2) [interquartile range: 1.87

33 Compared to placebo, levosimendan increased cardiac index (22%), stroke volume index (15%), and hear

34 patients had severe myocardial dysfunction (cardiac index 3 L/min per m(2) or less or left ventricul

35 ed (mean arterial pressure 70 [65-77] mm Hg, cardiac index 3.3 [2.7-4.0] L/min.m, and SvO2 68.3 [62.8

36 e, prone positioning significantly increased cardiac index (3.0 [2.3-3.5] to 3.6 [3.2-4.4] L/min/m(2)

37 nts with microvascular dysfunction had lower cardiac index (3.1+/-0.7 versus 3.5+/-0.7 L/min per m(2)

38 335 dyne . s . cm(-5) , P < 0.05) and higher cardiac index (3.3 +/- 0.9 versus 2.8 +/- 0.4 L/min/m(2)

39 ssure (median, 11 vs 15 cm H(2)O; P < .001), cardiac index (3.4 vs 3.8 L/min/m(2); P = .001), and glo

40 0.07 vs. 0.29 +/- 0.05, p < .05) and higher cardiac index (4.8 +/- 0.4 vs. 3.4 +/- 0.2 L.min-1.m-2,

41 Hepa Wash also enhanced cardiac index (4.94+/-0.32 vs. 3.36+/-0.25 l/min/m2, p=0

42 peripherally inserted central catheter (mean cardiac index, 4.2 vs 3.7 L/min/m; p = 0.043; bias, 0.51

43 sitivity, 68% specificity), and by change in cardiac index (69% sensitivity, 59% specificity), change

44 2] vs 33 mm Hg [95% CI, 30-36], p < 0.0001), cardiac index (76 mL/min/kg [95% CI, 63-91] vs 47 mL/min

45 lung water >/= 10% was predicted by baseline cardiac index (77% sensitivity, 98% specificity) and pul

46 The increased cardiac index accounted for the increased cerebral and p

47 ure variation and the change in stroke index/cardiac index after a fluid challenge.

48 me variation and the change in stroke volume/cardiac index after a fluid or positive end-expiratory p

49 < 0.05) were Crawford type 2, acuity, SFDN, cardiac index after unclamping, mean arterial pressure d

50 ressure, there was a significant increase in cardiac index and a slight but significant reduction in

51 HES improved liver microcirculation, cardiac index and DO(2)-I, but significantly increased I

52 With the exception of cardiac index and indexed systemic vascular resistance,

53 Microsphere-determined cardiac index and left ventricular myocardial blood flow

54 ours of reperfusion and resulted in elevated cardiac index and local cerebral blood flow compared wit

55 diac energy-dependent workload with improved cardiac index and lower vascular resistance, 2) upgraded

56 (1))apelin-13 (30 to 300 nmol/min) increased cardiac index and lowered mean arterial pressure and per

57 trahydrobiopterin prevented the decreases in cardiac index and mean arterial pressure.

58 jury, there is a high prior probability that cardiac index and mixed venous oxygen saturation are nor

59 vascular resistance index, and PVRI, whereas cardiac index and mixed venous oxygen saturation remaine

60 lar tone and oxygen extraction, whereas both cardiac index and oxygen delivery decreased for patients

61 ase in vascular tone and a reduction in both cardiac index and oxygen delivery.

62 ic left ventricular dysfunction and improved cardiac index and pulmonary and systemic vascular resist

63 pressure, and secondary end points comprised cardiac index and pulmonary arterial pressure at rest an

64 Baseline cardiac index and pulmonary blood volume index were high

65 ed with higher pulmonary pressures and lower cardiac index and pulmonary capacitance (all P<0.05).

66 tion improved cardiac performance, improving cardiac index and pulmonary capillary wedge pressure, bu

67 ry arterial pressure, right atrial pressure, cardiac index and pulmonary vascular resistance.

68 Cardiac index and QRS duration were improved by BDOO com

69 Hemodynamic variables such as cardiac index and right atrial pressure have consistentl

70 cular volume indexes or cardiac performance (cardiac index and stroke volume index).

71 is also associated with an acute decrease in cardiac index and systemic O(2) delivery.

72 RI) was estimated from the echocardiographic cardiac index and the mean arterial pressure.

73 s pressure and change in stroke volume index/cardiac index and the percentage of fluid responders.

74 disease were excluded, the relation between cardiac index and total brain volume remained (P=0.02).

75 Pulse contour analysis-derived cardiac index and velocity-time integral were measured d

76 worse systolic (lower EF, stroke volume, and cardiac index) and diastolic (shorter deceleration time

77 ressure, pulmonary capillary wedge pressure, cardiac index, and estimated glomerular filtration rates

78 l/min) renal function, despite similar PCWP, cardiac index, and LV ejection fraction.

79 ved measurements, mean right atrial pressure cardiac index, and mixed venous oxygen saturations.

80 unctional class, mean right atrial pressure, cardiac index, and mixed venous oxygen saturations.

81 ially decreased coronary perfusion pressure, cardiac index, and myocardial blood flow.

82 e systemic vascular resistance index (SVRI), cardiac index, and myocardial performance at a targeted

83 moderate-severe tricuspid regurgitation, low cardiac index, and raised right atrial pressure were ass

84 educes right heart hypertrophy, restores the cardiac index, and reduces pulmonary vascular remodeling

85 ial effusion, pulmonary vascular resistance, cardiac index, and right atrial pressure may be used to

86 Systolic blood pressure, cardiac index, and stroke volume index were significantl

87 lent improvement in systolic blood pressure, cardiac index, and stroke volume index, when the ITD alo

88 ors including pulmonary vascular resistance, cardiac index, and vasoreactivity.

89 rger left ventricular volumes, more impaired cardiac indexes, and the presence of cardiac resynchroni

90 evated intracardiac filling pressures, lower cardiac index, anemia, hypoalbuminemia, hyperbilirubinem

91 tment groups did not significantly differ in cardiac index, arrhythmias, peak lactate, inotropic scor

92 Blood samples for cardiac index (arterial and venous) and tissue Doppler (

93 Median cardiac index at 6 months was significantly lower than b

94 Median cardiac index at beginning of the treatment was 5.05 L/m

95 subjects and controls, with lower VO(2) and cardiac index at peak, and more severe dyspnea and fatig

96 es, LV end-systolic volumes, cardiac output, cardiac index, atrial volumes, and NT-proBNP were also s

97 The cardiac index averaged 0.73 +/- 0.19 mL/min/g and the st

98 ltiple logistic regression analysis baseline cardiac index, baseline pulmonary blood volume index, th

99 is associated with increased SVRI and lower cardiac index because of lower heart rate with unaffecte

100 No differences were observed for cardiac index before and after the operation; at the arr

101 ascular resistance and increased heart rate, cardiac index, blood urea nitrogen (BUN) level, creatini

102 ased heart rate, mean arterial pressure, and cardiac index but decreased tissue perfusion indicated b

103 EOV was associated with slightly lower mean cardiac index but other invasive hemodynamic variables w

104 olume expansion did not significantly change cardiac index, but the oxygen delivery decreased due to

105 Nitric oxide inhalation increased the cardiac index by 24 +/- 11% and the stroke volume index

106 DITPA increased cardiac index (by 18%) and decreased systemic vascular r

107 cardiac magnetic resonance imaging-assessed cardiac index (cardiac output divided by body surface ar

108 riables were related to cardiac MRI-assessed cardiac index (cardiac output/body surface area).

109 s, the Simplified Acute Physiology Score II, cardiac index, cardiac power index, and continuous hemod

110 output divided by body weight was defined as cardiac index; cardiac output divided by heart rate yiel

111 y pressure, in view of widening the range of cardiac index:central venous pressure measurements and i

112 The slope of the multipoint cardiac index:central venous pressure relationship incre

113 pulmonary blood volume index, the change in cardiac index, change in pulmonary blood volume index, a

114 volume index were higher, whereas change in cardiac index, change in pulmonary blood volume index, a

115 in WSES were not correlated with changes in cardiac index, changes in WSED correlated significantly

116 pulmonary capillary wedge pressure (PCWP) to cardiac index (CI) at peak exercise after 12 weeks.

117 pulmonary capillary wedge pressure and lower cardiac index (CI) but not Paco2 or Pao2.

118 = 0.706; P = 0.002) strongly correlated with cardiac index (CI) following transplantation.

119 It is widely believed that a reduced cardiac index (CI) is a significant contributor to renal

120 Cardiac index (CI) was significantly increased in both t

121 ulmonary capillary wedge pressure (PCWP) and cardiac index (CI), and by changes in those measures aft

122 n fraction (LVEF) and LV-Tei correlated with cardiac index (CI; P<0.001), and LV Tei was most frequen

123 PAP >/=35 mm Hg or mPAP >/=25 mm Hg with low cardiac index [CI <2.0 l/min/m(2)]; severe PH-COPD, seve

124 iac output was indexed to body surface area (cardiac index [CI]) for all analyses.

125 diography) and indexed to body surface area (cardiac index [CI]).

126 e, total peripheral vascular resistance, and cardiac index, compared with effects in sham rats.

127 e in 20 of 24 patients with normalization of cardiac index (complete response [CR]) in 3 of 24, parti

128 HTK and control hearts post-transplantation (cardiac index: control 49.5+/-6% and HTK 48.5+/-5% of ba

129 Low cardiac index correlated with cool extremities, high cen

130 Whereas cardiac index decreased (4.0 [3.5; 5.3] vs 3.1 [2.6; 3.9

131 increased 4 +/- 1 mm Hg (n = 27, p < 0.01), cardiac index decreased 0.4 +/- 0.1 L/min/m2 (n = 27, p

132 Simultaneously, cardiac index decreased from 3.47 +/- 0.86 L/min/m2 to 3

133 In the remaining patients, cardiac index did not change despite a significant decre

134 olume, left ventricular end-systolic volume, cardiac index, dP/dt max, -dP/dt min, and left ventricul

135 ed postresuscitation myocardial dysfunction (cardiac index, dP/dt40, -dP/dt) was observed with propra

136 up (left ventricular end-diastolic pressure, cardiac index, +dP/dt, -dP/dt, and time constant of expo

137 In Fontan patients, sildenafil improved cardiac index during exercise with a decrease in total p

138 We measured the changes in cardiac index during passive leg raising and after volum

139 duced left ventricular ejection fraction and cardiac index, elevated pulmonary capillary wedge pressu

140 iratory occlusion induced percent changes in cardiac index estimated by esophageal Doppler are taken

141 after infusing 500 mL of saline, we measured cardiac index estimated by esophageal Doppler before and

142 sum of absolute values of percent changes in cardiac index estimated by esophageal Doppler during bot

143 If the absolute sum of the percent change in cardiac index estimated by esophageal Doppler induced by

144 End-expiratory occlusion increased cardiac index estimated by esophageal Doppler more in re

145 001) and end-inspiratory occlusion decreased cardiac index estimated by esophageal Doppler more in re

146 piratory occlusion induced percent change in cardiac index estimated by esophageal Doppler with an ar

147 -1.00]) and with a threshold of 9% change in cardiac index estimated by esophageal Doppler, which is

148 .00) and a threshold value of 4% increase in cardiac index estimated by esophageal Doppler.

149 When coupled with low cardiac index, even mild elevations in PVRI identify pat

150 .5+/-4.9 to 19.8+/-7.0 mm Hg (P<0.0001), and cardiac index (excluding augmented aortic flow) increase

151 V1000 led to a significant overestimation of cardiac index, global end-diastolic index, extravascular

152 ed a passive leg raising-induced increase in cardiac index greater than or equal to 10% with a sensit

153 ed a passive leg raising-induced increase in cardiac index greater than or equal to 10% with a sensit

154 by a passive leg raising-induced increase in cardiac index greater than or equal to 10%.

155 test was defined as positive if it increased cardiac index greater than or equal to 10%.

156 s was defined by a fluid-induced increase in cardiac index greater than or equal to 15%.

157 end-expiratory occlusion-induced increase in cardiac index greater than or equal to 5% detected a pas

158 end-expiratory occlusion-induced increase in cardiac index greater than or equal to 6% detected a pas

159 30%), and those with normal PVRI and normal cardiac index (group 2, n=182, 70%).

160 en to identify fluid responders (increase in cardiac index > 15%).

161 Fluid increased cardiac index >/= 15% (44% +/- 39%) in 30 "responders."

162 Volume expansion increased cardiac index >/= 15% in 49% of patients ("volume-respon

163 rognostic features at follow-up, including a cardiac index >/=2.5 L.min(-1).m(-2), 6-minute walk dist

164 nary capillary wedge pressure <18 mm Hg, and cardiac index >2.2 L/(min.m(2)).

165 mics with right atrial pressure <8 mm Hg and cardiac index >2.5 mg/kg/min(2).

166 tion with right atrial pressure <8 mm Hg and cardiac index >2.5 to 3.0 l/min/m(2).

167 mL/100 mL per minute in those with preserved cardiac index (>/=2.5 L/min per m(2); P=0.003).

168 ded (n=184), individuals with clinically low cardiac index had a higher relative risk of both dementi

169 rdiac index, individuals with clinically low cardiac index had a higher relative risk of dementia (HR

170 01 for both), whereas patients with a higher cardiac index had better survival overall (HR, 0.384; 95

171 rocirculatory parameters (arterial pressure, cardiac index, heart rate, and pulse pressure variations

172 Cardiac index, heart rate, systemic oxygenation, plasma

173 After implantation, the cardiac index improved from median 0.52 (interquartile r

174 time and stroke volume, cardiac output, and cardiac index in all groups.

175 be a more appropriate treatment target than cardiac index in PAH.

176 nd-expiratory pressure significantly reduced cardiac index in passive leg raising responders (-27% [i

177 Higher cardiac index in the adenosine, lidocaine, and Mg/adenos

178 th concurrent increase of cardiac output and cardiac index in the overall population (p < 0.001).

179 terval and stroke volume, cardiac output, or cardiac index in the overall population.

180 rterial blood flow (corrected for changes in cardiac index) in response to left lung hypoxic challeng

181 Po2 improved (48 to 60 mm Hg, P=0.0004), and cardiac index increased (4.3 to 5.4 L/min per m2, P=0.00

182 Conversely, cardiac index increased (from 2.6 L.min(-1).m(-2) [IQR,

183 % and 15% groups (p < .05 vs. baseline), and cardiac index increased 130% (p < .05 vs. baseline) at 1

184 Cardiac index increased and left ventricular end-diastol

185 However, with treatment cardiac index increased at rest (P=0.006) and peak exerc

186 tension+ patients with fluid responsiveness, cardiac index increased by 10% +/- 14% during passive le

187 tension- patients with fluid responsiveness, cardiac index increased by 25% +/- 19% during passive le

188 Each 1-SD unit decrease in cardiac index increased the relative risk of both dement

189 Before sildenafil, cardiac index increased throughout exercise (4.0+/-0.9,

190 ehicle (distilled water), caffeine decreased cardiac index, increased systemic vascular resistance, r

191 In the 15 fluid responders (cardiac index increases >= 15%), fluid administration in

192 include decreased arterial tone even if the cardiac index increases.

193 Compared with individuals with normal cardiac index, individuals with clinically low cardiac i

194 ssure = 5 cm H2O, we measured the changes in cardiac index induced by end-expiratory occlusion and a

195 We also tested whether the changes in cardiac index induced by passive leg-raising and by an e

196 he changes in pulse contour analysis-derived cardiac index induced by passive leg-raising and end-exp

197 ints were 6-hour and peak cTnT, ECG changes, cardiac index, inotrope and vasoconstrictor use, renal d

198 nine model of severe septic shock with a low cardiac index, intra-aortic balloon counterpulsation pro

199 al and clinical research suggests that lower cardiac index is associated with abnormal brain aging, i

200 Lower cardiac index is associated with an increased risk for t

201 pressors and inotropes, and association with cardiac index, lactate, and central venous oxygen satura

202 Forearm blood flow, cardiac index, left ventricular dimensions, and mean art

203 Myocardial function, including cardiac index, left ventricular, dP/dt max (dP/dt max),

204 decreasing cardiac function, even at normal cardiac index levels, is associated with accelerated bra

205 Cardiac index, local cerebral blood flow, and hemodynami

206 a score, edema, positive fluid balance, high cardiac index, low PaO2/FIO2 ratio, and high levels of c

207 h ADHF admitted between 2000 and 2005 with a cardiac index < or =2 l/min/m(2) for intensive medical t

208 were adults with ejection fraction </= 25%, cardiac index </= 2.2 l/min/m(2) without inotropes or we

209 gen/fraction of inspired oxygen ratio </=55, cardiac index </=2.2, or ventricular tachycardia or fibr

210 Patients with cardiac index <2.2 L x min(-1) x m(-2) by catheterizatio

211 negative predictive value (93% and 86%) for cardiac index <2.5 and mixed venous oxygen saturation <6

212 s: those with high PVRI (>2 WU.m(2)) and low cardiac index <2.5 L min(-1) m(-2) (group 1, n=70, 30%),

213 significantly lower in patients with reduced cardiac index (<2.5 L/min per m(2)): 94+/-30 mL/100 mL p

214 Hemodynamics, including cardiac index, LV dP/dt40, LV negative dP/dt, and LV dia

215 in ejection fraction, fractional shortening, cardiac index, LV dP/dt40, LV negative dP/dt, and LV dia

216 l stress (LVESWS), LVESWS-index, and maximal cardiac index (MCI; a measure of cardiac output at peak

217 95% CI, 0.20-0.68 mmol/L; p = 0.0004), lower cardiac index (mean difference, -0.76 L/min/m; 95% CI, -

218 eratively, all patients had severely reduced cardiac index (mean, 2.1 L/min/m2).

219 We noninvasively monitored cardiac index, mean arterial blood pressure, heart rate,

220 were associated with pronounced increases in cardiac index, mean arterial pressure, and heart rate an

221 ctive of our study is to describe changes in cardiac index, mean arterial pressure, and their relatio

222 l to 15% were defined as "fluid responders." Cardiac index measured by the Pulse Contour Cardiac Outp

223 Patients in whom volume expansion increased cardiac index measured by transpulmonary thermodilution

224 s, renal failure, prolonged ventilation, low cardiac index, myocardial infarction, and stroke.

225 ed ejection fraction, 3D sphericity indices, cardiac index, normalized systolic volume, normalized LV

226 me expansion (500 mL of saline), we measured cardiac index, o2- and Co2-derived variables and lactate

227 ontrast to the dopamine-mediated increase in cardiac index observed at normothermia, high-dose dopami

228 aseline New York Heart Association class IV, cardiac index of 1.7 L/min per m(2), pulmonary capillary

229 i 13.0 (+/- 6.7) Wood Units/m(2) and reduced cardiac index of 2.21 (+/- 0.5) L/min/m(2) were recruite

230 the beginning of the treatment with a median cardiac index of 4.2 L/min/m(2) (range, 2.9-5.2; P < .00

231 n/kg, right atrial area of less than 18 cm2, cardiac index of greater than 2.5 L/min/m2, and absent o

232 In the goal-directed therapy arm, a cardiac index of greater than 3 L/min/m was targeted wit

233 recruitment, prone positioning increased the cardiac index only in patients with preload reserve, emp

234 es in systemic hemodynamic variables such as cardiac index or mean arterial pressure.

235 irculation are not useful for predicting low cardiac index or mixed venous oxygen saturation.

236 significantly lower heart rates (p < 0.05), cardiac index (p < 0.05), mean arterial pressure (p < 0.

237 Sildenafil increased cardiac index (P<0.0001) and stroke volume index (P=0.00

238 sistance, and increased resting and exercise cardiac index (P<0.05 for all) without altering mean art

239 uration gradient (P<0.05) and inversely with cardiac index (P<0.05) for both CHF patients and control

240 th LGSAS had reduced stroke volume index and cardiac index (P=0.003 for both).

241 o (n=101) had no significant effect on donor cardiac index (pooled mean difference, 0.15 L/min/m(2);

242 Cardiac index progressively increased in the device (2.0

243 Cardiac index, pulmonary artery pressure, and pulmonary

244 obtained by transpulmonary dilution such as cardiac index, pulmonary blood volume index, and extrava

245 ted the regression line between the pairs of cardiac index (pulse contour analysis) and central venou

246 The cardiac index, pulse oximetry, transcutaneous oxygen ten

247 etween changes in mean arterial pressure and cardiac index (r = 0.035, p = 0.79).

248 ravascular lung water correlated to baseline cardiac index (r = 0.17; p = .001), baseline pulmonary b

249 ed HHT, severe liver involvement, and a high cardiac index related to HHT.

250 Despite the reduction in heart rate, cardiac index remained unchanged under mild therapeutic

251 features of relative hypovolemia and reduced cardiac index reserve.

252 Cardiac index responders and mean arterial pressure-resp

253 - central venous pressure and the number of cardiac index-responders after fluid bolus were similar,

254 Cardiac index-responsiveness and mean arterial pressure-

255 predictable with a poor relationship between cardiac index-responsiveness and mean arterial pressure-

256 terin blunted the increase in heart rate and cardiac index seen in the control group without affectin

257 usly altered and dopamine failed to increase cardiac index since stroke index was reduced with increm

258 Cardiac MRI measurements included cardiac index, stroke volume index, global and regional

259 , cardiac power index (CPI), cardiac output, cardiac index, stroke volume, left ventricular work, lef

260 and secondary end points included mean SVRI, cardiac index, systolic function, and lactate levels.

261 olumes (P=0.04) than participants in the top cardiac index tertile (values >2.92).

262 ons revealed that participants in the bottom cardiac index tertile (values <2.54) and middle cardiac

263 diac index tertile (values <2.54) and middle cardiac index tertile (values between 2.54 and 2.92) had

264 levations in PVRI, and when coupled with low cardiac index, this would identify patients at increased

265 Passive leg raising increased cardiac index to a larger extent after (8% +/- 4%) than

266 tory pressure, passive leg raising increased cardiac index to a larger extent than at positive end-ex

267 in a more favorable index of forward output (cardiac index) to mechanical energy (pressure-volume are

268 Cardiac index, total peripheral resistance, and blood vo

269 We measured the response of cardiac index (transpulmonary thermodilution) to fluid a

270 mia, high-dose dopamine at 25 degrees C left cardiac index unchanged despite a concomitant increase i

271 GIK significantly increased cardiac index versus placebo (P=0.037).

272 s pressure and change in stroke volume index/cardiac index was 0.18 (95% CI, 0.1-0.25), being 0.28 (9

273 ular ejection fraction was 16% (10% to 30%), cardiac index was 1.3 L/min/m (0.7-2.2 ) and systemic re

274 Cardiac index was 1.39 +/- 0.43 L . min(-)(1) . m(-)(2),

275 AP) was 44 +/- 10 mm Hg (range 26-73 mm Hg), cardiac index was 3.5 +/- 0.9 L/min/m(2) , and pulmonary

276 ents with septic acute kidney injury, median cardiac index was 3.5 L/min/m2 (range 1.6-8.7), and medi

277 ry wedge pressure was 5.9 +/- 4.6 mm Hg, and cardiac index was 3.6 +/- 0.6 L . min(-)(1) . m(-)(2).

278 The combination of high PVRI and low cardiac index was an independent risk factor for Fontan

279 -5 mm Hg; P<0.001), whereas no difference in cardiac index was found between groups.

280 shorter for BDOO compared with CDOO, and the cardiac index was higher with BDOO compared with CDOO.

281 In multivariable-adjusted models, cardiac index was positively related to total brain volu

282 After surgery, the cardiac index was significantly higher in the OPCABG gro

283 Cardiac index was significantly higher when measured wit

284 Cardiac index was similar and most patients were WHO FC

285 left ventricular end-diastolic pressure, and cardiac index, was significantly improved in pentazocine

286 The cardiac index went from 3.6 +/- 0.4 to 2.9 +/- 0.6 L/min

287 pressure variation, and the change in stroke/cardiac index were 0.78, 0.72, and 0.72, respectively.

288 ar resistance, capillary wedge pressure, and cardiac index were also obtained at baseline in all pati

289 Cardiac output and cardiac index were increased significantly in 79% patien

290 fraction, left ventricular stroke index and cardiac index were initially (at H0 or H6 according to l

291 sure variation, stroke volume variation, and cardiac index were recorded at tidal volume 6 mL/kg pred

292 e 16 (4.2) and 22 (5.3) mm Hg (p = .02), and cardiac indexes were 4.6 (2.8) and 2.2 (0.6) L/min/m (p

293 Cardiac indexes were significantly higher with normotens

294 poreal flows (approximately = 9% of baseline cardiac index) were required in zoniporide and control g

295 avenous (Pyr(1))apelin-13 infusion increased cardiac index, whereas reducing mean arterial pressure a

296 n-13 infusion caused a sustained increase in cardiac index with increased left ventricular ejection f

297 d in WM (median 11-hour cardiac index/1-hour cardiac index: WM=27% versus non-WM=9.5%, P=0.022).

298 2.96, p = 0.307; Q = 44.88, I(2) = 95.54%), cardiac index (WMD: 0.05 L/min/m(2), 95%CI: -0.05, 0.15,

299 Age, baseline cardiac index, World Health Organization functional clas

300 esized that cardiac function, as measured by cardiac index, would be associated with preclinical brai